SAH Flashcards
Causes
- Most commonly, burst aneurysm in subarachnoid space, usually circle of willis
- Trauma
- AVM - arteriovenous malformations
- ICH with extension of bleed into SA space
What happens
Blood leaks into SA space, increasing ICP, therefore decreasing CPP, tissue ischemia, hemorrhagic stroke
Grading SAH from aneurismal burst
Grade 1 - 70% survival • Minimal or no bleed • Slight headache • No neuro deficits - watch and wait interventions
**Grade 2 – 60% survival • Mild bleed • Moderate-severe H/A • Awake and alert • Some nuchal rigidity
**Grade 3 – 50% survival • Drowsiness, confusion • Mild focal deficit(s)
** candidate for surgical intervention
~Grade 4 – 20% survival • Stupor • Mild-to-severe hemiparesis • Poss. early decerebrate rigidity
~Grade 5 – 10% survival • Deep coma • Decerebrate rigidity • Moribund appearance
~ basically gonna die, so usually no surgical
Surgical options for SA aneurysm
- clipping
- coiling
Named theory about intracranial pressures and components
Monroe-Kellie Hypothesis
Intracranial spaces filled with non-compressible components of brain, blood and CSF, increase one and the others have to decrease in volume to accommodate. Ie increase blood, brain herniated out the foremen magnum
- Brain can increase from Tumors Hematomas Abscesses Cerebral edema
- CSF increases in spina bifida, tumours, infection, inhibited CSF reabsortion (in SAH)
- Blood increases in hemorrhage, inc CO2, acidosis, hypoxia (vasodilation), changes in map in absense of autoregulation, venous outflow obstruction (inc PEEP, tight collars/ties)
Cerebral compliance
With good cerebral compliance, A change in volume will not change ICP.
In brain injury, a small change in volume, will change ICP dramatically. Poor cerebral compliance. Same as lungs.
Preop nursing responsibilities
- Control BP, slow/prevent further bleeding
- Adequate CVP for perfusion
- Anticipate fevers - blood in subarachnoid space is irritant
- Manage pain/stimulus to keep demand low
Post Op Nursing responsibilities
- Maintain MAP > 80, SBP 150-180, maintain CPP
- Statins in prevent vasospasms, promotes endothelial NO synthase
- Maintain CO2 within range to avoid vasodil/constrict
- Control ICP with EVD? CSF drain
- Monitor vitals
- HOB keep pressure okay
Symptoms and treatment of vasospasm
- Presents with stroke-like symptoms 3-12 days post op
- Triple H tx
- Hypervolemia - CVP 8-10 - increase blood flow
- Hemodilution - HCT 0.32-0.35 - dec viscosity
- Hypertension - increase MAP by 30% with levo to support CPP
- Nimodopine - Ca channel blocker to prevent and tx vasospasm
How do we manage ICP directly
IVC - intraventricular catheter- inserted directly to ventricle, allowing continuous ICP monitoring
W/ EVD - external ventricular drain - drain away CSF/blood to decrease ICP. Normally 5-15cc/hour out. If >30cc/hr or increase of 10cc/hr more than usual, notify neurologist
-levelled at foramen of Monroe, usually temple