RC 110 EXAM 2 Flashcards

1
Q

how to conduct pt interview

A

IDENTIFY PT

COMMUNICATION - CONSIDER PT EMOTIONAL STATE, ENVIRO. FACTORS, AND CULTURAL BELIEFS

AVOID LEADING QUESTIONS - USE OPEN ENDED STATEMENTS

EX WHEN DID SYMPTOMS START
WHAT MAKES IT BETTER OR WORSE

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2
Q

PARTS OF AN EFFECTIVE INTERVIEW

A

SUBJECTIVE INFO

OBJECTIVE (MEASURABLE)

PT IMPRESSION OF HEALTH - INCLUDING OF DESCRIPTION OF ONGOING AND PAST MED HX

ESABLISH RAPPORT

SHOW INTEREST AND UNDERSTANDING - WHICH ENHANCES PARTICIPATION IN IDENTIFYING CONDITIONS

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3
Q

NORMAL VALUES FOR VITALS

A

BP - 120/80
RR -12-20
HR - 60-100
SPO2 -95-100%
TEMP 37 C

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4
Q

What is the normal range for adult human body temperature in Celsius?

A

37 degrees Celsius.

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5
Q

True or False: A normal resting heart rate for adults is typically between 60 and 100 beats per minute.

A

True.

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6
Q

Fill in the blank: The normal blood pressure range for adults is typically less than _____ mmHg systolic and _____ mmHg diastolic.

A

120; 80.

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7
Q

What is the normal range for fasting blood glucose levels in mg/dL?

A

70 to 99 mg/dL.

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8
Q

Which of the following is considered a normal respiratory rate for adults? A) 10-15 breaths/min B) 12-20 breaths/min C) 20-25 breaths/min

A

B) 12-20 breaths/min.

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9
Q

What is the normal range for cholesterol levels in mg/dL?

A

Total cholesterol should be less than 200 mg/dL.

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10
Q

True or False: A normal range for adult hemoglobin levels is 12 to 15 g/dL for women and 14 to 16 g/dL for men.

A

True.

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11
Q

What is the normal range for serum creatinine levels in mg/dL?

A

0.6 to 1.2 mg/dL.

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12
Q

Fill in the blank: A normal body mass index (BMI) is between _____ and _____ kg/m².

A

18.5; 24.9.

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13
Q

What is the normal range for electrolytes like sodium in serum, in mEq/L?

A

136-142

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14
Q

True or False: Normal levels of potassium in serum are between 3.8 and 5.0 mEq/L.

A

True.

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15
Q

Which of the following is considered a normal range for triglycerides in mg/dL? A) Less than 150 B) Less than 200 C) Less than 250

A

A) Less than 150.

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16
Q

What is the normal range for white blood cell (WBC) count in cells per microliter?

A

4,500 to 11,000 cells/µL.

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17
Q

Fill in the blank: The normal range for platelet count is _____ to _____ cells per microliter.

A

150,000; 450,000.

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18
Q

True or False: A normal range for adult calcium levels in serum is 4.5 to 5.4.

A

True.

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19
Q

What is the normal range for thyroid-stimulating hormone (TSH) in mIU/L?

A

0.4 to 4.0 mIU/L.

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20
Q

Which of the following is a normal range for body pH? A) 7.0-7.2 B) 7.35-7.45 C) 7.5-7.7

A

B) 7.35-7.45.

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21
Q

Fill in the blank: The normal range for bilirubin levels in adults is _____ to _____ mg/dL.

A

0.1; 1.2.

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22
Q

What is the normal range for lactate in serum in mmol/L?

A

0.5 to 2.2 mmol/L.

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23
Q

True or False: A normal range for serum albumin levels is 3.5 to 5.0 g/dL.

A

True.

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24
Q

What is the normal range for serum magnesium levels in mg/dL?

A

1.7 to 2.2 mg/dL.

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25
Fill in the blank: A normal range for prothrombin time (PT) is _____ to _____ seconds.
11; 13.5.
26
What is the normal range for INR (International Normalized Ratio) for patients not on anticoagulants?
1.0.
27
True or False: A normal range for C-reactive protein (CRP) is less than 1.0 mg/L.
True.
28
What is the normal range for urine specific gravity?
1.005 to 1.030.
29
NAMES FOR ABNORMAL VITALS
30
What is the term for abnormally high blood pressure?
Hypertension
31
What is the opposite of hypertension?
Hypotension
32
True or False: Tachycardia refers to a heart rate that is slower than normal.
False
33
What is the medical term for an abnormally fast heart rate?
Tachycardia
34
What is the term for an abnormally slow heart rate?
Bradycardia
35
Fill in the blank: An elevated body temperature is called ______.
Fever
36
What is the medical term for low body temperature?
Hypothermia
37
True or False: A normal respiratory rate is typically between 12 to 20 breaths per minute.
True
38
What is the term for an abnormally high respiratory rate?
Tachypnea
39
What is the name for an abnormally low respiratory rate?
Bradypnea
40
What is the term for an abnormally high level of glucose in the blood?
Hyperglycemia
41
What is the opposite of hyperglycemia?
Hypoglycemia
42
True or False: Hyperthermia is the same as a fever.
False
43
What is the medical term for high cholesterol levels?
Hyperlipidemia
44
What is the term for abnormally low oxygen levels in the blood?
Hypoxemia
45
Fill in the blank: The term for excessive sweating is ______.
Hyperhidrosis
46
What is the term for abnormally low blood volume?
Hypovolemia
47
True or False: A normal pulse oximetry reading is typically above 95%.
True
48
What is the term for an abnormally high heart rate exceeding 100 beats per minute?
Tachycardia
49
What is the medical term for elevated levels of calcium in the blood?
Hypercalcemia
50
What is the term for low levels of calcium in the blood?
Hypocalcemia
51
Fill in the blank: The medical term for an abnormally high level of potassium in the blood is ______.
Hyperkalemia
52
What is the term for low levels of potassium in the blood?
Hypokalemia
53
True or False: A normal temperature range for adults is typically between 97°F and 99°F.
True
54
What is the term for an abnormally low respiratory rate, typically fewer than 12 breaths per minute?
Bradypnea
55
What is the name for a dangerously high body temperature, often exceeding 104°F?
Hyperpyrexia
56
WHAT IS AN ACUTE COUGH
A COUGH LESS THAN 8 WEEKS
57
WHAT IS A CHRONIC COUGH
A LONG LASTING COUGH, GREATER THAN 8 WEEKS INIDICATIVE OF AN UNDERLYING ISSUE
58
WHAT IS ANOTHER NAME FOR POST NASAL DRIP
UPPER AIRWAY SYNDROME
59
BROWN/DARK SPUTUM
OLD BLOOD
60
RED/FRESH SPUTUM
BLEEDING TUMOR/TB
61
CLEAR/TRANSLUCENT SPUTUM
NORMAL
62
COPIUOUS SPUTUM
LARGE AMOUNTS
63
FRANK HEMPPTYSIS SPUTUM
MASSIVE AMOUNT OF BLOOD
64
GREEN SPUTUM
STAGNANT GRAM NEG
65
GREEN FOUL SMEELING SPUTUM
PSEUDOMONAS ANAEROBIC
66
PINK AND FROTHY SPUTUM
PULM EDEMEA
67
PERCUSION OF THE WE ARE LOOKING FOR
SIZE BORDER CONSISTENCY
68
VICIOUS SPUTUM
STICKY THICK
69
TENACIOUS SPUTUM
STICKY SECRETIONS ADHEZIVE STICK TO ONE ANOTHER
70
YELLOW OR OPAQUE SPUTUM
WBC PRESENT OR INFECTION
71
KUSSMAUL BREATHING
INCREASED CO2 METABOLIC ACIDOSIS FAST DEEP EX. DKA
72
WHEEZE
AIRWAY OBSTRUCTION BRONCHOSPASM CONTINOUS HIGH PITCHED
73
STRIDOR
CONTINOUS LOUD HIGH PITCHED CAUSED BY UPPER AIRWAY OBSTRUCTION
74
PLEURAL FRICTION RUB
PLEURAL MEMEBRANES RESIST MOVEMENT CONTINOUS LOW COARSE CREAKING OR GRATING
75
DIMINISHED BREATH SOUNDS
CAN BE SHALLOW OR SLOW BS INTENSITY DECREASES
76
BIOT
FAST DEEP AND ABRUPT IRREG PAUASE EX. BRAIN INJURY
77
FINE CRACKLES
DISCONTIONOUS HIGH PITCH CRACKLING POPPING (BUBBLE WRAP) RAPID EQUALIZATION OF GAS PRESSURE WITH COLLAPSED ALVEOLI
78
COARSE CRACKLES
LOW PITCHED RUMBLING BUBBLING AIR MOVING THROUGH SECRETIONS
79
WHAT ARE THE ACCESSORY MUSCLES OF INSP
SCALENE STERNOCLEIDOMASTOID PECTORALIS TRAPEZIOUS
80
WHAT ARE THE ACCESSORY MUSCLES OF EXP
RECTUS ADBOMINUS EXT OBLIQUE INT OBLIQUE TRANSVERSE ABDOMINUS
81
PULSE LOCATIONS
TEMPORAL CAROTID APICAL BRACHIAL FEMORAL POPLITEAL PEDAL (DORSALIS PEDIS) POSTERIOR TIBIAL
82
WHAT HAPPENS DURING SYSTOLE
VENTRICULAR CONSTRATION
83
WHAT HAPPENS DURING DIASTOLE
VENTRICULE REST
84
WHAT IS AFEBRILE
NO FEVER
85
WHAT IS PYREXIA
TEMP ABOVE NORMAL
86
HYPERTHERMIA
TEMP ABOVE NORMAL RANGE
87
HYPERPYREXIA
AN EXCEPTIONALLY HIGH TEMP
88
HYPOTHERMIA
A TEMP BELOW NORMAL
89
PARTS OF A PT ASSESSMENT
COLLECTION FORMULATION SELECTION DOCUMENTATION
90
WHAT ARE THE NAMES FOR NORMAL BREATH SOUNDS
BRONCHIAL VESICULAR BRONCHOVESICULAR
91
WHAT IS EUPNEA
NORMAL BREATHING
92
WHAT IS BRADYPNEA
SLOW LABORED BREATHING
93
WHAT IS TACHYPNEA
FAST BREATHING
94
NAMES FOR ABNORMAL BREATHING PATTERNS
TACHYPNEA BRADYPNEA HYPOVENTILATION HYPERVENTILATION HYPERPNEA KUSSMAUL CHEYNE STOKES APNEA
95
PRIMARY BREATHING MUSCLES
DIAPHRAGM AND INTERCOSTALS
96
TYPES OF FEVERS
INTERMITTENT REMITTENT RELAPSING CONSTANT
97
METHOD OF TAKING TEMP
ORAL RECTAL AXILLARY TYMPANIC TEMPPORAL
98
WHAT IS DIFFERENCE BETWEEN SIGNS AND SYMPTOMS
SIGNS - OBJECTIVE DATA DESCRIBES CHARACTERISTICS OF A PARTICULAR DISEASE/DISORDER SYMPTOMS - SUBJECTIUVE DATA DESCRIBES CHARACTERISTICS OF A PARTICULAR DISORDER OR DYSFUNCTION
99
WHAT IS THE DIFFERENCE BETWEEN VASODILATION AND VASOCONSTRICTION
VASODILATION - AN INCREASE IN BODY TEMP CAUSES THE BLOOD VESSELS NEAR THE SKIN SURFACE TO DILATE VASOCONSTRICTION - DECREASE IN BODY TEMP WHICH WORKS TO KEEP WARMED BLOOD CLOSER TO THE CENTER
100
101
PARTS OF PT EXAMINATION
INSPECTION PALPATION PERCUSSION AUSCULATION
102
CONSOLIDATION MEANS
PUS OR FLUID IN LUNGS
103
ATELECTASIS
AIR TRAPPING
104
ERRORS ASSOC WITH AN ABG
PARALYTICAL ANALYTICAL INTERPRETAION POST ANALYTICAL
105
IS ATELECTSASIS CONSIDERED RESTRICTIVE OR OBSTRUCTIVE
OBSTRUCTIVE
106
EXAMPLES OF OBSTRUCTIVE DISEASES OR CONDITIONS
FLAIL CHEST MUCUC PLUGGING PLEURAL EFFUSION PNEUMOTHORAX
107
COOMON OF CAUSES OF ABNORMAL BLOOD GASES
RESPIRATIORY METABOLIC
108
SPIROMETER MEASURES
CAN ONLY BE USED TO EVALUATE VT IC IRV ERV VC
109
INDIRECT ARE USED TO TEST FOR
RV FRC TLC
110
WHAT ARE THE 4 LUNG VOL ARE TESTED
VT IRV ERV RV
111
4 LUNG CAPACITIES ARE TESTED VIA PFT
VC IC FRC TLC
112
WHAT ARE THE 3 INDIRECT METHODS TO TEST RV, FRC, TLC
1. CLOSED CIRCUIT HELIUM DILUTION TEST 2. OPEN CIRCUIT NITROGEN TEST 3. BODY PLETTHYSMOOGRAPHY
113
WHAT HAPPENS IN OBSTRUCTIVE DISEASES
LOW VENT RATE TIDAL VOL INCREASED HIGH LUNG COMPLIANCE AIR TRAPPING
114
WHAT HAPPENS IN RESTRICTIVE DISEASE
LOW LUNG COMPLIANCE INCREASED VENT RATE TIDAL VOL DECREASE RIGIDITY
115
WHAT DOES A PFT LOOKS AT
FVC FEV1 FEVT FEV1/FVC RATIO FEF 25%-75% FEF 200-1200 PEFR MVV FLOW VOL LOOP
116
DLCO MEASURES WHAT
THE AMOUNT OF CARBON MONOXIDE THAT MOVES ACROSS ACROSS THE ALVEOLAR -CAP MEMEBRANE
117
NORMAL DLCO RATE FOR A MALE RESTING 25 ML/MG
MALE - 170 L/MIN FEMALE 110 L/MIN
118
MVV IS WHAT
LARGEST VOL OF GAS THAT CAN BE BREATHED IN AND OUT OF LUNGS
119
IF PACO2 INCREASES BY 10 MMHG
PH WILL DECREASE BY 0.06UNITS HC03 WILL INCREASE 1 MEQ/L
120
IF PACO2 DECERASE BY 5 MMHG
PH WILL INCREASE BY O.O6 UNITS HCO3 WILL DECERASE BY 1 MEQ/L
121
WHAT IS PART PART OF CBC WITH DIFF
RBC HGB TOTAL WBC HCT
122
WHAT ARE THE ELECTROLYETES
SODIUM CHLORIDE CALCIUM POTASSIUM
123
WHAT ARE CARDIAC BIOMARKERS
CARDIAC TROPININ LESS THAN 0.01 CREATNINE KINASE 25-200 CREATNINE KINASE M8 - 0.4 MYGLOBIN - 25-72
124
WHAT IS POLYCYTHEMIA
DRIVEN BY HYPOEXEMIA TOO MANY RBCS MAKE BLOOD THICKER THIS CAN RASISE PULM HYPERTENSION BLOOD CLOTS STROKE RIGHT HEART STRAIN
125
TYPES OF WBC
ESOPHILS - 2%-4% NEUTROPHILS -60%-70% BASOPHILS - 0.5-1^ LYMPOCYTES =20%-25% MONOCYTES-3-8%
126
TRANSUDATE
CLEAR FLUID HEART + LUNGS
127
EXUDATE
CLOUDY FLUID
128
WHAT IS C&S
NOT ANY DOES IT CHECK FOR INFECTION HELPS TO DETERMINATION FLOW AND EFFECTIVENESS
129
WHERE ARE SPUTUM SAMPLES OBTAINED
FROM EXPECTORATION TRACHEAL SUCTION OR BRONCHOSCOPY
130
BRONCHOSCOPY IS
WELL ESTABLISHED DX AND THERAPEUTIC TOOL
131
TYPES OF THERAPIST DRIVEN PROTOCOLS
LUNG EXPANSION PROTOCOL AIRWAY CLEARANCE PROTOCOL AEROLIZAED MEDICATION MEDICATION THERAPY PROTOCOL
132
THORACENTISIS
USED TO DX AND TX THROCIC PROBLEM DONE IN UPDRIGHT POSITION
133
EBUS
EXAMINATION MAY BE PERFORMED DURING A BRONCHOSCOPY TO ESTBLISH OR STAGE LUNG CA OR THAT MAY HAVE SPREAD
134
WHAT IS GRAM STAINING
A STAINING PROCESS PERFOMRED TO CLASSIFY BACTERIA INTO GRAM POSITIVE OR NEG ABLE TO GUIDE THERAPY UNTIL CAND S TEST COME BACK
135
CALCULATE AIRWAY RESISTANCE
R = Δ P / V
136
CALCULATE LUNG COMPLIANCE
ompliance (C) = ΔV/ΔP.
137
CALCULATE PAO2
PAO2 = (Patm - PH2O) * FiO2 - PaCO2 / RQ PATCM = ATM PRESSURE ( 760) PH20 = 47 FIO2 = WILL BE GIVEN PACO2 = WILL BE GIVEN RQ = .80 UNLESS STATED UNIT OF MEASURE MMHG
138
CALCULATE P[A-a]02
P(A-a)O2 = PAO2 - PaO2 BUT NEED TO CALUALTE Alveolar Gas Equation: PAO2 = (FiO2 * (Patm - PH2O)) - (PaCO2 / RQ GET ANSWER FOR PA02 THEN SUBTRACT FROM PaO2 ANSWER WILL BE IN MMHG
139
CALCULATE CaO2
CaO2 = (Hb x 1.34 x SaO2) + (PaO2 x 0.003) UNIT OF MEASURE
140
CALCULATE (a-v)O2
START WITH CALCULATING CaO2 = (Hb x 1.34 x SaO2) + (0.003 x PaO2). THIS IS ARTERIAL BLOOD THEN CALCULATE VENOUS BLOOD CvO2 = (Hb x 1.34 x SvO2) + (0.003 x PvO2 THEN SUBTRACT
141
COR PULMALE
a condition where the right side of the heart becomes damaged or overworked due to underlying lung diseases
142
SINUS BRADY
143
SINUS TACHYCARDIA
144
2 SHOCKABLE RYTHMS
V TACH V FIB
145
VENTRICLULAR TACHY (V TACH)
146
V FIB
147
SINUS RHYTHM WITH PVC
148
ASYTOLE
NO ELECTRICAL ACTIVITY
149
V FIB
CHAOTIC ELECTRICAL ACTIVITY During ventricular fibrillation, the lower heart chambers contract in a very rapid and uncoordinated manner. As a result, the heart doesn't pump blood to the rest of the body.
150
V TACHY
P WAVE INDERCERNIBLE QRS WIDE AND BIZARRE
151
PVC
NOT PRECEDED BY P WAVE QRS IS WIDE AND BIZARRE REG HR ALTERED BY PVC
152
ATRIAL FIBRILALTION
ATRIAL CONTRACTIONS ARE DISORGANIZED AND INEFFECTIVE MAY REDUCE CO BY 20% P WAVE ABSENT QRS NORMAL
153
WHAT IS NORMAL AIRWAY RESISTANCE
0.5-2.5 CM H20/L/S
154
LABEL THE PARTS OF THE EKG
155
SINUS BRADYCARDIA
HR LESS THAN 60 BPM NORMAL P-QRS-T WAVE
156
SINUS TACHYCARDIA
HR OVER 100 BPM NORMAL P-QRS-T WAVE
157
SINUS ARRYTHMIA
PULSE WILL INCREASE WITH AN EXHALE INTERVALS GRPUPS CAN VARY BY 10% NORMAL P-QRS-T WAVE
158
ATRIAL FLUTTER
P WAVE ABSENT REPLACED BY 2 SAW TOOTH WAVES
159
HOW TO CALCULATE HR ON EKG STRIP IF IIREGULAR RYTHM
COUNT THE NUMBER OF QRS IN 6 SEC X 10
160
HOW TO CALCULATE HR ON EKG REGULAR
COUNT THE NUMBER OF BOXES BETWEEN QRS COMPLES AND DIVIDE BY 300
161
PULM ARTERY CATH IS ASLO CALLED
SWAN GANZ BALLOON TIPPED FLOW DIRECTED CATH MEASURES RAP PAP PCWP CO
162
SYSTEMIC ARTERIAL CATH
IS AN INDWELLING CATH MODE OF INVASIVE HEMODYNAMIC MONITORING A systemic catheter, or right heart catheterization, measures pressures and blood flow in the heart and lungs by threading a thin tube through a blood vessel and into the heart, allowing for the assessment of cardiac function and identification of abnormalities.
163
WHAT DOES THE SYSTEMIC ARTERIAL CATH ALLOW FOR
1. CONTINOUS AND PRECISE MEASUREMENTS OF SYSTOLIC, DIASTOLIC, AND MEAN BP 2. ACCURATE INFO OF FLUCTUATIONS OF BP 3. GUIDANCE IN THE DECISION TO GO UP, REGULATE OR GO DOWN IN REG THERAPY
164
CENTRAL VENOUS CATH
READILY MEAURES THE CVP AND VENTRICULAR FILLING PRESSURE 1. VERNTRICLE HEART FAILURE 2. ACCESS FOR POSTIVE PRESSURE 3. COR PULMALE 4. FLAIL CHEST
165
WHAT IS THERMODILUTION
a method of measuring cardiac output (CO) by injecting a known volume of a cold solution into the right atrium and measuring the temperature change in the pulmonary artery
166
FLOW OF BLOOD
SVC AND IVC R ATRIUM TRICUSPID VALVE R VENT PULM SEMI LUNAR VALVE PULM ARTERY LUNGS GAS EXCHANGE PULM VIEN L. ATRIUM MITRAL VALVE L. VENT AORTIC SEMI LUNAR VALVE AORTA BODY
167
V TACHY
HR RANGES FROM 150-250 BPM
168
WHAT IS THE DIFFERENCE BETWEEN TTE AND TEE
TTE (Transthoracic Echocardiogram) is a non-invasive heart ultrasound done by placing a probe on the chest, TEE (Transesophageal Echocardiogram) is a more invasive procedure where a probe is inserted down the esophagus for a clearer view of the heart.
169
CAPILLARY REFILL TEST
press firmly on the nail bed or fingertip until it turns white, then release the pressure and time how long it takes for the color to return, which should be less than 2 seconds for normal perfusion.
170
P WAVE IS
DEPOLARIZATIION OF ATRIA (CONTRACTION)
171
QRS COMPLES IS
DEPOLARIZATION OF VENTRICLES (CONTRACTION)
172
T WAVE IS
REPOLARIZATION OF VENTRICLES ( RELAX)
173
WHAT ARE THINGS THAT IMPACT O2 CONSUMPTION
Oxygen consumption is how much oxygen the body is using and needs to maintain metabolic equilibrium, and it varies based on certain states. Oxygen consumption increases with illness, fever, and exercise.
174
WHAT IS DISADVANTAGE OF AP VIEW RADIOGRAPH
THE HEART AND MEDIATSINUM ARE SIGNIFICANTLY MAGNIFIED LESS RESOLUTION
175
WHAT 2 VIEW IN RADIOGRAPH COMPLEMENT EACH OTHER
LATERAL AND PA
176
CALCULATE WOB
W=P X V
177
HISTOLOGIC HYPOXEMIA
THE IMPAIRED ABILITY OF THE TSSUE CELLS TO METABOLIZE O2
178
CIRCULATORY ANEMIA
BLOOD FLOW TO THE TISSUE IS INADEQUESTE
179
ANEMIC HYPOEMIA
INADEQUESTE O2 AT TEH TISSUE LEVEL CAUSED BY DECREASE IN ARTERIAL 02 TENSION
180
COR PULMOALE
R VENT HYPERTROPHY TERM TO DESCRIBE PULM ARTERIAL HYPERTENSION
181
WHAT IS THERMODILUITION USED FOR
TO MEASURE CO
182
NORMAL TIDAL VOL
500 ML 7-9 ML/KG
183
NORMAL MINUTE VENT
5-8
184
NORMAL VALUES FOR ABG
PH - 7.35-7.45 PACO2 - 45-35 HC03 - 22-26 PAO2 - 80-100
185
NORMAL VALUES FOR VBG
PH - 7.30-7.40 PACO2 - 48-44 HC03 - 24-30 PA02 - 35-35
186
MINUTE VENTILATION CALCULATION
MV= RR X VT
187
IN IOBSTRUCTUIVE DIS, WHAT PART OF A PFT ARE IMPACTED
DCERASE FEF INCREASE TLC INCREASE RV
188
IN RESTRICTIVE DIS, WHAT PARTS OF PFT ARE IMPACTED
DECREASE IN PEF
189
WHAT IS FEV1/FVC RATION
PERCENT OF THE PT TOTAL VOL OF AIR FORCE EXHALED ADULTS EXPEL 83% DECREASES WITH AGE
190
WHAT IS FEV 1
THE AMOUNT OF AIR EXPELLED IN 1 SEC
191
HISTOLOGY OF THE TRACHEOBRONCHIAL TRESS
LAMINA PROPIRA EPITHELIUM CARTILAGE Basal and ciliated as well as few goblet cells interspersed among the ciliated cells Ciliated pseudostratified columnar epithelium with goblet cells (mucus-producing cells Similar to the trachea, but with shorter epithelial cells, fewer goblet cells, and fewer seromucous glands
192
WHAT IS NORMAL PEFR VALUES FOR MALES AND FEMALES
MALE - 450-700 L/MIN FEMALE - 300-500 L/MIN
193
ANOTHER NAME FOR ACUTE RESP ALKALOSIS
ACUTE AVEOLAR HYPERVENT
194
ANOTHER NAME FOR COMPENSATED RESP ACIDOSIS
CHRONIC VENT FAILURE
195
ANIOTHER NAME OFR LACTIC ACIDOSIS
METABOLIC ACIDOSIS
196
WHAT IS BARREL CHEST CHEST SHAPE
AIR TRAPPING WITH LUNG HYPERINFLATION IN OBSTRUCTIVE DIS
197
WHAT IS H KYPHOSIS CHEST SHAPE
AN EXAGERETED POSTERIOR CURVATURE OF THORACIC "HUNCH BACK"
198
SCOLISOS CHEST SHAPE
A LATERAL S SHAPE CURVATURE OF THORACIC AND LUMBAR SPINE OFTEN WITH INVOLVED VERTEBRAL ROTATION
199
KYPHOSOCOLIOSIS CHEST SHAPE
THE COMBINATION OF BOTH KYPHOSIS AND SCOLIOSIS SEVERE DEFORMITY IMPAIRS THE CARDIO PULM FUNCTION
200
PECTUS EXCAVATUM CHEST SHAPE
A SUNKEN STERNUM AND ADJECENT CARTILAGES THE DEPRESSION BEGINS AT 2ND INTERCOSTAL SPACE CECOMES MORE DEPRESSED AT THE JUNCTION OF THE XIPHOID AND THE BODY STERNUM
201
PECTUS CARMITHTUM CHEST SHAPE
THE INWARD PROJECTIION OF THE STERNUM WITH RIBS STOPING AT THE OUTSIDE AND VERTICAL DEPRESSION ALOMNG THE COSTOCHRONDRAL "BIRD CHEST"
202
NORMAL IRV VALUES
MALE - 3400 FEMALE 1900
203
ERV VALUES
MALE -1200 FEMALE - 800
204
NORMAL TLC
4.5-6
205
WHAT IS THE HERRING BEUERE REFLEX
a reflex triggered to prevent the over-inflation of the lung Pulmonary stretch receptors, located in the walls of the bronchi and bronchioles, respond to excessive stretching of the lungs during large inspiration
206
J RECEPTORS
sensory nerve endings located in the alveolar walls near pulmonary capillaries, innervated by the vagus nerve, that respond to conditions like pulmonary edema and congestion, leading to increased breathing rate and potentially dyspnea. RESPOND TO FLUID BUILD UP RAPID SHALLOW BREATHING
207
IRRITANT REFLEX
The irritant reflex is a protective mechanism in the respiratory system triggered by irritants, leading to responses like cough, bronchoconstriction, and increased breathing rate, to expel or reduce the harmful substance. IN TRACHEA BRONCHI BRONCHIOLES
208
WHAT ARE PORES OF KAHN
ALVEOLUS HAVE OPENING TO GAS TO MOVE ALVEOLI TO ALVEOLI
209
WHAT ARE CANALS OF LAMBERT
OPENINGS THAT GAS EXCHANGE BETWEEN ALVEOLUS AND BRONCHIOLES NEARBY
210
ORTHOPNEA (OR POSTIONAL)
shortness of breath that occurs while lying flat and is relieved by sitting or standing
211
CARDIAC DYSPNEA
LABORED BREAHING CAUSED BY HEART DIS
212
EXERTIONAL DYSPNEA
PROVOKED BY PHYSICAL ACTIVITY
213
PAROXYSMAL NOCTURANL
A FORM OF RESP DISTRESS RESLATED TO POSTURE (RECLINING POSITION)
214
WHAT IS RENAL DYSPNEA
DYSPNEA RELATED TO KIDNEY DIS
215
WHERE ARE THE CONDUCTING AIRWAYS
UPPER AIRWAYS LOWER AIRWAYS
216
WHERE DOES GAS EXCHANGE OCCUR CONDUCTING AIRWAYS OR RESP ZONE
RESP ZONE
217
NORMAL PAO2 VALUES
NORMAL - 80-100 MILD - 79-60 MOD - 59-40 SEVERE - LESS THAN 40
218
NORMAL HGB AND HCT LEVELS
HGB - MALE - 14-16 HCT - MALE 45% HGB - FEMALE-12-15 HCT - FEMALE - 42%
219
WHAT IS A NORMAL 1/E RATION
1:2
220
WHAT ARE NORMAL SPO2 VALUES
NORMAL - 95-100 MILD - 91-94 MOD - 90-75 SEVERE LESS THAN 75
221
WHAT MAKES UP THE VC
VC=IRV+VT+ERV
222
WHAT MAKES UP THE IC
IC = VT+ IRV
223
WHAT MAKE SUP THE FRC
FRC= ERV +IRV
224
WHAT MAKES UP THE TLC
TLC = IC+ ERV+RV
225
WHAT IS THE RES VOL/TOTAL LUNG CAP RATION
1200/6000 =20% (MALE) 1000/4200 =0.238 25% (FEMALE)
226
LUNGS ARE CONSIDERED NORMAL IF WHAT ARE WITHIN 200 ML OF EACH OTHER
FVC AND VC
227
WHAT IS MORMAL IRV VALUES
MALE - 3100 FEMALE - 1900
228
WHAT IS NORMNAL ERV VALUES
MALE - 1200 FEMALE -800
229
WHAT IS NORMAL RV VALUES
MALE - 1200 FEMALE - 1000
230
TIDAL VOULME NORMALVALUES
MALE -500 FEMALE - 400-500
231
NORMAL VALUES FOR NA
136-142
232
NORMAL VALUES FOR K
3.8-5.0
233
NORMAL VALUE FOR CL
95-103
234
CA+ NORMAL VALUE
4.5-5.4
235
WHAT IS A NORMAL CVP PRESSURE
0-8
236
WHAT IS A NORMAL RAP PRESSURE
0-8
237
MPAP PRESSURE VALUE
10-20
238
PCWP VALUES
4-12
239
CO
4-6
240
HOW TO CALCULATE O2 DISSOLVED
PAO2 X0.03 (DISSOLVED 02) TO GET THE O2 BOUND TO HGB FIRST
241
HOW TO CALCULATE O2 BOUND TO HGB
1.34 X O2 X HGB = ANSWER PER 100 ML OF BLOOD
242
HOW TO CALCULATE CO
CO=SV X HR
243
WHEN IS DULL PERCUSSION HEARD
PLEURAL THICKENING PLEURAL EFFUSION ATELECATASIS CONSOLIDATION
244
HYPERRESONNAT TONE HEARD WHEN
AIR TRAPPING
245
HR IS DESCRIBED AS
RATE RYTHM AND STRENGTH
246
VESICULAR BREATH SOUND IS WHAT TYPE OF PITCH AND INTENSITY
PITCH - HIGH INTENSITY - SOFT
247
BROCNHIAL BREATH SOUNDS HAVE WHAT TYPE OF PITCH AND INTENSITY
PITCH - HIGH INTENSITY - LOUD
248
BRONCHOVESICULAR BREATH SOUNDS HAVE WHAT YYPE OF PITCH AND INTENSITY
PITCH - MOD INTENSITY - MOD
249
WHAT ARE THE STEP 1 APPROACH PER GINA FOR ASTHMA
DAYTIME SYMPTOMS LESS THAN OR EQUAL 2X A WEEK NOCTURNAL AWAKENINGS LESS THAN OR EQUAL TO 2/MONTH NORMAL FEV 1 EXACERBATIONS LESS THAN OR EQUAL TO 1/YR
250
WHAT ARE THE TREATMENT AND RECOMMENDATIONS PER STEPWISE STEP 1
LOW DOSE ICS WITH RAPID ONSET LABA PRN
251
WHAT ARE THE TREATMENT AND RECOMMENDATIONS PER STEPWISE STEP 2
LOW DOSE ICS WITH RAPID ONSET LABA PRN ASSESS FOR ADD ON THERAPIES
252
WHAT ARE THE TREATMENT AND RECOMMENDATIONS PER STEPWISE STEP 3
LOW DOSE ICS WITH RAPID ONSET LABA AS CONTROLLER LOW DOSE ICS WITH RAPID ONSET LABA PRN ASSESS FOR ADD ON THERAPY
253
WHAT ARE THE TREATMENT AND RECOMMENDATIONS PER STEPWISE STEP 4
MEDIUM DOSE ICS LAMA AS CONTROLLER SABA PRN ASSESS FOR ADD ON THERAPIES
254
WHAT ARE THE TREATMENT AND RECOMMENDATIONS PER STEPWISE STEP 5
MEDIUM DOSE ICS WITH RAPID ONSET LABA AS CONTROLLER LAMA AS CONTROLLER SABA PRN ASSESS FOR ADD ON THERAPY
255
WHAT ARE THE STEP 2 APPROACH PER GINA FOR ASTHMA
DAYTIME SYMPTOMS GREATER TO OR EQUAL 2 DAYS/WK BUT LESS THAN OR EQUAL TO 7 DAYS/WK NOCTURNAL AWAKENINGS 3-4 NIGHTS/MONTH MINOR INTERFERENCE WITH ACTIVITIES FEV1 WITHIN NORMAL RANGE EXACERBATIONS GREATER TO OR EQUAL 2/YR
256
WHAT ARE THE STEP 3 APPROACH PER GINA FOR ASTHMA
DAYTIME SYMPTOMS NOCTURNAL AWAKENINGS LESS THAN 1/WK DAILY NEED FOR SABA SOME ACTIVITY LIMITATION FEV1 60-80% EXACERBATIONS GREATER THAN OR EQUAL 2/YR
257
WHAT ARE THE STEP 4-6 APPROACH PER GINA FOR ASTHMA
SYMPTOMS ALL DAY NOCTURNAL AWAKENINGS NEED FOR SABA SEVERAL TIMES/DAY EXTREME LIMITATION IN ACTIVITY FEV1 LESS THAN OR EQUAL 60% PREDICTED EXACERBATIONS GREATER THAN OR EQUAL 2/YR
258
IF A MEDICATION IS NOT SCHEDULED BUT NEEDED FOR IMMEDIATE CONTROL
RELEIVER OR RESCUE
259
IF A MEDICATION IS SCHEDULED
MAINTENANCE OR CONTROLLER
260
TYPES OF ASTHMA
ALLERGIC (ATOPIC) NON-ALLERGIC LATE ONSET ASTHMA WITH FIXED AIRFLOW LIMITATION ASTHMA WITH OBESITY
261
LEUKOTRIENES DO WHAT
LTRA BLOCK THE AFFECTS OF LEUKOTRIENES preventing leukotrienes from binding and causing their effects.
262
LAMA DOES WHAT
PREVENTS MUSCARIRNIC RECEPTOR SMOOTH MUSCLE CONSTRICTION
263
MONOCLONAL ANTIBODIES DO WHAT
TARGETS IMMUNOGLOBINS OR INTERLEUKINS
264
METHYLXANTHINES DO WHAT
BRONCHODILATION
265
PEAK FLOW VALUES
RED - LESS THAN 50% YELLOW - 79-50% GREEN - 100-80%
266
WHAT IS EXTRINSIC ASTHMA
CHEMICAL MEDIATORS THE PARASYMPATHETIC NERVES: REFLEX BRONCHOCONSTRICTION INCREASED MUCUS PRODUCTION VASCULAR PERMEABILITY LEADING TO TISSUE DAMAGE
267
WHAT IS INTRINISC ASTHMA
CANNOT BE DIRECTLY LINKED TO A SPECIFIC ANTIGEN NORMAL SERUM IGE LEVELS OFTEN OCCURS AFTER THE AGE OF 40
268
IMMUNOLOGIC MECHANISM OF AN ASTHMA ATTACK ( OF EXTRINISC ASTHMA)
LINKED TO ANTIGEN EXPOSURE ANALPHYLATIC HYPERSENSITIVITY REACTION IMMUNOLOGIC MECHANISM . LYMPHOID TISSUE FOR IGE ANTIBODIES . IGE ANTIBODIES ATTACH TO MAST CELL . MAST CELL DEGRANULATION RELASING HISTAMINE, EOSINOPHIL CHEMOTAXIC FACTOR OF ANAPHAYLYSIX, NEUTROPHIL CHEMOTXIC FACTORS LEUKOTRIENES PROSTOGLANDINS PLATELET ACTIVATING FACTORS
269
ANATOMIC ALTERATIONS OF ASTHMA
SMOOTH MUSCLE CONSTRICTIION OF BRONCHIAL AIRWAYS (BRONSHOSPASM) EXCESSIVE PRODUCTION OF THICK, WHITISH BRONCHIAL SECRETIONS MUCUS PLUGGING HYPERINFLATION OF ALVEOLI (AIR TRAPPING) IN SEVERE CASE, ATEELACSIS CAUSED BY MUSCUS PLUGGING BRINCHIAL LEADING TO FIBROSIS (CAUSED BY REMODELING)
270
HOW IS THE ABG FOR ASTHMA
PACO2 AND BICARB GO UP
271
PFT FOR ASTHMA
FEV1 DOWN EVRYTHING \\GOES DOWN
272
HOW ARE BS FOR AN ASTHMA PT
EXPIRATORY PROLONGATION (I/E RATIO GREATER TAN 3.3) DECREASED TACYILE AND VOCAL FREMITUS HYPERRESONANT PERCUSSION NOTE DIM BS WHEEZE CRACKLES
273
HOW DOES AN ASTHMA RADIOGRAPH APPEAR
TRANSLUCENT DARK FIELDS (AIR TRAPPING) INCREASED ANTERIORPOSTERIOR DIAMETER (BARREL CHEST) DEPRESSED OR FLATTENED DIPHRAGM
274
WHAT ARE THE DIFFERENT FREQUENCIES FOR COPD
EXCERBATION SYMPTOM MEDCIATION 02 THERAPY
275
WHAT ARE THE SYMPTOMS AND EXCERBATION LEVEL FOR EACH GOLD GROUP
A- LOW SYMP/LOW EXCERABTION B- HIGH SYM/LOW EXACERBATION E. - HIGH EXACERBATION
276
HOW DOES A PFT LOOK FOR A COPD PT
VT - N OR INCREASE RV - NORMAL OR INCREASE FRC - INCREASE TLC - N OR INCREASE RV /TLC RATIO - N OR INCREASE IC - N OR DECREASE EVERYTHING ELSE DECERASE
277
HOW DOES A COPD PT CXR LOOK
TRANSLUCENT DARK LUNG FIELDS DEPRESSED OR FLATTENED DIAPHRAGM LONG NARROW HEART , PULLED DOWN BY DIAPHRAGM INCREASED RETROSTERNAL AIR SPACE
278
279
cystic fibrosis
FATAL DISORDER CHARACTERIZED BY EXCESSIVE PRODUCTION AND ACCUMULATION OF THE THICK MUCUS IN THE TRACHEOBROCHIAL TREE
280
CFTR
CYSTIC FIBROSIS TRANSMEMEBRANE CONDUCTANCE REGULATOR A GENE THAT, WHEN MUTATED, LEADS TO CF
281
BROCNHIAL OBSTRUCTION
A CONDITION WHERE BRONCHIAL AIRWAYS ARE BLOCKED WHICH CAN BE CAUSED BY MUCUS PLUGGING
282
ATELECTASIS
PARTIAL OR COMPLETE COLLAPSE OF THE LUNG OFTEN DUE TO BLOCKAGE OF THE AIR PASSAGES
283
MUCUS PLUGGING
ACCUMULATION OF THICK MUCUS IN THE AIRWAYS THAT OBSTRUCT AIRFLOW
284
SWEAT TEST
A DIAGNOSTIC TEST FOR CF THAT MEAUSRES THE AMOUNT OF SODIUM AND CHLORIDE IN SWEAT
285
ELEVATED SWEAT CHLORIDE
SWEAT CHLORIDE LEVELS GREATER THAN 60 MEQ/L ON TWO OCCSAIONS INIDCATE A DX OF CF
286
GENETIC COUNSELING
A PROCESS TO INFORM AND SUPPORT INDIVIDUALS REGARDING GENETIC ASPECTS OF CF INCLUDING RISKS AND INHERITANCE PATTERNS
287
PFT
TESTS THAT MEASURE LUNG FUNCTION AND CAPACITY OFTEN INDICATING THE SEVERITY OF CF
288
SYMPTOMS OF CF
Chronic cough, wheezing, sputum production, frequent respiratory infections, and failure to thrive.
289
Hematologic outcomes in CF
Increased hematocrit and hemoglobin levels, often in response to chronic hypoxia.
290
Nasal potential difference (NPD)
A measurement that assesses the transport of sodium and chloride across epithelial cell linings, indicative of CFTR function.
291
Pre-natal testing for CF
Screening for CF mutations in pregnant women to assess the risk of having a child with CF.
292
anatomic alterations of the lungs -cf
EXCESSIVE RODUCTION AND ACCULULATIONOF THICK TENACIOUS MUCUS IN THE TTRACHEA-BRONCHIAL TREE PARTIAL BRONCHIAL OBSTRUCTION (MUCUS PLUGGING) HYPERINFLATION OF ALVEOLI TOTAL BRONCHIAL OBSTRUCTION (MUCUS PLUGGING) ATELECTASIS
293
ETIOLOGY AND EPIDEMIOLOGY OF CF
AL GENE DISORDER CAUSED BY MUTATIONS IN APAIR OF GENES LOCATED ON CHROMOSOME 7 OVER 1700 DIFF MUTATIONS IN THE GENE THAT ENCODES FOR THE CF TRANSMEMBRANE CONDUCTANCE REGULATOR, CFTR.
294
THE ABNORMAL EXPRESSIONOF THE CFTR RESULTS IN
RESULTS IN THICK VICIOUS ACCULUMALTION IN THE LUNGS MUCUS BLOCKS THE PASSAGEWAYS OF THE PANCREAS WHICH PREVENTS ENZYMEABNORMLA TRANSPORT OF SODIUM AND CHLORIDE IONS ACROSS MANY TYPES OF EPIHERLIAL SURFACES S FROM THE PANCREAS FROM REACHING THE INTESTINE
295
6 CLASSES OF CFTR MUTATIONS
DIVIDED INTO 3 BROAD CATEGORIES: AFFECING THE QUALITY OF THE CFTR PROTIEN AFFECTING FUNCTION PF THE CFTR PROTEIN AS A GATING DEFECT (CLASSII) WHERE THE CHANNEL DOES NOT OPEN A CONDUCTANCE DEFECT (CLASS IV) WHERE THE ION FLOW IS IMPAIRED
296
HOW CF GENE IS INHERITED
A RECESSIVE GENE DISORDR THE CHILD MUST INHERIT 2 COPIES OF THE DEFECTIVE CG GENE TO HAVE DISESE IF BOTH PARENTS CARRY THE GENE, THE POSSIBILITY OF HEIR CHILDREN HAVING FOLLOW THE MENDELIAN inherited pattern of 1 in 4.
297
SCREENING AND DX OF CF
BASED ON THE CLINCAL MAIFESTATIONS ASSOCIATED WITH CF FAMILY HX OF CF LAB FINDINGS
298
2 CRITERIA MUST BE MET TO DX FOR CF
1) Clinical presentation consistent with CF symptoms . 2) Laboratory evidence of CFTR dysfunction or the presence of two CFTR mutations.
299
clinical eveidence of CF CFTR DYSFUNCTION
ELEVATED SWEAT CHLORIDE GREATER THAN 60 MEQ/L (ON 2 OCCASIONS) MOLECULAR DX - GENETIC TESTING ABNRMAL NASAL POTENTIAL DIFFERENCE
300
NEBORN SCREENING OF CF
A test performed on newborns to detect cystic fibrosis early, typically through the measurement of immunoreactive trypsinogen (IRT) levels.
301
HOW LONG HAS CF SCREENING IN NEWBORNS BEEN PRRACTICED IN ALL 50 STATES
2011
302
MOST INFANTS WITH CF HAVE AN ELEVATED
BLOOD LEVEL OF IMMUNOREACTIVE TRYPSINOGEN (IRT)
303
THE IMMUNOREACTIVE TRYPSIN IS MEASURED FROM
A blood sample taken from the newborn's heel. ON THE GUTHRIE CARDS
304
WHAT IS TEH SWEAT TEST
A diagnostic test used to measure the amount of chloride in sweat, helping to confirm a diagnosis of cystic fibrosis.
305
WHAT US THE GOLD STANADRD FOR DX TEST FOR CF
The sweat test is considered the gold standard diagnostic test for cystic fibrosis, measuring chloride levels in sweat to confirm the condition.
306
WHAT DOES THE SWEAT TEST MEASURE
The sweat test measures the concentration of chloride and sodium in sweat, which can indicate cystic fibrosis when elevated.
307
WHAT IS PILOCARPINA
a medication used to stimulate sweat production during the sweat test for cystic fibrosis. USUALLY DONE 2X
308
INFANTS WITH A + CF NEWBORN SCREENING RESULT SHOULD HAVE WHAT DONE
A diagnostic sweat test performed to confirm cystic fibrosis. AFTER 2 WEEKS OF AGE AND GREATER THAN 2 KG IF ASYMPOTAMTIC
309
SWEAT TESTS INTERPRETATIONS INFANTS LESS THAN 6 MONTHS OF AGE
LESS THAN OR EQUAL TO 29 MMOL/L - NORMAL 30-59 MMOL/L -INTERMEDIATE - POSSIBLE CF GREATER THAN OR EQUAL TO 60 MMOL/L - ABNRMAL - DX OF CF
310
SWEAT TESTS INTERPRETATIONS INFANTSOLDER 6 MONTHS OF AGE, CHILDREN AND ADULTS
LESS THAN OR EQUAL TO 39 MMOL/L - NORMAL 40-59 MMOL/L - INTERMEDIATE - POSSIBLE CF GREATER THAN OR EQUAL TO 60 MMOL/L - ABNORMAL - DX OF CF GREATER THAN 60MMOL/L ABNRONAL - DX OF CF
311
CLINICAL INDICATIONS JUSTIFYING THE INTIAL EVELAUTAION FOR CF
WHEEZE DISTAL INTEST. OBSTRUCTION SYNDROME CHRINIC COUGH PANCREAS INSUFFICIENT SPUTUM PRODUCTION PANCREATITIS PARASINUSITIS CHOLELITHIASIS GI ISSUES FAT SOLUBLE DEF (A, D, E, K) FREQ RESP INFECTIONS FAILURE TO THRIVE HYPOPROTEINEMIA HEPTOMEGALY LABNORMAL CT SCAN HEPTOBILARY PROLAPSE INFERTILITY MECONIUM ILEUS OBSTRUCTIVE AZOOSPERMIA MECONIUM PERITONITIS NASAL POLYPS FOCAL BILIARY CIRRHOSIS PARASINUSITIS CHOLELITHIASIS
312
MOLECULAR DX FOR CF
Molecular diagnosis for cystic fibrosis involves genetic testing to identify mutations in the CFTR gene, which is responsible for CF.
313
NASAL POTENTIAL DIFFERENCES OF CF
THE IMPAIRED TRANSPORT NA+ AND CL- ACROSS THE EPITHELIAL CELL LINING THE AIRWAY OF THE CF PT CAN BE MEASURED by nasal potential difference measurements. This reflects abnormal ion transport, characteristic of cystic fibrosis.
314
WHEN NA+ AND CL- MOVE ACROSS THE EPITHELIAL CELL MEMBRANE THEY GENERATE
an electrical potential difference, which can be measured to assess ion transport abnormalities in cystic fibrosis.
315
THE NASAL PASSAGES THE ELECTRICAL POTENTIAL DIFFERENCE IS CALLED
the nasal potential difference.
316
PRE NATAL TESTING FOR CF
PREG FEMALES OFFERED SCREENING FOR CF MUTATIONS IF BOTH PARENTS TEST + TEST FOR CF MUTATIONS THEN THE FETUS HAS A 1:4 CHANCE OF HAVING CF
317
STOOL FECAL FAT TEST FOR CF
a diagnostic test that measures the amount of fat in a stool sample, used to assess pancreatic function in cystic fibrosis patients. MEAUSRES THE AMOUNT OF FATE IN THE IN INFANTS STOOL AND THE % OF DIETARY FAT THAT IS NOT ABSORBED BY THE BODY
318
WHAT IS THE PURPOSE OF THE STOOL FECAL FAT TEST
USED TO EVEALUATE HOW THE LIVER, GALL BLADDER, PANCREASE, AND INTESTINES ARE FUNCTIONING
319
INFANTS WITH CF AND PANCREATIC INSUFF WILL HAVE A FECAL ELASTANCE OF LESS THAN
50 UG/G OF STOOL
320
WHAT IS THE NORMAL VALUES FOR INFANTS FOR THE STOOL FECAL FAT TEST
NORMAL IS GREATER THAN 300 UG/G OF STOOL
321
OVERVIEW OF TEH CARDIOPULMONARY CLINICAL MANIFESTATIONS OF CF
ATELECTASIS BRONCHOSPASM EXCESSIVE BRONCHIAL SECRETIONS
322
VITALS SIGNS FOR CF PT
INCREASED BP RR (TACHYPNEA) HR
323
HOW ARE ACCESSORY MUSCLES AND BREATHING IN CF
used during respiration to assist with breathing due to increased work of breathing and airway obstruction. ACCESSORY MUSCLES FOR INSPIRATION AND EXPIRATION AND PURSED LIP BREATHING BARREL CHEST COUGH AND SPUTUM PRODUCTIO AND HEMPPYTSIS CYANOSIS
324
CHEST ASSESMENT FOR CF
DECREASED TACTILE AND VOCAL FREMITUS HYPER-RESONANT DIM BS DIM HEART SOUNDS BRONCHIAL BREATH SOUNDS (OVER ATELECTSASIS) CRACKLES WHEEZE SPONTANEOUS PNEUMOTHORAX
325
PULM FUNCTION TEST FOR SEVERE CF
FEV1/FVC RATIO - D PEFR - D FEF 25-75 - VT - N OR I IC - N OR D IRV - N OR D FRC - I ERV - N OR D TLC - N OR I RV - I VC -D RV/TLC RATIO - N OR I FVC -D FEVt -D FEF 50% - D FEF200-1200 -D
326
ABG FOR NORMAL CF PT
PH - ELEVATED PACO2 - DECREASED HC03- - DECREASED PAO2 - DECREASED SAO2/SPO2 - DECREASED
327
ABG OF SEVERE STAGE CF
PH = NORMAL PACO2 - ELEVATED HC03- - ELEVATED PAO2 - DECREASED SAO2/SPO2 - DECREASED
328
HEMODYNAMICS INDICES FOR MODERATE TO SEVRE STAGES CF
CVP - I RAP - I PA - I CO - N SV - N PCWP - N CL - N RVSWI - I LVSWI N PVR -I SVR - N
329
ABNORmal testiung for cf- hematology
increased hct/hgb INCREASED WBC COUNT
330
ABNORMAL TESTING FOR ELECTROLYTES FOR CF
HYPOCHLOREMIA- (CHRONIC VENT FAILURE) INCREASED SERUM BICARBONATE
331
ABNORMAL TESTING SPUTUM FOR CF
GRAM + STAPH AUREAUS HAEMOPHILUS INFLUENZAE GRAM - PSEUDOMONAS AERUGINOSA STENTROPHOMONAS MALTOPHILIA BURkholderia cepacia
332
ABNORMAL CHEST RADIOGRAPH FOR CF
TRANSLUCENT (DARK) LUNG FIELDS DEPRESSED OR FLATTENED DIAPHRAGM RIGHT VENTRICULAR ENLARGEMENT MAY SHOW TRAM TRACKS PNEUMOTHORAX (SPONATEOUS) ABCESS FORMATION may show hyperinflation, atelectasis, and bronchiectasis.
333
COMMON NON-RESPIRATORY CLINCAL MANIFESTATIONS FOR CF
DIOS (Distal Intestinal Obstruction Syndrome) MALNUTRITION AND POOR BODY DEVELOPMENT DEF OF VIT A, D, E, AND K NASAL POLYPS AND SINUSITIS INFERTILITY (MALES)
334
GENETIC MANAGEMENT OF CF
THE PRIMARY GOALS ARE TO PREVENT PULM INFECTIOONS AND REDUCE THE AMOUNT OF THICK BRINCHIAL SECRETIONS IMPROVE THE AIR FLOW AND PROVIDE ADEQUEATE NUTRITION
335
RESP CARE TX PROTOCOLS FOR CF
O2 THERAPY AIRWAY CLEARANCE THERAPY LUNG EXPANSION THERAPY AERSOLOZISED MEDS MECH VENT PROTOCOL OTHER MEDICATIONS AND SPECIAL PROCESURES CFTR modulators and supplemental therapies. PATHOGENS AND ABX EXERCISE AND PULM REHAB ANTI INFLAMM THERAPY PREVENTION LUNG TRANSPLANT
336
THERAPY RECOMMENADATIONS FOR CF
BRONCHOPULM HYGIENE (VEST) NIV (IF APPROPRIATE) MECH VENT (IF APPROPRIATE) IS 10X/HR DB&C SURGERY (LUNG TRANSPLANT)
337
PHARM RECOMMENDATIONS FOR CF ACUTE
ACUTE - BROAD SPECTRUM ABX SABA PRN
338
PHARM RECOMMENDATIONS FOR CF MANAGEMENT
SABA PRN
339
PHARM RECOMMENDATIONS FOR CF ASSESS FOR
LABA LAMA ICS INHALED ABX DNA-ASE HYPERTONIC SALINE CFTR CORRECTOR/POTENTIATOR CFTR POTENTIATOR O2 THERAPY PANCREATIC LIPASE IBUPROFREN
340
TEST RECOMMENDATIONS FOR CF
PFT ABG (ACUTE AND BASELINE) CXR (ACUTE AND BASELINE) CBC WITH DIFF ELECTROLYET PANEL
341
WHAT ARE DIAGNOSTIC TEST RECOMMENDATIONS FOR CF
SWEAT CHLORIDE X2 (GREATER THAN 60MEQ/L MOLECULAR GENETIC TESTING (CFTR 2 MUTATIONS) NASAL POTENTIAL DIFFERENCE IMMUNOREACTIVE TRYPSIN STOOL FECAL FAT TEST
342
Pneumonia
An inflammatory process primarily affecting the gas exchange area of the lung, often resulting in alveolar consolidation and atelectasis.
343
Etiology (PNEUMONIA)
The study of the cause of a disease, in this case, pneumonia.
344
Community Acquired Pneumonia (CAP)
Pneumonia acquired from normal social contact, often caused by pathogens like Streptococcus pneumoniae.
345
Atypical Pneumonia
Pneumonia that does not present with classic symptoms and is typically caused by pathogens like Mycoplasma pneumoniae.
346
Hospital Acquired Pneumonia (HAP)
Pneumonia that occurs 48 hours or more after hospital admission and was not present at the time of admission.
347
Aspiration Pneumonia
A type of pneumonia caused by the inhalation of foreign materials, such as gastric fluids, into the lungs.
348
Mendelson's Syndrome
A condition associated with aspiration of acidic stomach contents, causing tachycardia, dyspnea, and cyanosis.
349
Fungal infections in pneumonia
Infections caused by fungi, such as Histoplasmosis and Coccidioidomycosis, that can lead to pneumonia.
350
Pneumocystis jirovecii (formerly Pneumocystis carinii)
A common opportunistic pathogen causing pneumonia in immunocompromised individuals, especially those with AIDS.
351
Nebulization Therapy
A treatment for pneumonia involving the use of nebulizers to deliver medication directly into the lungs.
352
Ventilator Associated Pneumonia (VAP)
Pneumonia that occurs more than 48 hours after endotracheal intubation.
353
Alveolar consolidation
The filling of air spaces in the lungs with fluid, pus, blood, or other material, disrupting normal gas exchange.
354
Diagnosis of pneumonia
Can include chest X-rays, sputum cultures, and blood tests to identify the causative organisms.
355
Streptococcus pneumoniae
A common bacterial pathogen that accounts for more than 80% of all bacterial pneumonias.
356
COPD
Chronic Obstructive Pulmonary Disease, a condition that can increase the risk of severe pneumonia.
357
Lobar pneumonia
A type of pneumonia that affects a large and continuous area of the lobe of a lung.
358
Pulmonary Function Tests
Tests that measure lung function and capacity, often with decreased values in pneumonia patients.
359
Chest radiograph findings
X-ray findings in pneumonia patients, which can show consolidation, atelectasis, and pleural effusion.
360
what happens in pneumonia
Structural changes in lung tissues due to pneumonia, including inflammation, consolidation, and impaired gas exchange. the result of an inflammation process that primarily affects the gas exchange
361
ANATOMIC ALTERATIONS OF LUNGS - PNEUMONIA
INFLAMMATION OF THE ALVEOLI ALVEOLI CONSOLIDATION ATELECTSASIS (ASPIRATION PNEUMONIA)
362
PNEUMONIA -ETIOLOGY AND EPIDEMIOLOGY
PNEUMONIA AND INFLUENZA COMBINED ARE THE 8TH LEADING CAUSE OF DEATH AMONG AMERICANS
363
IN WHOM IS PNEUMONIA BAD
WHOSE LUNGS ARE ALREADY DAMAGED BY COPD OR ASTHMA OR SMOKING
364
THE RISK OF DEATH FROM PNEUMONIA OR INFLUENZA IS HIGHER AMONG
PEOPLE WITH HEART DISEASE DIABETES OR WEAKENED IMMUNE SYSTEM
365
PNEUMONIA IS LEADING CAUSE OF MORTALITY AND MORBIDITY IN
CHILDREN
366
WHAT ARE CAUSES OF PNEUMONIA
BACTERIA FUNGI VIRUS PROTOZOA PARASITES TB ANAEROBIC ORGANISMS ASPIRATION INHALATION OF IRRITATING CHEMICALS (FOR EX. CHLORINE)
367
COMMON WAYS PNEUMONIA CAUSING AGENTS SPREAD
LAND DIRECTLY ON: MOUTH EYES NOSE LAND DIRECTLY ON NEARBY SRFACES
368
general pneumonia terminology
double pneumonia walking pneumonia
369
pneumonia is named for location such as the
bronchopneumonia lobarpneumonia interstitial pneumonia
370
what is community acquired pneumonia
pneumonia acquired from normal social contact
371
common causes of community acquired pneumonia
streptococcus pneumonaiae staphyloccocal pneumonia haemophilus influenza leginonella pneumophiia mycoplasma pneumoniae
372
STREPTOCCOUS PNEUMONAIE ACCOUNTS FOR
80% OF ALL BACTERIAL PNEUMONIA
373
STAPHYLOCCOSUS PNEUMONIA IS FROM WHAT BACTERIA
STAPHYOCCOUS AUREUES STEPHYLCOCUS ALBANS STAPHYLOCOCCUS EPIDERMIDIS
374
COMMON CAUSES OF COMMUNITY ACQUIRED PNEUMONIA ASSOCIATED WITH LOBAR PNEUMONIA
THE COMMON CAUSE IS MOXAXELLA CATARRHALIS
375
CLINICAL PRESENTATION IS OFTEN SUB ACUTE
PT PRESENTS WITH A VARIETY OF BOTH PULM AND EXTRAPULM FINDINGS MYCOPLASMA ORGANISMS IS THE MOST COMMON CAUSE CAUSES SIMILAR TO BOTH BACTERIAL AND VIRAL PNEUMONIA A COUGH THAT TENDS TO COME IN VIOLENT ATTACKS PRODUCING ONLY A SMALL AMOUNT OF WHITE MUCUS
376
ATYPICAL REFERS TO WHAT (PNEUMONIA)
THE ORGANISM ESCAPES IDENTIFICATION BY STANARD BACTERIOLOGIC TESTS GENERALLY ONLY A MODERATE AMOUNT OF EXPECTORANT SPUTUM THERE IS AN ABSENCE OF ALVEOLAR CONSOLIDATION THERE IS ONLY A MODERATE ELEVATION OF WBC COUNT THERE IS A LACK OF ALEVEOLAR EXUDATE
377
EXAMPLES OF ATYPICAL PNEUMONIA CAUSES
COXIella burnetii, Chlamydophila pneumoniae, Legionella pneumoniae
378
FACTS ABOUT COXIELLA BURNETTI
GRAM NEG BACTERIA CAUSES Q FEVER IN HUMANS
379
VIRUS ACCOUNT FOR
50% OF ALL PNEUMONIAS SEVERAL ARE ASSOCIATED WITH A COMMUNITY ACQUIRED ATYPICAL PNEUMONIA
380
VIRUS ASSOCIATED WITH ATYPICAL PNUEMONIA
RSV PARAINFLUENZA (CHILDREN) - RELATED TO MUMPS, RUBELLA, AND RSV INFLUENZA A AND B - MOST COMMON CAUSES OF VIRAL RESP TRACT INFECTIONS ADENOVIRUS HUMAN METAPNEUMOVIRUS - 2ND MOST COMMON CAUSE OF LOWER RESP INFECTIONS IN YOUNG CHILDREN
381
HOSPITAL ACQUIRED PNEUMONIA IS
A TYPE OF PNEUMONIA THAT OCCURS 48 HOURS OR MORE AFTER HOSPITAL ADMISSION AND THAT WAS NOT PRERSENT AT THE TIME OF ADMISSIONA TYPE OF PNEUMONIA THAT OCCURS 48 HOURS OR MORE AFTER HOSPITAL ADMISSION AND THAT WAS NOT PRESENT AT THE TIME OF ADMISSION.
382
MOST IMPORTANT PATHOGENS ASSOCIATED WITH HOSPITAL ACQUIRED PNEUMONIA
include Staphylococcus aureus, Pseudomonas aeruginosa, and Klebsiella pneumoniae. . MRSA
383
HEALTHCARE ASSOCIATED PNEUMONIA REFERS TO
A PT WHO HAVE RECENTLY BEEN HOSPITALIZED IN AN ACUTE CARE HOSPITAL WITH IN 90 DAYS OF THE INFECTION HAVE BEENRESIDING IN NURSING HOME OR LONG TERM CARE FACILITY AND HAVE RECIEVED PARENTERAL ANTIMICROBIAL THERAPY, CHEMOTHERAPY OR WOUND CARE WITH IN 30 DAYS OF PNEUMONIA
384
vent associated pneumonia is a pneumonia that
a pneumonia of infectious diseases origin that develops more than 48-72 hours after endotracheal intubation
385
common infections associated with vent associated pneumonia
include Staphylococcus aureus , Pseudomonas aeruginosa, Acinetobacter species. ENTERBACTER SPECIES S.MALTOPHILIA
386
CAUSES OF ASPIRATION PNEUMONIA
result from the inhalation of or aspiration of foreign material, such as food, liquid, or vomit, into the lungs, leading to infection. ASPIRATION OF GASTRIC FLUID OF 2.5 PH OR LESS can also cause significant lung injury and infection.
387
THE MAJOR CAUSE OF ASPIRATION PNEUMONIA - ASPIRATING ON GASTRIC JUICES IS THE MAJOR CAUSE OF
ANAEROBIC LUNG INFECTIONS
388
3 DISTINCTIVE FORMS OF ASPIRATION PNEUMONIA
TOXIC INJURY TO THE LUNG OBSTRUCTION BY FORGEIN BODY OR FLUID INFECTIONS
389
WHAT IS THE ISSUE WITH ASPIRATION PNEUMONIA
COMMONLY MISSED BECAUSE ACUTE INFLAMMATION REACTIONS MAY NOT BEGIN UNTIL SEVERAL HOURS OBSERVED ASPIRATION OF THE GASTRIC FLUID
390
THE INFLAMMATORY REACTION IN ASPIRATION PNEUMONIA GENERALLY
INCREASES IN SEVERITY 12-26 HOURS AND MAY PROGRESS TO ARDS
391
WHAT IS ARDS
Acute Respiratory Distress Syndrome, a serious condition characterized by widespread inflammation in the lungs, leading to respiratory failure.
392
MENDELSONS SYNDROME
a form of aspiration pneumonia resulting from the inhalation of gastric contents, leading to chemical pneumonitis.
393
SYMPTOMS OF MENDELSONS SYNDROME
TACHYCARDIA DYSPNEA CYANOSIS ASSOCIATED WITH ASPIRATION OF ACID STOMACH CONTENTS USUALLY CONFINED TO ASPIRATIONS PNEUMONITIS IN PREG WOMEN
394
WHAT IS GERD
THE REGULATION OF STOMACH CONTENTS INTO THE ESOPHAGUS
395
GERD IS SEEN 3X MORE IN WHAT PTS
ASTHMA
396
CHRONIC PNEUMONIA IS
TYPICALLY is a localized lesion in pts with a normal immune system with or without regional node involvement commonly seen in pt with tb and fungal diseases of the lungs pts usually have granulomatous inflammation
397
fungal infections - anatomic alterations of the lungs
alveolar consolidation alveolar capill;ary memebrane thickening/destruction caseous tubercles or granulomas cavity formation fibrosis and secondary calcification of the lung parencychema
398
primary pathogens of fungal diseases
histoplasmosis coccidiodmycosis blastomycosis paracoccidioidomycosis
399
facts about hisoplasmosis
most common fungal disease in the us - along river valleys of the midwest found in soil enriched with bird excreta
400
screening and diagnosis for histoplasmosis
fungal culture fungal stain - 100% accurate serology - relatively fast and accfurate test
401
how is histoplasmosis contracted
by inhaling the spores found in contaminated soil or dust.
402
what is coccididmycosis
caused by the inhalation ofCoccidioides immitis spores, often found in dry, arid regions.
403
screning and diagnosis for coccidiodomycosis
made by visaulization of distinctive spherules in microscopy of the pt: sputum tissue exudate biopsies spinal fluid
404
screening and diagnosis for blastomycosis
direct visaulaization of yeast in sputum smears culture of fungal infection
405
cryptococcus neoformans
caused from the high nitrogen content of pigeon droppings
406
aspergillus is found
in decaying vegetation and soil, and its spores are commonly inhaled.
407
Pneumonia in the immunocompromised host
Cytomegalovirus -members of herpes family Pneumocystis jirovecii - seen in pt who are profoundly immunosuppressed Mycobacterium avium complex - a series opportunistic infections that is caused by similar bacteria
408
Cytomegalovirus is common viral pulmonary complications of
aids
409
Pneumocystis jirovecii is an
Fatal form of pneumonia Seen in pt who are profoundly immunocompromised Major pulmonary infection seen in pts with HIV and AIDS
410
Mycobacterium avium complex is caused by what 2 bacteria
Mycobacterium avium Mycobacterium intercellulare Found in soil and dust particles
411
Other types of pneumonia impacting immunocompromised pt
Invasive aspergillosis Invasive candiasis Ricketsia Varicella Rubella Severe acute respiratory syndrome Lipoid pneumatic Avian influenza A
412
Lipoid pneumonitis is from
Aspiration of mineral oil
413
What is the mortality rate of varicella pneumonia is
About 20%
414
Where is mycobacterium avium complex found
Found in soil and dust particles
415
What is necrotizing pneumonia
The pneumonia that causes the death of lung tissue cells within the infected pulmonary parenchyma
416
What is the result of necrotizing pneumonia
In lung abscess
417
Major pathological or structural changes in necrotizing pneumonia
Alveolar consolidation Alveolar capillary and bronchial wall destruction Tissue necrosis Alveolar consolidation Alveolar capillary and bronchial wall destruction Tissue necrosis Cavity formation Fibrosis and calcification of the lung parenchychema Bronchial pleural fistula and emphysema Atelectasis Excessive airway secretions
418
What is a common complication of aspiration pneumonia
Lung abscess
419
What are pre disposing factors forming abscess for necrotizing pneumonia include
Alcohol abuse Seizure disorder General anesthesia Head trauma CVA Swallowing disorders
420
421
What are some of the reasons besides lung abscess associated with necrotizing pneumonia
Bronchial obstruction with secondary infection Vascular obstruction with tissue infraction Interstitial lung disorder with cavity formation Bullae or cyst that become infected Penetrating chest wounds lead to an infection
422
vital signs for pneumonia
chest pain cynanosis decreased lung expansionn cough and sputum production emopytsis
423
pulmonary function test for pneumonia
FVC -D FEV1T -N OR D FEV/FVC RATIO = N OR I FEF25-75 - N OR D FEF 50% N OR D FEF 200-1200 - N OR D FVC -D FEV1T -N OR D FEV/FVC RATIO = N OR I FEF25-75 - N OR D FEF 50% N OR D FEF 200-1200 - N OR D FRC - D TLC - D RV/TLC RATIO - N
424
WHAT TYPE OD DISEASE IS PNEUMONIA RESTRICTIVE OR OBSTRUCTIVE
RESTRICTIVE
425
ABG FOR SEVERE STAGES - PNEUMONA
PH - DECREASED PACO2 - ELEVATED HCO3- - ELEVATED PAO2-DECREASED SPO2/SAO2 -DECREASED
426
ABNORMAL TESTS FOR PNEUMONIA
ABNORMAL SPUTUM CHEST RADIOGRAPH (INCREASED DENSITY FROM CONSOLIDATION AND ATEECSTASIS AND INFILTRATES) ABNORMAL BRONCHOGRAM (MAY HAVE AIR OR FLUID FILLED CAVITIES) LUNG ABSCESS PLEURAL EFFUSIONS OR EMPHYSEMA
427
GENRAL MANAGEMENT OF PNEUMONIA
TH TX OF PNEUMONIA IS BASED ON THE SPECIFIC ETIOLOG OF THE PNEUMONIA THE SEVERITY OF THE SYMPTOMS BY THE PT O2 THERAPY LUNG EXPANISION LUNG EXPANISON THERAPY PROTOCL AIRWAY CLEARANCE THERANCE THORACENTIS
428
DIAGNOSTIC THORACENTIS
COLOR OROR RBC COUNT PROTIEN GLUCOSE LDH AMYLASE PH WRIGHTS, GRAM OR ACID FAST BACILLUS AFB STAIN AEROBOC, ANAAERBOIC, TB, AND FUNGAL CULTURES CYTOLOGY
429
RECOMMENDED THERAPY FOR PNEUMONIA
BRINCHIAL HYGIENE DB & C IS 10/HR
430
RECOMMENDED PHARM FOR PNEUMONIA
BROAD SPECTRUM ABX SABA PRN IF INDICATED ICS IF INDICATED O2 THERAPY IF INDICATED
431
RECOMMENDED TESTS FOR PNEUMONA
ABG CXR CBC WITH DIF SPUTUM WITH CULTURE AND SENSISTIBTY
432
pneumonia is said to be
PROCESS THAT PRIMARILY AFFECTS THE GAS EXCHANGE AREA OF THE LUNG
433
THE DEVELOPMENT OF PNEUMONIA OCCURS IN 4 STAGES
1. STAGE - OCCURS WITH IN 24 HOURS OF AN INFECTION. IN RESPONSE TO INFLAMM BLOOD FLOW TO ALEVOLI INCREASES 2. STAGE 2 - DAYS 2 -3 - CHARACTERIZED BY THE PRESENCE OF RBC, POLY MORPH ONUCLEAR LEUKOCYTES INCREASE TO ENGULF INDAING PATHOGENS 3. STAGE 3 - DAYS 4-6 - INCREASED NUMBER OF MACROPHAGES THAT MOVE INTO THE INFECTED AREA TO REMOVE CELLULAR DEBRIS 4. STAGE 4 - DAYS 7-100- CALLED THE RESOLIUTION STAGE. CHARACTERIZED BY THE REABSORPTION OF INFLAMM FLUIDS AND CELLULAR DEBRIS AND RE-ESTABLISHMENT OF NORMAL AIRWAYS AND ALVEOLI
434
WHEN IS ATELECTSASIS A NORMAL SIDE EFFECT
ASPIRATION PNEUMONIA
435
THE MAJOR PATHOLOGIC OR STRUCTUAL CHANGE ASSOCIATED WITH PNEUMONIA ARE AS FOLLOWS
1. INFLAMM OF THE ALVEOLI 2. ALVEOLI CONSOLIDATION 3. ATEECTASIS
436
WHAT DOES PNEUMONIA OFTEN MIMIC
A COMMON COLD
437
SOME BACTERIAL PNEUMONIAS ARE CALLED ATYPICAL BECUASE
THE PNEUMONIA CAUSED BY THESE (MYCOPLASMA PNEUMONIA, CHLAYMDIOPHILIA PNUEMONIA, AND LEGIONELLA ONEUMONIA) HAVE SLIGHTLY DIFFERENT SYMPTOMS, APPEAR DIIFERENT ON CXR OR RESPOND TO DIFFERENT ABX THAN TYPICAL PNUEMONIA
438
RISK FACTORS FOR ONUEMONIA
OVER 65 Y/O AGE ASPIRATION OFOROPHARYNGEAL SECRETIONS VIRAL RESP INFECTIONS CHRONIC ILLNESS CHRONIC RESP DISEASE (COPD, ASTHMA, CF) CANCER ESPECIALLY LUNG CA PROLONGED BED REST TRACHEOSTOMY OR ET TUBE ABDOMINAL OR THORACIC SURGERY RIB FRACTURE IMMUNOSUPPRESSIVE THERAPY AIDS
439
HOW IS PNUEMONIA TYPICALLY TRANSMITTED
PERSON TO PERSON THROUGH SMALL AEROLSIZED DROPLETS THAT ARE PRODUCED WHENAN INFECTED PERSON COUGH, SNEEZES, OR TALKS STRONGLY NEAR INFECTED PEOPLE
440
IF INFECTED DROPLETS LAND ON TEXTILES OR FABRICS HOW LONG CAN THEY LIVE
APPROX 26 DAYS ON COTTON APPROX 28 DAYS ON SILK APPROX 30 DAYS ON WOOL
441
HOW IS PNUEMONIA CLASSIFIED
1. THE PATHOGEN RESPONSIBLE 2. THE CLINCAL SETTING THE PNUEMONIA OCCURED
442
WHAT IS DOUBLE ONUEMONIA
BOTH LUNGS INVOLVED
443
WHAT IS WALKING PNEUMONIA
USED TO DESCRIBE A MILD CASE OF PNUEMONIA
444
BRONCHOPNEUMONIA REFERES TO
BY A PATCHY PATTERN PATTERN OF INFECTION THAT IS LIMITED TO TEH SEGEMNETAL BRONCHI AND SURROUNDING LUNG PARENCHYEMA USUALLY INVOLVES BOTH LOBES AND OFTEN SEEN IN LOWER LOBES
445
LOBAR PNEUMONIA REFERS TO
WIDESPREAD OR DIFFUSE ALVEOLAR INFLAMM AND CONSOLIDATION CONTINUED TO ONE OR MORE LOBES OF THE LUNG THE END RESULT OF SEVERE OR LONG TERM BRONCHOPNEUMONIA IN WHICH THE INFECTION SPREAD FROM ONE SEGEMNT TO ANOTHER UNTIL THE ENTIRE LOBE IS INVOLVED
446
INTERSTITIAL PNUEMONIA REFERS TO
USUALLY A DIFFUSE AND BILATERAL INFLAMM THAT PRIMARILY INVOLVES THE ALVEOLAR SEPTA AND INTERSTITIAL
447
COMMUNITY ACQUIRED PNA REFERS TO
PNEUMONIA FROM NORMAL SOCIAL CONTACT
448
STREPTOCOCCUS PNEUMONIAE IS
ACCOUNTS FOR 80% OF ALL TEH BACTERIAL PNUEMONIA IS GRAM + IS FOUND IN PARIS (DIPLOCOCCI) USUALLY TRANSMITTED FROM AEROSOL MOST ARE SENISTIVE TO PENICILLIN AND ITS DERIATIVES COMMONLY CULTURED FROM THE SPUTUM OF PATEINTS HAVING AN ACUTE EXACERBATIION OF CHRONIC BRINCHITIS
449
WHAT IS THE APPEARANCE OF STREPTOCOCCUS PNEUMONIAE
THE COCCI ARE ENCLOSED IN A SMOOTH, POLY SACCHARIDE CAPSULE THAT IS ESSENTIAL FOR VIRULENCE
450
STAPHLOCCOSUS HAS 2 MAJORS
1. STAPH AUREUS - RESPONSIBLE FOR MOST STAPH INFECTION 2. STAPH ALBUS/STAPH EPIDERMIDIS - PART OF NORMAL SKIN FLORA
451
STAPHYLOCCOCUS PNEUMONIA ARE
GRAM + BACTERIA FOUND SINGLY, AND IN IRREGULAR CLUSTERS OFTEN FOUND AFTER A PREDISPOSING VIRUS INFECTION AND OFTEN SEEN IN CHILDREN AND IMMUNOSUPPRESSED ADULTS
452
S. AUREUS IS OFTEN TRANSMITTED
BY CONTACT COMMON CAUSE OF HOSIPTAL ACQUIRED PNEUMONIA AND MRSA
453
HAEMOPHILUS INFLUENZAE IS
SOMMON HUMAN PHARYNGEAL FLORA ONE OF THE SMALLEST GRAM NEG. BACILLI APPEARS AS COCCOBACILLI THERE ARE 6 TYPES, LABELED A TO F MOST COMMON TYPE IS B -PATHOGENIC OFTEN SEEN IN CHILDREN 1 MONTH TO 6 YEARS OLD ALMOST ALWAYS THE CAUSE OF EPIGLOTITIS TRANSMITTED VIA AEROSOL OR CONTACT
454
WHICH PATEINTS IS H.INFLUENZAE COMMONLY CULTURED
SPUTUM OF PATIENTS HAVING ACUTE EXERABTION OF CHRONIC BRINCHITIS
455
RISK FACTORS FOR H. INFLUENZAE
COPD DEFECTS IN B CELL FUNCTION FUNCTIONAL AND ANATOMIC ASPLENIA HIV
456
LEGIONELLA PNUEMONIA IS
GRAM NEG BACILLUS AND ATYPICALL CONCENTRATION OF CERTAIN BRANCHED CHAIN LIPIDS
457
WHEN WAS THE FIRST CASE OF LEGIONELLA PNEUMONIA AND WHAT IS LEGIONAIRES DISEASE
IN JULY 1976 AT AMERICAN LEGION CONF. A SEVERE PNUEMONIA OUTBREAK OCCURED THE CAUSE EXCLUDED IDENTIFICATION FOR MANY MONTHS LEGIONAAIRES DIS - THE PNUEMONIA ASSOC WITH LEGIONIONLA HAVE NOW BEEN TERMED LEGIONAIRES
458
LEGIONELLA PNEUMONIA IS CLASSIFIED BY BOTH
1. BACTERIAL CAUSATIVE AGENT 2. AN ATYPICAL PNEUMONIA
459
WHERE DO MOST LEGIONALA PNUEMONIA RESISE
FREE LIVING ON SOIL AND WATER TEHY ACT AS A DECOMPOSER ORGANISMS MULTIPLES IN STANDING WATER SUCH AS CONTAMINATED MUD PUDDLES, ALRGE AIR CONDITIONING SYSTEMS AND WATER TANKS TRANSMITTED WHENIT BECOMES AIRBORNE AND ENTERES THE PT VOA AEROSOL
460
WHO DOES LEGIONELLA PNUEMONIA MOSTLY IMPACT
MIDDLE AGE MEN WHO SMOKE
461
KLEBISELLA PNUEMONIA IS PART OF WHAT BACTERIAL FAMILY
ENTERBACTERIACAE HE BACILUUS IS NAMED - FRIEDLANDER
462
WHAT IMPACT DOES KLEBISELLA PNEUMONIA NORMALLY HAVE
ASSOC WITH LOCAR PNUEMONIA
463
WHO IS AT RISK FOR KLEBSIELLA PNYEMONIA
MEN OLDER THAN 40 IN CHRONIC ALCOHOLICS OF BOTH GENDERS
464
WHAT KIND OF BACTERIA IS KLEBISELLA
GRAM NEG BACILLUS FOUND SINGLY, IN PAIRS, OR CHAINS NORMAL INHABITATES IN GI TRACT COOMON HOSPITAL ACQUIRED DISEASE NORMALLY TRANSMITTED VIA ROUTES SUCH AS: CLOTHING, IV ROUTES, AND HEALTH WORKER HANDS
465
HOW IS KLEBISELLA TRANSMITTED
DIRECTLY BY AEROSL OR INDIRECTLY BY CONTACT
466
WHAT IS THE MORTALITY RATE OF KLEBISELLA
VERY HIGH AS SEPTICEMIA IS A FREQUENT COMPLICATION
467
MORAXELLA CATARRHALIS IS WHAT TYPE OF BACTERIA
GRAM NEG DIPLOCOCCUS COLONIZES TO THE UPPER RESP TRACT COMMONLY IN CHILDREN IF IT OFTEN CUASED CASE OF OTOTIS MEDIA IN CHILDREN AND ASSOCIATED WITH EXACERBATIONS IN ADULTS WITH COPD
468
PSEUDOMONAS AERUGINOSA IS
HIGHLY MOBILE GRAM NEG BACILLUS OFTEN FOUND IN GI TRACT, BURNS, AND CATHERIZED URINARY TRACT OFTEN CULTURED FROM CHRINICALLY ILL PT AND PT WHO HAVE TRACH LEADING CAUSE OF HOSPITAL ACQUIRED PNEUMONIA THRIVES IN DAMPNESS
469
WHAT ARE RISK FACTORS FOR P. AERUGINOSA
NEUTROPENIA HIV INFECTION PRE-EXITING LUNG DISEASE ET TUBE PREVIOUS ABX USE
470
HOW IS P. AERUGINOSA TRANSMITTED
AEROSOL OR BY DIRECT CONTACT WITH FRESHLY CONTAMINATED ARTICLES
471
WHAT IS THE APPEARANCE OF P. AERUGINOSA
VERY MUCOID COLONIAL FORM
472
WHAT DOES THE SPUTUM LOOK LIKE IN PT WITH P. AERUGINOSA
GREEN AND SWEET SMELLINH
473
COMMON CAUSES OF COMMUNITY ACQUIRES ATYPICAL PNEUMONIA ARE
1. MYCOPLASM BACTERIA 2.COXIELLA BURNETTI 3. CHALMYDOPHILA SPP. 4. RSV 5. PARAINFLEUENZA 6. INFLUENZA A AND B 7. ADENOVIRUS 8. HUMAN METAPNEUMOVIRUS
474
WHAT DOES THE MYCOPLASMA BACTERIA LOOK LIKE
TINY CELL WALL CELL WALL DEFICIENT SMALLER THAN BACTERIA BUT LARGER THAN VIRUSES
475
THE PNUEMONIA CAUSED BY MYCOPLASMA ORGANISM IS OFTEN DESCRIBED AS
PRIMARY ATYPICAL PNEUMONIA THE AYTPICAL REFERS TO: 1. THE ORGANISM ESCAPES IDENTIFICATION 2. GENERALLY ONLY A MODERATE AMOUNT 3. THERE IS AN ABSENCE OF ALVEOLAR CONSOLIDATION 4. ONLY A MODERATE ELEVATION OF WBC COUNT 5. A LACK OF ALVEOLAR EXUDATE
476
HOW ARE THE SYMPTOMS OF MYCOPLASMA ORG
SIMILAR TO BACTERIAL AND VIRAL PNEUMONIA BUT MORE GRADUAL AND OFTEN MILDER COUGH THAT COMES IN VIOLENT ATTACKS IS COMMON SIGN PRODUCING ONLY A SMALL AMOUNT OF WHITE MCUCUS OFTEN SEEN IN PEOPLE WHO CONGREGATE OFTEN SAID IN PT THAT HAVE BEEN DX - AS WALKING PNEUMONIA
477
COXIELLA BURNETTI IS WHAT TYPE OF BACTERIA
GRAM NEG THAT CAUSES Q FEVER IN HUMANS MORE RESISTANT THAN OTHER RICKETSISAE PASSED VIA AEROSOL TO PERSON TO PERSON ACUTE PNUEMONIA AND CHRONIC ENDOCARDITIS ARE ALSO ASSCO WITH THIS SPECIES
478
CHLAMYDOPHILIA SPP IS FOUND WHERE NORMALLY
IN CERVIX, URETHRA RECTUM THROAT AND RESP TRACT FOUND IN THE FECES OF BIRDS
479
VIRUSES ACCOUNT FOR WHAT % OF ALL ONEUMONIAS
50% AND ARE ASSOC WITH A COMMUNITY ACQUIRED AATYPICAL PNEUMONIA
480
WHAT ARE THE SYMPTOMS OF VIRAL PNEUMONIA
FLU-LIKE SYMPTOMS UNPRODUCTIVE COUGH HEADACHE FEVER MUSCLE PAIN FATIGUE AS DISEASE PROGRESSES, PT MAY BECOME SHORT OF BREATH COUGH AND PRODUCE A SMALL AMOUNT OF CLEAR OR WHITE SPUTUM
481
WHAT POTENTIAL RISK DOES VIRAL PNEUMONIA CARRY
FOR THE DEVELOPMENT OF A SECONDARY BACTERIAL PNEUMONIA
482
HOW DO VIRUSES EXIST
THEY ARE PARASITIC AND DEPEND ON NUTRIENTS INSIDE THE CELL FOR THEIR METABOLIC AND REPRODUCTIVE NEEDS
483
WHAT % OF ACUTE RESP TRACT INFECTIONS ARE CAUSED BY VIRUSES
0.9
484
WHAT OTHER DISEASE BELONG TO THE RSV GROUP
PARAINFLUENZA MUMPS RUBELLA
485
WHO DOES RSV MOSTLY IMPACT
CHILDREN YOUNGER THAN 12 MONTHS AND OLDER ADULTS WITH UNDERLYING HEART OR PULM DISEASE
486
HOW IS RSV TRANSMITTED
AEROSOL ROUTE AND BY DIRECT CONTACT
487
THERE ARE 5 TYPES OF PARAMYXOINFLUENZA GROUP (AND RELATED TO MUMPS, RUBELLA AND RSV)
TYPES 1, 2, 3, 4a and 4b TYPES 1,2, AND 3 - ARE MAJOR CAUSES OF INFECTION IN HUMANS TYPE 1 IS CROUP TYPE 2-3 ARE ASSOCIATED WITH SEV INFECTION TYPES 1 -2 SEEN IN FALL AND LATE SPRING TRANSMITTED BY AEROSOL DROPLET AND DIRECT CONTACT
488
PARAINFLUENZA VIRUSES ARE KNOWN FOR THEIR ABILITY TO
SPREAD RAPIDLY
489
INFLUENZA A AND B
MOST COMMON CAUSES OF RESP TRACT INFECTIONS AND OCCUR IN EPIDEMIC IN WINTER MOST AT RISK CHILDREN AND OLDER ADULTS HABVE INCUBATION PERIOD OF 1-3 DAYS
490
HOW DO INFLUENZA A AND B SUSVIVE
IN CONDITIONS OF LOW TEMP AND LOW HUMIDITY
491
ADENOVIRUS SEROTYPE 7
HAS BEEN RELATED TO FATAL CASES OF PNEUMONIA IN CHILDREN
492
HOW IS ADENOVIRUS TRANSMITTED
AEROSOL
493
WHEN IS THE ADENOVIRUS MOST PREVALENT
FALL WINTER SPRING
494
HUMAN METPNEUMOVIRUS
NEG SINGLE STRAND RNA VRIUS ASSOC WITH A FAMILY OF VIRUSES THAT ASLO CAUS RSV AND PARAINFLEUNZA VIRUS TYPICALLY OCCURS IN YOUNG CHILDREN IS THE SECOND COMMON CAUSE OF LOWER RESP TRACT INFECTIONS IN YOUNG CHILDREM
495
HOSPITAL ACQUIRED PNUEMONIA IS DEFINED AS
PNEUMONIA THAT OCCURS 48 HOURS OR MORE AFTER HOSPITAK ADMISSION AND THAT WAS NOT PRESENT AT TIME OF ADMISSION
496
WHAT ARE THE MOST IMPORTANT PATHOGENTS ASSOC WITH HOSPITAL ASSOC PNEUMONIA
P.AERGUNIOSA MRSA METHICILLIN SENSISTIVE S.AUREUS ENTERBACTER SPP. KLEBESILLA PNEUMONIA ESCHERIA COLI SERRATIA MARCESCENS PROTEUS SPP. ACINBACTER SPP
497
THE PATIENT THAT DEVELOPS PNEUMONIA BETWEEN 4-7 DAYS AFTER OF HOSPITALIZATION, THE MOST COMMON PATHOGENS ARE
MRSA S. PNEUMONIOIAE H. INFLEUNZAE
498
IN PT HOSPITALIZED LONGER THAN 7 DAYS, WHICH PATHOGENS ARE RESPONSIBLE FOR PNEUMONIA
P. AERUGINOSA MRSA ENTERIC GRAM NEG ORGANISMS
499
WHAT IS HOSPITAP CARE ASSOCIATED PNUEMONIA
REFERES TO PT WHO HAVE BEEN HOSPITALIZED IN AN ACUTE CARE HOSPITAL WITHIN 90 DAYS OF AN INFECTION WHO RESIDE IN A NUSRING HOME OR LTCF WHO HAVE RECIEVED PARENTERAL ANTIMICROBIAL THERAPY CHEMO THERPY OR WOUND CARE WITHIN 30 DAYS OF PNEUMONIA
500
WHAT ARE COMMON PATHGOGENS OF HEALTH CARE ASSOC PNEUMONIA
COMMON PATHOEGNS INCLUDE MIXED AEROBIC AND ANAEROBIC MOUTH FLORA S.AUREUS ENTERIC GRAM NEG BACILLUS INFLUENZA M. TUBERCULOSIS
501
WHAT IS VENTILATO ASSOC PENUMONIA
CAN BE CATERGORIZE DAS A NOSCOSMIAL INFECTION DEFINED AS A PNEUMONIA THAT DEVELOPS MORE THAN 48 TO 72 HOURS AFTER ENDOTRACHEAL INTUBATION
502
WHAT PATHOGENS ARE ASSOC WITH VAP
P.AERUGINOSA ENTERBACTER KLEBISELLA ACINTEROBACTER SPP. STENOTROPHOMONAS MALTOPHILIA S. AUREUS
503
COMMON PATHOGENS ASSOC WITH ASPIRATION PENUMONIA ARE
INCLUDE ANAAEROBIC ORAL FLORA PEPTOSTREPTOCOCUS PEPTOCOCCI BACTERIODES FRAGILIS PREVOTELLA MELANINGENICA FUSOBACTERIUM SPP AEROIB BACTERIA KLEBISELLA STAPHYLOCOCCUS MYCOBACTERIUM TUBERCULOSIS
504
ASPIRATION PNUEMONI OCCURS BECUASE
ASPIRATION OF GASTRIC FLUID, IT HAS A PH OF 2.5 OR LESS CNA CAUSE A SERIOUS AND FATAL FORM OF PNUEMONIA
505
ASPIRATING GASTRIC FLUIDS IS MAJOR CAUSE OF
ANAEROBIC LUNG INFECTIONS
506
WHY IS ASPIRATION PNEUMONIA COMMONLY MISSED
BECAUSE ACUTE INFLAMM REACTION MAY NOT BEGIN UNTIL SEVERAL HOURS AFTER OBSERVED ASPIRATION OF THE GASTRIC FLUID
507
WHAT IS THE COMPLICATION WHICH CAN ARISE FROM ASPIRATION PNEUMONIA
ARDS THE INFLAMMATORY REACTION GENERALLY INCREASES SEVERITY FOR 12-26 HOURS AND MAY PROGRESS TO ARDS WHICH MAY INCLUDE INTRAALVEOLAR EDEMA INTRALVEOLAR HYALINE MEMBRANE FORMATION ATELECTATSIS
508
WHAT IS MENDELSON SYNDORME
DESCRIBED AS THE TACHYCARDIA, DYSPNEA, AND CYANOSIS ASSOCI WITH ASPIRATION USUALLY CONFINED TO ASPIRATION PNEUMONIA IN PREG WOMEN
509
HOW IS APRIRATION PNEUMONIA DEFINED
PULMONARY RESULT OF THE ENTRY OF MATERIAL FROM THE STOMACH OR UPPER RESP TRACT INTO THE LOWER AIRWAYS
510
3 DIDTSINCTIVE FORMS
1. TOXIC INJURY TO THE LUNG (CUASED BY GASTRIC JUICES) 2. OBSTRUCTION (FORGEIN BODY OR FLUID) 3. INFECTION
511
THERE IS A DIFFERENCE BETWEEN ASPIRATION OF GASTRIC CONTENTS AND THE ASPIRATION OF FOOD
ASPIRIARTION OF GASTRIC CONTENTS CAUSES INTIAL HYPOXEMIA REGARDLESS OF THE PH LEVEL OF THE ASPIRATE IF THE PH OF APSIRATE IS HIGH THE INTIAL INJURY IS REVERSIBLE OF THE PH OF THE ASPIRATE IS LOW THEN PARENCHYEMA DAMAGE MAY OCCUR WITH INFLAMM, EDMEA AND HEMORRHAGE
512
SIX CRANIAL NERVES WHICH CARRY MOTOR SIGNALS GENERATED BY CERBERAL AND BRAIN STEM ARE
V -TRIGEMINAL VII - FACIAL IX - GLOSSOPHARYNGEAL X -VAGUS XI - SPINAL ACCESORY XII HYPOGLOSSAL
513
DYSPHAGIA
THE RESULT OF AN ABNORMAL SWALLOW THAT CAN INVOLVE THE ORAL, PHARYNHEAL AND ESOPHAGEAL PHASES
514
WHAT TEST IS DONE TO TEST FOR DYSPHAGIA
EVANS BLUE DYE TEST
515
WHAT ARE THE NIRMAL SWALLOWING MECHANICS
1. ORAL PREP 2. ORAL 3. PHARYNGEAL 4. ESOPHEGEAL
516
TX OF DYSPHAGIA
IS SPECIFIC TO THE NATURE OF THE DISORDER VARIED METHODS OF PRESENTATION OF FOODS AND LIQUIDS, BLOUS VOLUMES AND CONSISTENCY, PSOTURAL MOVEMENTS AND FOOD TEMP CAN AFFECT THE DYNAMICS OF THE RELATION BETWEEN RESP AND SWALLOING
517
WHAT IS SILENT ASPIRATION
DEFINED AS THE ASPIRATION THAT DOES NOT EVOKE CLINICALLY OBSERVABLE ADVERSE SYMPTOMS SUCH AS: COUGHING ABD IMMEDIATE RESP DISTRESS
518
PT WITH TRACH ARE AT HIGH RISK FOR
SILENT ASPIRATION 55% TO 70% OF INTUBATED OF TRACH PT ASPIRATE
519
GRANULOMONAS ARE ASSOC
CHRONIC PENUMONIA AND FUNGAL DISEASE OF THE LUNG
520
BECAUSE FUNGI ARE _________ THE LUNGS ARE A PRIME LOCATION FOR FUNGAL INFECTIONS
AEROBES
521
HOW DOES FUNGI MAKE US SICK
WHEN SPORES INHALED THEY REACH THE LUNGS AND GERMINATE THEN THIS HAPPENS THE SPORES PROCUDE A FROTHY YEASTLIKE SUBSTANCE THAT LEADS TO AN INFLAMM RESPONSE
522
FUNGAL DISEASES OF THE LUNG CAUSE A CHRONIC __________ PULM DISORER
restrictive
523
when h.capsilulatum reaches the alveoli what happens
AT BODY TEMP, IT CONVERTS FROM ITS MYCELIAL FORM (MOLD) TO A PARASITIC YEAST FORM
524
WHAT ARE THE CLINICAL MANIFESTATIONS OF HISTOPLASMOSIS
ARE SIMILAR TO TB THE INCUBATION PERIOD IS ONLY 17 DAYS
525
WHAT ARE THE FORMS OF HSITOPLASMOSIS
1. ASYMPOTMATIC 2. ACUTE SYMPOTOMATIC PULM HISTOPLASMOSIS 3. CHRONIC SYMPTOMPMATIC PULM HISTOPLASMOSIS 4. DISSEMINATED HISTOPLASMOSIS
526
WHAT IS COCCIDIOMYCOSIS CAUSED FROM
INHALATION OF SPORES OF COCCIDIES INMITITIS WHICH IS SPHERICAL FUNGI CARRIED BY WINDBLOWN DUST PARTICLES ENDEMIC IN HOT REGIONS
527
BLASTOMYCOSIS
OCCURS IN 1 (OR 2) OUT OF EVERY 100,000 PEOPLE CAUSED BY B. DERMATIDIIS MOST COMMON IN PREG WOMEN AND MIDDLE AGED AFRICAN MEN PRIMARY PORTAL OF ENTRY OS THE LUNGS
528
OPPORTUNISTIC PATHOGENS
CANDIDIS ALBICANS CRYPTOCOCCUS NEOFORMATUS ASPERGILLUS
529
ALLERGIC BRONCHOPULMONARY ASPERGILLUS IS A FORM OF
ASTHMA
530
WHO IS CRYPTOCOCCUS NEOFORMANOUS USSUALLY SEEN
PTS WITH HIV
531
ONEUMOCYITIS ORGANISM IS WHAT TYPE OF ORGANISM
PROTOZOAN AND MORE RELATED TO FUNGI
532
LIPOID PNEUMOCYITIS
CAN DEVELOP IN THE LUNGS WHEN LIPIDS ENTER THE TRACHEAL BRONCHIAL TREE COMMON CAUSES INCLUDE EXOGENOUS SOURCES SUCH AS: INHLAED NOSE DROPS WITH AN OIL BASE OR THE ACCIDENTAL INHALATION OF COSMETIC OIL THE SEVERITY OF THE ONEUMONIA DEPENDS ON THE TYPE OF OIL ASPIRATED
533
WHEN MINERAL OIL IS ASPIRATED IN AEROSOL FORM
AN INTENSE PULM TISSUE REACTION OCCURS
534
WHAT IS THE RESOSONANCE DULL OR LOUD (HYPER)
DULL
535
536
what are pharmacological recommendations for cold per GOLD 2025
bronchodilators (laba, lama) ics if symptoms persist, then escalate LABA/LAMA OR LABA-ICS COMBO INTRODUCTION OF NEW THERAPIES SUCH AS : ENSIFENTRINE DUPILUMAB
537
WHAT ARE SURGICAL RECOMMENDATION FOR COPD PER GOLD 2025
LUNG VOLUME REDUCTION THERAPY BRONCHOSCOPIC LUNG VOLUME REDUCTION (I.E ENDOBRONCHIAL COILS OR ENDOBRONCHIAL VALVES) BULLECTOMY LUNG TRANSPLANATAION
538
WHAT ARE THERAPY RECOMMENDATIONS FOR COPD PER GOLD 2025
SMOKING CESSATION VACCINATIONS PULM REHAB PHARM TX BRONCHODILATORS ICS ENSIFENTRINE LUNG VOLUME REDUCTION SURGERY BRONCHOSCOPIC LUNG VOL REDUCTION O2 THERAPY
539
WHAT ARE THE NUTRITION RECOMMENDATIONS FOR COPD PER GOLD 2025
CHOOSE COMPLEX CARBS ADEQUEATE AMOUNT OF PROTIENS OPT FOR HEALTHY FATS ENSURE VITAMINS AND MINERALS (D, C, AND E AND POTASSIUM AND CALCIUM) LIMIT PORCESSED FOODS INCREASE HYDRATIONS
540
WHAT TESTING RECOMMENDATIONS FOR COPD PER GOLD 2025
SPIROMETRY (PRE/POST BRONCHODILATOR) LOW DOSE CT CARDIOVASCULAR EVAL PULM HYPERTENSION SCREENING
541
WHAT ARE THE DIFFERENT TYPES OF FREQUENCIES PER GOLD 2025 FOR COPD
EXACERBATION SYMPTOM MEDICATION USE 02 THERAPY
542
WHAT IS THE DIFFERENCE BETWEEN RESCUE OR RELIVER FOR COPD
USED INTERCHANGEABLY THESE MEDICATIONS ARE TYPICALLY SHORT ACTING THAT WORK QUICKLY TO RELAX THE MUSCLES AROUND THE AIRWAYS
543
PRN MEDICATIONS PER GOLD 2025 FOR COPD
GROUP A - LOW SYMPTOMS/LOW EXACERBATION GROUP B - HIGH SYMPTOMS/LOW EXACERBATION GROUP E - HIGH EXACERBATION
544
WHAT IS THE DIFFERENCE BETWEEN MAINTENANCE MEDS AND RESCUE/RELIEVER
MAINTENANCE - SCHEDULED TIME USE//DAILY AND REGULAR BASIS RESCUE/RELIEVER - AS NEEDED/PRN//FOR IMMEDIATE SYMPTOMS
545
WHAT ARE EXAMPLES OF MAINTENANCE THERAPY
LABA LAMA ICS
546
WHAT DO THE ABBREVIATIONS MEAN QD BID TID QID Q6H
QD - EVERY DAY BID - 2X A DAY TID - 3X A DAY QID - 4X A DAY Q6H - EVERY 6 HOURS
547
HOW DOES A ABG LOOK LIKE FOR COPD PT
PH - ACIDOIC PACO2 - ELEVATED PA02 - LOWER HC03- ELEVATED
548
HOW DOES A PFT LOOK LIKE FOR COPD PT
FVC -DECREASED FEV1 - DECREASED FEV1/FVC RATIO - DECREASED PEF - DECREASED PEF 50% - DECREASED PEF200-1200 -DECREASED TIDAL VOL -NORMAL OR INCREASED IPV - NORMAL OR DECREASED ERV - NORMAL OR DECREASED RV - NORMAL OR INACREASED VC -DECREASED IC - NORMAL OR DECREASED FRC - INCREASED TLC - NORMAL OR INCREASED RV/TLC RATIO NORMAL OR INCREASED
549
WHAT THERAPIES ARE USED TO ASSESS FOR IN ACUTE COPD EXACERBATION
SHORT ACTING BRONCHODILATORS (SABA FIRST LINE THERAPY) SAMA - IPATROPIUM (ATROVENT) COMBINATION THERAPY SABA + SAMA USED FOR BETTER SYMPTOM RELIEF AND INCREASED EFFECTIVENESS SYSTEMIC STEROIDS (ORAL OR IV) ABX - IF EXACERBATION IS LIKELY DUE TO A BACTERIAL INFECTION, PARTICULARLY IN PT WITH INCREASED SPUTUM PURULENCE OR WORSENG OF BASELINE SYMPTOMS O2 THERAPY - GOAL SPO2 88-92% EVALUATE FOR NIV ASSESS FOR MECH VENT
550
HOW DOES A CHEST XRAY LOOK LIKE FOR A COPD PT
COMMON FINDINGS TRANSLUCENT (DARK) LUNG FIELDS DEPRESSED OR FLATTENED DIAPHRAGM LONG AND NARROW HEART (PULLED DOWNWARD BY DIAPHRAGM) INCREASED RETROSTERNAL OR SPACE (LATERAL RADIOGRAPH)
551
WHAT LABORATORY TESTS ARE DONE FOR
HCT/HGB - POLYCYTHERMIA ELECTROLYTES (EARLY AND LATE STAGES) - HYPOCHLOREMIA HYPERNATREMIA (NA+) SPUTUM EXAM - SPRETOCOCUS PNEUMONAIE HEAMOPHILLUS INFLUENZAE MORAXELLA CATARRHALIS
552
BREATH SOUNDS FOR COPD PT
WHEEZE OFTEN SEEN EXPIRATION DECREASED BREATH SOUNDS - DUE TO HYPERINFLATION AND AIR TRAPPING CRACKLES OR RALES IN PT WITH CONSISTENT PULM INFECTIONS
553
WHAT IS A PINK PUFFER
A TERM USED TO DESCRIBE AN EMPHYSEMA OR COPD PT PT HAVE BARREL CHEST DUE TO HYPERINFLATION PUFFER REFERS TO THE FACT THESE PT ARE PURSED LIPPED PT OFTEN REFERS TO THE FACT THE PT APPEARS WELL OXYGENATION (PINK SKIN TONE)
554
WHAT IS A BLUE BLOATER
A TERM USED TO DECSRIBE COPD PT WITH PREDOMINATELY CHRONIC BRONCHITIS THESE PT TEND TO HAVE A CHRONIC COUGH, SPUTUM PORDUCTION AND CYANOSIS DUE TO SEVERE HYPOXEMIA MAY DEVELOP RIGHT HEART FAILURE DUE TO LONG TERM PULM HYPERTENSION FROM HYPOXEMIA
555
HOW DOES THE BP LOOK FOR COPD PT
PULSE PRESSURE CAN BE WIDENED WHICH MAY INDICATE CARDIOVASCULAR STRAIN ELEVATED SYSTOLIC BP
556
WHAT ARE THE 2 TYPES OF EMPHYSEMA
1. PANACINAR - ABNORMAL WEAKENING AND ENLARGEMENT OF ALL ALVEOLI DISTAL TO THE TERMINOLE BROCHIOLES 2. CENTRIACINAR - INVOLVE THE RESP BRONCHIOLES IN THE PROXIMAL (CENTRAL) PORTION OF THE ACINUS
557
MAJOR PATHOLOGIC OR STRUCTURAL CHANGES THAT ARE ASSOCIATED WITH CHRONIC BRONCHITIS
1. CHRONIC INFLAMMATION AND THICKENING OF THE WALLS OF THE PERIPHERAL AIRWAYS 2. EXCESSIVE MUCUS PRODUCTION AND ACCUMULATION
558
WHAT IS CHRONIC BRONCHITIS
THE PRESENCE OF A COUGH AND SPUTUM PRODUCTION FOR 3 MONTHS IN EACH CONSECUTEIVE YEARS, REMAIN A CLINICALLY AND EPIDEMICOLOGICALLY USEFUL BUT IT IS PRESENT IN A MINORITY OF SUBJECTS
559
WHAT IS EMPHYSEMA
CHARACTERIZED BY A WEAKENING AND PERSISTENT ENLARGEMENT OF THE AIR SPACES DISTAL TO THE TERMINAL BRONCHIOLES AND BY THE DESTRUCTION OF THE ALVEOLI WALLS
560
WHAT HAPPENS TO THE DISTAL AIRWAYS IN EMPHYSEMA
WEAKENED HAVE MUCUS BUILD UP AND TEND TO COLLAPSE DURING EXPIRATION IN RESPONSE TO INCREASED INTRAPLEURAL PRESSURE
561
WHAT ARE THE RISK FACTORS FOR COPD
GENETIC FACTORS - ALPHA 1 ANTITRYPSIN DEF AGE AND GENDER - AS A PERSON AGES THE RISK INCREASES GREATER AMONG MEN LUNG GROWTH AND DEVELOPMENT - ISSUES DURING GESTATION AND CHILDHOOD HAS POTENTIAL TO IMPACT FOR COPD EXPOSURE TO PARTICLES: TOBACCO OCCUPTAIONAL EXPOSURE INDOOR/OUTDOOR AIR POLLUTION SOCIOECONIMIC STATUS - POVERTY IS A CLEARLY A RISK FACTOR FOR COPD BRONCHIAL/ASTHMA HYPERSENSITIVITY CHRONIC BRONCHITIS RESP INFECTIONS DYSPANAPSIS
562
WHAT IS DYSPANPSIS
HAS BEEN INTRODUCED TO REFER TO A MISMATCH BETWEEN THE NORMAL CALIBER OF TEH AIRWAYS AND THE LUNG PARENCHYCHEMA CAPACITY
563
WHAT TEST IS REQUIRED TO DX COPD
SPIROMETRY SHOWING A POST BRONCHIDILATOR FEV1/FVC OF LESS THAN 0.7
564
WHAT 3 MAIN SPIROMETRY TESTS ARE USED TO MEASURE THE SEVERITY OF AIRFLOW LIMITATION
FVC FEV1 FEV1/FVC RATIO
565
WHAT ARE THE QUESTIONANIRES USED TO ASSESS SYMPTOMS
mMRC BREATHLESSNESS SCALE COPD ASSESSMENT TEST
566
ATS DEFINITION OF PT WITH COPD
BRONCHITIS - BASED ON MAJOR CLINICAL MANIFESTATIONS ASSOCIATED WITH THE DISEASE EMPHYSEMA - IS BASED ON PATHOLOGY
567
PT WITH COPD WHAT IS NORMALLY PRESENT
BRONCHITIS AND EMPHYSEMA
568
ANATOMIC ALTERATIONS ASSOCIATED WITH BRONCHIATITS
CHRONIC INFLAMM ABD THICKENING OF THE WALL OF THE PERIPHERAL AIRWAYS EXCESSIVE MUCUS PRODUCTION AND ACCUMULATION PARTIAL OR TOTAL MUCUS PLUGGING SMOOTH MUSCLE CONSTRICTION OF THE BRONCHIAL AIRWAYS AIR TRAPPING AND HYPERINFLATION OF ALVEOLI
569
ANATOMIC ALTERATIONS ASSOCIATED WITH EMPHYSEMA
PERSISTENT ENLARGEMENT AND DESTRUCTION OF THE AIR SPACES DISTAL TO THE TERMINAL BRONCHIOLES DESTRUCTION OF ALVEOLAR CAP MEMBRANE WEAKENING OF THE DISTAL AIRWAYS, PRIMARILY THE RESP BRONCHIOLES AIR TRAPPING AND HYPERINFLATION
570
ETIOLOGY AND EPIDEMIOLOGY OF COPD
ESTIMATED BETWEEN 10-15 MILLION PEOPLE IN THE US STATES EITHER HAVE CHRONIC BRONCHITIS, EMPHYSEMA, OR A COMBINATION OF BOTH
571
RISK FACTORS ACCORDING TO GOLD
GENETIC PRE-DISPOSITION AGE AND GENDER CONDITIONS THAT AFFECTS NORMAL LUNG GROWTH EXPOSURE TO PARTICLES SOCIOECONIMIC STATUS ASTHMA/BRONCHIAL HYPERSENSITIVITY CHRONIC BRONCHITIS RESP INFECTIONS TB
572
KEY INDICATORS FOR CONDISERING COPD IN PT OVER 40
DYSPNEA CHRONIC COUGH CHRONIC SPUTUM HX OF EXPOSURE TO RISK FACTORS FAMILY HX OF COPD
573
GOLD RECOMMENDS COMBINING ALL THE ABOVE ASSESSMENTS TO DETERMINE COPD
SYMPTOM ASSESMENT AIR FLOW LIMITATION ASSESMENT RISK OF EXACERBATION ASSESSMENT
574
ADDITIONAL SCREENING TOOLS TO DX COPD
BODE INDEX CHEXT RADIOGRAPHS CT OF CHEST LUNG VOL AND DIFFUSING CAPACITY OXIMETRY AND ABG ALPHA 1 ANTITRYPSIN DEF SCREENING EXERCISE TESTING
575
INSPECTION SIGNS FOR COPD
PURSED LIP BREATHING COUGH MUCUS PRODUCTION
576
HOW DOES THE PALPATION OF THE CHEST IN EMPHYSEMA
DECREASED TACTILE FREMITUS DECREASED CHEST EXPANSION PMI SHIFTS TO THE EPIGASTRIC AREA
577
HOW DOES THE PALPATION OF THE CHEST IN CHRONIC BRONCHITIS
NORMAL
578
HOW IS THE PERCUSSION OF THE CHEST IN EMPHYSEMA
HYPERRESONANCE DECREASED DIAPHTAGMATIC EXCURSION
579
HOW IS THE PERCUSIION OF THE CHEST CHRINIC BRIONCHITIS
NORMAL
580
HOW IS THE AUSCULATAION OF THE CHEST FOR EMPHYSEMA
DIMINISHED BREATH SOUNDS PROLONGED EXPIRATION DIMINISHED HEART SOUNDS
581
HOW IS AUSCULTATION OF THE CHEST FOR CHRONIC BRONCHITIS
CRACKLES WHEEZE
582
WHAT IS A TELL TALE SIGN OF EMPHYSEMA
DECREASED DLCO
583
WHAT ARE 2 DANGEROUS ABG SITUATIONS FOR EMPYSEMA AND CHRONIC BRONCHITIS
1.ACUTE ALVEOLAR HYPERINFLATION SUPERIMPOSED ON A CHRONIC VENTILATIORY FAILURE 2. ACUTE VENTILATORY FAILURE (ACUTE HYPOVENTILATION)SUPERIMPOSED ON CHRONIC VENTILATORY FAILURE
584
HOW DOES HCT/HGB LOOK LIKE IN CHRONIC BRINCHITIS
POLYCYTHEMIA
585
HOW DOES ELECTROLYTES LOOK LIKE CHRONIC BRONCHITIS
HYPOCHLOREMEIA HYPERNATREMIA (NA+)
586
HOW IS SPUTUM IN EMPHYSEMA
NORMAL
587
HOW IS SPUTUM IN CHRONIC BRONCHITIS
STREPTOCOCCUS PNEUMONIAE HAEMOPHILUS INFUENZAE MORAXELLA CATARRHALIS
588
HOW DOES A BRONCHOGRAM LOOK LIKE FOR CHRONIC BRONCHITIS
SMALL SPIKELIKE PROTRUSIONS
589
HOW DOES CHEST RADIOGRAPH LOOK LIKE FOR CHRONIC BRONCHITIS
LUNGS MAY BE CLEAR IF ONLY IN LARGE BRONCHIOLES OCCASSIONALLY TRANSLUCENT DEPRESSED OR FLATTENED DIAPHRAGM
590
HOW DOES THE MANAGEMENT OF STABLE COPD LOOK LIKE
REDUCE EXPOSURE TO RISK FACTORS PHARMALOGIC TX NON PHARMOLOGIC TX
591
WHAT ARE THE COPD GRADES BASED ON SEVERITY OF AIRFLOW
GARDE 1 - MILD = FEV1 GREATER OR EQUAL TO 80% GRADE 2 - MODERATE = FEV1 50% LESS THAN 80% GRADE 3 - SEVERE = 30% LESS THAN 50% THAN PREDICTED GRADE 4 - VERY SEVERE = FEV 1 LESS THAN 30%
592
TX FOR GRADE 1 COPD
LABA + LAMA GROUP A - A BRONCHODILATOR GROUP B - LABA/LAMA
593
WHAT IS THE PHARMACOLOGIC MANAGEMENT OF COPD
GROUP A - SABA/LABA PRN GROUP B - LABA-LAMA MAINTENANCE SABA PRN GROUP E - LABA-LAMA (ASSESS FOR ICS) MAINTENANCE SABA PRN
594
cystic fibrosis
FATAL DISORDER CHARACTERIZED BY EXCESSIVE PRODUCTION AND ACCUMULATION OF THE THICK MUCUS IN THE TRACHEOBROCHIAL TREE
595
CFTR
CYSTIC FIBROSIS TRANSMEMEBRANE CONDUCTANCE REGULATOR A GENE THAT, WHEN MUTATED, LEADS TO CF
596
BROCNHIAL OBSTRUCTION
A CONDITION WHERE BRONCHIAL AIRWAYS ARE BLOCKED WHICH CAN BE CAUSED BY MUCUS PLUGGING
597
ATELECTASIS
PARTIAL OR COMPLETE COLLAPSE OF THE LUNG OFTEN DUE TO BLOCKAGE OF THE AIR PASSAGES
598
MUCUS PLUGGING
ACCUMULATION OF THICK MUCUS IN THE AIRWAYS THAT OBSTRUCT AIRFLOW
599
SWEAT TEST
A DIAGNOSTIC TEST FOR CF THAT MEAUSRES THE AMOUNT OF SODIUM AND CHLORIDE IN SWEAT
600
ELEVATED SWEAT CHLORIDE
SWEAT CHLORIDE LEVELS GREATER THAN 60 MEQ/L ON TWO OCCSAIONS INIDCATE A DX OF CF
601
GENETIC COUNSELING
A PROCESS TO INFORM AND SUPPORT INDIVIDUALS REGARDING GENETIC ASPECTS OF CF INCLUDING RISKS AND INHERITANCE PATTERNS
602
PFT
TESTS THAT MEASURE LUNG FUNCTION AND CAPACITY OFTEN INDICATING THE SEVERITY OF CF
603
SYMPTOMS OF CF
Chronic cough, wheezing, sputum production, frequent respiratory infections, and failure to thrive.
604
Hematologic outcomes in CF
Increased hematocrit and hemoglobin levels, often in response to chronic hypoxia.
605
Nasal potential difference (NPD)
A measurement that assesses the transport of sodium and chloride across epithelial cell linings, indicative of CFTR function.
606
Pre-natal testing for CF
Screening for CF mutations in pregnant women to assess the risk of having a child with CF.
607
anatomic alterations of the lungs -cf
EXCESSIVE RODUCTION AND ACCULULATIONOF THICK TENACIOUS MUCUS IN THE TTRACHEA-BRONCHIAL TREE PARTIAL BRONCHIAL OBSTRUCTION (MUCUS PLUGGING) HYPERINFLATION OF ALVEOLI TOTAL BRONCHIAL OBSTRUCTION (MUCUS PLUGGING) ATELECTASIS
608
ETIOLOGY AND EPIDEMIOLOGY OF CF
AL GENE DISORDER CAUSED BY MUTATIONS IN APAIR OF GENES LOCATED ON CHROMOSOME 7 OVER 1700 DIFF MUTATIONS IN THE GENE THAT ENCODES FOR THE CF TRANSMEMBRANE CONDUCTANCE REGULATOR, CFTR.
609
THE ABNORMAL EXPRESSIONOF THE CFTR RESULTS IN
RESULTS IN THICK VICIOUS ACCULUMALTION IN THE LUNGS MUCUS BLOCKS THE PASSAGEWAYS OF THE PANCREAS WHICH PREVENTS ENZYMEABNORMLA TRANSPORT OF SODIUM AND CHLORIDE IONS ACROSS MANY TYPES OF EPIHERLIAL SURFACES S FROM THE PANCREAS FROM REACHING THE INTESTINE
610
6 CLASSES OF CFTR MUTATIONS
DIVIDED INTO 3 BROAD CATEGORIES: AFFECING THE QUALITY OF THE CFTR PROTIEN AFFECTING FUNCTION PF THE CFTR PROTEIN AS A GATING DEFECT (CLASSII) WHERE THE CHANNEL DOES NOT OPEN A CONDUCTANCE DEFECT (CLASS IV) WHERE THE ION FLOW IS IMPAIRED
611
HOW CF GENE IS INHERITED
A RECESSIVE GENE DISORDR THE CHILD MUST INHERIT 2 COPIES OF THE DEFECTIVE CG GENE TO HAVE DISESE IF BOTH PARENTS CARRY THE GENE, THE POSSIBILITY OF HEIR CHILDREN HAVING FOLLOW THE MENDELIAN inherited pattern of 1 in 4.
612
SCREENING AND DX OF CF
BASED ON THE CLINCAL MAIFESTATIONS ASSOCIATED WITH CF FAMILY HX OF CF LAB FINDINGS
613
2 CRITERIA MUST BE MET TO DX FOR CF
1) Clinical presentation consistent with CF symptoms . 2) Laboratory evidence of CFTR dysfunction or the presence of two CFTR mutations.
614
clinical eveidence of CF CFTR DYSFUNCTION
ELEVATED SWEAT CHLORIDE GREATER THAN 60 MEQ/L (ON 2 OCCASIONS) MOLECULAR DX - GENETIC TESTING ABNRMAL NASAL POTENTIAL DIFFERENCE
615
NEBORN SCREENING OF CF
A test performed on newborns to detect cystic fibrosis early, typically through the measurement of immunoreactive trypsinogen (IRT) levels.
616
HOW LONG HAS CF SCREENING IN NEWBORNS BEEN PRRACTICED IN ALL 50 STATES
2011
617
MOST INFANTS WITH CF HAVE AN ELEVATED
BLOOD LEVEL OF IMMUNOREACTIVE TRYPSINOGEN (IRT)
618
THE IMMUNOREACTIVE TRYPSIN IS MEASURED FROM
A blood sample taken from the newborn's heel. ON THE GUTHRIE CARDS
619
WHAT IS TEH SWEAT TEST
A diagnostic test used to measure the amount of chloride in sweat, helping to confirm a diagnosis of cystic fibrosis.
620
WHAT US THE GOLD STANADRD FOR DX TEST FOR CF
The sweat test is considered the gold standard diagnostic test for cystic fibrosis, measuring chloride levels in sweat to confirm the condition.
621
WHAT DOES THE SWEAT TEST MEASURE
The sweat test measures the concentration of chloride and sodium in sweat, which can indicate cystic fibrosis when elevated.
622
WHAT IS PILOCARPINA
a medication used to stimulate sweat production during the sweat test for cystic fibrosis. USUALLY DONE 2X
623
INFANTS WITH A + CF NEWBORN SCREENING RESULT SHOULD HAVE WHAT DONE
A diagnostic sweat test performed to confirm cystic fibrosis. AFTER 2 WEEKS OF AGE AND GREATER THAN 2 KG IF ASYMPOTAMTIC
624
SWEAT TESTS INTERPRETATIONS INFANTS LESS THAN 6 MONTHS OF AGE
LESS THAN OR EQUAL TO 29 MMOL/L - NORMAL 30-59 MMOL/L -INTERMEDIATE - POSSIBLE CF GREATER THAN OR EQUAL TO 60 MMOL/L - ABNRMAL - DX OF CF
625
SWEAT TESTS INTERPRETATIONS INFANTSOLDER 6 MONTHS OF AGE, CHILDREN AND ADULTS
LESS THAN OR EQUAL TO 39 MMOL/L - NORMAL 40-59 MMOL/L - INTERMEDIATE - POSSIBLE CF GREATER THAN OR EQUAL TO 60 MMOL/L - ABNORMAL - DX OF CF GREATER THAN 60MMOL/L ABNRONAL - DX OF CF
626
CLINICAL INDICATIONS JUSTIFYING THE INTIAL EVELAUTAION FOR CF
WHEEZE DISTAL INTEST. OBSTRUCTION SYNDROME CHRINIC COUGH PANCREAS INSUFFICIENT SPUTUM PRODUCTION PANCREATITIS PARASINUSITIS CHOLELITHIASIS GI ISSUES FAT SOLUBLE DEF (A, D, E, K) FREQ RESP INFECTIONS FAILURE TO THRIVE HYPOPROTEINEMIA HEPTOMEGALY LABNORMAL CT SCAN HEPTOBILARY PROLAPSE INFERTILITY MECONIUM ILEUS OBSTRUCTIVE AZOOSPERMIA MECONIUM PERITONITIS NASAL POLYPS FOCAL BILIARY CIRRHOSIS PARASINUSITIS CHOLELITHIASIS
627
MOLECULAR DX FOR CF
Molecular diagnosis for cystic fibrosis involves genetic testing to identify mutations in the CFTR gene, which is responsible for CF.
628
NASAL POTENTIAL DIFFERENCES OF CF
THE IMPAIRED TRANSPORT NA+ AND CL- ACROSS THE EPITHELIAL CELL LINING THE AIRWAY OF THE CF PT CAN BE MEASURED by nasal potential difference measurements. This reflects abnormal ion transport, characteristic of cystic fibrosis.
629
WHEN NA+ AND CL- MOVE ACROSS THE EPITHELIAL CELL MEMBRANE THEY GENERATE
an electrical potential difference, which can be measured to assess ion transport abnormalities in cystic fibrosis.
630
THE NASAL PASSAGES THE ELECTRICAL POTENTIAL DIFFERENCE IS CALLED
the nasal potential difference.
631
PRE NATAL TESTING FOR CF
PREG FEMALES OFFERED SCREENING FOR CF MUTATIONS IF BOTH PARENTS TEST + TEST FOR CF MUTATIONS THEN THE FETUS HAS A 1:4 CHANCE OF HAVING CF
632
STOOL FECAL FAT TEST FOR CF
a diagnostic test that measures the amount of fat in a stool sample, used to assess pancreatic function in cystic fibrosis patients. MEAUSRES THE AMOUNT OF FATE IN THE IN INFANTS STOOL AND THE % OF DIETARY FAT THAT IS NOT ABSORBED BY THE BODY
633
WHAT IS THE PURPOSE OF THE STOOL FECAL FAT TEST
USED TO EVEALUATE HOW THE LIVER, GALL BLADDER, PANCREASE, AND INTESTINES ARE FUNCTIONING
634
INFANTS WITH CF AND PANCREATIC INSUFF WILL HAVE A FECAL ELASTANCE OF LESS THAN
50 UG/G OF STOOL
635
WHAT IS THE NORMAL VALUES FOR INFANTS FOR THE STOOL FECAL FAT TEST
NORMAL IS GREATER THAN 300 UG/G OF STOOL
636
OVERVIEW OF TEH CARDIOPULMONARY CLINICAL MANIFESTATIONS OF CF
ATELECTASIS BRONCHOSPASM EXCESSIVE BRONCHIAL SECRETIONS
637
VITALS SIGNS FOR CF PT
INCREASED BP RR (TACHYPNEA) HR
638
HOW ARE ACCESSORY MUSCLES AND BREATHING IN CF
used during respiration to assist with breathing due to increased work of breathing and airway obstruction. ACCESSORY MUSCLES FOR INSPIRATION AND EXPIRATION AND PURSED LIP BREATHING BARREL CHEST COUGH AND SPUTUM PRODUCTIO AND HEMPPYTSIS CYANOSIS
639
CHEST ASSESMENT FOR CF
DECREASED TACTILE AND VOCAL FREMITUS HYPER-RESONANT DIM BS DIM HEART SOUNDS BRONCHIAL BREATH SOUNDS (OVER ATELECTSASIS) CRACKLES WHEEZE SPONTANEOUS PNEUMOTHORAX
640
PULM FUNCTION TEST FOR SEVERE CF
FEV1/FVC RATIO - D PEFR - D FEF 25-75 - VT - N OR I IC - N OR D IRV - N OR D FRC - I ERV - N OR D TLC - N OR I RV - I VC -D RV/TLC RATIO - N OR I FVC -D FEVt -D FEF 50% - D FEF200-1200 -D
641
ABG FOR NORMAL CF PT
PH - ELEVATED PACO2 - DECREASED HC03- - DECREASED PAO2 - DECREASED SAO2/SPO2 - DECREASED
642
ABG OF SEVERE STAGE CF
PH = NORMAL PACO2 - ELEVATED HC03- - ELEVATED PAO2 - DECREASED SAO2/SPO2 - DECREASED
643
HEMODYNAMICS INDICES FOR MODERATE TO SEVRE STAGES CF
CVP - I RAP - I PA - I CO - N SV - N PCWP - N CL - N RVSWI - I LVSWI N PVR -I SVR - N
644
ABNORmal testiung for cf- hematology
increased hct/hgb INCREASED WBC COUNT
645
ABNORMAL TESTING FOR ELECTROLYTES FOR CF
HYPOCHLOREMIA- (CHRONIC VENT FAILURE) INCREASED SERUM BICARBONATE
646
ABNORMAL TESTING SPUTUM FOR CF
GRAM + STAPH AUREAUS HAEMOPHILUS INFLUENZAE GRAM - PSEUDOMONAS AERUGINOSA STENTROPHOMONAS MALTOPHILIA BURkholderia cepacia
647
ABNORMAL CHEST RADIOGRAPH FOR CF
TRANSLUCENT (DARK) LUNG FIELDS DEPRESSED OR FLATTENED DIAPHRAGM RIGHT VENTRICULAR ENLARGEMENT MAY SHOW TRAM TRACKS PNEUMOTHORAX (SPONATEOUS) ABCESS FORMATION may show hyperinflation, atelectasis, and bronchiectasis.
648
COMMON NON-RESPIRATORY CLINCAL MANIFESTATIONS FOR CF
DIOS (Distal Intestinal Obstruction Syndrome) MALNUTRITION AND POOR BODY DEVELOPMENT DEF OF VIT A, D, E, AND K NASAL POLYPS AND SINUSITIS INFERTILITY (MALES)
649
GENETIC MANAGEMENT OF CF
THE PRIMARY GOALS ARE TO PREVENT PULM INFECTIOONS AND REDUCE THE AMOUNT OF THICK BRINCHIAL SECRETIONS IMPROVE THE AIR FLOW AND PROVIDE ADEQUEATE NUTRITION
650
RESP CARE TX PROTOCOLS FOR CF
O2 THERAPY AIRWAY CLEARANCE THERAPY LUNG EXPANSION THERAPY AERSOLOZISED MEDS MECH VENT PROTOCOL OTHER MEDICATIONS AND SPECIAL PROCESURES CFTR modulators and supplemental therapies. PATHOGENS AND ABX EXERCISE AND PULM REHAB ANTI INFLAMM THERAPY PREVENTION LUNG TRANSPLANT
651
THERAPY RECOMMENADATIONS FOR CF
BRONCHOPULM HYGIENE (VEST) NIV (IF APPROPRIATE) MECH VENT (IF APPROPRIATE) IS 10X/HR DB&C SURGERY (LUNG TRANSPLANT)
652
PHARM RECOMMENDATIONS FOR CF ACUTE
ACUTE - BROAD SPECTRUM ABX SABA PRN
653
PHARM RECOMMENDATIONS FOR CF MANAGEMENT
SABA PRN
654
PHARM RECOMMENDATIONS FOR CF ASSESS FOR
LABA LAMA ICS INHALED ABX DNA-ASE HYPERTONIC SALINE CFTR CORRECTOR/POTENTIATOR CFTR POTENTIATOR O2 THERAPY PANCREATIC LIPASE IBUPROFREN
655
TEST RECOMMENDATIONS FOR CF
PFT ABG (ACUTE AND BASELINE) CXR (ACUTE AND BASELINE) CBC WITH DIFF ELECTROLYET PANEL
656
WHAT ARE DIAGNOSTIC TEST RECOMMENDATIONS FOR CF
SWEAT CHLORIDE X2 (GREATER THAN 60MEQ/L MOLECULAR GENETIC TESTING (CFTR 2 MUTATIONS) NASAL POTENTIAL DIFFERENCE IMMUNOREACTIVE TRYPSIN STOOL FECAL FAT TEST
657
Pneumonia
An inflammatory process primarily affecting the gas exchange area of the lung, often resulting in alveolar consolidation and atelectasis.
658
Etiology (PNEUMONIA)
The study of the cause of a disease, in this case, pneumonia.
659
Community Acquired Pneumonia (CAP)
Pneumonia acquired from normal social contact, often caused by pathogens like Streptococcus pneumoniae.
660
Atypical Pneumonia
Pneumonia that does not present with classic symptoms and is typically caused by pathogens like Mycoplasma pneumoniae.
661
Hospital Acquired Pneumonia (HAP)
Pneumonia that occurs 48 hours or more after hospital admission and was not present at the time of admission.
662
Aspiration Pneumonia
A type of pneumonia caused by the inhalation of foreign materials, such as gastric fluids, into the lungs.
663
Mendelson's Syndrome
A condition associated with aspiration of acidic stomach contents, causing tachycardia, dyspnea, and cyanosis.
664
Fungal infections in pneumonia
Infections caused by fungi, such as Histoplasmosis and Coccidioidomycosis, that can lead to pneumonia.
665
Pneumocystis jirovecii (formerly Pneumocystis carinii)
A common opportunistic pathogen causing pneumonia in immunocompromised individuals, especially those with AIDS.
666
Nebulization Therapy
A treatment for pneumonia involving the use of nebulizers to deliver medication directly into the lungs.
667
Ventilator Associated Pneumonia (VAP)
Pneumonia that occurs more than 48 hours after endotracheal intubation.
668
Alveolar consolidation
The filling of air spaces in the lungs with fluid, pus, blood, or other material, disrupting normal gas exchange.
669
Diagnosis of pneumonia
Can include chest X-rays, sputum cultures, and blood tests to identify the causative organisms.
670
Streptococcus pneumoniae
A common bacterial pathogen that accounts for more than 80% of all bacterial pneumonias.
671
COPD
Chronic Obstructive Pulmonary Disease, a condition that can increase the risk of severe pneumonia.
672
Lobar pneumonia
A type of pneumonia that affects a large and continuous area of the lobe of a lung.
673
Pulmonary Function Tests
Tests that measure lung function and capacity, often with decreased values in pneumonia patients.
674
Chest radiograph findings
X-ray findings in pneumonia patients, which can show consolidation, atelectasis, and pleural effusion.
675
what happens in pneumonia
Structural changes in lung tissues due to pneumonia, including inflammation, consolidation, and impaired gas exchange. the result of an inflammation process that primarily affects the gas exchange
676
ANATOMIC ALTERATIONS OF LUNGS - PNEUMONIA
INFLAMMATION OF THE ALVEOLI ALVEOLI CONSOLIDATION ATELECTSASIS (ASPIRATION PNEUMONIA)
677
PNEUMONIA -ETIOLOGY AND EPIDEMIOLOGY
PNEUMONIA AND INFLUENZA COMBINED ARE THE 8TH LEADING CAUSE OF DEATH AMONG AMERICANS
678
IN WHOM IS PNEUMONIA BAD
WHOSE LUNGS ARE ALREADY DAMAGED BY COPD OR ASTHMA OR SMOKING
679
THE RISK OF DEATH FROM PNEUMONIA OR INFLUENZA IS HIGHER AMONG
PEOPLE WITH HEART DISEASE DIABETES OR WEAKENED IMMUNE SYSTEM
680
PNEUMONIA IS LEADING CAUSE OF MORTALITY AND MORBIDITY IN
CHILDREN
681
WHAT ARE CAUSES OF PNEUMONIA
BACTERIA FUNGI VIRUS PROTOZOA PARASITES TB ANAEROBIC ORGANISMS ASPIRATION INHALATION OF IRRITATING CHEMICALS (FOR EX. CHLORINE)
682
COMMON WAYS PNEUMONIA CAUSING AGENTS SPREAD
LAND DIRECTLY ON: MOUTH EYES NOSE LAND DIRECTLY ON NEARBY SRFACES
683
general pneumonia terminology
double pneumonia walking pneumonia
684
pneumonia is named for location such as the
bronchopneumonia lobarpneumonia interstitial pneumonia
685
what is community acquired pneumonia
pneumonia acquired from normal social contact
686
common causes of community acquired pneumonia
streptococcus pneumonaiae staphyloccocal pneumonia haemophilus influenza leginonella pneumophiia mycoplasma pneumoniae
687
STREPTOCCOUS PNEUMONAIE ACCOUNTS FOR
80% OF ALL BACTERIAL PNEUMONIA
688
STAPHYLOCCOSUS PNEUMONIA IS FROM WHAT BACTERIA
STAPHYOCCOUS AUREUES STEPHYLCOCUS ALBANS STAPHYLOCOCCUS EPIDERMIDIS
689
COMMON CAUSES OF COMMUNITY ACQUIRED PNEUMONIA ASSOCIATED WITH LOBAR PNEUMONIA
THE COMMON CAUSE IS MOXAXELLA CATARRHALIS
690
CLINICAL PRESENTATION IS OFTEN SUB ACUTE
PT PRESENTS WITH A VARIETY OF BOTH PULM AND EXTRAPULM FINDINGS MYCOPLASMA ORGANISMS IS THE MOST COMMON CAUSE CAUSES SIMILAR TO BOTH BACTERIAL AND VIRAL PNEUMONIA A COUGH THAT TENDS TO COME IN VIOLENT ATTACKS PRODUCING ONLY A SMALL AMOUNT OF WHITE MUCUS
691
ATYPICAL REFERS TO WHAT (PNEUMONIA)
THE ORGANISM ESCAPES IDENTIFICATION BY STANARD BACTERIOLOGIC TESTS GENERALLY ONLY A MODERATE AMOUNT OF EXPECTORANT SPUTUM THERE IS AN ABSENCE OF ALVEOLAR CONSOLIDATION THERE IS ONLY A MODERATE ELEVATION OF WBC COUNT THERE IS A LACK OF ALEVEOLAR EXUDATE
692
EXAMPLES OF ATYPICAL PNEUMONIA CAUSES
COXIella burnetii, Chlamydophila pneumoniae, Legionella pneumoniae
693
FACTS ABOUT COXIELLA BURNETTI
GRAM NEG BACTERIA CAUSES Q FEVER IN HUMANS
694
VIRUS ACCOUNT FOR
50% OF ALL PNEUMONIAS SEVERAL ARE ASSOCIATED WITH A COMMUNITY ACQUIRED ATYPICAL PNEUMONIA
695
VIRUS ASSOCIATED WITH ATYPICAL PNUEMONIA
RSV PARAINFLUENZA (CHILDREN) - RELATED TO MUMPS, RUBELLA, AND RSV INFLUENZA A AND B - MOST COMMON CAUSES OF VIRAL RESP TRACT INFECTIONS ADENOVIRUS HUMAN METAPNEUMOVIRUS - 2ND MOST COMMON CAUSE OF LOWER RESP INFECTIONS IN YOUNG CHILDREN
696
HOSPITAL ACQUIRED PNEUMONIA IS
A TYPE OF PNEUMONIA THAT OCCURS 48 HOURS OR MORE AFTER HOSPITAL ADMISSION AND THAT WAS NOT PRERSENT AT THE TIME OF ADMISSIONA TYPE OF PNEUMONIA THAT OCCURS 48 HOURS OR MORE AFTER HOSPITAL ADMISSION AND THAT WAS NOT PRESENT AT THE TIME OF ADMISSION.
697
MOST IMPORTANT PATHOGENS ASSOCIATED WITH HOSPITAL ACQUIRED PNEUMONIA
include Staphylococcus aureus, Pseudomonas aeruginosa, and Klebsiella pneumoniae. . MRSA
698
HEALTHCARE ASSOCIATED PNEUMONIA REFERS TO
A PT WHO HAVE RECENTLY BEEN HOSPITALIZED IN AN ACUTE CARE HOSPITAL WITH IN 90 DAYS OF THE INFECTION HAVE BEENRESIDING IN NURSING HOME OR LONG TERM CARE FACILITY AND HAVE RECIEVED PARENTERAL ANTIMICROBIAL THERAPY, CHEMOTHERAPY OR WOUND CARE WITH IN 30 DAYS OF PNEUMONIA
699
vent associated pneumonia is a pneumonia that
a pneumonia of infectious diseases origin that develops more than 48-72 hours after endotracheal intubation
700
common infections associated with vent associated pneumonia
include Staphylococcus aureus , Pseudomonas aeruginosa, Acinetobacter species. ENTERBACTER SPECIES S.MALTOPHILIA
701
CAUSES OF ASPIRATION PNEUMONIA
result from the inhalation of or aspiration of foreign material, such as food, liquid, or vomit, into the lungs, leading to infection. ASPIRATION OF GASTRIC FLUID OF 2.5 PH OR LESS can also cause significant lung injury and infection.
702
THE MAJOR CAUSE OF ASPIRATION PNEUMONIA - ASPIRATING ON GASTRIC JUICES IS THE MAJOR CAUSE OF
ANAEROBIC LUNG INFECTIONS
703
3 DISTINCTIVE FORMS OF ASPIRATION PNEUMONIA
TOXIC INJURY TO THE LUNG OBSTRUCTION BY FORGEIN BODY OR FLUID INFECTIONS
704
WHAT IS THE ISSUE WITH ASPIRATION PNEUMONIA
COMMONLY MISSED BECAUSE ACUTE INFLAMMATION REACTIONS MAY NOT BEGIN UNTIL SEVERAL HOURS OBSERVED ASPIRATION OF THE GASTRIC FLUID
705
THE INFLAMMATORY REACTION IN ASPIRATION PNEUMONIA GENERALLY
INCREASES IN SEVERITY 12-26 HOURS AND MAY PROGRESS TO ARDS
706
WHAT IS ARDS
Acute Respiratory Distress Syndrome, a serious condition characterized by widespread inflammation in the lungs, leading to respiratory failure.
707
MENDELSONS SYNDROME
a form of aspiration pneumonia resulting from the inhalation of gastric contents, leading to chemical pneumonitis.
708
SYMPTOMS OF MENDELSONS SYNDROME
TACHYCARDIA DYSPNEA CYANOSIS ASSOCIATED WITH ASPIRATION OF ACID STOMACH CONTENTS USUALLY CONFINED TO ASPIRATIONS PNEUMONITIS IN PREG WOMEN
709
WHAT IS GERD
THE REGULATION OF STOMACH CONTENTS INTO THE ESOPHAGUS
710
GERD IS SEEN 3X MORE IN WHAT PTS
ASTHMA
711
CHRONIC PNEUMONIA IS
TYPICALLY is a localized lesion in pts with a normal immune system with or without regional node involvement commonly seen in pt with tb and fungal diseases of the lungs pts usually have granulomatous inflammation
712
fungal infections - anatomic alterations of the lungs
alveolar consolidation alveolar capill;ary memebrane thickening/destruction caseous tubercles or granulomas cavity formation fibrosis and secondary calcification of the lung parencychema
713
primary pathogens of fungal diseases
histoplasmosis coccidiodmycosis blastomycosis paracoccidioidomycosis
714
facts about hisoplasmosis
most common fungal disease in the us - along river valleys of the midwest found in soil enriched with bird excreta
715
screening and diagnosis for histoplasmosis
fungal culture fungal stain - 100% accurate serology - relatively fast and accfurate test
716
how is histoplasmosis contracted
by inhaling the spores found in contaminated soil or dust.
717
what is coccididmycosis
caused by the inhalation ofCoccidioides immitis spores, often found in dry, arid regions.
718
screning and diagnosis for coccidiodomycosis
made by visaulization of distinctive spherules in microscopy of the pt: sputum tissue exudate biopsies spinal fluid
719
screening and diagnosis for blastomycosis
direct visaulaization of yeast in sputum smears culture of fungal infection
720
cryptococcus neoformans
caused from the high nitrogen content of pigeon droppings
721
aspergillus is found
in decaying vegetation and soil, and its spores are commonly inhaled.
722
Pneumonia in the immunocompromised host
Cytomegalovirus -members of herpes family Pneumocystis jirovecii - seen in pt who are profoundly immunosuppressed Mycobacterium avium complex - a series opportunistic infections that is caused by similar bacteria
723
Cytomegalovirus is common viral pulmonary complications of
aids
724
Pneumocystis jirovecii is an
Fatal form of pneumonia Seen in pt who are profoundly immunocompromised Major pulmonary infection seen in pts with HIV and AIDS
725
Mycobacterium avium complex is caused by what 2 bacteria
Mycobacterium avium Mycobacterium intercellulare Found in soil and dust particles
726
Other types of pneumonia impacting immunocompromised pt
Invasive aspergillosis Invasive candiasis Ricketsia Varicella Rubella Severe acute respiratory syndrome Lipoid pneumatic Avian influenza A
727
Lipoid pneumonitis is from
Aspiration of mineral oil
728
What is the mortality rate of varicella pneumonia is
About 20%
729
Where is mycobacterium avium complex found
Found in soil and dust particles
730
What is necrotizing pneumonia
The pneumonia that causes the death of lung tissue cells within the infected pulmonary parenchyma
731
What is the result of necrotizing pneumonia
In lung abscess
732
Major pathological or structural changes in necrotizing pneumonia
Alveolar consolidation Alveolar capillary and bronchial wall destruction Tissue necrosis Alveolar consolidation Alveolar capillary and bronchial wall destruction Tissue necrosis Cavity formation Fibrosis and calcification of the lung parenchychema Bronchial pleural fistula and emphysema Atelectasis Excessive airway secretions
733
What is a common complication of aspiration pneumonia
Lung abscess
734
What are pre disposing factors forming abscess for necrotizing pneumonia include
Alcohol abuse Seizure disorder General anesthesia Head trauma CVA Swallowing disorders
735
736
What are some of the reasons besides lung abscess associated with necrotizing pneumonia
Bronchial obstruction with secondary infection Vascular obstruction with tissue infraction Interstitial lung disorder with cavity formation Bullae or cyst that become infected Penetrating chest wounds lead to an infection
737
vital signs for pneumonia
chest pain cynanosis decreased lung expansionn cough and sputum production emopytsis
738
pulmonary function test for pneumonia
FVC -D FEV1T -N OR D FEV/FVC RATIO = N OR I FEF25-75 - N OR D FEF 50% N OR D FEF 200-1200 - N OR D FVC -D FEV1T -N OR D FEV/FVC RATIO = N OR I FEF25-75 - N OR D FEF 50% N OR D FEF 200-1200 - N OR D FRC - D TLC - D RV/TLC RATIO - N
739
WHAT TYPE OD DISEASE IS PNEUMONIA RESTRICTIVE OR OBSTRUCTIVE
RESTRICTIVE
740
ABG FOR SEVERE STAGES - PNEUMONA
PH - DECREASED PACO2 - ELEVATED HCO3- - ELEVATED PAO2-DECREASED SPO2/SAO2 -DECREASED
741
ABNORMAL TESTS FOR PNEUMONIA
ABNORMAL SPUTUM CHEST RADIOGRAPH (INCREASED DENSITY FROM CONSOLIDATION AND ATEECSTASIS AND INFILTRATES) ABNORMAL BRONCHOGRAM (MAY HAVE AIR OR FLUID FILLED CAVITIES) LUNG ABSCESS PLEURAL EFFUSIONS OR EMPHYSEMA
742
GENRAL MANAGEMENT OF PNEUMONIA
TH TX OF PNEUMONIA IS BASED ON THE SPECIFIC ETIOLOG OF THE PNEUMONIA THE SEVERITY OF THE SYMPTOMS BY THE PT O2 THERAPY LUNG EXPANISION LUNG EXPANISON THERAPY PROTOCL AIRWAY CLEARANCE THERANCE THORACENTIS
743
DIAGNOSTIC THORACENTIS
COLOR OROR RBC COUNT PROTIEN GLUCOSE LDH AMYLASE PH WRIGHTS, GRAM OR ACID FAST BACILLUS AFB STAIN AEROBOC, ANAAERBOIC, TB, AND FUNGAL CULTURES CYTOLOGY
744
RECOMMENDED THERAPY FOR PNEUMONIA
BRINCHIAL HYGIENE DB & C IS 10/HR
745
RECOMMENDED PHARM FOR PNEUMONIA
BROAD SPECTRUM ABX SABA PRN IF INDICATED ICS IF INDICATED O2 THERAPY IF INDICATED
746
RECOMMENDED TESTS FOR PNEUMONA
ABG CXR CBC WITH DIF SPUTUM WITH CULTURE AND SENSISTIBTY
747
pneumonia is said to be
PROCESS THAT PRIMARILY AFFECTS THE GAS EXCHANGE AREA OF THE LUNG
748
THE DEVELOPMENT OF PNEUMONIA OCCURS IN 4 STAGES
1. STAGE - OCCURS WITH IN 24 HOURS OF AN INFECTION. IN RESPONSE TO INFLAMM BLOOD FLOW TO ALEVOLI INCREASES 2. STAGE 2 - DAYS 2 -3 - CHARACTERIZED BY THE PRESENCE OF RBC, POLY MORPH ONUCLEAR LEUKOCYTES INCREASE TO ENGULF INDAING PATHOGENS 3. STAGE 3 - DAYS 4-6 - INCREASED NUMBER OF MACROPHAGES THAT MOVE INTO THE INFECTED AREA TO REMOVE CELLULAR DEBRIS 4. STAGE 4 - DAYS 7-100- CALLED THE RESOLIUTION STAGE. CHARACTERIZED BY THE REABSORPTION OF INFLAMM FLUIDS AND CELLULAR DEBRIS AND RE-ESTABLISHMENT OF NORMAL AIRWAYS AND ALVEOLI
749
WHEN IS ATELECTSASIS A NORMAL SIDE EFFECT
ASPIRATION PNEUMONIA
750
THE MAJOR PATHOLOGIC OR STRUCTUAL CHANGE ASSOCIATED WITH PNEUMONIA ARE AS FOLLOWS
1. INFLAMM OF THE ALVEOLI 2. ALVEOLI CONSOLIDATION 3. ATEECTASIS
751
WHAT DOES PNEUMONIA OFTEN MIMIC
A COMMON COLD
752
SOME BACTERIAL PNEUMONIAS ARE CALLED ATYPICAL BECUASE
THE PNEUMONIA CAUSED BY THESE (MYCOPLASMA PNEUMONIA, CHLAYMDIOPHILIA PNUEMONIA, AND LEGIONELLA ONEUMONIA) HAVE SLIGHTLY DIFFERENT SYMPTOMS, APPEAR DIIFERENT ON CXR OR RESPOND TO DIFFERENT ABX THAN TYPICAL PNUEMONIA
753
RISK FACTORS FOR ONUEMONIA
OVER 65 Y/O AGE ASPIRATION OFOROPHARYNGEAL SECRETIONS VIRAL RESP INFECTIONS CHRONIC ILLNESS CHRONIC RESP DISEASE (COPD, ASTHMA, CF) CANCER ESPECIALLY LUNG CA PROLONGED BED REST TRACHEOSTOMY OR ET TUBE ABDOMINAL OR THORACIC SURGERY RIB FRACTURE IMMUNOSUPPRESSIVE THERAPY AIDS
754
HOW IS PNUEMONIA TYPICALLY TRANSMITTED
PERSON TO PERSON THROUGH SMALL AEROLSIZED DROPLETS THAT ARE PRODUCED WHENAN INFECTED PERSON COUGH, SNEEZES, OR TALKS STRONGLY NEAR INFECTED PEOPLE
755
IF INFECTED DROPLETS LAND ON TEXTILES OR FABRICS HOW LONG CAN THEY LIVE
APPROX 26 DAYS ON COTTON APPROX 28 DAYS ON SILK APPROX 30 DAYS ON WOOL
756
HOW IS PNUEMONIA CLASSIFIED
1. THE PATHOGEN RESPONSIBLE 2. THE CLINCAL SETTING THE PNUEMONIA OCCURED
757
WHAT IS DOUBLE ONUEMONIA
BOTH LUNGS INVOLVED
758
WHAT IS WALKING PNEUMONIA
USED TO DESCRIBE A MILD CASE OF PNUEMONIA
759
BRONCHOPNEUMONIA REFERES TO
BY A PATCHY PATTERN PATTERN OF INFECTION THAT IS LIMITED TO TEH SEGEMNETAL BRONCHI AND SURROUNDING LUNG PARENCHYEMA USUALLY INVOLVES BOTH LOBES AND OFTEN SEEN IN LOWER LOBES
760
LOBAR PNEUMONIA REFERS TO
WIDESPREAD OR DIFFUSE ALVEOLAR INFLAMM AND CONSOLIDATION CONTINUED TO ONE OR MORE LOBES OF THE LUNG THE END RESULT OF SEVERE OR LONG TERM BRONCHOPNEUMONIA IN WHICH THE INFECTION SPREAD FROM ONE SEGEMNT TO ANOTHER UNTIL THE ENTIRE LOBE IS INVOLVED
761
INTERSTITIAL PNUEMONIA REFERS TO
USUALLY A DIFFUSE AND BILATERAL INFLAMM THAT PRIMARILY INVOLVES THE ALVEOLAR SEPTA AND INTERSTITIAL
762
COMMUNITY ACQUIRED PNA REFERS TO
PNEUMONIA FROM NORMAL SOCIAL CONTACT
763
STREPTOCOCCUS PNEUMONIAE IS
ACCOUNTS FOR 80% OF ALL TEH BACTERIAL PNUEMONIA IS GRAM + IS FOUND IN PARIS (DIPLOCOCCI) USUALLY TRANSMITTED FROM AEROSOL MOST ARE SENISTIVE TO PENICILLIN AND ITS DERIATIVES COMMONLY CULTURED FROM THE SPUTUM OF PATEINTS HAVING AN ACUTE EXACERBATIION OF CHRONIC BRINCHITIS
764
WHAT IS THE APPEARANCE OF STREPTOCOCCUS PNEUMONIAE
THE COCCI ARE ENCLOSED IN A SMOOTH, POLY SACCHARIDE CAPSULE THAT IS ESSENTIAL FOR VIRULENCE
765
STAPHLOCCOSUS HAS 2 MAJORS
1. STAPH AUREUS - RESPONSIBLE FOR MOST STAPH INFECTION 2. STAPH ALBUS/STAPH EPIDERMIDIS - PART OF NORMAL SKIN FLORA
766
STAPHYLOCCOCUS PNEUMONIA ARE
GRAM + BACTERIA FOUND SINGLY, AND IN IRREGULAR CLUSTERS OFTEN FOUND AFTER A PREDISPOSING VIRUS INFECTION AND OFTEN SEEN IN CHILDREN AND IMMUNOSUPPRESSED ADULTS
767
S. AUREUS IS OFTEN TRANSMITTED
BY CONTACT COMMON CAUSE OF HOSIPTAL ACQUIRED PNEUMONIA AND MRSA
768
HAEMOPHILUS INFLUENZAE IS
SOMMON HUMAN PHARYNGEAL FLORA ONE OF THE SMALLEST GRAM NEG. BACILLI APPEARS AS COCCOBACILLI THERE ARE 6 TYPES, LABELED A TO F MOST COMMON TYPE IS B -PATHOGENIC OFTEN SEEN IN CHILDREN 1 MONTH TO 6 YEARS OLD ALMOST ALWAYS THE CAUSE OF EPIGLOTITIS TRANSMITTED VIA AEROSOL OR CONTACT
769
WHICH PATEINTS IS H.INFLUENZAE COMMONLY CULTURED
SPUTUM OF PATIENTS HAVING ACUTE EXERABTION OF CHRONIC BRINCHITIS
770
RISK FACTORS FOR H. INFLUENZAE
COPD DEFECTS IN B CELL FUNCTION FUNCTIONAL AND ANATOMIC ASPLENIA HIV
771
LEGIONELLA PNUEMONIA IS
GRAM NEG BACILLUS AND ATYPICALL CONCENTRATION OF CERTAIN BRANCHED CHAIN LIPIDS
772
WHEN WAS THE FIRST CASE OF LEGIONELLA PNEUMONIA AND WHAT IS LEGIONAIRES DISEASE
IN JULY 1976 AT AMERICAN LEGION CONF. A SEVERE PNUEMONIA OUTBREAK OCCURED THE CAUSE EXCLUDED IDENTIFICATION FOR MANY MONTHS LEGIONAAIRES DIS - THE PNUEMONIA ASSOC WITH LEGIONIONLA HAVE NOW BEEN TERMED LEGIONAIRES
773
LEGIONELLA PNEUMONIA IS CLASSIFIED BY BOTH
1. BACTERIAL CAUSATIVE AGENT 2. AN ATYPICAL PNEUMONIA
774
WHERE DO MOST LEGIONALA PNUEMONIA RESISE
FREE LIVING ON SOIL AND WATER TEHY ACT AS A DECOMPOSER ORGANISMS MULTIPLES IN STANDING WATER SUCH AS CONTAMINATED MUD PUDDLES, ALRGE AIR CONDITIONING SYSTEMS AND WATER TANKS TRANSMITTED WHENIT BECOMES AIRBORNE AND ENTERES THE PT VOA AEROSOL
775
WHO DOES LEGIONELLA PNUEMONIA MOSTLY IMPACT
MIDDLE AGE MEN WHO SMOKE
776
KLEBISELLA PNUEMONIA IS PART OF WHAT BACTERIAL FAMILY
ENTERBACTERIACAE HE BACILUUS IS NAMED - FRIEDLANDER
777
WHAT IMPACT DOES KLEBISELLA PNEUMONIA NORMALLY HAVE
ASSOC WITH LOCAR PNUEMONIA
778
WHO IS AT RISK FOR KLEBSIELLA PNYEMONIA
MEN OLDER THAN 40 IN CHRONIC ALCOHOLICS OF BOTH GENDERS
779
WHAT KIND OF BACTERIA IS KLEBISELLA
GRAM NEG BACILLUS FOUND SINGLY, IN PAIRS, OR CHAINS NORMAL INHABITATES IN GI TRACT COOMON HOSPITAL ACQUIRED DISEASE NORMALLY TRANSMITTED VIA ROUTES SUCH AS: CLOTHING, IV ROUTES, AND HEALTH WORKER HANDS
780
HOW IS KLEBISELLA TRANSMITTED
DIRECTLY BY AEROSL OR INDIRECTLY BY CONTACT
781
WHAT IS THE MORTALITY RATE OF KLEBISELLA
VERY HIGH AS SEPTICEMIA IS A FREQUENT COMPLICATION
782
MORAXELLA CATARRHALIS IS WHAT TYPE OF BACTERIA
GRAM NEG DIPLOCOCCUS COLONIZES TO THE UPPER RESP TRACT COMMONLY IN CHILDREN IF IT OFTEN CUASED CASE OF OTOTIS MEDIA IN CHILDREN AND ASSOCIATED WITH EXACERBATIONS IN ADULTS WITH COPD
783
PSEUDOMONAS AERUGINOSA IS
HIGHLY MOBILE GRAM NEG BACILLUS OFTEN FOUND IN GI TRACT, BURNS, AND CATHERIZED URINARY TRACT OFTEN CULTURED FROM CHRINICALLY ILL PT AND PT WHO HAVE TRACH LEADING CAUSE OF HOSPITAL ACQUIRED PNEUMONIA THRIVES IN DAMPNESS
784
WHAT ARE RISK FACTORS FOR P. AERUGINOSA
NEUTROPENIA HIV INFECTION PRE-EXITING LUNG DISEASE ET TUBE PREVIOUS ABX USE
785
HOW IS P. AERUGINOSA TRANSMITTED
AEROSOL OR BY DIRECT CONTACT WITH FRESHLY CONTAMINATED ARTICLES
786
WHAT IS THE APPEARANCE OF P. AERUGINOSA
VERY MUCOID COLONIAL FORM
787
WHAT DOES THE SPUTUM LOOK LIKE IN PT WITH P. AERUGINOSA
GREEN AND SWEET SMELLINH
788
COMMON CAUSES OF COMMUNITY ACQUIRES ATYPICAL PNEUMONIA ARE
1. MYCOPLASM BACTERIA 2.COXIELLA BURNETTI 3. CHALMYDOPHILA SPP. 4. RSV 5. PARAINFLEUENZA 6. INFLUENZA A AND B 7. ADENOVIRUS 8. HUMAN METAPNEUMOVIRUS
789
WHAT DOES THE MYCOPLASMA BACTERIA LOOK LIKE
TINY CELL WALL CELL WALL DEFICIENT SMALLER THAN BACTERIA BUT LARGER THAN VIRUSES
790
THE PNUEMONIA CAUSED BY MYCOPLASMA ORGANISM IS OFTEN DESCRIBED AS
PRIMARY ATYPICAL PNEUMONIA THE AYTPICAL REFERS TO: 1. THE ORGANISM ESCAPES IDENTIFICATION 2. GENERALLY ONLY A MODERATE AMOUNT 3. THERE IS AN ABSENCE OF ALVEOLAR CONSOLIDATION 4. ONLY A MODERATE ELEVATION OF WBC COUNT 5. A LACK OF ALVEOLAR EXUDATE
791
HOW ARE THE SYMPTOMS OF MYCOPLASMA ORG
SIMILAR TO BACTERIAL AND VIRAL PNEUMONIA BUT MORE GRADUAL AND OFTEN MILDER COUGH THAT COMES IN VIOLENT ATTACKS IS COMMON SIGN PRODUCING ONLY A SMALL AMOUNT OF WHITE MCUCUS OFTEN SEEN IN PEOPLE WHO CONGREGATE OFTEN SAID IN PT THAT HAVE BEEN DX - AS WALKING PNEUMONIA
792
COXIELLA BURNETTI IS WHAT TYPE OF BACTERIA
GRAM NEG THAT CAUSES Q FEVER IN HUMANS MORE RESISTANT THAN OTHER RICKETSISAE PASSED VIA AEROSOL TO PERSON TO PERSON ACUTE PNUEMONIA AND CHRONIC ENDOCARDITIS ARE ALSO ASSCO WITH THIS SPECIES
793
CHLAMYDOPHILIA SPP IS FOUND WHERE NORMALLY
IN CERVIX, URETHRA RECTUM THROAT AND RESP TRACT FOUND IN THE FECES OF BIRDS
794
VIRUSES ACCOUNT FOR WHAT % OF ALL ONEUMONIAS
50% AND ARE ASSOC WITH A COMMUNITY ACQUIRED AATYPICAL PNEUMONIA
795
WHAT ARE THE SYMPTOMS OF VIRAL PNEUMONIA
FLU-LIKE SYMPTOMS UNPRODUCTIVE COUGH HEADACHE FEVER MUSCLE PAIN FATIGUE AS DISEASE PROGRESSES, PT MAY BECOME SHORT OF BREATH COUGH AND PRODUCE A SMALL AMOUNT OF CLEAR OR WHITE SPUTUM
796
WHAT POTENTIAL RISK DOES VIRAL PNEUMONIA CARRY
FOR THE DEVELOPMENT OF A SECONDARY BACTERIAL PNEUMONIA
797
HOW DO VIRUSES EXIST
THEY ARE PARASITIC AND DEPEND ON NUTRIENTS INSIDE THE CELL FOR THEIR METABOLIC AND REPRODUCTIVE NEEDS
798
WHAT % OF ACUTE RESP TRACT INFECTIONS ARE CAUSED BY VIRUSES
0.9
799
WHAT OTHER DISEASE BELONG TO THE RSV GROUP
PARAINFLUENZA MUMPS RUBELLA
800
WHO DOES RSV MOSTLY IMPACT
CHILDREN YOUNGER THAN 12 MONTHS AND OLDER ADULTS WITH UNDERLYING HEART OR PULM DISEASE
801
HOW IS RSV TRANSMITTED
AEROSOL ROUTE AND BY DIRECT CONTACT
802
THERE ARE 5 TYPES OF PARAMYXOINFLUENZA GROUP (AND RELATED TO MUMPS, RUBELLA AND RSV)
TYPES 1, 2, 3, 4a and 4b TYPES 1,2, AND 3 - ARE MAJOR CAUSES OF INFECTION IN HUMANS TYPE 1 IS CROUP TYPE 2-3 ARE ASSOCIATED WITH SEV INFECTION TYPES 1 -2 SEEN IN FALL AND LATE SPRING TRANSMITTED BY AEROSOL DROPLET AND DIRECT CONTACT
803
PARAINFLUENZA VIRUSES ARE KNOWN FOR THEIR ABILITY TO
SPREAD RAPIDLY
804
INFLUENZA A AND B
MOST COMMON CAUSES OF RESP TRACT INFECTIONS AND OCCUR IN EPIDEMIC IN WINTER MOST AT RISK CHILDREN AND OLDER ADULTS HABVE INCUBATION PERIOD OF 1-3 DAYS
805
HOW DO INFLUENZA A AND B SUSVIVE
IN CONDITIONS OF LOW TEMP AND LOW HUMIDITY
806
ADENOVIRUS SEROTYPE 7
HAS BEEN RELATED TO FATAL CASES OF PNEUMONIA IN CHILDREN
807
HOW IS ADENOVIRUS TRANSMITTED
AEROSOL
808
WHEN IS THE ADENOVIRUS MOST PREVALENT
FALL WINTER SPRING
809
HUMAN METPNEUMOVIRUS
NEG SINGLE STRAND RNA VRIUS ASSOC WITH A FAMILY OF VIRUSES THAT ASLO CAUS RSV AND PARAINFLEUNZA VIRUS TYPICALLY OCCURS IN YOUNG CHILDREN IS THE SECOND COMMON CAUSE OF LOWER RESP TRACT INFECTIONS IN YOUNG CHILDREM
810
HOSPITAL ACQUIRED PNUEMONIA IS DEFINED AS
PNEUMONIA THAT OCCURS 48 HOURS OR MORE AFTER HOSPITAK ADMISSION AND THAT WAS NOT PRESENT AT TIME OF ADMISSION
811
WHAT ARE THE MOST IMPORTANT PATHOGENTS ASSOC WITH HOSPITAL ASSOC PNEUMONIA
P.AERGUNIOSA MRSA METHICILLIN SENSISTIVE S.AUREUS ENTERBACTER SPP. KLEBESILLA PNEUMONIA ESCHERIA COLI SERRATIA MARCESCENS PROTEUS SPP. ACINBACTER SPP
812
THE PATIENT THAT DEVELOPS PNEUMONIA BETWEEN 4-7 DAYS AFTER OF HOSPITALIZATION, THE MOST COMMON PATHOGENS ARE
MRSA S. PNEUMONIOIAE H. INFLEUNZAE
813
IN PT HOSPITALIZED LONGER THAN 7 DAYS, WHICH PATHOGENS ARE RESPONSIBLE FOR PNEUMONIA
P. AERUGINOSA MRSA ENTERIC GRAM NEG ORGANISMS
814
WHAT IS HOSPITAP CARE ASSOCIATED PNUEMONIA
REFERES TO PT WHO HAVE BEEN HOSPITALIZED IN AN ACUTE CARE HOSPITAL WITHIN 90 DAYS OF AN INFECTION WHO RESIDE IN A NUSRING HOME OR LTCF WHO HAVE RECIEVED PARENTERAL ANTIMICROBIAL THERAPY CHEMO THERPY OR WOUND CARE WITHIN 30 DAYS OF PNEUMONIA
815
WHAT ARE COMMON PATHGOGENS OF HEALTH CARE ASSOC PNEUMONIA
COMMON PATHOEGNS INCLUDE MIXED AEROBIC AND ANAEROBIC MOUTH FLORA S.AUREUS ENTERIC GRAM NEG BACILLUS INFLUENZA M. TUBERCULOSIS
816
WHAT IS VENTILATO ASSOC PENUMONIA
CAN BE CATERGORIZE DAS A NOSCOSMIAL INFECTION DEFINED AS A PNEUMONIA THAT DEVELOPS MORE THAN 48 TO 72 HOURS AFTER ENDOTRACHEAL INTUBATION
817
WHAT PATHOGENS ARE ASSOC WITH VAP
P.AERUGINOSA ENTERBACTER KLEBISELLA ACINTEROBACTER SPP. STENOTROPHOMONAS MALTOPHILIA S. AUREUS
818
COMMON PATHOGENS ASSOC WITH ASPIRATION PENUMONIA ARE
INCLUDE ANAAEROBIC ORAL FLORA PEPTOSTREPTOCOCUS PEPTOCOCCI BACTERIODES FRAGILIS PREVOTELLA MELANINGENICA FUSOBACTERIUM SPP AEROIB BACTERIA KLEBISELLA STAPHYLOCOCCUS MYCOBACTERIUM TUBERCULOSIS
819
ASPIRATION PNUEMONI OCCURS BECUASE
ASPIRATION OF GASTRIC FLUID, IT HAS A PH OF 2.5 OR LESS CNA CAUSE A SERIOUS AND FATAL FORM OF PNUEMONIA
820
ASPIRATING GASTRIC FLUIDS IS MAJOR CAUSE OF
ANAEROBIC LUNG INFECTIONS
821
WHY IS ASPIRATION PNEUMONIA COMMONLY MISSED
BECAUSE ACUTE INFLAMM REACTION MAY NOT BEGIN UNTIL SEVERAL HOURS AFTER OBSERVED ASPIRATION OF THE GASTRIC FLUID
822
WHAT IS THE COMPLICATION WHICH CAN ARISE FROM ASPIRATION PNEUMONIA
ARDS THE INFLAMMATORY REACTION GENERALLY INCREASES SEVERITY FOR 12-26 HOURS AND MAY PROGRESS TO ARDS WHICH MAY INCLUDE INTRAALVEOLAR EDEMA INTRALVEOLAR HYALINE MEMBRANE FORMATION ATELECTATSIS
823
WHAT IS MENDELSON SYNDORME
DESCRIBED AS THE TACHYCARDIA, DYSPNEA, AND CYANOSIS ASSOCI WITH ASPIRATION USUALLY CONFINED TO ASPIRATION PNEUMONIA IN PREG WOMEN
824
HOW IS APRIRATION PNEUMONIA DEFINED
PULMONARY RESULT OF THE ENTRY OF MATERIAL FROM THE STOMACH OR UPPER RESP TRACT INTO THE LOWER AIRWAYS
825
3 DIDTSINCTIVE FORMS
1. TOXIC INJURY TO THE LUNG (CUASED BY GASTRIC JUICES) 2. OBSTRUCTION (FORGEIN BODY OR FLUID) 3. INFECTION
826
THERE IS A DIFFERENCE BETWEEN ASPIRATION OF GASTRIC CONTENTS AND THE ASPIRATION OF FOOD
ASPIRIARTION OF GASTRIC CONTENTS CAUSES INTIAL HYPOXEMIA REGARDLESS OF THE PH LEVEL OF THE ASPIRATE IF THE PH OF APSIRATE IS HIGH THE INTIAL INJURY IS REVERSIBLE OF THE PH OF THE ASPIRATE IS LOW THEN PARENCHYEMA DAMAGE MAY OCCUR WITH INFLAMM, EDMEA AND HEMORRHAGE
827
SIX CRANIAL NERVES WHICH CARRY MOTOR SIGNALS GENERATED BY CERBERAL AND BRAIN STEM ARE
V -TRIGEMINAL VII - FACIAL IX - GLOSSOPHARYNGEAL X -VAGUS XI - SPINAL ACCESORY XII HYPOGLOSSAL
828
DYSPHAGIA
THE RESULT OF AN ABNORMAL SWALLOW THAT CAN INVOLVE THE ORAL, PHARYNHEAL AND ESOPHAGEAL PHASES
829
WHAT TEST IS DONE TO TEST FOR DYSPHAGIA
EVANS BLUE DYE TEST
830
WHAT ARE THE NIRMAL SWALLOWING MECHANICS
1. ORAL PREP 2. ORAL 3. PHARYNGEAL 4. ESOPHEGEAL
831
TX OF DYSPHAGIA
IS SPECIFIC TO THE NATURE OF THE DISORDER VARIED METHODS OF PRESENTATION OF FOODS AND LIQUIDS, BLOUS VOLUMES AND CONSISTENCY, PSOTURAL MOVEMENTS AND FOOD TEMP CAN AFFECT THE DYNAMICS OF THE RELATION BETWEEN RESP AND SWALLOING
832
WHAT IS SILENT ASPIRATION
DEFINED AS THE ASPIRATION THAT DOES NOT EVOKE CLINICALLY OBSERVABLE ADVERSE SYMPTOMS SUCH AS: COUGHING ABD IMMEDIATE RESP DISTRESS
833
PT WITH TRACH ARE AT HIGH RISK FOR
SILENT ASPIRATION 55% TO 70% OF INTUBATED OF TRACH PT ASPIRATE
834
GRANULOMONAS ARE ASSOC
CHRONIC PENUMONIA AND FUNGAL DISEASE OF THE LUNG
835
BECAUSE FUNGI ARE _________ THE LUNGS ARE A PRIME LOCATION FOR FUNGAL INFECTIONS
AEROBES
836
HOW DOES FUNGI MAKE US SICK
WHEN SPORES INHALED THEY REACH THE LUNGS AND GERMINATE THEN THIS HAPPENS THE SPORES PROCUDE A FROTHY YEASTLIKE SUBSTANCE THAT LEADS TO AN INFLAMM RESPONSE
837
FUNGAL DISEASES OF THE LUNG CAUSE A CHRONIC __________ PULM DISORER
restrictive
838
when h.capsilulatum reaches the alveoli what happens
AT BODY TEMP, IT CONVERTS FROM ITS MYCELIAL FORM (MOLD) TO A PARASITIC YEAST FORM
839
WHAT ARE THE CLINICAL MANIFESTATIONS OF HISTOPLASMOSIS
ARE SIMILAR TO TB THE INCUBATION PERIOD IS ONLY 17 DAYS
840
WHAT ARE THE FORMS OF HSITOPLASMOSIS
1. ASYMPOTMATIC 2. ACUTE SYMPOTOMATIC PULM HISTOPLASMOSIS 3. CHRONIC SYMPTOMPMATIC PULM HISTOPLASMOSIS 4. DISSEMINATED HISTOPLASMOSIS
841
WHAT IS COCCIDIOMYCOSIS CAUSED FROM
INHALATION OF SPORES OF COCCIDIES INMITITIS WHICH IS SPHERICAL FUNGI CARRIED BY WINDBLOWN DUST PARTICLES ENDEMIC IN HOT REGIONS
842
BLASTOMYCOSIS
OCCURS IN 1 (OR 2) OUT OF EVERY 100,000 PEOPLE CAUSED BY B. DERMATIDIIS MOST COMMON IN PREG WOMEN AND MIDDLE AGED AFRICAN MEN PRIMARY PORTAL OF ENTRY OS THE LUNGS
843
OPPORTUNISTIC PATHOGENS
CANDIDIS ALBICANS CRYPTOCOCCUS NEOFORMATUS ASPERGILLUS
844
ALLERGIC BRONCHOPULMONARY ASPERGILLUS IS A FORM OF
ASTHMA
845
WHO IS CRYPTOCOCCUS NEOFORMANOUS USSUALLY SEEN
PTS WITH HIV
846
ONEUMOCYITIS ORGANISM IS WHAT TYPE OF ORGANISM
PROTOZOAN AND MORE RELATED TO FUNGI
847
LIPOID PNEUMOCYITIS
CAN DEVELOP IN THE LUNGS WHEN LIPIDS ENTER THE TRACHEAL BRONCHIAL TREE COMMON CAUSES INCLUDE EXOGENOUS SOURCES SUCH AS: INHLAED NOSE DROPS WITH AN OIL BASE OR THE ACCIDENTAL INHALATION OF COSMETIC OIL THE SEVERITY OF THE ONEUMONIA DEPENDS ON THE TYPE OF OIL ASPIRATED
848
WHEN MINERAL OIL IS ASPIRATED IN AEROSOL FORM
AN INTENSE PULM TISSUE REACTION OCCURS
849
WHAT IS THE RESOSONANCE DULL OR LOUD (HYPER)
DULL
850
when a sample of arterial blood is analyzed for the presence of oxygen (Pao2) the value comes from the a. blood plasma b. leukocyte c. hemoglobin d. erythocyes
A. BLOOD PLASMA
851
OXYGEN CONSUMPTION 1. INCREASE WITH EXERCISE 2. IS THE AMOUNT OF OXYGEN USED BY THE BODY 3. IS INVERSELY RELATED T CRABONDISOXIDE 4. IS ABOUT 250 ML PER MIN IN THE RESTING ADULT
1, 2, 4
852
WHICH IOF THE FOLLOWING VALUES IS CONSIDERED A NORMAL HGB LEVEL IN A HEALTHY ADULT A. O.OO3-1.34 MEQ/L B. 12-16 G/DL C. 96% - 100% D, 14-20 G/DL
B. 12-16
853
A SAMPLE OF BLOOD HAS BEEN TAKEN FROM A PT PULM ARTERY . WHAT MIXED VENOUS OXYGEN SATURSATION (SV02) WOULD INDICATE THAT THE PT IS NORMAL A. 40 MMHG B. 95 MMHG C. 75% D. 97%
C. 75%
854
POLYCYTHEMIA IS 1. A CONDITION OF TOO MANY RED BLOOD CELLS 2. A CONDITION OF TOO FEW RED BLOOD CELLS 3. CAUSED BY A LACK OF IRON IN THE DIET 4. THE BODY'S RESPONSE TO CHRONIC HYPOXEMIA
1, 4
855
THE RT IS REVIEWING A PT EMR TO TRY TO VERIFY IF THE PT HAS TISSUE HYPOXIA. WHICH OF THE FOLLOWIN LABAORTORY VALUES IS MOST LIKELYTO CORRELATE WITH TISSUE HYPOXIA A. METABOLIC ACIDOSIS B. THROMBOCYOPNEIA C. HYPOKALEMIA D. BILLERUBEMIA
A. METABOLIC ACIDOSIS
856
A PT IS MILDLY HYPOEXEMIC. WHAT OF THE FOLLOWING SIGNS WOULD THE RT EXPECT TO FIND IN THIS PT. A. AN INCREASE IN THE T BREATHING RATE AND HR B. A DECREASE IN SYSTEMIC BP C. CARDIAC DYSRHYTHMIAS D. ANEMIA
A. AN INCREASE IN PT BREATHING RATE AND HR
857
YOU ARE WORKING WITH THEPHYSICIAN TO DETERMINE THE ANTIBIOYIC BESTSUITED TO TREAT YOUR PT PNEUMONIA. WHICH TEST WOULD YOU SUGGEST? A. AN EBUS EXAM B. CYTOLOGY EXAM C. BAL D. C & S TESTS
D. C& S
858
DURING A VATS, HOW IS THE THORASCOPE INSERTED A. VIA AN ET TUBE B. THROUGH A NEEDLE INSERTED THROUGH THE CHEST WALL C. VIA AN ARETRIAL CATH D. THROUGH A SMALL INCICION IS MADE IN THE CHEST WALL
D. THROUGH A SMALL INSICION IS MADE IN THE CHEST WALL
859
DIAGNOSTIC THORACENTISIS CAN BE USED: A. TO WITHDRAW A SCRETION SAMPLE FROM THE LUNG FOR A SPUTUM SMEAR B. WITHDRAW A SECRETION SAMPLE FROM THE LUNG FOR A GRAM STAIN C. DETERMINE THE ETIOLOGY OF A PLEURAL EFFUSION D. REMOVE AIR FROM THE PLEURAL SPACE
D. TO REMOVE AIR FROM TEH PLEURAL SPACE
860
THE RT IS CARING FOR A PT WHO IS RECIEVING A DIEUERTIC THERAPY AS PART OF THE MEDICATION REGIMEN FOR HER CONGESTVE HEART FAILURE, WHICH OF THE FOLLOWING WOULD THE THERAPIST MOST LIKELY MONITOR IN WATCHING FOR COMMON SIDE EFFECTSASSOCIATED WITH DIURETIC THERPAY A.HYPOKALEMIA B. HYPERCHLOREMIA C. HYPERBILRUBEMIA D. HYPONATREMIA
A. HYPOKALEMIA
861
AFTER A THORACENTSESIS PROCEDURE, IT IMPORTANT TO MONITOR THE PT FOR 1. BRONCHOSPASM 2. HEMOPYTSIS 3. INCREASED BUN 4. PNEUMOTHORAX
D. PNEUMOTHORAX
862
IN A NORMAL DIFFERENTIAL WBC COUNT WHICH OF THE FOLLOWING WOULD HAVE THE HIGHEST NUMBER A. NEUTROPHILS B. BASOPHILS C. EOSINPHILS D. LYMPHOCYTES
A. NEUTROPHILS
863
A 14 Y/O PT HAS HAD A SERIOUS ASTHMA ATTACK . HER WHITE BLOOD CELL COUNT CAN BE EXPECTED TO SHOW A. DECREASED NEUTROPHILS B. DECREASED RBC C. INCREASED EOSINOPHILS D, INCREASED MONOCYTES
C. INCREASED EOSINPHILS
864
IN RESPONSE TO A VIRAL INFECTION, WHICH OF THE FOWLLOING CAN BE EXPECTED TO BE SEEN IN THE WHITE BLOOD CELLS A. INCREASED LYMPHOCYTES B. DECREASED MONOCYTES C. INCREASED EOSINOPHILS D. DECREASED PLATELETS
A. INCREASED LYMPHOCYTES
865
WHICH OF THE FOLLOWING TEST WOULD SUPPORT THE DIAGNOSIS OF ACUTE RENAL FAILURE A. ALT VALUES THAT ARE LOWER THAN THE NORMAL B. BUN AND CREATININE VALUES THAT ARE HIGHER C. LDH VALUES THAT ARE HIGHER D. EOSIPILS THAT ARE HIGHER
B. BUN AND CREATNINE
866
WHEN PERFORMING A THERAPIST DRIVEN PROTOCOL , TEH SEVERITY ASSESMMENT DETERMINES A. THE FREQUENCY OF PERFORMING A TX MODALITY B. THE BASELINE DYSPNEA INDEX C. THE NUMBER OF HOSP DAYS COVERED BY INSURANCE CARRIER D. HOW URGENTLY THE PHYSICIAN WANTS THE PT TREATED
A. THE FREQ OF PERFORMING A TX MODALITY
867
POSTURAL DRAINAGE, PERCUSSION AND VIBRATION ARE PART OF WHAT TDP A. LUNG EXPANSION PROTOCOL B. AIRWAY CLEARANCE THERAPY PROTOCOL C. AEROLSIZED MEDICATION THERAPY PROTOCOL D. O2 THERAPY PROOCOL
B. AIRWAY CLEARANCE THERAPY PROTOCOL
868
A FEMALE PT HAD UPPER ABDOMINAL SURGERY 2 DAYS EARLIER. HE HAS A WEAK NONPRODUCTIVE COUGH AND A PULSE OXIMETER (SPO2) READING 84% ON ROOM AIR WHICH THERAPIST DRIVEN PROTOCOLS SHOULD BE IMPLEMENTED 1. AIRWAY CLEARANCE HYGIENE THERAPY 2. LUNG EXPANSION 3. O2 THERAPY 4. AEROSOLIZED MEDICATION THERAPY
2, 3
869
WHICH OF THE FOLLOWING COMMUNICATION TOOLS SIMPLIFIES AND REINFORCES, INTERCATIONS BETWEEN TEAM MEMBERS REGARDING OF THE PT CONDITIONS 1. POMR 2. SBAR 3. SOAPER 4. TDP
2. SBAR
870
YOU HAVE APT RECIEVING 40% 02 VIA A NASAL CANNULA, BP PRESSURE IS 700 MMHG AND WATER VAPOR PRESSURE 45MM HG. THE PT RESP QUOTIENT IS 0.8. ABG IS: PH - 7.38 PACO2 - 40 PAO2 - 90 HCO2 - 22 CALCULATE THE PT PAO2 WITH CORRECT UNIT OF MEASURE
PAO2 - 211.2 MMHG
871
YOUR PT SPO2 IS READING 95% ON ROOM AIR. A CBC SHOWS A HGB IS 15G/DL PAO2 IS 90 CALCULATE THE CAO2 WITH CORRECT UNITS
19.37 ML/DL %
872
YOUR PT HAS A TIDAL VOL OF 600 ML AND A TRANSAIRWAY PRESS OF 3 CMH20 CALCULATE LUNG COMPLIANCE
0.2 L/CMH20
873
WHICH OF THE FOLLOWING WILL LIKELY TO BE SEEN IN TEH ARTERIAL BLOOD GAS VALUES OF A PT WITH ACUTE VENT FAILURE 1. ACIDIIC PH 2. NEAR NORMAL HCO3 3. ALKLOTIC PH 4. HIGH CO2 LEVEL
2,3,4
874
COMMON CAUSES OF METABOLIC ACIDOSIS ARE 1. DKA 2. SHALLOW BREATHING FROM A SEDATIVE OVERDOSE 3. LACTIC ACIDOSIS 4. RENAL FAILURE
1,3, 4
875
WHAT ARE COMMON CAUSES OF METABOLIC ALKAOSIS INCLUDE 1. RENAL FAILURE 2. VOMITING 3. EXCESSIVE BICARBONATE ADMINISTRATION 4. GASTRIC SUCTIONG
2, 3,4
876
THE MOST COMMON CAUSE OF ACUTE ALVEOLAR HYPERVENTILATION IS
HYPOXEMIA
877
PULM FUNCTIONS TESTS ARE DONE FOR WHICH FOF THE FOWLLOING MEASUREMENTS 1. CO LEVELS 2. LUNG VOL AND CAPACITIES 3. PULM DIFFUSION CPACITY 4. FORCED EXP FLOW RATES
2, 3, 4
878
WHICH OF THE FOLLOWING PULM FUNCTION TEST OR STUDY REQUIRES A SPECIAL INDIRECT MEASUREMENT PROCDURE TO OBTAIN A. IRV B. RV C. ERV D. IC
B
879
IN A HEALTHY INDIV, WHICH IS CONDIFDERED A NORMAL EXP TIME NECESARY TO PERFORM A FORCED VITAL CAPCITY
4 TO 6 SEC
880
OVERALL CHARACTERISTICS OF PULM FUNCTION TESTS RESULTS ON A PT WITH OBSTRUCTIVE LUNG DIS INCLUDE THAT THE FEV1 A. A REDUCED AND FEV1 % IS NORMAL B. AND FEV1% ARE BOTH INCREASES C. AND FEV1% ARE BOTH REDUCED D. IS INCREASED AND FEV 1% IS DECREASED
C
881
A PHYSICIAN ASK YOU TO SUGGEST THE BEST MANEUEVER TO EVELAUATE THE FLOW IN A PT MEDIUM SIZE TOSMALL AIRWAYS WHICH OF THE FOLLOWING WOULD YOUSUGGEST A. TLC B. MVV C. FEV1% D. FEF25-75%
D
882
883
WHICH OF THE FOLLOWING ARE TRUE OF THE PEAK EXP FLOW RATE TEST 1. IT IS EFFORT DEPENDENT 2. IT IS TAKEN FORM THE FVC TEST RESULTS 3. IT IS TAKEN FROM THE MVV 4. IT ASSSESS LARGE UPPER AIRWAYS
1, 2, 3
884
VITAL CAPACITY IS COMPROMISED OF WHAT LUNG VOL 1. IRV 2. TIDAL VOLME 3. ERV 4. RV
1, 2, 3
885
TLC IS COMPRISED OF IRV TIDAL VOL ERV RV
IRV ERV RV
886
FRV IS COMPRISED OF WHAT LUNG VOLUMES IRV TIDAL VOL ERV RV
ERV AND RV
887
IC IS COMPROMISED OF WHAT LUNG VOL IRV TIDAL VOL ERV RV
IRV TIDAL VOL
888
WHICH ARYTHMIAS IS THE ATRIAL RATE FASTER THAN THE VENT RATE
ATRIAL FIB ATRIAL FLUTTER
889
THE PULMONATY ARTERY CATH (SWAN GANZ) MEASURES WHAT PRESSURE 1. R. ARTRIAL PRESSURE 2. PULM ARTERIAL PRESSURE 3. DIASTOLIC BP 4.. CENTRAL VENOUS PRESSURE 5. LEFT ARTERIAL PRESSURE
1,2 , 5
890
EXTRINSIC ASTHMA IS ASSOCIATED WITH
A SPECIFIC ANTIGEN
891
A PT WITH CHRONIC INFLAMM DUE TO ASTHMA WHICH OF THE FOWLLOIN IS USED TO DESCRIBE ANATOMIC ALTERATIONS RESULTING IN DMAGED CILIA AND FIBROSIS OF THE BASEMENT LAYER
REMODELING
892
IF A BETA 2 AGONIST AGENT AND AN ANTICHOLINERGIC AGENT WERE ADMINSTERED CONCURRENTLY TO A PT DURING AN ACUTE ASTHMA EPISODE WHICH WOULD BE EXPECTED
BRONCHIAL SMOOTH MUSCLES RELAXTION WOULD OCCUR
893
WHAT IS CONSISTENT WITH OCCUPATIONAL ASTHMA
IGE MEDIATED ALLERGIC REACTION AND CELL MEDIATED
894
WHICH OF THE FOLLOWING WOULD BE EXPECTED WHEN A CHEST ASSESMENT IS PERFORMED ON APT DURING AN ASTHMATIC EPIDOSDE 1. INVERSE I/E RATIO 2. DECREASED VOCAL FREMITUS 3. INCREASED VESICULR BREATH SOUNDS 4. HYPERRESONANT PERCUSSION NOTE
2 4
895
Which of the following are anatomic alterations that occur when a person has a pneumothorax? 1. The lung on the affected side collapses. 2. The visceral and parietal pleura separate. 3. The visceral pleura adheres to the parietal pleura. 4. The chest wall moves outward.
C) Disseminated TB
896
Mycobacterium tuberculosis is particularly damaging to the lungs because of the: A) healing of a tubercle. B) formation of micropulmonary emboli. C) increased mucus production. D) irreversible bronchospasm that it causes.
A) healing of a tubercle.
897
A patient has postprimary TB. What are the major pathologic or structural changes associated with it? 1. Cavity formation 2. Laryngeal edema 3. Dilated and distorted bronchi 4. Fibrosis of lung parenchyma
D) 1, 3, 4
898
A patient who has an uncontrolled tuberculosis infection will show: 1. weight loss. 2. high fever. 3. bloody sputum. 4. night sweats.
D) 1, 3, 4
899
A negative tuberculin test would be demonstrated by an induration (wheal) of what size? A) Less than 2 mm B) Less than 5 mm C) Greater than 8 mm D) Greater than 10 mm
B) Less than 5 mm
900
Pulmonary function testing results on a patient with an advanced case of TB will display which of the following? A) Decreased RV B) Increased IRV C) Increased VC D) Increased TLC
DECREASEDRV
901