revision Flashcards

1
Q

asymmetry:

minor:
measurements:
comparison:
no of pts with it:
most important area:
A

minor:

  • desirable
  • little inconsistencies are perceived aesthetically pleased

asymmetry of:
5 mm and below -> unnoticed
10 mm -> significant

The greater the degree of asymmetry, the more noticeable and the greater the desire for correction

34% of patients with dentofacial deformities were found to have clinically apparent facial asymmetry

most important area:

  • lower area of face, reflected in the position of the chin (74%)
  • then midface and then upper face
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2
Q

hemifacial microsomia:

clinical features:
results from:
main etiopathogenic units:
etiology:
soft tissue defects:
classification system:
difference in features from Goldenhar syndrome:
tx: (*SOS*)
A

clinical features:

  • mostly unilateral condylar underdevelopment (can be bilateral)
  • abnormalities of the external and middle ear
  • midline deviation, crossbite, deviation of chin and tilting of the occlusal plane, disorders of skin, eye and ear
  • can vary from slight asymmetry to complete absence of one ear, small ipsilateral face, facial nerve palsy and cleft of mouth corner
  • It can be also manifested at both sides of the face simultaneously in 10-15% of patients

results from:
malformation of the 1st and 2nd branchial arches
-occurs in 1/3500 = 1/5600 births

main etiopathogenic units:
condyle and angle

etiology:

  • heterogeneous
  • congenital disease
soft tissue defects:
•skin tags
•facial clefts
•cranial nerve function
•soft palate function
•ear abnormalities
•bulk of subcutaneous soft tissue
•muscles of mastication and facial expression
•macrostomia

OMENS:
= orbit, mandible, ear, nerve and soft tissue

Goldenhar syndrome:
addition of epibulbar dermoids and vertebral anomalies

tx:

  • ortho and surgery
  • if mild with sufficient muscle mass and good mandibular mobility: orthopedics and orthodontics only
  • must start 6-8y old; early tx
  • it varies on: patient’s age, degree of facial deformity, and degree of skeletal deformity
  • extend of TMJ involvement determines timing and type of treatment
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3
Q

hemimandibular hyperplasia:

how common is it?
clinical features:
when does it begin:

A

PANORAMIC X-RAY
-excessive growth of the condyle in one of the two sides

uncommon maxillofacial deformity

clinical features:

  • increase in ramus height
  • rotated facial appearance
  • kinking at the mandibular symphysis
  • prominence of the lower border of the mandible
  • maxillary and mandibular alveolar bone overgrowth
  • compensatory canting of occlusal plane
  • serious functional malocclusion
  • diffuse enlargement of the condyle, the condylar neck, the ramus, and the body of the mandible
  • clear hyperactivity in the condyle
  • actively proliferating cartilage !!

begins:
usually before puberty

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4
Q

condylar fractures:

if left undiagnosed:
% of cases that can cause this:
tx:

A

can lead to:
•Face asymmetries
•Severe malocclusion
•TMJ ankylosis

25% (75% doesn’t produce asymmetry)

tx:

  • IMF for a few days
  • physiotherapy
  • non-surgical condyle manipulation or open surgery
  • orthopedics-orthodontics

-> aims to minimize the destructive influence of fracture and surgery to future mandibular growth

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5
Q

tmj akylosis:

=   (*SOS*)
classification:
caused by:
most common cause?
clinical features:
tx: (*SOS*)
goals of surgical tx:
A

Chronic Hypomobility and Growth Disorders

Intracapsular adhesions or ossification between the disc and temporal articular surface that attach the disc-condyle complex to the articular eminence

classification:

  • degree of limitation (partial or complete)
  • union location (intracapsular or extracapsular)
  • tissue types involved (fibrous, osseous, fibro-osseous)
caused by:
»Trauma (fracture / hemarthrosis) 
»Previous joint surgery
»Systemic or local infections
»Tumors
»Systemic diseases

most common:
trauma

clinical features:

  • limited mouth opening w/o pain
  • present for a long time
  • pt does not feel that it poses a significant problem
  • condyle can still rotate, with some degree of restriction on the inferior surface of the disc
  • decreased opening
  • lateral movements are restricted
  • opening pathway deflects to the ipsilateral side

tx:

  • Immediate surgical release of the joint
  • Use of functional appliances for dentoalveoral influence and symmetrical function and growth

goals of surgical tx:

  • Restore range of motion and function
  • Avoid development of malocclusion
  • Prevent recurrence of ankylosis
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6
Q

juvenile rheumatoid arthritis:

what does it cause?
what does it affect:
history should include:
how do we assess the occlusal plane tilting?

A
  • produces bone resorption -> which causes: posterior rotation of the mandible, anterior open bite and if it is asymmetrical it produces also facial asymmetry
  • has an effect on both bone and muscle because it is an inflammatory disorder
  • damage to joint -> joint function change -> muscular function change -> mandibular morphology development changes -> unstable occlusion -> mandibular function decreases
history should include:
•face trauma
•bone & muscle inflammatory disorders
•continuously progressing face & dentoalveolar asymmetry
•syndrome or craniofacial anomaly

occlusal plane tilting assessed with:
with a spatula

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7
Q

why the patient in the max opening deviates to lateral deviation? /what is the dd of this opening of this person? (picture)

A

anatomical
functional (locking of joint, inability of full translation)
muscular (spasm)

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8
Q

MRI uses:

A
  • only when we want to assess intra-articular problems relating to disc, condyle and fossa (condyle disc fossa relationship)
  • only for TMJ internal assessment
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9
Q

hypodontia:

=
classification:
most common tooth missing, excluding the M3:
syndromes associated:
dental anomalies commonly associated:
diagnosis:
impact:
A

= developmental absence of 1 or more deciduous/permanent teeth, excluding M3

classification:
mild: 1-2 missing teeth
moderate: 3-6 missing teeth
severe/oligodontia: > 6 missing teeth
anodontia: complete absence of teeth in 1 or both dentitions

most common tooth missing:
lower P2

syndromes associated:
•Cleft lip and palate
•Ectodermal dysplasias
•Oral-facial-digital syndromes
•Down syndrome
•Wiktop syndrome
•Van der Woude syndrome
•Ehlers-Danlos syndrome
•Incontinentia pigmenti

dental anomalies:

  • hypoplastic enamel
  • ectopic maxillary canine tooth position
  • microdontia
  • molar taurodontism
  • conical crown shape (peg shaped I2)
  • transposition !!
  • supernumerary teeth !!
  • infra-occlusion
  • delayed teeth eruption
diagnosis:
HISTORY –CLINICAL EXAMINATION
•Abnormalities in the eruption of teeth
•Prolonged retention of deciduous teeth
•Inconsistencies in eruption timing between contralateral teeth more than 6 months
•Deviation from the normal eruption sequence
•Generalized delay in dental development
RADIOGRAPHIC EXAMINATION
•OPG

impact:
reduces quality of life for those affected due to detrimental effects on appearance (esthetics), function (mastication), and psychosocial well-being

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10
Q

Adjunctive orthodontic treatment:

=
examples:

A

= tooth movement carried out to facilitate other dental procedures necessary to control disease and to restore function

examples:

  1. Uprighting of abutment teeth
    - tooth loss -> drifting into space
    - with this you can facilitate placement of replacement of prosthetic teeth
  2. Redistribution or closure of spaces
    - replace the space with an abutment tooth
    - roots may need to be repositioned to permit implant placement
  3. Intrusion of over-erupted teeth
    - tooth loss side effect: over-eruption of opposing teeth
    - can interfere with restoration of space so we can intrude the over-erupted tooth
  4. Extrusion of fractured teeth
    - in order to bring the fracture line supragingivally to allow placement of a restoration
    - there is a limit to this, as excess extrusion will reduce the amount of tooth supported by bone, reducing the crown-to-root ratio
  5. Alignment of anterior teeth
    - alignment of healthy teeth with restorations is not an acceptable procedure
    - conservative tx options (ortho, bleaching, enameloplasty) should be offered
    - a clinician should present only treatment options that involve predictable, conservative restorations or that preserve healthy tooth structure
  6. Crossbite correction (functional with trauma)
  7. Adjunctive ortho applications in implantology
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11
Q

risks of orthodontic tx:

A
  1. root resorption
  2. loss of periodontal support
  3. demineralization
  4. enamel damage
  5. intra-oral soft tissue damage
  6. pulpal injury
  7. extra-oral damage
  8. failure to achieve tx objectives

(study slide 152)

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12
Q

root resorption:

etiology:
types:
post-tx:
risk factors:

A

etiology:
multifactorial
-> apical displacement tx duration and genetics play a role

(study slide 116 of revision)

types: (it is not a single entity)
1. moderate generalized resorption:
- inevitable
- 1-2 mm resorption during tx
- affects almost all teeth
- no consequences
- no action needed

  1. severe generalized resorption:
    - unknown etiology (related to auto-immune response?)
    - doesn’t matter if you do ortho or not
    - rare
    - not orthodontist fault
    - risk of tooth mobility -> tx: follow up and give instructions
  2. severe localized resorption:
    - loss of more than ¼ of the root length of some teeth, usually maxillary incisors
    - in 2-3% of ortho pts
    - related to ortho tx
    - one cause is movement of root apices against the lingual cortical plate
    - risk of tooth mobility -> tx: follow up and give instructions

post-tx:
x-ray exam is mandatory, and the patient and referring dentist should be informed if a root resorption has occurred

Long-term, a tooth with a short root has a very favorable prognosis and need not be extracted and replaced by an implant or other restoration

risk factors:

  • shortened roots w/ evidence of previous root resorption
  • pipette-shaped or blunted roots
  • teeth with previous trauma
  • patient habits (nail biting)
  • iatrogenic: excessive forces, intrusion, prolonged tx time
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13
Q

loss of periodontal support:

risk of harmful effects with tooth movement explanation:
cortical laminae thickness explanation:
with a perio patient we need to have:

A

-as long as a tooth can be moved within the envelope of the alveolar process, risk of the development of harmful side effects in the marginal tissue is minimal regardless of the dimensions and quality of the soft tissue

The thickness of the cortical laminae varies in different locations. In the incisor and canine region, the cortical bone plate at the labial aspect of the teeth is considerably thinner than at the lingual aspect

need:
adequate medical and dental history, appropriate intraoral x-rays and complete perio chart

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14
Q

most important iatrogenic effect of fixed appliance orthodontic therapy:

A

Decalcification of enamel

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15
Q

preventive dentistry:

explain it:
measures to be taken:

A

To prevent the development of lesions and assure proper tx, the clinician must be familiar with the major aspects of caries process and current principles of it

Unesthetic discoloration or resin remnants and discolored caries lesions formed during ortho tx

measures to be taken:

  • Good OH
  • Fluoride dentifrice (x2 daily) and rinse (x1 daily)
  • Avoidance of carbonated soft drinks (may induce enamel erosion)
  • OH monitoring (ex: gingival bleeding)
  • Complex appliances that complicate plaque removal should be avoided on the maxillary anterior teeth
  • Topical F solns, gels or varnishes
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16
Q

enamel damage:

A
  1. enamel color alterations:
    - color of natural teeth is changed in various ways after fixed appliances ortho tx both short-term and long-term
    - resulted from the irreversible penetration of resin tags
    - associated with bonding, debonding and cleaning procedures
    - extrinsic and intrinsic discoloration of the remaining adhesive material
  2. tooth wear:
    - especially with ceramic brackets
    - ceramic brackets are contraindicated on the mandibular anterior teeth in occlusions with deep overbite and minimal overjet
    - during maxillary incisor retraction, the overbite should be reduced first so that the maxillary incisors do not contact the mandibular ceramic brackets
    - care should also be taken not to bond ceramic brackets on the mandibular canines when they are in Class II relationship
    - Band seaters, band removers, and brackets removal can cause fracture of enamel , or even whole cusps in heavily restored teeth
    - During removal of adhesives, the debonding burs can cause enamel damage, particularly if used in a high-speed handpiece
    - Certain components of orthodontic appliances can cause wear to opposing teeth if there is heavily occlusal contact during function
17
Q

Intra oral soft tissue damage:

A
  • Ulceration can occur during tx as a result of direct trauma from both fixed and removable orthodontic appliances. Lesions generally heal within a few days without lasting effect
  • Intra-oral allergic reactions to ortho components are rare but have been reported in relation to Ni, latex, and acrylate
  • Management depends on the location and severity of the allergic rxn and the scope for modifying tx
  • Orthodontic wires and brackets contain Ni and Ni allergy is increasing in frequency. Its prevalence has been reported to be approximately 10%, being more common in females
  • Intraoral signs are non-specific and have been reported to include erythematous areas and severe gingivitis despite good OH
18
Q

pulpal injury:

A
  • Excessive apical root movement can lead to a reduction in blood supply to the pulp and even pulpal death
  • Teeth which have undergone a previous episode of trauma appear to be particularly susceptible, probably because the pulpal tissues are already compromised
  • Any teeth that have previously suffered trauma or are judged to be at risk of pulpal injury require thorough examination prior to ortho tx, and any therapy should be delivered with light force and monitoring
19
Q

extra-oral damage:

A
  • It is important that when tx planning to correct malocclusion, the impact on overall facial appearance is considered
  • Recoil injury from the elastic components of headgear poses a rare but potentially severe risk of damage to the eyes
  • Contact dermatitis is reported in approximately 1% of the population and allergic rxns may be seen on facial skin in response to components of appliances, usually Ni
20
Q

Failure to achieve tx objectives:

operator factors:
patient factors:

A

operator factors:

  • error in diagnosis
  • error in tx planning
  • anchorage loss
  • technique error
  • poor communication
  • inadequate experience/training

patient factors:

  • poor OH/diet
  • failure to wear appliances/elastics
  • repeated appliance breakage
  • failure to attend appointments
  • unexpected unfavorable growth
21
Q

Impacted canines - adverse effects:

A
  • Root resorption of adjacent teeth was detected in more than 2/3 of 60 ppl
  • Extraction of primary canines in the mixed dentition may increase the chance of subsequent eruption of palatally displaced permanent canines in the long-term
22
Q

The American Association of Orthodontists recommends that all children have a check-up with an orthodontic specialist no later than:

A

7 years old

23
Q

TMD:

=
etiology:
may be associated with:
most common clinical markers:
can be divided into:
can ortho cause it?
strategies to control TMD pain:
occlusion is a contributory factor in the development in TMD signs and symptoms by:
A

= a collective term embracing a no of clinical problems that involve the masticatory musculature, the TMJs or both

etiology: multifactorial !
psychological
hormonal
traumatic
occlusal factors
genetic
sleep disorders
depression
stress
-> parafunctional activity can contribute to muscle pain and spasm
may be associated with:
•stressful habits
•emotional disorders
•structural malrelationships
•trauma to the face or head
•occlusal disharmonies
•headaches
•non-painful muscle hypertrophy
•abnormal occlusal wear
•osseous alterations of the joints

most common clinical markers:
•limited range of mandibular motion
•muscle and TMJ tenderness
•TMJ sounds

can be divided into:

  • internal derangements of disc and related structures
  • disorders of chewing muscles
  • degenerative joint and bone conditions
  • developmental abnormalities

ortho tx, either alone or in combination with extractions, cannot be reliably shown to either “cause” or “cure” TMD

  • The development of TMD cannot be predicted
  • Once TMD is present, TMD cures cannot be assumed or assured
  • No method of TMD prevention has been demonstrated
  • TMD are primarily a pain management problem

strategies to control TMD pain:
•teaching the patient how to reduce oral habits, stress and anxiety -biofeedback
•applying physical therapy and exercises to restore function (neck, shoulders, TMJs)
•using acetosalicylic acid or ibuprofen for their analgesic and anti-inflammatory effects
•use of intraoral splints

occlusion is a contributory factor in the development in TMD signs and symptoms by:

  • Initiating
  • Perpetuating
  • Predisposing

A lot of persons who are considered as TMD patients have other problems. They are misdiagnosed because they mimic TMD

24
Q

internal derangements of disc and related structures:

Anterior disc displacement with reduction:
Vs
Anterior disc displacement without reduction:

SOS

A

Anterior disc displacement w/ reduction:
/clicking
= disc displaces anteriorly when the mouth is closed, the patient opens the mouth and click happens and this takes position between the condyle and eminence but when the mouth closes again it displaces

Vs

Anterior disc displacement w/o reduction:
/locking
= disc is permanently displaced anteriorly when the mouth is closed, the mouth tries to open and disc is still displaced anteriorly and stays there for the whole period
-if it is acute then the mouth cannot open too much
-if chronic the pt has learned to push the disc and open a little more

25
Q

splints - mechanism of action:

A
  • occlusal disengagement
  • vertical dimension
  • maxillomandibular realignment
  • TMJ repositioning
  • cognitive awareness
26
Q

optimum goal of TMJ tx:

A
  • Optimum Condyle / Disk Position
  • Optimum Integrated Muscle Activity
  • Maximal Occlusal Stability
27
Q

screening examination:

A

All patients candidates for orthodontic therapy, independently of the type of malocclusion, must get the same attention during screening

•No evidence of TMD:
proceed with ortho tx
•Evidence of TMD:
differential diagnosis
•Recommend ortho tx:
observe TMD

-Stabilize TMD before any ortho tx

28
Q

medical history:

special attention must be given to:

A
  • inflammatory bone & muscle disorders
  • face and head trauma
  • chronic facial pain
29
Q

clinical examination for TMD - search for:

A
  • Pain
  • Limitations
  • Noises
  • Deviations
30
Q

dentists should deal only with:

SOS

A
  • TMJ internal derangements of the disc and related structures
  • Disorders of the chewing muscles and myogenic type of pain
  • Tension type headaches