Review: OAT GI patient Flashcards
How do you determine whether an SD is primarily MSK or secondary to viscerosomatic reflex?
Failure of SD to respond to OMT points to viscerosomatic problem
Pain patterns help to make a ddx in pts with GI complaints. In the GI tract, ____ ____ and ______ are found in walls of viscera and are highly sensitive to stretch, spasm, inflammation, and ischemia
Pacinian corpuscles; free nerve endings
Describe visceral pathology as a result of a viscerosomatic reflex
Increased stretch/irritation to GI nerves —> increased afferent signals to CNS —> afferent fiber synapse in dorsal horn of spinal cord
Prolonged afferent activity leads to facilitation of the neurons and corresponding spinal segments
Visceral pathology results in somatic changes paraspinally as paravertebral TTA and increased tenderness based on increased sensitivity of segment from spinal facilitation [pattern usually reflexes to soma on side of organ]
What is the percutaneous reflex of Morley?
Direct transfer of inflammatory irritation from viscera to peritoneum (not a visceral afferent reflex!)
Ex: appendicitis —> peritonitis; morley reflex responsible for abdominal wall rigidity, pain, and rebound tenderness
Sympathetics components of GI system
Thoracic splanchnic n —> celiac and superior mesenteric ganglion
Lumbar splanchnic n —> inferior mesenteric ganglion
Celiac ganglion is T5-T9 = distal esophagus, stomach, proximal duodenum, liver, gallbladder, spleen, portions of pancreas
SMG is T10-11 = distal duodenum, portions of pancreas, jejunum, ascending colon, proximal 2/3 of transverse colon
IMG is T12-L2 = distal 1/3 of transverse colon, descending colon, sigmoid colon, rectum
Parasympathetic components of GI system
Vagus n., pelvic splanchnic nn (S2-4)
[note anterior vagal trunk = left vagus n., posterior vagal trunk = right vagus n.]
Right vagus n. = lesser curvature of stomach, liver/gallbladder, small bowel, right colon to mid-transverse colon
Left vagus n. = greater curvature of stomach, ends at duodenum
Pelvic splanchnics = descending colon, sigmoid colon, rectum
Possible sympathetic-related pathologies of GI tract
Ileus
Constipation/flatulence (increased water absorption)
Abdominal distention with hypoactive bowel sounds
Possible parasympathetic pathologies associated with GI tract
Increased secretion of all GI glands
Diarrhea/fecal incontinence (decreased water absorption); hyperactive bowel sounds
Conditions associated with GI autonomic neuropathy
Gastroparesis, GERD, achalasia, cyclic vomiting syndrome, IBS, reflux esophagitis, etc
During inhalation, as the thoracic diaphragm contracts and moves _____, the pelvic diaphragm moves _____
Inferiorly; inferiorly
[reversed with exhalation — thoracic and pelvic diaphragm both expand and move superiorly — acts as a pump for movement of vasculature and lymphatic fluids]
Significance of vasculature and lymphatics in the abdomen, particularly in reference to acute abdominal etiologies
Acute etiologies often require surgical intervention. Potentially resulting vasculature and lymphatic stasis can cause bacterial overgrowth, leading to systemic sepsis
Inflammation and infection increase metabolic process — more ILs and other cytokines
Lymphatic congestion leads to accumulation of waste, decreased medicine distribution, decreased cell nutrition, increased fibrosis/scarring, worse prognosis of IBD, bloating, cramps, reduced oxygenation
3 large collecting intestinal nodes and where they drain
Celiac (drains stomach, duodenum, spleen, liver)
Superior mesenteric (drains jejunum, ileum, ascending/transverse colon)
Inferior mesenteric (drains descending/sigmoid colon, rectum)
These large intestinal nodes drain into cisterna chyli (L1-L2 area, lies on abdominal aorta) —> thoracic duct —> L subclavian v.
What are some GI disorders influencing the metabolic energetic model?
Crohns
Celiac
Food sensitivity (lactose intolerance)
Sleep disorders (OSA)
Thyroid d/o
Inflammatory conditions, malabsorption d/o, inability to eliminate metabolic waste
Metabolic changes associated with diarrhea
Hyperthyroidism
Hyperkalemia
Diarrhea may lead to metabolic acidosis (loss of sodium bicarb)
Metabolic changes associated with constipation
Hypothyroidism
Hypercalcemia and hypokalemia
Note that geriatrics are predisposed to constipation d/t decreased physical activity