Respiratory Conditions Flashcards

1
Q

What is respiratory compromise

A

Occurs when the blood contained in the capillaries of both lungs stops or interrupts its normal process of oxygen absorption.
Blood isn’t oxygenated.

This leads to lack of oxygen in the blood stream - vital organs and body tissues receive inadequate supply - hypoxia. They will ultimately fail and cease to function.
Doesn’t happen straight away. But danger of brain damage within 2-3 mins

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2
Q

Respiratory compromise affects the…

A
Respiratory tract (nose to lungs)
Respiratory mechanism (ribs, Intercostal muscles and diaphragm)
Respiratory centre (in the brain).
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3
Q

What are the 5 Causes of respiratory compromise?

A

Obstruction of air passages
Chest or lung trauma (injury as opposed to medical cause)
Paralysis of respiratory nerves (don’t get to where they are supposed to be or don’t get sent)
Non oxygen atmosphere
Lung disease and illness

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4
Q

Obstruction of air passages - how and what happens

A

Obstruction by…
Tongue during unconsciousness
Maxillo facial injuries (injuries to front of face)
Blood and vomit
Inhaled foreign bodies e.g food and false teeth
Suffocation
External pressure to the neck (hanging or strangulation )
Drowning
Internal swelling of the throat tissues (burns, scalds, stings, anaphylactic reaction, disease)

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5
Q

What can Chest or lung trauma result from?

A

External compression - sand, masonry, damaged vehicle and crowd pressure
Blast injury

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6
Q

Paralysis of respiratory nerves and muscles may arise from…

A
Electrocution
Head or spinal injuries 
Poisons such as cyanide, week killers and toxic fumes
Drugs such as barbiturates and morphine
Disease such as tetanus and polio
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7
Q

Non oxygen atmospheres can result from…

A

Gases e.g carbon monoxide are absorbed by the bloodstream and progressively replace oxygen.
Chemical fumes
Smoke
Altitude

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8
Q

Lung disease and illness can lead to chronic respiratory failure…

A
Emphysema 
Pulmonary oedema 
Acute pneumonia 
Asthma/bronchitis
Continuous fits
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9
Q

How do we recognise what is going on? Obstruction

A
Difficult and noisy breathing (disponea)
Cyanosis (skin colour starts to go blue)
Dilated pupils (getting bigger)
Congestion of the veins in the head and neck 
Fits
Gradual loss of consciousness 
Petechial haemorrhage
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10
Q

Signs (when there is a paralytic origin)

A

Progressive deterioration in rate and depth of breathing (quiet unless obstructed by tongue).
Increasing cyanosis
Dilated pupils
Gradual loss of consciousness

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11
Q

Management

A

Ensuring an open airway (using appropriate position techniques)
Commencing cardiopulmonary rescuitation if neccessary
Administer high flow oxygen /(target SP02 94-98%)
Placing the patient in the recovery position if unconscious or semi recumbent if conscious
Reassure the patient

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12
Q

Respiratory diseases cause hypoxia how?

A

Block airway

Or impaired perfusion of the alveoli. (Messes up external respiration)

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13
Q

Obstructive airway conditions

A

COPD (chronic obstructive pulmonary disease)
ASTHMA

These conditions cause obstruction in the pulmonary tissues reducing free flow of air into the lungs.

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14
Q

What is COPD?

A

Airway obstruction mainly in smoker or ex smokers
E.g
Chronic Bronchitis
Emphysema

Approx 3.7 million people with COPD in UK but only 90,000 diagnosed.
Fifth biggest killer kills about 30,000 a year.

Chronic = over time, a,ways there?
Acute =

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15
Q

Sign vs symptom

A

Symptom - something the patient reports

Sign - something you can see

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16
Q

COPD sign and symptoms

A

Not all patients may have these and don’t have all of them

Easily fatigued
Frequent respiratory infections 
Use of accessory muscles to breathe
Orthopneic (difficulty when lying down)
Thin in appearance 
Wheezing 
Dyspnea 
Chronic cough (long term)
Digital clubbing -
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17
Q

What is bronchitis?

A

Inflammation of the mucous membranes in the bronchial tubules, caused by bacterial or viral infections.
Have more goblet cells than a normal person.
Acute bronchitis can be common in very young and the elderly and normally short lived.

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18
Q

What is acute bronchitis

A

In previously healthy individuals it is often viral.
Bacterial infections with organisms such as strep. Pneumonia and H influenza is a common sequel to viral infections in smokers and those with COPD.

Damage caused by irritation of the airways leading to inflammation and neutrophils (WBC) infiltrating the lung tissue.
Mucosal hylersection is promoted by a substance released by neutrophils.
Further obstruction to airways caused by goblet cells.

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19
Q

Things to look for in acute bronchitis

A

Initially irritating, unproductive cough together with discomfort behind sternum.
Tightness of the chest with associated respiratory wheeze and shortness of breath.
Cough becomes productive, the sputum being yellow suggestive of live neutrophils or green suggesting dead neutrophils.
Mild fever
Crackles heard upon auscultation (in bronchi region about T5)

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20
Q

What is chronic bronchitis

A

Productive cough on most days for at least 3 months of the year for more than one year.

Inflammation of the bronchus and bronchioles leading to enlargement of mucous secretion glands which can block airways and only cleared by patient coughing the obstruction up.

Blue and bloated
May not appear breathless but will be even after minimal effort.
May have pitting Odema.
If hypoxia is severe then levels of consciousness may be decreased.

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21
Q

What is ACVPU?

Measure levels of conscious

A

A - alert
C - new confusion
V - verbal (you as a paramedic talking to your patient e.g. hello can you hear me
P - pain. Only allowed to illicit pain - give pain response to two areas. Trapezius muscle you can pinch this or the ear lobe.
U - unconscious

22
Q

Typical bronchitic patient

A
Airway flow problem due to mucous and inflammation
Cyanosis (blue colour)
Recurrent cough and sputum production
Hypoxia
Respiratory acidosis. 
Increased haemoglobin level - as body is trying to increase oxygen carry. Even though no oxygen to carry but doesn’t realise.
Exertional dyspnea. 
Ask history of smoking 
Digital clubbing 
Increase respiration rate 
Cardiac enlargement
Use of accessory muscles to breathe
Can lead to right sided heart failure and then bilateral pedal edema.
23
Q

What is emphysema?

A

Distension of the alveoli and destructive changes in their membranes. (Get larger and break open, so lose their large surface area which they need for gas exchange).

24
Q

Emphysema stimulus to breathe

A

Not normally cyanotic
Hypoxia is still the driving factor of the respiratory system.

Our stimulus to breath is high levels of c02. Emphysema patients have to recognise low levels of 02 instead to start breathing. So only give 28% oxygen (normally) as if we were too give them too high oxygen the body wouldn’t recognise that they need to breathe as not detecting low levels of 02.

25
Q

Emphysema signs

A

Breathe out through pursed lips
Thin
Normally maintain colour as still diffusion still happening unlike bronchitis.
Tachycardia (fast HR above 100)
Tachpnoea (fast respiration rate e.g. more than 20 per min
Dyspnoea
Blood pressure elevated
Use of accessory muscles with difficulty healing
Maybe cyanosis in acute attack
Confusion
Abnormally large chest
Oedema
Cardiac dysrhythmias may be evident on ECG.
Elderly
Hyper resonance on chest percussion

26
Q

Treatment/management of COPD

A

SCENE/SMART - scene safety, mechanism of injury, airway?, triage
Ensure open airway, use suction if neccessary.
Follow JRCALC UK ambulance services clinical practie guidelines oxygen administration. Aim for oxygen saturation 88-92%
Consider salbutamol for wheeze or exacerbation
Consider ipratropium bromide
Place or allow patient to be in most comfortable position to aid breathing.
Be prepared to assist ventilations or full CPR.
calm patient to maintain and reduce effort of breathing
Document everything that you do
Hypoxia presents a greater risk than hypercapnia. Do not withold high conc 02 from a COPD patient who needs it.

27
Q

What is the hypoxic drive theory

A

In some patients the respiratory centre may be relatively insensitive to c02 and respiration is driven by hypoxaemia.

28
Q

What is asthma?

A

One of the most common medical conditions

Acute episodes (short lived reversible) can be brought on by many factors such as infection, environmental factors and hyperactive airways responding to inhaled irritants.

Most calls occur when patients own attempts haven’t worked.

29
Q

What happens with asthma

A

Chronic airway inflammation and increased hyper responsiveness

Inhalation of allergen leads to spasm and bronchi constriction

Increased bronchial mucus production (goblet cells)

Swelling of the mucosal cells that line the bronchioles

30
Q

Asthma signs can be divided into

A

Moderate asthma exacerbation

Acute or severe asthma attack

Life threatening asthma attack

31
Q

Moderate asthma

A

Increasing symptoms
Peak flow >50-75% of best/predicted
No features of severe asthma attack

32
Q

Acute/severe asthma

A

Unable to complete sentences in one breath

Respiratory rate >25% in adult and pulse rate above 110BPM

Peak flow 33% -50% of best/predicted

33
Q

Life threatening attack

A
Exhaustion
Bradycardia
Hypotension
Peak flow <33%
SPO2 less than 92%
Cyanosis
Confusion 
Silent chest
34
Q

Asthmatic patients are often prescribed two types of medication

A

Prevent - Anti inflammatory drugs includimg steroids. Include beclomethoasone and fluticasone. Work on smooth muscle and with regular work can eradicate symptoms.

Relievers - drugs such as salbutamol, terbutaline, titropium and ipatropium bromide. These work rapidly on bronchi and bronchioles to relax smooth muscle.

35
Q

Asthma management

A

Scene/smart
Open airway
Position patient upright
02 as per JRCALC
adrenaline 1:1000 in life threatening asthma as per JRCALC
nebulised salbutamol as per JRCALC
nebulised ipratropium bromide as per JRCALC
Try to achieve SP02 levels >94%
Calm patient
Remove possible intrinsic/extrinsic causes
Be prepared to assist ventilations or full CPR
Document all interventions and basic observations
Rapid transportation.

36
Q

Asthma standards

A

Need to record respiratory rate - no exceptions to this
Need to record peak flow (not doing this at the moment)
Need to record SP02 (before treatment)
Salbutamol administered
Oxygen administered

37
Q

Signs and symptoms of airway obstruction

A

Productive cough (green/yellow sputum)
Cyanotic
Disponea
Etc.

38
Q

Impaired perfusion if alveoli

A

Many conditions cause gas exchange between alveolus and pulmonary blood capillary to become restricted.

39
Q

Pneumonia

A

Inflammatory condition if e lung, especially of the alveoli,.

Usually cause by a range of bacteria

Either localised, such as when whole or one love is affected or diffuse when they primarily affect the lobules of both lungs, often in association with the bronchi and bronchioles

40
Q

Pneumonia clinical features

A

Varies according to the immune state of 5e patient and the infecting agent

Often a preceding history of a viral infection with the patient rapidly becoming ill with a high temp up to 39.5 degrees cel, pleuritic pain and a dry cough which develops a day or two later into rusty coloured sputum.

The patient breathes rapidly and shallowly >30 BPM. The affected side of the chest moved left and signs of consolidation may be present with hypo-resonance.

41
Q

Pleurisy

A

Inflammation of the pleural lining which leads to the drying up of serous fluid found between the pleura.

Often occurs in conjunction with pneumonia

Symptoms includ sharp chest pain, worse on breathing and movement and a dry cough.

May be relieved through shallow breathing. But this means wont be able to remove bacteria on chest

Pleuritic rub may be heard upon auscultation over the affected area.

42
Q

What is pulmonary embolism?

A

Blood clot that sits in the pulmonary artery. Prevents blood from going to get oxygenated.

Blockage of artery in the lungs by a substance that has travelled from elsewhere in the body via the blood stream (embolism).

Symptoms include pain, shortness of breath, sometimes coughing up blood.

43
Q

Risk factors

A
Extended travel
Prolonged bed rest
Obesity
Oral contraceptives
Varicose veins - blood sits and gets stale and clots
Surgery of throat, abdomen, pelvis and legs
Fractures of pelvis and legs 
Diabetes
Trauma
Pregnancy
44
Q

Pulmonary embolism

A

Lungs ventilated but not perfused

Right heart failure as trying to bump blood heard

45
Q

Things to look for in PE

A
Dyspnoea
Pleuritic chest pain
Tachypnoea
Pleural rub
Hyporesonance upon percussion 

In big PE
Severe chest pain, evidence of shock, pale, sweaty, tachycardia and tachypnoea, absent breath sounds, syncope,

46
Q

Management of a PE

A
Scene/smart
Open airway
Administer oxygen at high flow rate
Cardiac monitor
Prepare to ventilate or perform CPR
monitor 
Document
47
Q

What is tuberculosis?

A

Is the worlds leading cause of death from a sunken infection disease, resulting from
Inadequate programs for infection control
Multiple drug resistance
Co-infection with HIV
rapid rise in young adult population

Often symptomless
Lymph node enlargement can suppress the bronchi leading to collapse of segments or lobes of the lungs

48
Q

Features of TB

A

Patients may complain of aching chest
Pleural effusion or pneumonia
Finger clubbing

49
Q

Industrial lung diseases

A
Disease as a result of work 
E.g.
Coal dust (pneumoconiosis)
Silicosis (quarry dust)
Asbestosis 
Byssinosis (cotton, flax and hemp fibres)

Symptoms: Cough, sputum (quality, colour, any solid material in there, taste or smell)

50
Q

What Is hyperventilation syndrome

A

A rate of breathing that exceeds metabolic needs and higher than that required to maintain normal c02 levels in the blood.

It can occur in a number of life threatening sistuations including PE, asthma, diabetic ketoacidosis
Also anxiety and stress

51
Q

Signs of hyperventilation syndrome

A
Acute anxiety
Tetany (cramps including sharp bending of wrists and ankle joints)
Numbness and tingling of mouth and lips
Aching*of chest muscles
Light headed