Respiratory System Flashcards

1
Q

Types of breathing

A

Diaphragmatic: typical for men, diaphragm mainly involved

Costal/thoracic type: typical for women , intercostal muscles are more involved

Costal diaphragmatic:

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2
Q

What diseases cause difficulties in intercostal-diaphragmatic breathing
What breathing do they do then

A

Diaphragm diseases, emphysema, pleuritis, ascites, flatulence, pregnancy, tumours in abdominal cavity

Breathing mainly thoracic/costal

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3
Q

What happens when a patient has difficulty in thoracic breathing
When does the is occur

A

They are forced to breathe diaphragmatically

In inspiratiry muscle paralysis, broken ribs, emphysema, pneumofibrosis

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4
Q

What is normal respiratory rate

A

Number of inspirations and exhalations in one min
12-20 breaths per min

Impaired breathing may include changes in frequency, depth,rhythm and minute ventilation

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5
Q

What are some quantitative changes in breathing

A

Bradypnea- slow breathing RR<10 breaths /min

Tachypnea- rapid breathing RR>20breaths/min

Apnea-temporary cessation of breathing. Interruption of airflow in upper respiratory tract by more than 90% of normal

Hyperpnea- deep rapid breathing.respiratory volume increases

Hypopnea-reduced depth of breathing due to decreased sensitivity of respiratory center , weakness in respiratory muscles,loss of alveolar volume

Hyperventilation: increased ventilation of lung due to accelerated and deep breathing. Decreases co2

Hypoventilation: increase in partial pressure of co2

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6
Q

When does bradypnea occur

A

Hypothyroidism
Increases intracranial pressure
Brain tumours
Menignitints
Intoxication w opioids
Uremia/diabetic coma

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7
Q

When does tachypnea occur

A

Physical overload
Fever
Pneumonia
Resp and heart failure
Pulmonary embolism

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8
Q

When does apnea occur

A

Sleep related breathing disorders
Alcohol
Brain damage
Poisoning w opiates
Asphyxia

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9
Q

When does hyperpnea occur

A

Hypoxemia
Shock
Ketoacidosis
Renal failure
Sepsis

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10
Q

When does hyponea occur

A

Obstructive sleep apnea- oro nasal airflow is decreases by 50%

Myasthenia gravis
Neuromuscular dystrophic desases
Multiple sclerosis
Respiratory distress syndrome
Alveolar edema

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11
Q

When does hyperventilation occur

A

Panic attacks

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12
Q

When does hypoventilstion occur

A

Obesity
Respiratory disorders during sleep

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13
Q

What are some qualitative changes in breathing

A

Cheyne-stokes : alternating cycles of hyperpnea and hypopnea then apnea. During apnea pupils narrow, pulse slows, cyanosis, loss of consciousness

Kussmaul breathing : pathological deep breathing, noisy. Form of hyperventilation

Biots breathing : similar to cheyne stokes. Sudden start

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14
Q

What is the difference between cheyne stokes and biots breathing

A

In biots, the apnea stage is longer lasting hot to 1 min
In cheyne stokes, apnea phase is 15-30 sec

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15
Q

When can you have cheyne stokes breathing
When can you have biots breathing
When can you have kussmaul bretahing

A
  1. Severe heart failure, ischemic stroke, cerebral hemorrhage, brain tumours, drug abuse, hyperglycemia
  2. Brain tumours, brain hemorrhages, encephalitis, meningitis
  3. Ketoacidosis, diabetes
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16
Q

What is dyspnea
What can cause it

A

Shortness of breath

Obstruction in resp tracts
Ventilation perfusion mismatch
Impaired gas exchange through alveolar capillary membrane
Disturbed ventilation

17
Q

Types of dyspnea

A

Inspiratory: obstruction/ narrowing of upper respiratory tract ( larynx,trachea) due to tumours , infections
Prolonged inhalation 2:2
On chest, there will be depression in jugular and supraclavicular fossae (inspiratory tirage)

Expiratory: obstruction/ narrowing of lower airways like bronchial asthma,COPD, tumours in bronchi
Prolonged exhalation 1:3
Sitting position=arms support body

Restrictive: both phase of breathing disturbed ratio 1:1
Occurs in acute pulmonary diseases and chronic parchymal lung diseases, pleuritis, pneumothorax,chest wall diseasss
So decreased elasticity and fibrosis of lung

18
Q

What is cyanosis

A

Bluish discolouration of skin and visible mucus membranes

Due to non oxidised hemoglobin increases in capillary blood > 3.1 mmol/l

19
Q

What is central cyanosis
What is peripheral cyanosis

A

Central= decrease in oxygen saturation of arterial blood
Seen as bluish grey discolouration of skin, lips tongue,
Ear lobes, sublingual muscoa
In COPD, tuberculosis, severe pneumonia, pulmonary edema

Peripheral= increased oxygen consumption by tissues therefore decreases
O2 in blood (hypoxemia) and in tissues ( hypoxia)
Bluish violet skin, skin cold, expressed on fingers, tip of nose,
ear clams ,lips NOT expressed well tongue and sublingual
In shock, congestive heart failure, vessel clots

20
Q

Do lungs have pain receptors

A

No

21
Q

What is chest pain
Types

A

Occurs due to pathological changes in pleura, heart, oesophagus, mediastinum, sometimes abdominal organs

Parietal pain: originates from chest wall and parietal pleura then propagates along intercostal nerves. Pain is superficial and localised . Pain intensifies during coughing ir deep breathing
Diseases associated with pleura

Acromial pain is a type of parietal pain which caused by pleura, peritoneum, ant diaphragm. Translated by phrenic nerve so pain felt in scapula, shoulder, treaoszius muskxe

Visceral pain: diseases of heart, pericardium, large blood vessels, oesophagus, mediastinum
Deep pain usually behind sternum and radiates to shoulders neck etc