schizophrenia Flashcards
what does the term ‘schizophrenogenic’ mean
schizophrenia causing
what is family dysfunction (SZ)
psychologists have tempted to link childhood and adult experiences of living in a dysfunctional family to SZ the three explanations the schizophrenogenic mother double-bind theory expressed emotion (EE)
the schizophrenogenic mother (key ideas)
- they are cold, rejecting and controlling
- create tension and secrecy
- can lead to distrust, developing into paranoia and delusions
double-bind theory (key ideas)
- the communication style in family
- children feel trapped in situations and fearful of wrong doings, making them feel unable to comment on fairness
- children then get punished through withdrawal of love which leave the children confused
- can lead to disorganised thinking
expressed emotion (EE / key ideas)
level of (typically) negative emotions towards a patient by a carer
- verbal criticism, accompanied by violence
- hostility towards patient, anger, rejection
- emotional over-involvement in life
- high levels of EE in carer are a serious source of stress and prone to relapses. although they may already be vulnerable due to genetic makeup
cognitive explanations (overview)
- focuses on roles of mental processes
- reduced processing in the ventral striatum is associated with negative symptoms
reduced processing of info in the temporal and cingulate gyri is associated with hallucinations
Frith et al (1920)
identified two kinds of dysfunctional thought processing:
- metarepresentations
- central control
what is metarepresentations?
- ability to reflect on thoughts and behaviours
- interpret the actions of others
- dysfunction would disrupt our ability to recognise our own actions and thoughts
- explains hallucinations of voices and delusions
what is central control?
- ability to suppress the automatic responses whilst preforming deliberate actions instead
- disorganised speech and thought disorder could result from inability to suppress automatic thoughts and speech triggered by other thoughts.
drug theory AO1
most common treatment is anti psychotics
two main: typical anti and atypical anti
typical antipsychotics (1950s)
worked as a dopamine antagonist: chlorpromazine
Antagonists reduce action of neurotransmitter dopamine
by blocking dopamine receptors in synapses of the brain
this normalises the neurotransmission in keys areas of brain (reducing hallucinations)
chlorpromazine is sedative and is first given to people admitted to hospitals
atypical antipsychotics (1970s)
the aim was to improve the effectiveness of drugs in suppressing symptoms and minimising side effects
two types: clozapine and risperidone
clozapine AO1
developed 1960s but withdrawn in 1970s due to risk of agranulocytosis
it was then remarketed as it was tougher and more effective than TA (but patients do need regular blood tests)
binds to dopamine receptors and acts on serotonin and glutamate receptors
risperidone (1990s) AO1
aim was to produce a drug just as effective as czp but less side effects
small dosage initially given
binds most strongly to dopamine receptors
SZ drug therapy AO3 - evidence for effectiveness
research compared effects for chlorpromazine compared with a placebo (experiences were identical) and found that chlorpromazine was associated with better overall functioning and reduced symptom severity (relapse rate also lower). thus effective (30-50% effective in treatment-resistant cases where typical anti’s have failed.