Revision - Heart Block Flashcards

1
Q

Define a prolonged PR interval

A

> 0.2s

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2
Q

Define 1st degree heart block

A

Consistent prolongation of PR interval due to delayed conduction via AV node

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3
Q

What is the PR inverval on an ECG?

A

From the beginning of the P wave to the beginning of the QRS complex

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4
Q

What does the PR interval represent?

A

The time between atrial depolarisation and ventricular depolarisation

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5
Q

What drugs can cause 1st degree heart block? (4)

A

1) beta blockers

2) rate-limiting CCBs e.g. diltiazem

3) digoxin

4) magnesium

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6
Q

What occurs in Mobitz type I AV block?

A

There is progressive prolongation of the PR interval until eventually the atrial impulse is not conducted, and the QRS complex is dropped.

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7
Q

Why can an inferior MI cause AV block?

A

As the RCA supplies the AV node

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8
Q

Mx of Mobitz type I if patient is asymptomatic?

A

No mx required

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9
Q

Mx of Mobitz type I if patient is symptomatic?

A

Pacemaker may be considered

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10
Q

What happens in Mobitz type II?

A

There is consistent PR interval duration with intermittently dropped QRS complexes due to a failure of conduction.

The intermittent dropping of the QRS complexes typically follows a repeating cycle of every 3rd (3:1 block) or 4th (4:1 block) P wave.

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11
Q

Is Mobitz type II sometimes a normal variant?

A

No - always pathological

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12
Q

ECG findings in second-degree AV (type 2)?

A

1) Rhythm: irregular (may be regularly irregular in 3:1 or 4:1 block)

2) P wave: present but there are more P waves than QRS complexes

3) PR interval: consistent normal PR interval duration with intermittently dropped QRS complexes

4) QRS complex: normal (<0.12 seconds) or broad (>0.12 seconds)

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13
Q

When will the QRS complex be broad in second-degree AV block (type 2)?

A

The QRS complex will be broad if the conduction failure is located DISTAL to the bundle of His.

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14
Q

Symptoms & exam findings in second-degree AV block (type 2)?

A

Symptoms:
- palpitations
- pre-syncope
- syncope

Exam:
- ‘regularly irregular’ pulse

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15
Q

Management of second-degree AV block (type 2)?

A

1) Patients should be placed on a cardiac monitor as soon as possible (due to risk of progression to complete AV block).

2) Investigate underlying cause

3) Temporary pacing or isoprenaline may be required if the patient is haemodynamically compromised due to bradycardia.

4) A permanent pacemaker is usually inserted if there are no reversible causes identified.

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16
Q

What 2 types of escape rhythms can 3rd degree (complete) heart block consist of?

A

1) narrow complex escape rhythms

2) broad complex escape rhythms

17
Q

Narrow complex escape rhythms in complete heart block:

a) QRS interval?
b) where do they originate?
c) typical HR?

A

a) <0.12s
b) above bifurcation of bundle of His
c) >40bpm

18
Q

Narrow complex escape rhythms in complete heart block:

a) QRS interval?
b) where do they originate?
c) typical HR?

A

a) >0.12s
b) below bifurcation of bundle of His
c) <40bpm (more significant clinical features e.g. heart failure, syncope)

19
Q

What 3 infections can cause complete heart block?

A

1) Lyme disease

2) Chagas disease

3) Infective endocarditis

20
Q

ECG findings in 3rd degree heart block?

A

1) Rhythm: variable

2) P wave: present but not associated with QRS complexes

3) PR interval: absent (as there is atrioventricular dissociation)

4) QRS complex: narrow (<0.12 seconds) or broad (>0.12 seconds) depending on the site of the escape rhythm (see introduction)

21
Q

Management of 3rd degree heart block?

A

1) Place on cardiac monitor

2) Transcutaneous pacing/temporary pacing wire or isoprenaline infusion may be required. Some rhythms (particularly narrow-complex escape rhythms) may respond to atropine.

3) A permanent pacemaker is usually required.

22
Q

What is the main complication of 3rd degree heart block?

A

Sudden cardiac death due to ventricular arrhythmias

23
Q

What do the following ECG components represent:

1) p wave
2) PR interval
3) QRS complex
4) T wave

A

1) Atrial depolarisation

2) Conduction through the AVN to the ventricles (i.e. time between atrial and ventricular depolarisation)

3) Ventricular depolarisation

4) Ventricular repolarisation

24
Q

What is the main feature of bundle branch blocks?

A

Broad QRS complexes

25
Q

What are the causes of damage to the right bundle branch?

(i.e. causing RBBB)

A

1) lung pathology:
- COPD
- pulmonary emboli
- cor pulmonale

2) Primary heart muscle disease (ARVC)

3) Congenital heart disease (e.g. ASD)

4) Ischaemic heart disease

5) Primary degeneration of the right bundle

26
Q

What does the left bundle branch split into?

A

Anterior & posterior fascicles

27
Q

Each branch of the left bundle branch may be damaged in isolation.

Is anterior or posterior fascicular block more common?

A

Anterior

28
Q

In the mx of angina in patients already taking a beta blocker, but a CCB is contraindicated, what can be given next?

A

A long-acting nitrate, ivabradine, nicorandil or ranolazine

29
Q

When is aortic valve replacement considered in aortic stenosis?

A

1) patient is symptomatic

2) if asymptomatic but valvular gradient >40 mmHg and with features such as LV systolic dysfunction

30
Q
A