Respiratory emergencies Flashcards

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1
Q

Principal function of lungs

A

Respiration (O2 and CO2 exchange)

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2
Q

Where does gas exchange take place

A

Alveoli

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3
Q

Respiration

A

Brainstem senses level of CO2 in arterial blood

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4
Q

What happens when CO2 levels are too low

A

Slower rate of breathing, and less deep- less CO2 expired helping carbon dioxide levels in blood return to normal

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5
Q

Normal breathing rates

A

Adult: 12 to 20 breath/min
Child: 15 to 30 breath/min
Infant: 30 to 60 breath/min

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6
Q

Signs of normal breathing

A
  • Normal inhalation and exhalation
  • Clear and equal bilateral breath sounds
  • Regular and equal chest rise and fall
  • Adequate tidal volume (depth)
  • Unlabored: w/o abnormal breath sounds (wheezing, stridor)
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7
Q

Signs of inadequate breathing

A
  • Pt reports difficulty or shortness of breath
  • Pt has AMS w shallow or slow breathing
  • Adult pt appears anxious or restless
  • Respiratory rate too fast or too slow
  • rhythm irregular
  • Skin: pale, cool, clammy, cyanotic
  • Pursed lips, nasal flairing
  • Tripod position
  • Very little speaking
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8
Q

Hypoxic drive

A

Chronically low levels of O2 in blood that stimulate respiratory drive- seen in pt w chronic lung disease

*Use caution when administering O2 to these pt

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9
Q

Dyspnea

A

Shortness of breath

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10
Q

Causes of dyspnea

A
  • Pulmonary edema
  • Hay fever
  • Pleural effusion
  • Obstruction of airway
  • Hyperventilation syndrome
  • Environmental/industrial exposure
  • Carbon monoxide poisoning
  • Drug overdose
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11
Q

Dyspneic pt may have

A

Gas exchange between alveoli and pulmonary circulation obstructed by fluid in lungs, infection, or collapsed alveoli

  • alveoli damaged and cannot trnaport gas properly across the walls
  • air passes obstructed by muscle spasm, mucus, or weakened
  • blood flow to lungs obstructed by blood clot
  • pleural space filled w air or excess fluid (lungs cannot properly expand)
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12
Q

Dyspnea common w

A

Cardiopulmonary diseases

-CHF causes heart to pump ineffectively and deprive body of O2- pulmonary edema common w CHF

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