SCORE Flashcards
A 37-year-old woman has severe intense pain that lasts seconds when the right chin is stimulated by either tactile or thermal stimuli. The pain radiates to her right ear. She denies any traumatic, dental, medical, or surgical etiology. The results of a cone-beam CT study are normal. The pain is resolved with 600 mg of carbamazepine three times daily. These findings are consistent with typical trigeminal neuralgia of the right mandibular (V3) distribution. What is the next diagnostic step for this patient and why?
A. MRI of the brain without contrast to determine if there is compression of the right trigeminal nerve or evidence of demyelination disease
B. CT scan with 1-mm cuts to determine if there is any occult pathology of the right posterior and body of the mandible
C. Nerve blocks of the right inferior alveolar nerve to determine if the pain source is peripheral or central
D. Indomethacin challenge to determine if the pain is associated with hemicrania continua
A. MRI of the brain without contrast to determine if there is compression of the right trigeminal nerve or evidence of demyelination disease
MRI of the brain with and without contrast helps distinguish secondary causes of trigeminal neuralgia from the idiopathic form. It is the imaging modality of choice and is indicated for patients younger than age 60 who present with trigeminal neuralgia, principally to exclude a tumor, vessel compression, or demyelinating disease. CT, nerve blocks, or an indomethacin challenge would not be indicated for this patient with a history and clinical examination consistent with pharmacologically responsive trigeminal neuralgia.
ou are operating on a patient with middle face fractures, including alveolar, Le Fort, infraorbital rim, medial orbital wall, orbital floor, and nasal bone fractures. The fracture happened about 2 weeks ago, and you are having difficulty mobilizing the maxilla to properly reduce it. What should you do next? (NOTE: OMFS curriculum)
A. Use osteotomes to refracture the consolidated areas to obtain adequate mobility of the maxilla before reduction.
B. Use Rowe forceps, paying attention to the alveolar and nasoorbitoethmoid (NOE) components.
C. Put the patient into maxillomandibular fixation (MMF) to see if adequate occlusion can be obtained with the current maxillary position.
D. Choose a top-bottom approach, and start with the orbits before mobilizing the maxilla.
E. Perform an intraoperative CT scan to determine the points of resistance.
B. Use Rowe forceps, paying attention to the alveolar and nasoorbitoethmoid (NOE) components.
Using Rowe forceps is the easiest way to mobilize a fracture that is only 2 weeks old, but the surgeon must avoid propagating the alveolar bone fracture. Most importantly, if the Le Fort fracture is in combination with an NOE fracture, the surgeon must acknowledge the risk of a cranial base fracture at the level of the ethmoid plate, and evaluate the CT scan to confirm the level of the Le Fort fracture. Osteotomes are usually not necessary for a 3-week-old fracture. Putting the patient into MMF without adequate mobilization will tend to dislocate the condyles and lead to malocclusion. Starting with the orbit will not help mobilize the maxilla, and mobilizing the maxilla after fixating the orbital component might damage the fixation. There is no point in performing an intraoperative CT scan when the problem can be seen on the open surgical field. Transoperative CT is used at the end of skeletal fixation to confirm the adequate position of areas that are not easily accessed by surgical approaches, for example, evaluating the adequate reconstruction of the orbital floor.
A 40-year-old man presents as a level 1 trauma patient to the ED after being involved in a work-related accident. A clinical examination reveals a full-thickness scalp laceration involving the temporal region, and the patient has significant bleeding. His medical history is significant for a mechanical heart valve for which he is prescribed warfarin for anticoagulation. His international normalized ratio is 5. Which therapy will predictably reverse the patient’s coagulopathy in this acute, nonelective surgical setting? (NOTE: OMFS curriculum)
A. IV vitamin K and four-factor prothrombin complex concentrates
B. Fresh frozen plasma
C. Platelets
D. Packed red blood cells
A. IV vitamin K and four-factor prothrombin complex concentrates
This combination will provide the quickest reversal of the patient’s warfarin-induced coagulopathy. The IV route is the fastest, and the four-factor prothrombin complex concentrates are superior to fresh frozen plasma in reversing coagulopathy secondary to warfarin. Platelet and blood transfusions do not provide the necessary components to reverse the coagulopathy.
A 25-year-old woman presents to your office stating, “I don’t like my crooked nose.” You complete a thorough clinical examination and determine that the patient has a deviated septum and dorsal hump and will require nasal tip work. The patient asks, “What will you do to straighten my nose?” What is your response? (NOTE: OMFS curriculum)
A. “I will avoid disrupting the perpendicular plate of the ethmoid bone.”
B. “I will mobilize and reattach the septum to the anterior nasal spine.”
C. “I will perform septoplasty in a closed fashion followed by open rhinoplasty for remaining issues.”
D. “I will resect the septum leaving an L strut of at least 10 mm.”
D. “I will resect the septum leaving an L strut of at least 10 mm.”
The nasal septum is the key structure in the deviated nose. It is a supporting structure, particularly for the lower two-thirds of the nose. It forms a fixed tripod with the conjoined upper lateral cartilages. Floating over this fixed tripod is the so-called “floating tripod” of the lower lateral (alar) cartilages, which are attached by various ligaments. These include the lateral scroll ligament and interdomal sling, supported by muscular structures such as the dilator nasi, the pyriform ligament, and the transversalis muscle. Without a proper foundation and with a central septum supported by upper lateral cartilages, there is little hope of achieving a symmetric and stable nose. The key area overlooked with rhinoplasty surgeons who are unfamiliar with the anatomy of the posterior septum is the chondrovomerine spur, which must be released to allow the back of the septum to swing back to the midline. Failure to release this area and the adjacent ethmoid plate results in an invariable postoperative twist. Many surgeons have blamed this postoperative complication on so-called “cartilage memory,” when actually it is a failure to mobilize and stabilize the posterior and inferior margins of the quadrilateral cartilage. The basic premise to achieve a straight nose has to be a central nasal septum with symmetric nasal sidewalls. In correcting a deviated nose, the greatest deficit always occurs on the more vertically placed nasal sidewall, particularly distally, where there is a deficiency in the anterior height of the upper lateral cartilage. Septal deviation is corrected using a variety of techniques, including posterior caudal septal resection, leaving an L-strut with at least 10 mm of cartilage anteriorly. Depending on the type of septal deformity, cartilaginous scoring, partial mobilization of the perpendicular plate and quadrangle cartilage, and anterior nasal spine osteotomy may become necessary.
A 5-year-old boy is referred to your practice by his pediatrician. His mother reports that he has a very sore mouth and decreased oral intake over the past 2 days. She states he has some blisters in his mouth that are causing him pain in addition to his fevers and malaise. His clinical examination is positive for cervical lymphadenopathy in addition to the numerous ulcerations on the buccal and labial mucosa and the tongue. No other lesions are noted elsewhere during the evaluation. Given this patient’s presentation, which condition is most likely? (NOTE: OMFS curriculum)
A. Cat scratch disease
B. Herpangina
C. Hand, foot, and mouth disease
D. Acute herpetic gingivostomatitis
D. Acute herpetic gingivostomatitis
The patient’s age and clinical presentation most align with acute herpetic gingivostomatitis. There are no reported lesions on his hands or feet, which rules out hand, foot, and mouth disease. Herpangina will most commonly present with lesions on the pharynx and tonsils, not the mucosa. Cat scratch disease will present with cervical lymphadenopathy, but not with any oral lesions.
A 14-year-old girl with maxillary hypoplasia and mandibular hyperplasia and a resulting severe class III malocclusion presents to your office with her parents reporting that she wants corrective jaw surgery. You, the patient, and the patient’s family agree to schedule the surgery as soon as possible, but given the patient’s young age and her mandibular hyperplasia, you are concerned that her future growth could lead to a relapse of the planned mandibular setback. The patient’s parents are nervous about any unnecessary radiation and seem displeased when you suggest a hand-wrist radiograph to help assess the patient’s future growth, so you decide to use her serial cephalometric imaging to assess her C-spine score. You note that the C2, C3, and C4 vertebrae have pronounced concavities on the inferior border that do not appear changed from the previous lateral cephalometric radiograph. You also note that the vertebrate of C3 and C4 have lengthened vertically and are becoming more like vertical rectangles when compared with the previous lateral cephalometric radiograph. What is this patient’s likely C-spine score, and what is your decision on surgical timing? (NOTE: OMFS curriculum)
A. Stage 2: Delay surgery.
B. Stage 3: Delay surgery.
C. Stage 4: Delay surgery.
D. Stage 5: Proceed with surgery.
E. Stage 6: Proceed with surgery.
E. Stage 6: Proceed with surgery.
The C-spine score described in this clinical scenario is most consistent with stage 6, or the final stage of cervical spine maturation. There is a general trend of deepening concavities along the inferior border of the C2, C3, and C4 vertebrae, and the vertebrae will transition from wedges to horizontal rectangles to vertical rectangle-shaped vertebrae. In the final stage, when adolescent growth is essentially complete, the C2, C3, and C4 vertebrae will have pronounced inferior borders, and C3 and C4 will start to take on a more square or vertical rectangular shape. This signals the completion of adolescent growth, which means that continued mandibular growth altering surgical outcomes is unlikely. Proceeding with surgery is therefore a reasonable decision.
While analyzing a patient for orthognathic surgery, you note that the patient’s genial region is deficient clinically. What cephalometric measurement of the skeletal position of the chin should you use to determine if genioplasty may be indicated for this patient? (NOTE: OMFS curriculum)
A. Wits analysis
B. Holdaway ratio
C. Maxillomandibular (Mx/Md) measurement
D. Andrews analysis
B. Holdaway ratio
The Holdaway ratio measures the position of the labial surface of the lower incisors to the NB line compared with the pogonion of the chin to a line NB. The distance should be equal; if not, genioplasty may be indicated. The Wits analysis determines the anteroposterior position of the jaws relative to one another. Mx/Md compares relative position and size. The Andrews analysis determines the esthetic position of the maxilla using forehead angulation and upper incisor angulation.
A 24-year-old man presents to your office with concerns about the appearance of his jaws and chin line. He feels like everything below his nose is “too prominent.” You perform the appropriate imaging and radiographic studies to assess the position of the jaws and find him to have a proper class I occlusion. You also perform two lateral cephalometric tracings and analyses, which reveal that the jaws are in clinically acceptable positions with no overt signs of prognathism. On the lateral cephalometric radiograph, you note that the soft tissues on the lips and overlying the chin are thicker than usual. Looking at your clinical photos, you see that the upper and lower lips extend anterior to the area demarcated by a line through the nasal tip and soft tissue pogonion as well as a line through the subnasal and soft tissue pogonion. What are you using to assess the soft tissue positioning? (NOTE: OMFS curriculum)
A. Subnasale vertical
B. S-line (Steiner analysis)
C. Riedel plane
D. Z-line (profile line of Merrifield)
E. Burstone-Ricketts triangle
E. Burstone-Ricketts triangle
The Burstone-Ricketts triangle includes the Ricketts E-line, which goes from the nasal tip to the soft tissue pogonion, and the Burstone Line, which connects the subnasale with the soft tissue pogonion. These two lines in combination with the columella form a triangle in which the upper and lower lips should reside in a normal population. The upper lip should be slightly anterior to the lower lip. All the remaining measures are single lines and do not outline an area.
A 65-year-old woman presents to your office 1 year after an “all-on-4” placement of zygomatic implants. She reports that in the past few weeks she has experienced several episodes of moderate sinus pressure and rhinorrhea. She has also experienced multiple headaches. How would you initially manage this patient? (NOTE: OMFS curriculum)
A. Provide supportive care.
B. Prescribe augmentin, guaifenesin, and pseudoephedrine HCl.
C. Refer the patient to an otolaryngologist for possible sinus exploration and surgery.
D. Remove the implants.
B. Prescribe augmentin, guaifenesin, and pseudoephedrine HCl.
In patients with acute allergy or sinus infection after integration of zygomatic implants, simple oral measures are a reasonable first-line treatment. Guaifenesin is useful for thinning the mucosa. Pseudoephedrine HCl decreases edema within the mucosa. Augmentin is used for antibiotic coverage. Levaquin may be prescribed for recurrent infections or episodes that do not resolve. This patient requires more than just supportive care. The zygomatic implant is generally well tolerated by the sinus and is rarely the cause of sinus infection. There is no indication that the implants need to be removed.
A 46-year-old woman has persistent pain attributable to the bilateral temporomandibular joints (with no limitations in mandibular range of motion) despite rigorous conservative measures and excellent compliance. Arthrocentesis only resulted in transient relief. The patient decides to undergo bilateral temporomandibular joint discectomy. Intraoperatively, significant bleeding is noted immediately after excising the disc from its posterior attachments. What is likely the source of the bleeding? (NOTE: OMFS curriculum)
A. Retrodiscal tissues
B. Deep temporal artery
C. Maxillary artery
D. Middle meningeal artery
E. Pterygoid venous plexus
A. Retrodiscal tissues
The retrodiscal tissues attach to the articular disc posteriorly. This vascular structure can often cause significant bleeding if not thoroughly controlled. The deep temporal arteries are medial and anterior to the retrodiscal tissue area. Bleeding from the maxillary and middle meningeal arteries and the pterygoid venous plexus occurs when the medial boundaries are violated.
An 11-month-old boy with sagittal suture craniosynostosis is brought to your office by his parents for a consultation about repair. What treatment do you recommend? (NOTE: OMFS curriculum)
A. Physical therapy and repositioning instructions (“tummy time”)
B. Endoscopic sagittal suturectomy with barrel stave osteotomies and postoperative orthotic molding
C. Strip craniectomy
D. Sagittal suturectomy and posterior cranial vault reconstruction
E. Sagittal suturectomy with fronto-orbital advancement and anterior cranial vault reconstruction
D. Sagittal suturectomy and posterior cranial vault reconstruction
Patients with scaphocephaly and sagittal suture craniosynostosis may be treated with one of two typical treatments: (1) endoscopic sagittal suturectomy, barrel stave osteotomies, and postoperative head molding with an orthotic device; or (2) sagittal suturectomy and posterior cranial vault reconstruction. Because the patient is older than the age at which a good outcome can be expected from an endoscopic approach, formal posterior vault reconstruction is the only reasonable choice.
71-year-old man with DM type 1 and a recent history of right maxillary sinus symptoms is referred to you for extraction of tooth #2. During your evaluation, you notice necrosis of the soft tissue and bone in the right maxilla. Surgically, you debride the area and remove several teeth. The specimen that you send to pathology returns with the result of irregular, wide, fungal hyphae with frequent right-angle branching. What is your diagnosis? (NOTE: OMFS curriculum)
A. Aspergillosis
B. Candidiasis
C. Actinomycosis
D. Mucormycosis
D. Mucormycosis
Knowledge of the hyphae branching patterns is critical to answer this question. Mucormycosis has nonseptate, irregular, wide fungal hyphae with right-angle branching. Actinomyces are gram-positive filamentous or rod-shaped bacteria. Aspergillus has septae that branch at acute angles. Candida grows as a yeast, an elongated form without hyphae, and true hyphae with septa form.
A 28-year-old man with a muscular build who has a history of deep vein thrombosis and pulmonary embolism is scheduled for a surgically assisted rapid palatal expansion in the office with deep sedation. The patient’s warfarin is discontinued before the surgery. During the administration of the anesthetic, the patient develops a laryngospasm. Positive pressure ventilation is unsuccessful in breaking the spasm. As the oxygen saturation drops to 80%, a low dose of succinylcholine is administered, and the patient is able to be ventilated. Spontaneous respirations return, and the procedure is completed. In the recovery area, the patient is unable to maintain his oxygen saturation above 88%. What is the most likely cause of the decreased oxygen saturation? (NOTE: OMFS curriculum)
A. Residual sedation
B. Postobstructive pulmonary edema
C. Pulmonary emboli
D. Pneumothorax
B. Postobstructive pulmonary edema
This is a presentation of postobstructive pulmonary edema, which has been classically described in athletic patients. The patient who develops an upper airway obstruction attempts to overcome the obstruction by increasing respiratory effort. When the upper airway obstruction cannot be overcome with increased respiratory effort, the increased negative intrathoracic pressure causes an increase in venous return. This results in a hydrostatic pressure gradient between the intravascular and extravascular compartments, leading to pulmonary edema. A manifestation of pulmonary edema is a reduction in oxygen saturation.
A 27-year-old patient presents to your office for removal of a surgical splint 4 weeks after undergoing a maxillary advancement. Upon removal of the splint, you discover a 5-mm unplanned unilateral posterior open bite on the right side. What is the initial treatment step for this patient? (NOTE: OMFS curriculum)
A. Place elastics, and follow the patient closely.
B. Remove the posterior plate and possibly the anterior plate on the right, and use elastics.
C. Bring the patient back to surgery to correct the malocclusion.
D. Monitor the patient closely to follow the self-correction of the open bite.
A. Place elastics, and follow the patient closely.
Although it may be necessary to remove the plates in the office or take the patient back to surgery, it is possible to correct the open bite with elastic traction. If elastics fail to correct the malocclusion, plates should be removed and elastics reapplied. If the malocclusion is severe or if the first two options are unsuccessful, it may be necessary to take the patient back to the operating room.
You are examining a 3-month-old boy who has rotation of the head to the right along with an increased tension of the right sternocleidomastoid (SCM) muscle that is consistent with congenital muscular torticollis (CMT). This patient is at risk for which condition? (NOTE: OMFS curriculum)
A. Midface hypoplasia
B. Deformational plagiocephaly
C. Unicoronal craniosynostosis
D. Lambdoid craniosynostosis
B. Deformational plagiocephaly
A patient with CMT will have overactivation of the SCM. At mild degrees, this restriction will cause an infant to have the head turned in a constant direction when lying supine. This causes a constant deformational force that ultimately molds the head into a deformational plagiocephalic condition. Craniosynostosis is an intrauterine event, and as such, it is extremely unlikely to have synostosis develop after birth regardless of whether the patient has CMT or not. Patients with more severe forms of CMT not only have deformational abnormalities of head shape because of supine sleeping problems, but they also have cranial base abnormalities secondary to the pull of the SCM on the mastoid process. The downstream effects can cause facial asymmetries; however, midface hypoplasia is not one of the primary outcomes.
A 35-year-old woman presents to your office for consultation regarding surgical treatment for her obstructive sleep apnea (OSA). She relates a history of excessive daytime sleepiness, nonrefreshing sleep, HTN, and type II DM. A physical examination shows a morbidly obese woman (BMI is 40 kg/m²) with mandibular retrognathia and normal temporomandibular joint (TMJ) function. She recently underwent a sleep study, which showed severe OSA (apnea-hypopnea index was 85). After the sleep study, the patient was prescribed continuous positive airway pressure (CPAP) therapy, but she was unable to tolerate it, even after 3 months of trying various modifications to improve her compliance. What treatment recommendation would you provide to this patient? (NOTE: OMFS curriculum)
A. Continue current therapy with CPAP.
B. Use an oral appliance (OA).
C. Obtain a consultation for bariatric surgery.
D. Undergo maxillomandibular advancement (MMA) surgery.
E. Undergo hypoglossal nerve stimulation (HNS).
C. Obtain a consultation for bariatric surgery.
With a BMI of 40 kg/m², the patient may be a candidate for bariatric surgery, likely as an initial surgical procedure, and thus a consultation with a bariatric surgeon would be indicated. Consideration for other surgical procedures (MMA or HNS) would occur after treatment of severe morbid obesity if the patient continues to have OSA. Continuation of CPAP is not a treatment option because the patient has failed CPAP. The patient is not a good candidate for an OA because OAs are more effective in less obese patients with mild-to-moderate OSA.
A 35-year-old woman at 20 weeks’ gestation comes to your office for evaluation after an ultrasound reveals a diagnosis of cleft palate for the fetus. The patient reports that she had been taking valproic acid for a diagnosis of epilepsy but she has since discontinued that medication. In addition, she states that she was not ready for pregnancy and has been anxious, leading her to continue habits such as alcohol consumption and tobacco use. She also reports she has not been taking prenatal vitamins. Which factor in this patient’s history placed her at highest risk for having a child with an isolated cleft palate? (NOTE: OMFS curriculum)
A. Maternal exposure to tobacco smoke
B. Ethanol
C. Folic acid deficiency
D. Valproic acid
A. Maternal exposure to tobacco smoke
Maternal exposure to tobacco smoke has been reported as the teratogen agent with the strongest risk factor for isolated cleft palate. Although other agents have been associated with the development of cleft palate, such as the use of valproic acid, folic acid deficiency, ethanol, excess of vitamin A, poor nutrition, and viral infections, maternal exposure to tobacco smoke is the factor that places the fetus at highest risk.
ou are called to see a patient with an upper eyelid avulsion. Upon examination, you see that it is a partial-thickness defect and you need to determine if the wound can be closed primarily or if it needs to be grafted. What percentage of tissue loss makes it unlikely that you will be able to close the wound primarily? (NOTE: OMFS curriculum)
A. 10%
B. 20%
C. 30%
D. 40%
E. 50%
E. 50%
This is not a closed number, and clinical aspects such as patient age and tissue conditions will play an important role in the final decision, but a wound with a 50% loss of tissue is usually not amenable to primary closure.
A 10-month-old girl who underwent surgery to treat a large complete bilateral cleft palate is brought to the 3-month follow-up visit by her parents. They ask you if her speech will be normal or if she will require additional surgery. What should you explain to the parents about detecting velopharyngeal insufficiency (VPI) after palatal repair surgery? (NOTE: OMFS curriculum)
A. VPI can be properly assessed in toddlers 3 months after primary palatal repair, and if it is present, it should be addressed as soon as possible to decrease the risk of compensatory misarticulations.
B. Cleft-related VPI is typically diagnosed after 3 years of age or when a child has adequate word inventory to provide a diagnostic speech sample.
C. When primary cleft palate repair is completed before 12 months of age, patients present with a higher risk of compensatory misarticulations than can confound the diagnosis of VPI.
D. Patients with VPI after primary palatoplasty present with a palatal fistula, making the diagnosis clinically obvious.
B. Cleft-related VPI is typically diagnosed after 3 years of age or when a child has adequate word inventory to provide a diagnostic speech sample.
The diagnosis of VPI is based on perceptual speech assessment and evidence of incomplete velopharyngeal closure. VPI cannot be readily assessed in most toddlers because of their inadequate word inventory and the subsequent inability of the OMS to get an adequate speech sample. In addition, at least 6 months of palatal healing should be allowed before additional surgery is considered. Patients undergoing palate repair before 12 months of age have a lower risk of compensatory misarticulations. Not all patients with VPI have a palatal fistula, and a palatal fistula is not always associated with VPI.
A 9-year-old boy who sustained a right condylar fracture yesterday is referred to your office. He has a 3-mm open bite on his left side. The condylar fragment is moderately displaced out of the fossa. He has no other injuries and is healthy. How would you manage this patient? (NOTE: OMFS curriculum)
A. Soft diet and exercise
B. Closed reduction with 2 weeks of maxillomandibular fixation (MMF)
C. Open reduction and internal fixation (ORIF) of the condyle
D. Application of MMF appliances, training elastics, and full function
E. Closed reduction with 4 weeks of MMF
D. Application of MMF appliances, training elastics, and full function
Application of MMF appliances, training elastics, and full function is the treatment currently favored by surgeons. The patient has a malocclusion, which will not be managed with a soft diet. Closed reduction with 2 or 4 weeks of MMF is a dated treatment. Condylar ORIF is rarely indicated in children.
A 62-year-old woman presents to your office for surgical evaluation of her severe obstructive sleep apnea (OSA). Her medical history is significant for anxiety and depression, hyperlipidemia, and HTN. Her BMI is 35 kg/m², and her neck circumference is 16 inches (40.6 cm). Compared with a similar person without OSA, which anatomic finding would this patient most likely have? (NOTE: OMFS curriculum)
A. Significantly more lateral pharyngeal wall collapse
B. High hyoid position
C. Bimaxillary protrusion
D. Increased upper airway volume
E. Incompetent internal and external nasal valves
A. Significantly more lateral pharyngeal wall collapse
People with OSA tend to have a greater amount of lateral pharyngeal wall collapse, which contributes to nocturnal obstruction during deep sleep. The other anatomic findings would tend to lead to an increase in upper airway volume and a decrease in upper airway obstruction during deep sleep.
A 44-year-old man presents to the clinic reporting swelling on the left side of his neck. The patient states the swelling has been present for 2 weeks, has slowly enlarged, and is occasionally mildly tender. He reports having a fever of 100 ºF (37.7 ºC) and throat pain 2 days before the swelling became visible. He denies any trouble breathing or swallowing. The patient also denies any medical problems and states that he is not on any medications and has no allergies. A physical examination reveals a 3 × 3-cm sessile nodule of the left neck in level IIA. It is nontender to palpation and has no overlying skin changes. It is soft, and upon manipulation, it is not fixed. What is the next step in the management of this patient? (NOTE: OMFS curriculum)
A. Aspirate the lesion, and send it for culture and flow cytometry.
B. Order CT of the neck with contrast.
C. Perform flexible nasopharyngoscopy.
D. Schedule the patient for an open biopsy.
E. Send the patient for a fine-needle aspiration biopsy (FNAB).
C. Perform flexible nasopharyngoscopy.
Flexible nasopharyngoscopy should be part of the physical examination of all unknown neck masses and should be performed before any invasive diagnostic procedures or advanced imaging. It can provide valuable information and help guide the diagnostic process. Cultures would be sent if an infectious process was suspected, and flow cytometry would be sent if a hematologic malignancy such as lymphoma was suspected. Imaging studies of the mass are certainly warranted; however, they would be done after a thorough physical examination. CT, MRI, ultrasonography, angiography, plain films, and positron emission tomography are all possibilities, depending on the type of lesion suspected or seen on physical examination. An open biopsy may be necessary later in the diagnostic process; however, a thorough physical examination, noninvasive diagnostic studies (ie, endoscopic examination), advanced imaging, and, possibly, FNAB should all be done before this.
A 50-year-old woman comes to your office with concerns about her smile, “long-face,” and anterior open bite. She has been in orthodontic treatment for the past 8 months and feels she is ready for surgery. You evaluate the patient with the appropriate measurements and imaging and determine that she has relative anteroposterior maxillary hypoplasia with vertical maxillary excess with a gummy smile, a steep mandibular plane angle with premature posterior occlusion, and an anterior open bite. You also note that she has a particularly steep forehead. You use the Andrews 6 elements of orofacial harmony philosophy to plan the maxillary advancement. What will be the main determinant as to the amount of advancement you plan to perform? (NOTE: OMFS curriculum)
A. 1 mm posterior to the forehead anterior limit line (FALL)
B. Maxillary tooth show at repose
C. Goal anterior limit line (GALL)
D. Andrews element I
E. Andrews element V
In the Andrews 6 elements analysis, element II is concerned with the proper placement of the jaws in the anteroposterior dimension. The measurement in question is the GALL, which is a function of the FALL. The ideal maxillary position in an Andrews element II maxilla is for the facial surface of the most anterior maxillary central incisor to coincide with the GALL. The GALL is determined by the angle the forehead makes with the FALL. In a patient with a steep forehead or a large angle between the forehead inclination and the FALL, the GALL will be further anterior. The most anterior the GALL can be is up to the true vertical line that goes through the soft tissue glabella. Therefore, when a patient has a steep forehead angle, it is likely that the GALL will coincide with the glabella. There are times where the FALL and GALL are equal; however, placing the jaws 1 mm behind the FALL would not be ideal. Andrews element I addresses the proper orientation of the teeth within the alveolus. Andrews element V involves the optimal chin prominence based on the inclination of the lower incisors and the ideal occlusal plane.
An 18-year-old athletic woman with asthma presents for the removal of four impacted third molars. She receives a general anesthetic consisting of midazolam, fentanyl, and propofol with 50% nitrous oxide and 50% oxygen administered via a nasal hood. The patient’s oxygen saturation is noted to have decreased from 99% to 92% after the extraction of the second tooth. The surgeon opts to discontinue nitrous oxide and administer 100% oxygen via the nasal hood. The surgeon continues with the procedure without interruption but then notes that the patient’s saturation continues to decrease to 87%. Her mouth is suctioned, the pharyngeal curtain is removed, and the pharynx is suctioned. Her chest is auscultated without audible wheezing. Oxygen 100% is administered via a face mask. Ventilation is achieved with ease. The patient is subsequently intubated after the oxygen continues to decrease. Ventilation is again achieved with ease. Albuterol is administered via the endotracheal tube. The patient develops ventricular fibrillation. Emergency intervention is provided, but the patient is not resuscitated. What is the most likely contributing factor in this patient’s outcome? (NOTE: OMFS curriculum)
A. The patient experienced a bronchospastic episode that was refractory to intervention.
B. The patient had undiagnosed hypertrophic cardiomyopathy.
C. The patient’s oxygen line lacked an oxygen sensor.
D. The patient underwent esophageal intubation.
C. The patient’s oxygen line lacked an oxygen sensor.
Anesthetic and surgical gas lines are frequently hidden within walls, delivering the gases from a central storage area to each operatory. It may be assumed that the lines are plumbed correctly because there are various regulations designed to ensure such; however, it is ultimately the surgeon’s responsibility to ensure that the lines are functioning correctly. When office renovations are made, it is important to verify that the gas lines are plumbed accurately. This can be done by tracing the line from the central reserve to each operatory. Because oxygen is the most critical of these gases, correct oxygen line plumbing may alternatively be verified by inserting an oxygen sensor at the oxygen outflow in each operatory. All anesthetic machines used in a Joint Commission operating facility have oxygen sensors incorporated into the machine; however, most oral and maxillofacial surgery (OMS) office anesthetic units lack an oxygen sensor. The lack of an oxygen sensor is not inappropriate in open airway anesthesia, which is typical in most OMS offices, but it should be used when advanced airways (eg, laryngeal mask airways, endotracheal tubes) are used. Therefore, in an OMS office using an open airway anesthetic technique, an oxygen sensor need not remain fixed within the oxygen line. The decrease in oxygen in this patient started before intubation, so esophageal intubation is not likely to be the contributory factor. Undiagnosed hypertrophic cardiomyopathy can contribute to sudden death. An increase in cardiac contractility can worsen outflow, resulting in decompensation and ventricular arrhythmias. Respiratory compromise is usually not the initial manifestation. A bronchospastic event can cause deteriorating oxygen saturation. However, the patient was not wheezing, and ventilatory efforts were achieved with ease. The more common potential etiologies are not consistent with the presentation. Most OMS offices will not experience an event like this, but it has happened. When a major event occurs, it impacts not only the patient but also the doctor and the staff. In a 2012 survey conducted by the American Society of Anesthesiologists, 70% of practitioners said they experienced guilt and reliving of the event, 80% required time to recover emotionally, 19% never fully recovered, and 12% considered a career change.