Quiz 2 Flashcards

1
Q

Apraxia is a difficulty with

A

motor planning and programming

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2
Q

How is CAS caused?

A

1) Known neuro impairment
2) Complex behavioral disorder
3) Idiopathically neurogenic

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3
Q

3 Key features of CAS speech

A

1) Inconsistent sound errors
2) Lengthened and disrupted co-articulatory transitions between sounds and syllables
3) Inappropriate prosody (stress)

*all with CAS have these features, but it is not enough for diagnosis

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4
Q

Suprasegmental definition

A

1) Rate
2) Pitch
3) Loudness

Also called prosodic feature, in phonetics, a speech feature such as stress, tone, or word juncture that accompanies or is added over consonants and vowels

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5
Q

Motor speech development trajectory (Hodge, 2000)

A

Development in order

1) Respiration and phonation
2) Velopharyngeal and laryngeal control (can send air through either mouth or nose) —> oral nasal contrast
3) Mandible (up and down first in the vertical plane)
4) Lips (horizontal plane with lip rounding)
5) Tongue (position, front-back, high or low)
5+) Lax vowels, liquids, refinement of grading movements

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6
Q

CAS Differential diagnosis Indicators (4 Things)

A

1) Difficulty planning/programming volitional movements for speech
2) Absence of neuromuscular deficits
3) Errors in prosody
4) DDK rates (AMRs and SMRs) are inconsistent

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7
Q

Dysarthria differential diagnosis indicators (4 Things)

A

1) Paralysis, weakness, abnormal tone, incoordination of speech muscles
2) Phonation, respiration, resonance, and articulation difficulties
3) Speech movements impaired in force, timing, endurance, direction
4) Slurred speech, weakness, low volume ect.

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8
Q

Phonological disorder

A

Phono disorders have patterns of rule-bound errors: fronting all velars, stopping of fricatives, reducing consonant clusters, etc.

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9
Q

Articulation disorder

A

You will often see that the client has 1 (or a few) individual sound errors, but doesn’t necessarily have trouble with entire classes of sounds (eg all fricatives, all clusters, etc). Articulation disorders are a result of not understanding what to do with your mouth more than not understanding phonological rule

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10
Q

Similarities between CAS and Phonological Disorders (6 Things)

A

1) Inventory constraints
2) Omissions in segments and structures
3) Segmental errors
4) Altered prosody
5) Increased errors with increased utterance length or complexity
6) Use of simple but not complex syllable and word shapes

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11
Q

Features of AOS speech (6 features)

A

1) Articulatory groping
2) Inconsistent errors (vowels)
3) Blurring
4) Slower rate
5) Stress and intonation errors
6) Word retrieval (agrammatism)

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12
Q

Definition of AOS

A

Neurologic MSD that reflects and impaired capacity to plan or program commands necessary for the positioning and sequencing of speech movements for volitional speech

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13
Q

AOS damage typically in the _______ or ________ lobe.

A

Frontal or parietal lobe (in the perisylvian area near Broca’s)

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14
Q

AOS has an impaired capacity for

A

Planning and programming volitional speech

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15
Q

The motor plan is

A

What to do

  • Higher level
  • Goal oriented
  • Specific movements of articulators
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16
Q

The motor program is

A

How to do it

  • Lower level
  • Procedure specific
  • Specific movement of each muscle
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17
Q

Most common areas for lesion in AOS

A

1) Posterior frontal lobe
2) Parietal lobe
3) Insula
4) Basal ganglia

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18
Q

OME results for AOS

A

1) Nonverbal oral apraxia (NVOA)
2) Non-speech DDKS (open and closing mouth)
3) Trouble following instructions

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19
Q

Are people with AOS aware of their mistakes (possess awareness of sound errors)

A

Yes, and they self-correct

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20
Q

Ideomotor apraxia (instructional)

A

Not sure of making volitional movements even when told to do so

21
Q

Ideational apraxia (kettle)

A

Movement errors and hesitations. Also errors are made and then self corrected.

22
Q

What might we use as an assessment task to determine CAS or AOS?

A

Repetition task

23
Q

Which is more impacted in AOS; volitional or automatic speech?

A

Volitional

24
Q

OME for AOS: will a person improve with a model or unison cues?

A

Yes

25
Q

What kind of aphasia often co-occurs with AOS

A

Broca’s aphasia

26
Q

OME results for AOS (4 things)

A

1) No weakness of face or oral musculature
2) Normal gag
3) NVOA = unable to smile, or pucker when requested
4) Breakdown of non-speech DDKs

27
Q

Do people with AOS have receptive language troubles

A

Yes then can; receptive language deficits, and trouble following directions is common.

28
Q

Apraxia Battery for Adults (ABA-2)

A

Standardized assessment for AOS

29
Q

NVOA stands for

A

Non verbal oral apraxia

volitional pucker and kiss

30
Q

CAS evaluation (criterion referenced)

A

Dynamic evaluation of motor speech skill (DEMSS)

31
Q

Other CAS assessment

A

Kaufman speech praxis text

32
Q

Non-speech clinical characteristics of apraxia (5 main ones)

A

1) NVOA
2) Hemiplegia/hemiparesis
3) Limb apraxia
4) Decreased sensation
5) Spasticity

33
Q

Syllabicity effects of apraxia

A

Fewer syllables are easier

34
Q

Production of stressed/unstressed syllables in apraxia

A

Stressed are easier to produce

35
Q

Is CAS due to neuromuscular deficits?

A

No

36
Q

Parent friendly CAS

A

There is a disruption between the message plan sent from the brain to the speech muscles

37
Q

Cas may occur with other neurologic disorders and/or with:

A

1) Expressive language impairments
2) Phonological disorders
3) Phonological awareness and literacy challenges
4) Social communication impairments
5) Fluency disorders

38
Q

Motor suprasegmental development trajectory (Hodge, 2000)

A

1) Contrast syllable, number, and stress
2) Introduce syllable shape
3) Diphthongs
4) Sequencing C & V across more than one plane
5) Elaboration / refinement of rate and rhythm

39
Q

Essential ax for CAS

A

Word shapes

v
cv
vc
cvcv

ect.

40
Q

Dynamic Evaluation of Motor Speech Skill (DEMSS)

A
  • One part of a comprehensive assessment
  • Criterion referenced
  • Vowels, prosody, overall articulatory accuracy and consistency
41
Q

Madison Speech Assessment Protocol

A
  • Very intensive
  • 2 hour battery
  • CAS differential diagnosis
  • Utilizes speech disorder classification system
42
Q

Motor programs for CAS for treatment

A
PROMPT
Kaufman
Nuffield
Speech Motor Chaining
DTTC 
Drill (make is fun)
43
Q

Primary behavioral AOS treatment

A

Articulatory kinematic techniques

44
Q

Stages of motor learning

A

1) Cognitive: understanding the nature of the problem (make sure the client understands what they’re doing)
2) Associative: starting to practice and becoming automatic
3) Automatic: skills performed with little conscious effort

45
Q

Specificity principle

A

Make the tasks as similar to speech as possible

46
Q

Motor learning promotes plasticity which is facilitated by _____________. (three things)

A

1) Stimulus selection
2) Structure of practice
3) Nature of feedback

47
Q

Task hierarchy for motor learning

A

Begin with smallest task possible and move on at 80%

48
Q

Perfection approximation

A

Perfect practice makes perfect

49
Q

Distributed practice

A

More sessions with less time per session leads to automaticity faster