Random Topics Flashcards
Exposure to an emotionally salient stimulus causes a physiological reaction which, in turn, is perceived as an emotion (experience of emotion follows physiological arousal)
James-Lange Theory of Emotion
Experience of an emotion and physiological arousal occur together when an environmental stimulus causes the thalamus to simultaneously send signals to the cortex and sympathetic NS (views all emotions as involving similar physiological arousal and differences in emotional reactions cannot be attributed to differences in the nature of physiological arousal
Cannon-Bard Theory of Emotion
Experience of emotion is the result of physiological arousal followed by an attribution (“cognitive appraisal”) for that arousal; proposes that differences in the experience of emotion are due to differences in the attributions for the arousal, which depend on the external cues
Schachter and Singer’s Two-Factor Theory of Emotion
Proposes that differences in emotional reactions to events are due to different appraisals of those events; also assumes that physiological arousal follows cognitive appraisal
Lazarus’s Cognitive Appraisal Theory of Emotion
Proposes that fear consists of two separate but interacting systems that both respond to threatening stimuli (a) subcortical system (survival system) and (b) cortical system (conscious emotional system)
LeDoux’s Two-System Theory of Emotion/Fear
hypersecretion of thyroid hormones, symptoms include increased rate of metabolism, elevated body temperature, heat intolerance, increased appetite with weight loss, accelerated heart rate, insomnia, emotional lability, and reduced attention span
Hyperthyroidism
Hyposecretion of hormones and involves a decreased rate of metabolism, reduced appetite with weight gain, slowed heart rate, lowered body temperature, cold intolerance, depression, lethargy, decreased libido, confusion, and impaired concentration and memory
Hypothyroidism
Low level of antidiuretic hormone due to some factor that causes frequent and excessive urination, extreme thirst, dehydration, constipation, weight loss, and low blood pressure
Central Diabetes Insipidus
chlorpromazine, haloperidol, thioridazine, and fluphenazine
First Generation Antipsychotics
Used to treat schizophrenia and other disorders with psychotic symptoms; dopamine antagonists that exert their therapeutic effects primarily by blocking dopamine (D2) receptors; most effective for treating positive symptoms
First Generation Antipsychotics
Major side effects of First Generation Antipsychotics
Anticholinergic side effects, Extrapyramidal side effects, Neuroleptic malignant syndrome
starts with involuntary, rhythmic movements of the tongue, face, and jaw and, over time, may also affect limbs and trunk; irreversible for some patients and generally treated by gradually withdrawing the antipsychotic, administering a benzodiazepine, or switching to a second-generation antipsychotic
Tardive dyskinesia
clozapine, risperidone, olanzapine, and quetiapine
Second-Generation Antipsychotics
dopamine-serotonin antagonists: alleviate positive symptoms primarily by blocking dopamine receptors and negative/cognitive symptoms primarily by blocking serotonin receptors
Second-Generation Antipsychotics
aripiprazole, brexpiprazole, and cariprazine
Third-Generation Antipsychotics
Referred to as dopamine-serotonin stabilizers and are categorized as partial agonists even though they have both antagonist and agonist effects
Third-Generation Antipsychotics
fluoxetine, fluvoxamine, paroxetine, sertraline, citalopram, and escitalopram
SSRI’s (Antidepressants)
Combining an SSRI with an MAOI or lithium can cause _____ which involves extreme agitation, confusion, autonomic instability, hyperthermia, tremor, seizures, and delirium
Serotonin syndrome
venlafaxine, duloxetine, desvenlafaxine, and levomilnacipran
SNRIs (Antidepressants)
used to treat MDD and assist with smoking cessation and exerts its therapeutic effects by inhibiting the reuptake of norepinephrine and dopamine at synapses
NDRIs (ex. bupropion)
Include tertiary amines (amitriptyline, imipramine, clomipramine, and doxepin) and secondary amines (nortriptyline and desipramine)
TCAs (Tricyclic Antidepressants)
phenelzine, isocarboxazid, and tranylcypromine
MAOIs (Monoamine Oxidase Inhibitors)
diazepam, alprazolam, and lorazepam
Benzodiazepines
These drugs increase GABA activity by acting as agonists at GABA receptors and are used to treat short-term anxiety, insomnia, seizures, and alcohol withdrawal
Benzodiazepines