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1
Q

SBAR

A

Situation
Background
Assessment
Recommendation

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2
Q

Situation

A

Identify yourself, client, location, diagnosis, and specific current situation

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3
Q

Background

A

Explain significant medical history and overview of current treatment

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4
Q

Assessment

A

Provide current vital signs and critical current assessment data, your clinical impression, and any concerns

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5
Q

Recommendation

A

Make suggestions; clarify expectations; make recommendations as appropriate to ensure client safety and satisfaction, care continuity, and best outcomes

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6
Q

Process for Medication Reconciliation

A
  1. Obtain a list of current medications from the client.
  2. Develop an accurate list of newly prescribed medications.
  3. Compare new medications to the list of current medications.
  4. Identify and investigate any discrepancies and collaborate with the PHCP as necessary.
  5. Communicate the finalized list with the client, caregiver(s), PHCP, and other team members.
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7
Q

5 Rights of Delegation

A
Right Task
Right Circumstances
Right Person
Right Direction/Communication
Right Supervision/Evaluation
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8
Q

Nurse Practice Act

A

Defines which aspects of care can be delegated.

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9
Q

UAP Assigned Tasks (Generally)

A
Noninvasive Interventions:
Skin Care
ROM Exercises
Ambulation
Grooming
Hygiene Measures
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10
Q

LPN Tasks (Generally)

A

Invasive Tasks:
Dressing Changes
Suctioning
Urinary Catheterization
Medication Admin (PO, Sub Q, IM, ID, PR, Selected Piggyback Meds)
Plus UAP-assigned tasks and review of teaching plans initiated by RN

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11
Q

Third Spacing

A
  • The accumulation or sequestration of trapped extracellular fluid in an actual or potential body space as a result of disease or injury.
  • Trapped fluid represents a volume loss and is unavailable for normal physiological processes.
  • Potential spaces: Pericardial, Pleural, Peritoneal, or Joint Cavities; the bowel; the abdomen; or within soft tissues after trauma or burns
  • Gathering data may be difficult; intravascular fluid loss may not be reflected in I/O or wt changes. May not become apparent until after organ malfunction occurs.
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12
Q

Edema

A

Cause:
Localized- traumatic injury (accidents or surgery), local inflammatory processes, or burns

Generalized (aka Anasarca)- cardiac, renal, or liver failure

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13
Q

Fluid Imbalance

A

Infants and Older Adults are at higher risk for fluid-related problems.

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14
Q

Insensible Fluid Loss

A

Water loss through Persperation or Respiration.

Unnoticeable and unmeasurable.

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15
Q

Fluid Volume Deficit

A

Causes: Vomiting/Diarrhea; Cont GI Irrigation; GI Suctioning; Ileostomy or Colostomy Drainage; Draining Wounds/Burns/Fistulas; Inc Urine Output from the use of diuretics

Data Collection: Thirst; Poor skin turgor; Dry mucus membranes; Inc HR, thready pulse, dyspnea, postural hypotension; Wt loss; Flat neck or hand veins; Dizziness or weakness; DEC IN URINE VOL AND DARK/CONCENTRATED URINE; Inc specific grav of urine; confusion; Inc hematocrit level

Tx: Treat the cause; Monitor VS/Resp/Neuro Status closely; Admin prescribed O2; Check mucus mem/Skin turgor; daily wt; I/O; Urine Spec Grav; Hem/electrolyte level monitoring and correction if needed.

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16
Q

Lithium

A

Hyponatremia precipitates lithium toxicity. (Hyponatremia can cause diminished lithium excretion.)

17
Q

Labeling IV Bag

A

Do not write directly on the bag: use ballpoint pen on a label

18
Q

IV Extension Tubing

A
  • Children
  • Restless Patients
  • Patients with Special Mobility Needs
19
Q

Phlebitis and Thrombophlebitis

A

Phlebitis: inflammation of the vein from mechanical or chemical trauma or local infection. Vein not hard, swelling proximal to site.

Thrombophlebitis: development of a clot as a result of phlebitis. Vein is hard/cord-like

Both: Heat; redness; site tenderness; IV infusion sluggish

20
Q

Disseminated Intravascular Coagulation (DIC)

A
  • The rapid and extensive formation of clots that occurs in DIC causes the platelets and clotting factors to be depleted.
  • Results in bleeding and potential vascular occlusion of organs from thromboemoblus formation