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week 60 > Red & White Lesions- Dr. Flores > Flashcards

Red & White Lesions- Dr. Flores Flashcards

1
Q

Frictional Hyperkeratosis

A
  • occurs in areas that are commonly traumatized
    • chronic rubbing or friction
    • lips
    • lateral tongue
    • buccal mucosa
  • macrophages & epithelial cells produce IL-8
    • causes overexpression of keratinocyte chemokine (KC)
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2
Q

What condition?

A

Frictional Keratosis

(hyperkeratosis without dysplasia)

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3
Q

Management of a white lesion suspected to be from mechanical irritation

A
  • remove source of irritation
    • will regress in absence of stimulus
  • patient returns in 2-3 weeks
  • If lesion has not regressed=biopsy
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4
Q

What condition?

A

Frictional Hyperkeratosis

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5
Q

Leukoplakia

A
  • when you cannot
    • identify source of white lesion
    • diagnose as any other condition
  • White lesion that does not rub off
  • Clinical TERM only
    • no diagnostic meaning
      • never seen on biopsy report as diagnosis
  • most common premalignant oral manifestation-85%
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6
Q

Benign Alveolar Ridge Keratosis

A
  • on edentulous alveolar ridge
    • Hyperkeratosis w/o dysplasia
      • similar to frictional hyperkeratosis
    • usually in patients who do not wear dentures
      • retromolar pod
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7
Q

Hairy Tongue

A
  • overgrowth of the dorsal tongue
    • filiform papillae
  • Predisposing factors:
    • Broad spectrum antibiotics
    • oxygenating mouth rinses
    • smoking
    • radiation
    • stem cell transplant
  • Management:
    • identify & eliminate predisposing factor
    • improve oral health
    • tongue scraper
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8
Q

What condition?

A

Benign Alveolar Ridge Keratosis

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9
Q

What condition?

A

Hairy Tongue

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10
Q

What condition?

A

Hairy Tongue

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11
Q

Smokeless Tobacco Keratosis

A
  • Associated with
    • Squamous Cell Carcinoma
      • most common malignancy from smokeless tobacco
    • Verrucous carcinoma
      • snuff dipper’s canncer
      • uncommon
      • low-grade malignancy
  • Clinical features
    • stretching reveals snuff pouch (tobacco pouch)
    • soft fissured gray-white lesion that blends in with surrounding mucosa
    • induration (hardened), pain, and ulceration are NOT associated
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12
Q

What condition?

A

Smokeless Tobaccoo Keratosis

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13
Q

What condition?

A

Smokess Tobacco Keratosis

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14
Q

What condition?

A

Smokeless Tobacco Keratosis

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15
Q

Oral Submucous Fibrosis

A
  • Pale, Marble-like stiff mucous in betel quid users
  • precancerous condition
  • Chief complain
    • trismus and generalized oral burning sensation
    • intolerance to spicy foods
  • Betel Chewers Mucosa
    • not precancerous
    • Brown-red mucosa
  • Betal quid lichenoid lesions
    • tooth staining
    • attrition
    • perio
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16
Q

What condition?

A

Oral Submucous Fibrosis

17
Q

Nicotinic Stomatitis

A
  • aka Smoker’s Palate
  • associated with tobacco smoking
    • develops in response to the heat
  • Dry mud apperance
    • Palatal mucosa is grey w/numerous pappules w/red center
  • Reverse smoker’s palate
    • should resolve w/stopping in 1 month
18
Q

What condition?

A

Nicotinic Stomatitis (Aka Smoker’s Palate)

19
Q

What condition?

A

Nicotinic Stomatits (smoker’s palate)

20
Q

What condition?

A

Nicotinic Stomatitis (smoker’s palate)

21
Q

Actinic Cheilitis

A
  • Actinic Cheilosis
    • farmer’s lip
    • sailor’s lip
  • common premalignant alteration of LOWR LIP Vermilion
  • results from long-term UV exposure
  • Clinical:
    • rare in younger than 45 y.o.
    • Males 10:1
    • slow developing lesion
    • overtime Scaly lesion develops sometimes w/ulceration
      • suggests SCC
22
Q

What condition?

A

Actinic Cheilitis

23
Q

What condition?

A

Actinic Cheilitis

24
Q

White Sponge Nevus

A
  • aka Cannon Disease
  • mutation- Keratin 4 & 13
  • white plaque on buccal mucosa, nasal, and anal mucosa
  • Unique:
    • eosinophilic condensation in perinuclear region of cells in superficial layers of epithelium
25
Q

What condition?

A

White sponge nevus

26
Q

What condition?

A

White Sponge Nevus

27
Q

Hereditary Benign intraepithelial Dyskeratosis

A
  • Primarily affects decendents of triracial people originally from NORTH CAROLINA
  • Duplication in chromosome 4q35
  • developed during childhood
  • Oral lesions:
    • thick, corrugated white plaques in buccal & labial mucosa
  • Occular lesions
    • gelatinous plaques of bulbar conjuctiva
      • adjacent to cornea
28
Q

What condition?

A

Hereditary Benign Intrapeithelial Dyskeratosis

29
Q

Oral Hairy Leukoplakia

A
  • Related to Epstein-Barr Virus infection (EBV)
  • common oral manifestation of imunosuppression due to HIV
  • Etiology:
    • HIV infections
    • Hematologic neoplasms
    • post-transplant proliferative disorders
    • Prolonged used of corticosteroids
30
Q

Candidiasis

A
  • Caused by Candida Albicans
    • apart of normal oral flora in 30-50%
    • Dimorphism
      • Yeast form
      • Hyphal form-invasion of host tissue
  • 3 factors of infection:
    • immune status
    • oral mucosal enviroment
    • C. Albicans strain
31
Q

Pseudomembranous Candidiasis

A
  • aka Thrush
  • exposed to broad-spectrum antibiotics or immunocompromised
    • affects infants
  • Clinical presentation:
    • buccal mucosa, palate, dorsal tongue
    • white plaques that resemble cottage cheese
    • scrape off to remove
  • Symptoms:
    • burning sensation or unpleasant taste
32
Q

What condition?

A

Pseudomembranous Candidiasis (Thrush)

33
Q

How do you manage a dental lesion that is able to be scraped off?

A
  • Antifungals
34
Q

Hyperplastic Candidiasis

A
  • aka Candidal Leukoplakia
  • idiopathic
  • immunosuppression
  • locations:
    • tongue
    • anterior buccal mucosa
35
Q

Erythroleukoplakia

A
  • red & white patches
36
Q

What condition?

A

Erythroleukoplakia

week 60 (9 decks)

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