Respiratory Assesment Flashcards
What to Look for/What patients tell us?
wheezing, chocking, spluttering, shortness of breath, coughing (productive-produces mucus or non-productive). Pain when inhaling/exhaling, fatigue, night sweats
4 Different Types of Respiration Rhythms and What Causes them
Tachypnoea - Rapid Breathing Caused by; cold weather, pain, drugs, shock, anxiety, exercise, chest, infection
Bradypnea - Slow Breathing Caused by; drugs usually opioid overdose
Apnea - Not Breathing Caused by; cardiac arrest, chocking
Cheyne-Stokes Respiration Respiration increase in rate/depth then become shallow and slow, Apneas may occur. Seen at end of Life
Kussmaul Respirations Caused by; Hyperventilating, Metabolic Issues
Observations in Effort of Breathing (7)
- Bent Forward
- Tripoding
- Use of Accessory Muscles
- Fatigue/Tiredness
- Hypernea - Abnormal rapid or deep
- Diaphragmatic - Belly breathing
- Costal Breathing - Intercostal Driven
What are we Listening For? (Normal Sounds)
- Bronchial Sound (loud, snorkel like sound)
- Bronchovesicular (Medium, sound of narrowing airway)
- Vesicular (Soft, low pitch - lung bases/soft high pitch - apical)
What are we Listening For? (Adbnormal Sounds)
Adventitious Sounds:
- Wheeze
- Narrow airways or turbulent airflow
- Upper Respiratory/broncho & Broncho-vesicular areas
- Fluid
- Lung Bases
Walking on Snow Sound = Pleurisy
What Respiratory Support can we Offer?
- Give oxygen
- Pain relief
- Coach breathing
- Change positioning
Different Oxygen Delivery Devices (8)
- Nasal cannula
- Face mask
- Face tent
- Oxymizer
- Venturi mask
- Non-rebreather
- High flow nasal cannular
- Bipap
Saturation Levels in Patients (Healthy vs Type 2 Respiratoy Failure)
- If 94%+ in healthy patient good
- If a patient is at risk of type two respiration failure should get oxygen levels up to 88% to 92%
2 Types of Respiratory Failure
Type1 - Hypoxic Failure Caused by damage to lungs eg infection, trauma, oedema therefore respond well to oxygen therapy
Type 2 - Hypercapnic Failure Caused by ventilation being inadequate to clear CO2 therefore doesn’t respond well to oxygen therapy
What happens when you ventilate COPD Patients
- In COPD patients, O2 exchange is reduced due to damage to alveolar sacs
- The body responds by remodelling the capillary beds by pulmonary vasoconstriction to the sacs
- This means that they don’t get O2 nor can’t expel CO2
- So when we ventilate the blood vessels will dilate in the presence of O2
- AAR more CO2 will build and any O2 will be expelled backwards
- Secondarily the Haldane effect when blood increases acidity, the ability to hold O2 decreases while the affinity for O2 decreases (changes)