SBAs in Surgery Flashcards

1
Q

Other name for Hartmann’s?

A

Proctosigmoidectomy.
Resection of the rectosigmoid colon with closure of the rectal stump and formation of an end/terminal colostomy. Used to be common for diverticular disease and cancer.

Can be reversed in 60-70% cases

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2
Q

Chylothorax

A

A chylothorax (or chyle leak) is a type of pleural effusion. resulting from lymph formed in the digestive system (chyle) accumulating in the pleural cavity due to either disruption or obstruction of the thoracic duct, which transports up to 4 L of chyle per day, allowing a rapid and large accumulation of fluid in the chest.

50% malignant (60% lymphoma, esp NHL); 25% trauma (esp cardiothoracic surgery)

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3
Q

The best form of fluid therapy is to replace ____

A

Like with like

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4
Q

Initial first line fluid therapy for trauma patients according to ATLS?

A

2L warmed Hartmann’s solution

Colloids are useful as osmotically active and stay IV, but are used after initial crystalloid

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5
Q

Typical requirements of fluid and electrolytes in a healthy adult

A

1-2.5 L H2O
1-2 mmol/kg Na
0.5-1 mmol/kg K
30-40 kcal/kg/day

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6
Q

Third space losses in bowel obstruction (remember ITU pt laparotomy with Mr Tsochazis)

A

Patients with bowel obstruction sequester huge amounts of fluid within bowel (3rd space). This is salt rich fluid - approx 130 mmol/L Na, 110 Cl, and 10 K

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7
Q

Glasgow Scale Pancreatitis

A
PaO2 < 8kPa (60mmhg)
Age > 55 years
Neutrophils: (WBC >15 x109/l
Calcium < 2mmol/l
Renal function: (Urea > 16mmol/l)
Enzymes: (AST/ALT > 200 iu/L or LDH > 600 iu/L)
Albumin < 32g/l
Sugar: (Glucose >10mmol/L) 

3+ Indicate SEVERE disease

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8
Q

ALI in severe pancreatitis

A

Acute lung injury (ALI) is a complication of systemic inflammation where increased endothelial and epithelial barrier permeability results in leakage of a protein-rich exudate into the alveolar space and interstitial tissues, thus compromising oxygenation and gas exchange. Therefore lungs become susceptible to fluid overload. Presents similarly to acute heart failure.

Bilateral pulmonary oedema, normal cardiac filling pressures, and a ratio of arterial oxygen pressure and inspiratory oxygen concentration (PaO2/FiO2 < 300 mmHg for ALI and < 200 mmHg for ARDS.

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9
Q

CVP fluid challenge monitoring and what is normal?

A

No defined normal value for CVP. increased value in hypervolaemia and cardiac failure.

If after fluid challenge CVP doesn’t change pt is hypovolaemic. If it increases 2-4 mmH20 and goes back down within 30 min = euvolaemia. A sustained increase >5 mm H2O = overload/CF

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10
Q

Define massive blood transfusion

A

Replacement of individuals entire circulating volume within 24h

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11
Q

Complications of massive blood transfusion

A

Stored blood deficient in V and VII.
Additional plt and cryoprecipitate needed to avoid DIC and bleeding
Blood stored at low temp, therfore pt prone to hypothermia
Stored blood high in K, ++K can be a problem
Hypocalcaemia - stored blood anticoagulated with citrate, which Ca ions thus preventing coagulation. In massive transfusion citrate may overwhelm circulating Ca

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12
Q

Hartmanns Composition

A
Na 131 (all mmol/L)
Cl 111
HCO3 29
K 5
Ca 2
Lactate 29
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13
Q

When is Hartmann’s useful, when is it not?

A

First choice resus fluid according to ATLS.

However lower Na than 0.9 saline (131 vs 150) and inability to add extra potassium (as in 0.9 saline) in pts experiencing salt losses makes its value more limited in medical patients.

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14
Q

SIADH

A
Hyponatreamia
Inappropriately high urine osmolality (>200 mEq/L)
Excessive urine Na losses (>30 mEq/L)
Decreased osmolality
Euvolaemic patient w/o signs of oedema
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15
Q

What should be measured daily in TPN patients?

A
Blood glucose
Urea
Creatinine
K
Na
Mg
PO4 - hypophosphate esp problematic in TPN pts
FBC - prone to sepsis
Daily weights and fluid balance charts

** LFTs twice weekly for cholestatic jaundice and fatty hepatitis

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16
Q

In TPN, of which is there more, fats or sugar?

A

Lipid > Carbohydrate (glucose converted to CO2 resulting in more resp work for the acute surgical pt)

Also 14 g Nitrogen as L-AA

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17
Q

Pneumothorax or Hemo/Chylo thorax?

A

With a very resonant percussion note - pneumo

With a dull percussion note - something solid must be filling that hole, either blood or chyle

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18
Q

How often should a central line be changed?

A

Every 5 days

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19
Q

Foramen of Winslow (Omental foramen)

A

Communication between greater and lesser sac of the abdomen. “Behind” CBD

Anterior: hepatoduodenal ligament (two layers containing CBD, HA and PV)
DAVE: Duct, Artery, Vein, Epiploic foramen.

Posterior: peritoneum covering IVC

Superior: peritoneum covering caudate lobe of liver

Inferior: the peritoneum covering the duodenum and the hepatic artery

Left lateral: gastrosplenic ligament and splenorenal ligament

As the portal vein is the most posterior structure in the hepatoduodenal ligament, and the inferior vena cava lies under the posterior wall, the epiploic foramen can be remembered as lying between the two great veins of the abdomen.

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20
Q

Where is portal triad contained anatomically?

A

Within hepatoduodenal ligament

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21
Q

Risk factors for gastric cancer

A
H pylori
Pernicious Anaemia
Previous gastrectomy
Dried fish and cured meats
Smoking and Drinking
Blood group A (wierd)
Atrophic Gastritis
Low social status
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22
Q

What are the branched of the internal carotid before it reaches the cranial vault

A

Trick question - there are none

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23
Q

Oesophagus - structure and blood supply

A

Top 1/3 is stricated muscle supplied by inferior thyroid artery and drained by inferior thyroid veins and deep cervical lymph nodes

Middle 1/3 blend of striated and smooth muscle supplied by descending aorta, drains via azygous vein and posterior mediastinal LN

Bottom 1/3 smooth muscle, supplied by oesophageal branches of left gastric artery and drained via left gastric vein into portal circulation, and LN around ceoliac plexus.

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24
Q

Define achalasia

A

Degenration of the Auerbach plexus leads to aganglionosis and failure to relax of the lower oesophageal sphincter. Idiopathic, most likely between 25-50

Similar to trypanosma cruzi (Chagas disease)

Will present as progressive dysphagia to fluids before solids (opposite to cancer). Reason being is that fluids reach LOS quicker and therfore not enough time for relax. Slow eating of food makes more efficient use of LOS. Weight loss late presentation

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25
Q

Plummer Vinson

A

Severe Iron Deficiency (Koilonychia) + Hyperkeratinasation of proximal 1/3 oesophagus leading to formation of oesophageal web and dysphagia.

26
Q

Describe the components of the gastro oesophageal sphincter

A

3 parts*

  1. LOS - 4cm segment of hypertrophied smooth muscle continous with the distal oesophagus. Maintains pressure 25mmH2O over resting intragastric pressure
  2. Extrinsic sphincter - skeletal muscle fibres of the right crus of diaphragm which slings around oesophagus. Supports LOS at rest but also contracts with diaphragm during inspiration and abdominal straining
  3. Physiological - oesophagus projects 2-3 cm into abdominal cavity therefore rise in intra-abdominal pressure will compress this. In addition angle of His provides a flap valve.
    * Knowledge important when considering effects of Hiatus hernia which will leave LOS without support
27
Q

How common is a hiatus hernia?

A

30% of population >50
50% of those will complain about GORD

8% of hernias are sliding and do not require surgery

Rolling hiatus hernia (paraoesophageal) may require repair due to risk of strangulation.

28
Q

Los Angeles classification of Oesophagitis severity

A

Grade 1 - small mucosal breaks, limited to <2 mucosal folds

Grade 2 - mucosal break >5mm long, limited to <2 mucosal folds

Grade 3 - >2 mucosal folds but <75% oesophageal circumference

Grade 4 - circumferential mucosal breakdown >75% of circumference

29
Q

Complications of oesophagitis

A

Bleeding (usually small volume chronic)
Barrett’s
Malignancy

  1. Conservative (smoke, drink, diet)
  2. Medical (PPI & H2 antagonists) - e.g. omeprazole and ranitidine. These are complimentary therapies
  3. Endoscopy to avoid missing early malignancy may be required
  4. Surgery if symptoms life threatening or v. severe
30
Q

Complications of partial gastrectomy / gastrectomy

A
\++ Risk of malignancy
Obstructions 
Malabsorbtive symptoms - B12, Iron, B9
Early satiety (often desired outcome)
Dumping syndrome - uncontrolled release of large volume of chyme into proximal bowel leads to rapid large volume fluid shifts (transient) causeing hot flushes, palpitations, syncope. Feels similar to hypo attack
31
Q

What drug do you give in oesophageal varices?

A

Propranolol

32
Q

What is the name of the balloon inserted into oesophagus to put pressure on variceal bleed?

A

Sengstaken - Blakemore tube

33
Q

Why can you get RIF pain in perforated PUD?

A

As fluid tracks into peritoneal gutter (gravity), causing inflammation there

34
Q

Kocher’s incision

A

Mostly used in open biliary surgery. Incision 3-5 cm below costal margin.
Anterior rectus sheath exposed and divided along line of incision exposing rectus muscles.
These are divided with care to ensure haemostasis when branches of superior gastric vessels sacrificed.
Eighth and Ninth intercostal nerves lie between internal oblique and transverse muscles - often 8 is sacrificed

35
Q

Lanz Incision

A

Transversely crosses McBurney’s point (2/3 between pubic symphysis and right ASIS. Incision starts 2 cm inferomedial to the right ASIS and extends medially for 5–7 cm.

Good access to appendix and caecum.

Its transverse lie tends to make the iliohypogastric and ilioinguinal nerves more susceptible to division. This can predispose the patient to later inguinal herniation.

36
Q

Inguinal Incision

A

Incision 1 cm above and parallel to the inguinal ligament, from the inner to the outer inguinal ring.

Through the SC fat and Camper’s fascia to expose the aponeurosis of the external oblique

Incision of the aponeurosis of the external oblique from the external inguinal ring to the level of the internal inguinal ring.

ID and protect ileoinguinal nerve

Blunt mobilization of the spermatic cord

37
Q

Blood supply to the stomach

A

All blood derived from coeliac plexus (T12).

Lesser curve supplied by L and R gastric vessels.

Greater curve by R and L gastroepiploic vessels

R gastroepiploic a branch of gastroduodenal, which passes just underneath D1, and is susceptible to erosion by DU (rapid bleed and high mort)

38
Q

How long can a pt have malaena after bleeding stopped

A

As long as 72h (even longer with constipation)

39
Q

Zollinger-Ellison

A

Gastrin-like hormone secreting tumour of the pancreas. Hyperacidity of stomach and therefore severe and extensive ulcers

40
Q

Gallstone composition

A

15% cholesterol
5% pigment stones
80% mixed

41
Q

Most common cause of pancreatitis

A

Gallstone (45%)
Alcohol (25%)
Rest

42
Q

Risk factors for gallstones to elicit in Hx

A
Race (black and asian)
Hypercholesterolaemia (or v. fatty diet)
Obesity
Oestrogen state - COCP, prego, PCOS
Haemolytic states
Crohn's - after surgery loss of terminal ileum
43
Q

Mirizzi’s syndrome

A

Common hepatic duct obstruction caused by an extrinsic compression from an impacted stone in the cystic duct or Hartmann’s pouch of the gallbladder

44
Q

Causes of cholecystectomy postoperative jaundice

A

Gallstone retention
Recurrence
Biliary sepsis
Fibrotic stricturing (usually if too enthusiastic with electrocautery - blunt disssection preferable)
Inappropriate ligation of cystic duct - anatomy of that region displays high variability

45
Q

CC and PBC

A

20-30% PSC pts will develop CC
Majority (vast) of CC pts will have PSC and IBD underlying

Liver flukes
Opisthorchis viverrini
Clonorchis sinensis

46
Q

Whipple’s procedure

A

Cancer of the head of pancreas. Removal of gastric antrum, pylorus, proximal duodenum, gallbladder, cystic duct, head of pancreas.

common hepatic duct preserved and anastamosed to blind loop of jejunum. Jejunum anastamosed to remant pancreas and stomach

47
Q

Blood film post splenectomy

A

Howell Jolly bodies - DNA remnants
Target cells - RBC with membrane abnormality
Ecchinocytes - RBC with membrane abnormality
Pappenheimer bodies - granules in iron loading cells

48
Q

Why is pancreatitis common after splenectomy?

A

Tail of pancrease shares blood supply with spleen

49
Q

Why do pts need antiplatelet therapy post splenectomy?

A

Paradoxical increase in plt count. High risk of thrombosis in first 3 weeks (can persist occasionally).
Aspirin ± Dipyridamole

50
Q

Vaccinations in hyposplenism

A

Encapsulated bacteria

Step Pneumo
Meningococcus B and C
HiB

+ Influenza
± prophylactic penicillin V

51
Q

How much pancreatic juice secreted in one day

A

1500 mL

52
Q

Endocrine functions of pancreas

A

4

Insulin (B cells)
Glucagon (A cells)
Somatostatin (D cells)
Gastrin (G cells)

53
Q

Pancreatic pseudocysts

A

Late complication of pancreatitis, occurs approx 20% cases. ++ chance with %% aetiology
6-8 weeks after initial attack

Persistantly raised amylase with no systemic upset and pyrexia. DDx chronic pancreatitis

Collection of pancreatic juices enclosed by non-epithelial (hence pseudocyst) fibrous layer. Most commonly in lesser sac.
Can become infected (abscess), rupture, erode, pressure (duodenal obstruction)

If small, conservative
If large (>6cm), US/CT guided drainage or open pancreatectomy
54
Q

Villous adenoma vs tubular adenoma

A

Villous colonic adenomas characteristically secrete large amounts of K rich mucus. Only symptoms may be mucous and hypokalaemia. Far higher malignant potential due to level of epithelial dysplasia

55
Q

Liver resection in colonic cancer

A

As long as mets confined to single lobe (gadolinium enhanced MRI to confirm), liver resection and agressive management increases survival to 30% (remember 5yr prognosis for Dukes D cancer 5%)

56
Q

Excising tumours of the transverse colon

A

Technical difficulty in creating colon:colon anastamosis means that left hemicolectomy and transverse colectomy have fallen out of favour. If possible, extended R hemicolectomy

57
Q

Post stoma formation and electrolytes

A

Immediately following stoma formation, large quantities of Na rich secretions may be lost = hypokalaemic metabolic alkylosis as Na conserved in exchange for K and H in renal tubules.
Short term complication

58
Q

Managing sigmoid volvulus

A

Sigmoidoscopy and insertion of flatus tube.

If failure or bowel infarction - laparotomy and colectomy

59
Q

Is OP a complication of CD?

A

No, although can be caused by long term steroid use. Not disease itself though

60
Q

Saint’s triad

A

Association between diverticula, cholelithiasis and hiatus hernia which occurs more frequently in western societies

61
Q

Where is the site of most diverticula and why not the rectum?

A

95% sigmoid colon
Outpouchings between taenia coli which are not true diverticula.

Within rectum, taenia coli become fused.

62
Q

What do you give a IBD patient intolerant to azathioprine?

A

6-mercaptopurine

Although this is the active metabolite of azathioprine (which is not tolerated by 1/3 pts), 50% of those intolerant to azathioprine will respond well to 6mp

First line mesalazine/sulphasalazine

Second line azathioprine/6mp

Third line therapy is methotrexate for CD and ciclosporin for UC

4th line therapy is biologics - infliximab