Quiz 4 Flashcards
strabismus
cross eyed lazy eye, not necessarily visually impaired, just struggle with alignment
ptosis
droopy eye
cataracts
cloudy lens, usually just one eye
amblyopia
‘lazy eye’- developmental problem, nerve pathways in one eye are under stimulated and the cells begin to favor the other eye
monocular deprivation studies
reveal that deprived eye would be largely incapable of stimulating cells in the visual cortex, but fine at stimulating cells in the optic nerve and retina.
binocular deprivation studies showed near normal cortical and retinal stimulation.
binocular competition hypothesis
eyes compete for cortical dominance in development- they should balance each other out creating predominantly binocular cortical cells. However, if one eye is inhibited in sending signals, the other will take over a cell
periphery study for binocular competition
deprived eye can drive cells accounting for the visual field in its periphery, but not in cells whose visual fields overlap with the nondeprived eye
retinal lesion study for binocular competition
lesion in non deprived eye’s retina- then, the deprived eye cannot drive cells overlapping with the non deprived eye except in the area that accounts for the lesion because there is no competition there
oblique effect
our brains are better at seeing vertical and horizontal lines than oblique ones
hemianopia
part of visual field is blinded due to stroke or brain injury- essentially an agnosia in v1 area, but called hemianopia
ERG
electroretinogram, measures evoked potential of the eye
middle temporal
visual area that codes for movement- sensitive to speed and direction
Brodmann
identified many brain areas in 1800s based on physical differences and density differences
aphasia
language disorder, usually caused by stroke, many different types cause different impairments
2 major types of stroke
ischemic- clogged blood vessel, hemorrhagic- burst blood vessel
agnosia
damage to higher visual areas, dorsal damage- where is the stimulus, ventral- what is the stimulus. prosopagnosia- can’t recognize faces. hemifield neglect- clock thing
apraxia
motor disability from brain damage (or developmental in rare instances)- construction apraxia, can’t do things in the right order
Charles Gross
first to discover facial area- began to study brain reactivity from complex stimuli. He found that certain cells with much larger receptive fields that overlapped in foveal area and were not retinotopically organized were driven by specific complex stimuli like faces
anosognosia
condition of confabulation and unawareness
acute vs chronic schizophrenia
acute has much better outcome and comes in an episode out of nowhere
chronic has much poorer predicted outcomes, strange behavior for years prior to onset
Giacomo Rizzolatti
discovered ‘mirror cell/neurons’- neurons fired when monkey did an action and also when monkey saw someone else doing the same action
Denis Levi
studied EE rats vs regular rate- EE rats had larger cells, thicker cortices, and more complex dendritic trees. EE rats had smaller of these factors compares to wild rats
schizophrenia
leading hypothesis holds that there is an overabundance in schizophrenics.
antipsychotics block d2 dopamine receptors, they work better in acute schizophrenia
tardive dyskinesia
potential long-term side effect of antipsychotic drugs
strange uncontrollable motor movements most commonly in tongue and mouth
could be explained by denervation supersensitivity