Respiratory Mechanics CPBC Flashcards

1
Q

Fill in the gaps

A
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2
Q

what 3 areas of the brain participate in the control of breathing?

A
  • medulla
  • pons
  • cortex, limbic system
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3
Q

What are the 2 respiratory groups of the medulla?

A

ventral respiratory groups

  • pre-boetzinger complex –> central pattern generator
  • boetzinger complex (aka SA node for breathing) –> expiratory neurons

dorsal respiratory groups

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4
Q

What are the 2 respiratory centers of the pons?

A

apneustic center –> stimulates inspiratory neurons of the dorsal and ventral respiratory group (medulla)

pneumotaxic center –> regulates volume and rate

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5
Q

how does ICH/TBI affect the breathing pattern

A

stimulation of the apneustic center of the pons –> gasping type breathing

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6
Q

What is the function fo the cortex/limbic system in control of respiration?

A

voluntary control/ emotions

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7
Q

where are the central chemoreceptors for breathing located and how are they stimulated?

A

in the medulla

blood CO2 diffuses into the CSF and increase [H+]/ decreased pH –> increase respiratory rate

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8
Q

where are the peripheal respiratory chemoreceptors located and how do they affect breathing?

A

cortid and aortic bodies

sense hypoxemia, hypercarbia, acidemia or decreased perfusion –> increase ventilation via the glossopharyngeal nerve

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9
Q

Where are the baroreceptors located and how do contribute to ventilation

A

carotid and aortic bodies

mostly concerned with circulation –> but sever hypotension will induce hyperventilation

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10
Q

what are the three types of breathing receptors located within the lungs?

A
  • stretch receptors
  • irritant receptors
  • J receptors
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11
Q

what are the stretch receptors within the lungs?

A

located within the smooth muscles of the airways –> distention –> vagus nerve stimulated –> inhibits apneustic center

= inhibits respiration, prevents overdistention via the Hering-Breuer Reflex

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12
Q

how do the irritant receptors within the lungs affect respiration?

A

lay between airway epithelial cells

detect noxious gases, cigarette smoke, dust, cold air

cause bronchoconstriction and hyperpnea (=increased depth of breathing)

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13
Q

How do J receptors affect respiration?

A

lay in juxtacapillary and alveolar walls

stimulated by interstitial fluid or engorged pulmonary capillaries –> stimulates rapid, shallow breathing

are very sensitive –> even mild pulmonary edema not showing up on radiographs will stimulate recpetors –> first sign of volume overload is increased respiratory rate

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14
Q

Fill the gaps

A
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15
Q

Fill the gaps

A
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16
Q

Fill the gaps

A
17
Q

Fill the gaps

A
18
Q

Fill the gaps

A
19
Q

What is the normal atmospheric pressure at sea level?

A

760 mmHg

20
Q

The atmospheric pressure ___ as altitdue increases

A

decreases

21
Q

What is the average pleural (thoracic) pressure?

A
  • 4 cm H2O (but changes from top to bottom)
22
Q

how is the lung distorted by its weight?

A

alveoli on top are stretched and expanded by the lung underneath

alveoli at the bottom are compressed by lung above

23
Q

what is the range from pleural pressure from top to bottom of the lungs?

A

ranges from -10 to -2.5 H2O

24
Q

what is the transpulmonary pressure?

A

airway pressure minus pleural pressure

25
Q

fill the gaps

A
26
Q

what is resistance?

A

force that slows motion, here: gases

primarily determined by the diameter of the airways

resistance = change of ressure/flow

27
Q

what is compliance?

A

the ability to stretch while under pressure = distensibility

compiance = change in volume/change in pressure

28
Q

fill the gaps with increased, decreased and normal

A
29
Q

what are “fast alveoli”?

A

alveoli higher up in the lungs, they fill up quicker and need less volume to increase pressure (low compliance)

30
Q

what are slow alveoli?

A

lower alveoli in the lungs, more diffuclt to inflate but can take more volume per unit of pressure = higher compliance

31
Q

What is Hysteresis?

A

= the reluctance or delay of elastic structures to accept the deformation imposed by an applied stress

–> compliance is not the same during inspiration and expiration –> lung volume at any given pressure lower during inspiration than expiration

most of this elastic recoil force is caused by surface tension

32
Q

What is “flow”?

A

change in pressure/ resistance

33
Q

what is resistance?

A

change in pressure/ flow

34
Q
A