Section 4 Flashcards
Hyperkalemia
Hyper-K-alemia
High potassium
Caused by renal disease, acidosis, meds
Hyperkalemia EKG Progression
- T waves peak
- Loss of P wave, further T wave peaking
- Sine Wave
Hypokalemia
Hypo-K-alemia
Low potassium
Caused by dietary deficiency, alkalosis, excess mineralocorticoids, meds
Hypokalemia EKG findings
T wave flattening
U wave appears in ONLY the anterior leads (between T and P wave)
Hypocalcemia
Low Calcium
Caused by malabsorption, vit d deficiency, hypoparathyroidism
Hypocalcemia EKG findings
Prolonged QT interval
Short PR interval
T wave flattening
Risk of V Tach
R on T phenomenon
Prolonged QT from hypocalcemia turning into Vtach. T wave encroaches onto the next R wave.
Hypercalcemia
High Calcium
Caused by malignancies, granulomatous diseases, medication induced, primary hyperparathyroidism
Hypercalcemia EKG findings
Shorted QT inervals
Hypothermia
Cold temp
Slows metabolism
Hypothermia EKG Changes
Sinus Brady
Prolonged segments
Distinct ST elevation: J Wave or Osborne Wave
Digitalis Effect
Drug Digoxin causes it
Normal at therapeutic levels
Slows AV node conduction
Digitalis EKG
ST Depression w/ downslope
(Looks like mustache)
T wave flattening or inversion
Digitalis Toxicity
Happens at supratherapeutic levels
Sinus Node suppression
AV Conduction blocks
Tachyarrhythmias
Paroxysmal Atrial Tach
Enhanced automaticity of ectopic atrial focus
Or from re-entrant circuit in atria
Usually looks like SVT, but can have P waves if slow enough
100-200 BPM
Associated with Digitalis Toxicity