secondary care Flashcards

1
Q

what is loss of RG colour vision and indicator of

A

optic neuritis

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2
Q

direct/consensual light pathway

A

optic nerve -> pretectal nucleus -> edinger-westphal nucleus (ipsilaterally and contralaterally) -> CN III (ipsi+contra) -> pupil constriction

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3
Q

what pupil reaction is seen in neurosyphilis

A

pupil doesn’t react well to light (i.e. doesn’t constrict) but does accommodate

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4
Q

what is the effect of cocaine on pupils (and how does it occur)

A

blocked the reuptake of NA in sympathetic neurons => repeated sympathetic stimulation -> pupils dilate

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5
Q

2 types of CN III palsy and which is an emergency

A

pupil sparing vs pupil involving

pupil involving is an emergency

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6
Q

superior vs inferior quadrantinopia lesions

A

PITS - parietal inferior, temporal superior

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7
Q

what diplopia is seen in 4th nerve palsy an what might a pt do to compensate

A

vertical/tilted diplopia -> pt may have a compensatory head tilt

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8
Q

3 things to comment on about the optic nerve

A

cup, colour, contour

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9
Q

what drug class is a risk factor for glaucoma

A

steroids

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10
Q

what is a pinhole used to check in a visual acuity exam

A

whether the problem is refractive (gets better w pinhole, fixable w glasses) or due to non-refractive factors (corneal problems, glaucoma etc.)

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11
Q

what 3 things are tested for in pupil exam

A
  1. direct and consensual light reflex
  2. swinging light test
  3. accommodation
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12
Q

function of ocular oblique muscles

A

look in

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13
Q

what symptom may be seen in lateral rectus palsy

A

compensatory head turn -> head turned inorder to diminish diplopia

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14
Q

why is the eye in the down and out position in CN III palsy

A

only LR and SO function -> LR is stronger than SO and so pulls the eye out (even tho SO pulls it in) and then SO pulls it down

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15
Q

what is the primary function of the superior oblique (when looking straight ahead)

A

intorsion

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16
Q

what is the likely cause for a CN IV palsy

A

trauma to the head (the nerve has a long journey through the brain)

17
Q

5 conditions that may cause and increase in size of the blind spot

A
  1. glaucoma
  2. papilloedema
  3. CRVO
  4. CRAO
  5. optic neuritis
18
Q

how to tell where in the optic tract tell is a lesion causing HH arises from

A

field defect congruity -> if less symmetrical between the eyes then more likely to be an anterior lesion, if symmetrical then more likely to be posteior lesion

19
Q

what visual field defect is likely to arise from a pit gland tumour

A

bitemporal superior quadrantinopia

20
Q

5 causes of asymmetrical hemifield loss

A
  1. optic neuritis
  2. inf. BRVO/BRAO
  3. glaucoma
  4. inf. space occupying lesion
  5. retinal detachment
21
Q

what daily function may someone w CN IV palsy struggle with?

A

reading or walking down stairs -> can’t brings eyes in + down

22
Q

4 fundoscopy findings of hypertensive retinopathy

A
  1. AV nipping;
  2. hard exudates/cotton wool spots;
  3. silver wiring;
  4. optic disc swelling
23
Q

what is retinitis pigmentosa

A

pigment deposit in the periphery of the retinal casing damage to rods

24
Q

where is the macula found on fundoscopy

A

the area between the temporal branch retinal veins

25
Q

what does retinitis pigmentosa present with

A

night blindness - loss of rods

26
Q

retinitis pigmentosa fundoscopy findings (3)

A

classic clinical triad:
1. arteriolar attenuation;
2. retinal pigmentary changes (could be either hypopigmentation and/or hyperpigmentation in form of bone-spicule and pigment clumpings)
3. waxy disc pallor

27
Q

causes of papilloedema (7)

A

papilloedema must arise from raised ICP - this can be due to many causes:

  1. malignant HTN
  2. glaucoma
  3. chronic CO2 retention
  4. venous sinus thrombosis
  5. metabolic hypocalcaemia;
  6. acute altitude sickness;
  7. zero gravity