Rehab Exam 4 Review Flashcards

1
Q

Life expectancy of someone with MS

A
  • No change in life expectancy with proper maintenance
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2
Q

Pathophysiology of MS

A
  • T-lymphocytes attach to and destroy myelin sheath
  • Disruption causes demyelination
  • Neural transmission is slowed or stopped; nerves fatigue rapidly
  • Inflammation, edema, infiltrates surround acute lesion and interfere with nerve conductivity
  • Most susceptible areas: Optic nerves, periventricular white matter, spinal cord, cerebellar peduncles
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3
Q

Exacerbation (regarding MS patient)

A
  • Significant decline in functional capability, sudden increase in clinical signs (8 manifestations - sensory, pain, visual, motor, speech and swallowing, cognitive, affective, autonomic)
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4
Q

2 factors that can contribute to an exacerbation

A
  • Heat

- Stress

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5
Q

Strategies that can be used during interventions involving strengthening and conditioning exercises

A
  • Start interventions early
  • Alternate days and during times of lower core temps (am) to prevent rapid fatigue (outpatient)
  • Balance exercise with rest periods
  • Incorporate resistance/circuit training
  • Manage core temp and prevent overheating
  • Consider cog deficits when implementing program
  • Integrate functional training and closed chain
  • Incorporate group exercise
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6
Q

Most likely cause of pain in MS patient

A
  • Demyelination (demyelinating lesions) of pain tracts (most common in spinothalamic tracts)
  • 80% pts Chronic neuropathic pain
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7
Q

Causes of depression in MS patient

A
  • Loss of function
  • Disease itself (related directly to MS)
  • Side effect of medication
  • Lesion located - whatever affect is controlled can lead to depression
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8
Q

Which affect characteristic is more common in later stages of MS?

A
  • Euphoria
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9
Q

Focus of PT interventions for MS patients

A
  • Education (increase awareness of sensory deficits)
  • Compensation (for sensory loss)
  • Neuroplasticity
  • Depends if pt is exacerbated or not
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10
Q

More appropriate for MS pt that has declined significantly in function - power of manual w/c

A
  • Power w/c: Need to conserve energy, pt fatigues faster
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11
Q

Pathology of Parkinson’s Disease

A
  • Abnormality of basal ganglia
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12
Q

PD is due to a decline in the production of dopamine - true or false?

A
  • True. Degeneration of neurons producing dopamine.
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13
Q

Impairments characteristic of PD

A
  • Rigidity
  • Pill-rolling tremor
  • Difficulty initiating movement (bradykinesia)
  • Postural instability
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14
Q

“On-Off” State in PD

A
  • Refers to medication (Levodopa) - when on, it causes increase in dopamine and a return to normal function, eventually shuts off when L-Dopa wears off)
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15
Q

Posture of PD pt

A
  • Presents with flexed, stooped posture; increased kyphosis; COM forward to limits of stability
  • Narrow BOS
  • Increased sway
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16
Q

Clinical presentation of sensation in PD patient

A
  • No primary sensory loss (ex: light touch)
  • Restlessness
  • Impaired proprioception
17
Q

Ortolani’s Sign

A
  • Attempts to reduce hip dislocation by relocating the hip
  • Postiive sign: Positive for hip dysplasia - hip is subluxed or dislocated and can be reduced - palpate hip moving over acetabular ridge
  • Negative sign: Hip cannot be relocated (hip dislocation)
18
Q

Barlow Maneuver

A
  • PTA flex, adduct hip
  • Attempts to dislocate/sublux the hip
  • Femoral head exits acetabulum; Positive is click with flex/add, also with abd as hip clicks back in
19
Q

Causes of hydroencephalus

A
  • Overproduction of CSF
  • Failure of absorption of CSF
  • Obstruction of the CSF flow
20
Q

Arnold Chiari Malformation

A
  • Posterior cerebellum herniates through the foramen magnum
  • Brainstem displaced caudally
  • Obstructs CSF flow in the 4th ventricle
  • Traction on lower CN
21
Q

Most severe type of Spina Bifida

A
  • Myelomeningocele
22
Q

Syringomyelia (Hydromyelia)

A
  • CSF collects in pockets along SC and creates areas of pressure
  • Signs include rapidly progressing scoliosis, progressive UE weakness, hypertonia
  • Can occur with SB or in isolation
23
Q

Post-conceptual age of newborn (5 wks old) who was birthed at 28 weeks

A
  • AA prior to achieving term age
  • GA + CA
  • 33 PCA
24
Q

Adjusted Term Age of Newborn in prior example

A
  • CA - #wks missed in utero

- 5 - 14 = -9 TAA

25
Q

NICU Levels

A
  • Level 1: Minimal observation or care; small community hospital; “Well-baby nursery”
  • Level 2: Step down from Level III; continued care from Level III, IV meds or alimentation, tube feedings, O2; regional or community hospitals
  • Level 3: Highly specialized services for sickest and most fragile infants - complex, medical interventions, advanced diagnostic testing, surgery, resp. support; Teaching hospitals and affiliated with medical schools
  • Level 4: Level III plus ECMO (extracorporeal membrane oxygenation)
26
Q

Reasons pre-term baby might not feed

A
  • Learned oral aversion resulting from NICU care
  • Lack of flex/ext balance and chin tuck to assist with sucking, swallowing, breathing
  • Residual lung disease may cause tachynea; interferes with sucking and swallowing
  • Lack of self-regulation; unable to calm self with environmental stressors
  • Apnea
  • Bradycardia
  • Unable to coordinate sucking, swallowing, breathing, and becomes physiologically unstable
  • Immature GI Tract
27
Q

Importance of surfactant

A
  • Lowers the surface tension in lungs and allows alveoli to maintain its shape
  • Prevents respiratory distress syndrome (RDS)
28
Q

Osteopenia

A
  • Decrease in bone density
  • Occurs because of immature bone development
  • 80% of bone formed between 24 and 40 weeks; 3rd semester essential for bone formation
29
Q

Meconium Aspiration Syndrome

A
  • During delivery, fetus may pass BM into amniotic fluid; as infant gasps for first breath, may aspirate meconium-tainted fluid
  • Meconium particles obstruct airway, interfere with gas exchange; leads to respiratory distress
30
Q

Brachial Plexus Injury - Position of baby’s arm

A
  • Baby’s arm in “waiter tip” position
  • Shoulder ADD, IR, elbow ext, pronation, wrist and finger flexion
  • UE limp at infant’s side