RT230: Clinical Medicine II ( 230 study guide ) Flashcards
Physiological effects of Peep/CPAP
All baseline pressers increase
MAP increases
FRC increases
Intrathoracic pressure increases
Increase Compliance
Goals of peep/CPAP
Increase tissue oxygenation
Maintain a PA02 >60 mmHg
Increase Lung compliance
Improve work of breathing
Prevent alveoli collapse and end-expiration
Hazards of PEEP/CPAP
Increase intrathoracic pressure
Decrease venous return
Decrease blood pressure
Decrease cardiac output, loss of the thoracic pump
Increase PVR
Increase ADH
Decrease lung compliance
Indications of PEEP/CPAP
Cardiogenic pulmonary edema
ARDS
Refractory hypoxemia
Recurrent atelectasis
Assist breath triggering with auto-peep
Contraindications of PEEP/CPAP
Unilateral lung disease
Hypovolemia
Hypotension
Increase ICP
Untreated tension pneumothorax
Indications of NIV
hypercapnic respiratory failure secondary to copd
Use of accessory muscles
Respiratory rate >25
Moderate to severe dyspnea
Exclusion criteria NIV
Apnea
Hemodynamic unstable
Uncooperative patient
Facial burns
Facial trauma
High risk of aspiration
Copious secretions
Anatomic abnormalities that interfere with gas delivery
Goals of NIV
Improve gas exchange
Rest respiratory muscles
Increase lung compliance
Avoid intubation
Decrease mortality, length of time on the ventilator, hospitalization, VAP, relieve symptoms of respiratory distress, improve patient-ventilator synchrony
Max patient comfort
Relieve/improve symptoms
Enhance the quality of life
Avoid hospitalization
Increase survival, mobility
Complications of NIV
Discomfort, facial skin necrosis, claustrophobic, nasal bride ulceration
Nasal congestion, sinus or ear pain
Nasal or oral dryness, eye irritation or gastric insufflation, air leaks
Aspiration pneumonia, hypotension, pneumothorax
- *Respiratory acidosis ( lungs retaining too much Co2 )**
- *how do kidneys compensate**
- *pH <7.35, CO2 >45**
Kidneys excrete excess hydrogen & retain Bicarb
- *Respiratory acidosis**
- *Causes**
Drugs ( opioids & sedatives )
Edema ( fluid in the lungs)
Pneumonia
Respiratory center of brain is damaged
Emboli
Spasms of bronchial
Sac elasticity damage( COPD & emphysema )
- *Respiratory Acidosis**
- *Signs & symptoms**
Blood pressure
Respiration rate
Heart rate
Restlessness
Confusion
Headache
Sleepy/coma
- *Respiratory acidosis**
- *Interventions**
Administer O2
Semi Fowler position
Pneumonia: administer antibiotics
Monitor potassium level ( 3.5-5.0 range)
- *Respiratory Alkalosis ( the lungs are losing too much CO2 )**
- *How do the kidneys compensate**
- *pH > 7.35, CO2 < 35**
The kidneys excrete excess Bicarb & retain Hydrogen
Causes of Respiratory Alkalosis
Increase in body temperature
Aspirin toxicity
Hyperventilation
Signs & Symptoms of Respiratory Alkalosis
Respiratory rate > 20
increase heart rate
Confused & tired
Tetany
EKG changes
(+) Chvosteke sign ( twitching of the facial muscles when tapping the facial nerve in response to hypocalcemia
Interventions for Respiratory Alkalosis
Provide emotional support
Fix the breathing problem
Encourage good breathing patterns
Rebreathing into a paper bag
Give anti-anxiety medications or sedatives to decrease the breathing rate
Monitor K+ and Ca levels
Flow of blood
Superior/inferior Vena Cava, right atrium, tricuspid valve, right ventricle, pulmonary valve, pulmonary artier, pulmonary capillaries, pulmonary veins, left atrium, mitral(bicuspid) valve, left ventricle, aortic valve, aorta, systemic
How to fix auto-peep
Decrease respiratory rate
Decrease tidal volume
decrease I time on I:E ratio ( increase E time )
( decrease minute ventilation which is the respiratory rate and tidal volume )
What are the three kinds of dead space
Anatomical
Alveolar
Physiological
What are three ways to improve ventilation
Tidal volume (the first thing you look at )
Respiratory rate
Dead space
What are three ways to improve oxygenation
FiO2
PEEP/CPAP/EPAP
MAP
What are the two methods of collection for end-tidal CO2 ( PETCO2 )
Mainstream uses an in-line analyzer placed between the airway and the ventilator circuit that heats the air and monitors the gas using an infrared source.
Sidestream uses a sampling tube to pump a small volume of gas continually from the ventilator circuit to an external device that measures the CO2
What are the uses of PETCO2
Assess trends in alveolar ventilation
Detect V/Q imbalance
Measure physiologic dead space
Detect esophageal intubation
Assess blow flow during cardiac arrest
Purpose of ventilator waveforms
Detect auto-PEEP, monitor patient-ventilator asynchrony, assess patient trigger effort, troubleshoot ventilator, determine breathe type, identify lung over-distention, minimize WOB, determine appropriate peep, assess effectiveness of bronchodilator administration, monitor patients disease status
Indications for CMV
Apnea, Inability to protect airway, hypoxemic respiratory failure, hypercapnia respiratory failure, impending respiratory failure, inadequate lung expansion, inadequate muscle strength, increase work of breathing
Goals of CMV
Support/manipulate gas exchange, maintain alveolar ventilation and arterial oxygenation, increase FRC, reduce WOB, minimize cardiovascular impairment, ensure patient-ventilatory synchrony, avoid lung damage, reverse hypoxemia, reverse acute respiratory acidosis, prevent or reverse atelectasis, reverse ventilator muscle dysfunction, decrease systemic or myocardial O2 consumption, maintain/improve cardiac output reduce icp, stabilize the chest
Negative effects of CMV
increase mean intrathoracic pressure
Ventilator-associated lung injury
Ventilator-associated pneumonia
oxygen toxicity
Positive effect of CMV
Decrease WOB
increase FRC
improve acid base imbalances
MODE OF VENTILATION ( in order from least to most imposed WOB )
CMV/AC
SIMV
PSV
CPAP
Tidal Volume(TV)
Volume of air inhaled or exhaled during each normal breath
Inspiratory Reserve Volume(IRV)
maximum volume of air that can be inhaled over and above the inspired tidal volume