SAH Flashcards

1
Q

Causes

A
  • Most commonly, burst aneurysm in subarachnoid space, usually circle of willis
  • Trauma
  • AVM - arteriovenous malformations
  • ICH with extension of bleed into SA space
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2
Q

What happens

A

Blood leaks into SA space, increasing ICP, therefore decreasing CPP, tissue ischemia, hemorrhagic stroke

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3
Q

Grading SAH from aneurismal burst

A

Grade 1 - 70% survival • Minimal or no bleed • Slight headache • No neuro deficits - watch and wait interventions
**Grade 2 – 60% survival • Mild bleed • Moderate-severe H/A • Awake and alert • Some nuchal rigidity
**Grade 3 – 50% survival • Drowsiness, confusion • Mild focal deficit(s)
** candidate for surgical intervention
~Grade 4 – 20% survival • Stupor • Mild-to-severe hemiparesis • Poss. early decerebrate rigidity
~Grade 5 – 10% survival • Deep coma • Decerebrate rigidity • Moribund appearance
~ basically gonna die, so usually no surgical

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4
Q

Surgical options for SA aneurysm

A
  • clipping

- coiling

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5
Q

Named theory about intracranial pressures and components

A

Monroe-Kellie Hypothesis
Intracranial spaces filled with non-compressible components of brain, blood and CSF, increase one and the others have to decrease in volume to accommodate. Ie increase blood, brain herniated out the foremen magnum

  • Brain can increase from Tumors Hematomas Abscesses Cerebral edema
  • CSF increases in spina bifida, tumours, infection, inhibited CSF reabsortion (in SAH)
  • Blood increases in hemorrhage, inc CO2, acidosis, hypoxia (vasodilation), changes in map in absense of autoregulation, venous outflow obstruction (inc PEEP, tight collars/ties)
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6
Q

Cerebral compliance

A

With good cerebral compliance, A change in volume will not change ICP.

In brain injury, a small change in volume, will change ICP dramatically. Poor cerebral compliance. Same as lungs.

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7
Q

Preop nursing responsibilities

A
  • Control BP, slow/prevent further bleeding
  • Adequate CVP for perfusion
  • Anticipate fevers - blood in subarachnoid space is irritant
  • Manage pain/stimulus to keep demand low
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8
Q

Post Op Nursing responsibilities

A
  • Maintain MAP > 80, SBP 150-180, maintain CPP
  • Statins in prevent vasospasms, promotes endothelial NO synthase
  • Maintain CO2 within range to avoid vasodil/constrict
  • Control ICP with EVD? CSF drain
  • Monitor vitals
  • HOB keep pressure okay
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9
Q

Symptoms and treatment of vasospasm

A
  • Presents with stroke-like symptoms 3-12 days post op
  • Triple H tx
    • Hypervolemia - CVP 8-10 - increase blood flow
    • Hemodilution - HCT 0.32-0.35 - dec viscosity
    • Hypertension - increase MAP by 30% with levo to support CPP
  • Nimodopine - Ca channel blocker to prevent and tx vasospasm
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10
Q

How do we manage ICP directly

A

IVC - intraventricular catheter- inserted directly to ventricle, allowing continuous ICP monitoring

W/ EVD - external ventricular drain - drain away CSF/blood to decrease ICP. Normally 5-15cc/hour out. If >30cc/hr or increase of 10cc/hr more than usual, notify neurologist
-levelled at foramen of Monroe, usually temple

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