Quiz 4 Flashcards

1
Q

strabismus

A

cross eyed lazy eye, not necessarily visually impaired, just struggle with alignment

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2
Q

ptosis

A

droopy eye

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3
Q

cataracts

A

cloudy lens, usually just one eye

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4
Q

amblyopia

A

‘lazy eye’- developmental problem, nerve pathways in one eye are under stimulated and the cells begin to favor the other eye

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5
Q

monocular deprivation studies

A

reveal that deprived eye would be largely incapable of stimulating cells in the visual cortex, but fine at stimulating cells in the optic nerve and retina.
binocular deprivation studies showed near normal cortical and retinal stimulation.

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6
Q

binocular competition hypothesis

A

eyes compete for cortical dominance in development- they should balance each other out creating predominantly binocular cortical cells. However, if one eye is inhibited in sending signals, the other will take over a cell

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7
Q

periphery study for binocular competition

A

deprived eye can drive cells accounting for the visual field in its periphery, but not in cells whose visual fields overlap with the nondeprived eye

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8
Q

retinal lesion study for binocular competition

A

lesion in non deprived eye’s retina- then, the deprived eye cannot drive cells overlapping with the non deprived eye except in the area that accounts for the lesion because there is no competition there

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9
Q

oblique effect

A

our brains are better at seeing vertical and horizontal lines than oblique ones

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10
Q

hemianopia

A

part of visual field is blinded due to stroke or brain injury- essentially an agnosia in v1 area, but called hemianopia

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11
Q

ERG

A

electroretinogram, measures evoked potential of the eye

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12
Q

middle temporal

A

visual area that codes for movement- sensitive to speed and direction

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13
Q

Brodmann

A

identified many brain areas in 1800s based on physical differences and density differences

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14
Q

aphasia

A

language disorder, usually caused by stroke, many different types cause different impairments

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15
Q

2 major types of stroke

A

ischemic- clogged blood vessel, hemorrhagic- burst blood vessel

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16
Q

agnosia

A

damage to higher visual areas, dorsal damage- where is the stimulus, ventral- what is the stimulus. prosopagnosia- can’t recognize faces. hemifield neglect- clock thing

17
Q

apraxia

A

motor disability from brain damage (or developmental in rare instances)- construction apraxia, can’t do things in the right order

18
Q

Charles Gross

A

first to discover facial area- began to study brain reactivity from complex stimuli. He found that certain cells with much larger receptive fields that overlapped in foveal area and were not retinotopically organized were driven by specific complex stimuli like faces

19
Q

anosognosia

A

condition of confabulation and unawareness

20
Q

acute vs chronic schizophrenia

A

acute has much better outcome and comes in an episode out of nowhere
chronic has much poorer predicted outcomes, strange behavior for years prior to onset

21
Q

Giacomo Rizzolatti

A

discovered ‘mirror cell/neurons’- neurons fired when monkey did an action and also when monkey saw someone else doing the same action

22
Q

Denis Levi

A

studied EE rats vs regular rate- EE rats had larger cells, thicker cortices, and more complex dendritic trees. EE rats had smaller of these factors compares to wild rats

23
Q

schizophrenia

A

leading hypothesis holds that there is an overabundance in schizophrenics.
antipsychotics block d2 dopamine receptors, they work better in acute schizophrenia

24
Q

tardive dyskinesia

A

potential long-term side effect of antipsychotic drugs
strange uncontrollable motor movements most commonly in tongue and mouth
could be explained by denervation supersensitivity

25
denervation supersensitivity
denerve muscles in a frog and administer acetylcholine. Then, measure the degree of contraction. After a week of no stimulation to the muscle, it will contract much more with the same amount of acetylcholine because the muscle cell created more cholinergic receptors in the absence of acetylcholine
26
botox
inhibits release of acetylcholine
27
phantom limbs
brain areas that surround the area of the phantom limb will take over the region when it stops receiving input from the missing limb. face region is next to arm, so when face region takes over, it feels like you are touching their missing arm when you are touching their face
28
memory
input-> storage-> retrieval mechanism storage from short term to long term is called consolidation and is driven by many factors including motivation (the 2 modes of memory storage) procedural memory has to do with motor tasks, whereas declarative memory has to do with information like names
29
HM
Henry Molson- lost parts temporal lobe and hippocampus. Seizures got better but he lost the ability to consolidate from short term to long term memory because of hippocampal damage. Only affected declarative memory, couldnt remember who people were shortly after they introduced themselves.
30
aerobic exercise in AD
moderate gain in functional ability, not in memory executive function and depressive symptoms greater peak VO2 associated with changes in memory and greater hippocampal volume
31
music therapy in ASD
better social communication, better connectivity between auditory and motor areas, reduced overconnectivity
32
lucid dreaming
higher gray matter volume in BA 9 and 10, hippocampus, right anterior cingulate, left supplementary motor area
33
facial recognition in humans and macaques
fusiform face area important in humans, other important regions like AFP 1 and 2 and anterior inferotemporal face area reproducible face systems in both humans and macaques possible homology in face areas between humans and macaques