questions cours Flashcards

1
Q

À quelle niveau retrouve-t-on des nodules de Schmorl?

A

L1-L2 et L2-L3

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2
Q

Quelles sont les 3 structures les plus atteintes dans les lombalgies non spécifiques?

A

Le disque (25-30 ans le +), les facettes articulaires (60 ans le +) et l’articulation sacro-iliaque (même que facettes)

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3
Q

Quelles sont les niveaus de dégénerescence facettaire les plus atteints?

A

L4-L5 et L5-S1 (encore plus)

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4
Q

Quelles signes et symptômes clinique nous font penser à une atteinte facettaire?

A

pas de valsalva, douleur + debout ou marche que assis ou coucher, pas de centralisation, ext fait + mal, flex ne fait pas + mal, limitations des mvts va avec les mvts facettaires
facette mal en fin de flex, disque mal en mi-flex

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5
Q

Causes des lombalgie avec atteinte neuro

A

ostéophytes, disque, spondylolisthésis

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6
Q

Causes de lombalgie sans atteinte neuro

A

disque, facette, sacro-iliaque, dure-mère sans neuro, ligaments, muscles

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7
Q

Central Stenosis

A

Narrowing of the spinal cord space as a result of a space-occupying lesion may lead to spinal cord compression.
The lesion is commonly caused by osteophytes, disc herniation, ligament thickening or calcification, or tumor

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8
Q

Degenerative lumbar joint disease (DJD)

A

Breakdown of the lumbar spine facet articular surfaces with a resultant loss of cartilage.
Can occur with DDD in which there is a loss of disc height. the breakdown of articular cartilage leads to a resultant inflammatory cascade that can redult in bone spurs or osteophyte formation

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9
Q

Interforaminal stenosis

A

Loss of interforaminal area.
this narrowing can result in impingement of the nerve root.
the most common sources of lesion that decrease the space are loss of intervertebral disc height, inflammation, osteophytes, disc herniation, ligament thickening, calcification, or tumor.

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10
Q

Lumbar disc pathology

A

disc herniation or disc bulges are synonymous terms used to describe a condition in which disc material extends beyond the perimeter of the normal disc space
four common classes: protrusion (np bulges outward into the annulus but no damage is done), prolapse (np bulges outward into the annulus and annular lamina are damaged), extrusion (np breaks past the outer annulus and into the space beyond), sequestration (np breaks free of annulus)

slr très sensible
contra lat slr est spécifique

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11
Q

Lumbar instability

A

excess motion in the lumbar spine segments in which a normal external force or load causes hypermobility and pain.
Excessive motion is defined as an excessive rotational segmental angle or segmental translation greater than 3mm.

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12
Q

Lumbar ligament strain

A

tearing of the fibers of a lumbar ligament.
if complete, it could lead to a lumbar segmental instability.
a partial tar can be as small as a microtear in the substance of the ligament.

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13
Q

Lumbar muscle strain

A

microtear of a lumbar muscle or tendon, most commonly at either the z line or the musculotendinous junction.
the most common strain is the erector spinae muscle.

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14
Q

Lumbar radiculopathy

A

not a cause itself of LBP but a result of another lumbar pathology on the lumbar nerve root(s).
if a lumbar nerve root is impinged or inflammed and causes neuro symptoms, the result is a lumbar radiculopathy.
causes pain, numbness, tingling, and weakness in the lower extremities in the dermatomes/myotomes.

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15
Q

Lumbar spondylolysis/spondilolisthesis

A

Defect in the pars interarticularis.
If the fracture defect widens and allows the superior segment to slip on the inferior segment, the result is a spondylolisthesis.
means spine slippage, therefore spondylolisthesis refers to a slippage of one spine segment in relationship to its adjacent segment

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16
Q

Ankylosing spondylitis

A

Chronic inflammatory disease of unknown ethology and is classified as a spondyloarthropathy since it affects the joints of the spine.
it typically affects fibrous tissue such as the entheses (insertions of ligaments, tendons and capsules into bone)

17
Q

Iliolumbar ligament strain

A

from the L5 proc. trans to ilium (2 parts).
its function is to restrain motion at the L5-S1 segment, especially flexion and lateral bending.
severing this ligament leads to increased sagittal plane motion if the sacroiliac joint, therefore this ligament may have a role in stability of the sacroiliac joint as well.

18
Q

Sacroiliac joint pathology (SJP)

A

pelvic pain is a rather complex term with many joints and structures that can refer pain to its region. Sacroiliac joint pathology is typically a subclassification of this broader term.
SJP refers to dysfonction of the innominate articulations that exclusively creates pelvic pain. Injectiosna are the gold standard to verify if the articulation is the source of the pain.

19
Q

caractéristiques de l’irritabilité

A

intensité de l’activité qui donne la douleur, temps de la douleur après la fin de l’activité et intensité de la douleur

léger, modéré ou sévère

20
Q

treatment based classification (4)

A

stabilisation, traction, exercices et mobilisations, manipulation

21
Q

treatment based classification - exercices et mobilisations

A

centralisation
atteinte du disque
mvts répétés aident

22
Q

treatment based classification - manipulation (et exercices de mobilité)

A
moins de 16 jours depuis l'apparition
pas de symptome en bas du genoux
moins de 19 points au FABQ-W
PA's centraux hypomobiles
rot interne d'une des deux hanches au moins en dec ventral est + de 35°

douleur fin de ROM dans tous les sens
hypomobilité
pas de direction pref
aigu

23
Q

treatment based classification - stabilisation

A

moins de 40 ans
SLR + de 90°
prone stability test +
mvt aberrant en flex lombaire

douleur en fin de ROM souvent
souvent hypermobilité

24
Q

treatment based classification - traction

A

atteinte neuro
centralisation en flex/périphérisation en ext
SLR croisé +
douleur dans la jambe

25
Q

classification de McKenzie

A

syndrome de dérangement (centralisation)
syndrome de dysfonction (pas de centralisation, douluer en fin de ROM qui ne varie pas avec la répétition)
syndrome postural (douleur durant une posture prolongée)
prob sacro-iliaque
prob de hanche

26
Q

queue de cheval

A

en bas de L2

incontinence
anesthésie en selle
diminution tonus rectal et périnéal
faiblesse au MIs
diminution sensibilité dermatomes
hyporéflexie/aréflexie
27
Q

compression de la moelle

A
paresthésie bi ou quadra
démarche spathique
faiblesse
diminution de coordination
hyper réflexie
spasticité
babinski + 
chaddock +
oppenheimer +
hoffman +
lhermitte +
dysfonction urinaire
douleur extra segmentaire
inversion des réflexes
28
Q

3 caractéristiques cliniques les + importantes pour une fracture

A

truama majeur
âge
corticostéroïdes

aussi contusion, sexe féminin, douleur ++

29
Q

2 causes les + fréquentes de radiculopathie lombaire

A

hernie discale

ostéophyte (sténose)

30
Q

mécanisme de lésion des nerfs

A
lésion 
inflammation
fibrose
baisse mobilité
augmentation tension
31
Q

ordre dégénérescence disque

A

plaque puis noyau puis anneaux

32
Q

phases du processus de dégénérescence

A

dysfonction –> instabilité –> stabilisation

dysfonction: post trauma, baisse ROM, hypertonie musculaire
instabilité: hypermobile, laxité capsulaire et discale
stabilisation: ostéophytes, fibrose

33
Q

étapes dégénérescence disque

A

fissure circonférentielles –> fissures radiales –> internal disruption –> résorption du disque (affaissement) –> ostéophytes

34
Q

étapes dégénérescence facettes

A

synpvites et hypomobile –> dégénérescence progresse et cartilage s’amincit –> laxité capsulaire –> subluxation –> ostéophytes