Pulmonology Flashcards
Acute respiratory distress syndrome
Non-cardiogenic pulmonary edema
Cor pulmonale
Right-heart failure
Hemoptysis
Coughing up blood
Orthopena
Difficulty breathing while lying down
Paroxysmal nocturnal dyspnea
Difficulty breathing at night
Positive end-expiratory pressure (PEEP)
Extrinsic PEEP uses an impedance valve to increase volume of air remaining in lungs at end of expiration to improve gas exchange
Subcutaneous emphysema
Crackling under the skin upon palpitation due to trapped air. Typically found in chest, neck, or face
Tidal volume
Volume of air inhaled or exhaled with each breath; normal adult tidal volume is about 500mL
Ventilation
Mechanical process that moves air in and out of lungs
Inspiration
Active process of ventilation (requires energy)
Exhalation
Passive process of ventilation
External respiration
Movement of oxygen from the alveoli into the bloodstream and movement of CO2 from the blood stream to the alveoli
Internal respiration
The exchange of gasses (O2 & CO2) between the bloodstream and the tissues in the body
Minute volume
Respiratory rate x tidal volume
CO2 drive
The primary system for monitoring breathing status
Monitors CO2 levels in blood and cerebral spinal fluid
Chemoreceptors in the brain detect increased CO2 and rapidly trigger increased respiratory rate
Hypoxic drive
Backup to CO2 drive
Monitors oxygen levels in plasma
Prolonged exposure to high concentration oxygen in hypoxia drive patients can cause respiratory depression
May be present in end-stage COPD patients
Acid-base disorders
Respiratory acidosis
Respiratory alkalosis
Metabolic acidosis
Metabolic alkalosis
Respiratory acidosis
Low pH and elevated CO2
PaCO2 greater than 45mmHg
Respiratory alkalosis
Elevated pH and low CO2
PaCO2 less than 35mmHg
Metabolic acidosis
Low pH and low HCO3
HCO3 below 22mmHg
Metabolic alkalosis
Elevated pH and elevated HCO3
HCO3 greater than 26mmHg
Normal arterial blood gas values
- pH: 7.35-7.45
- PaO2: 80-100 mmHg
- PaCO2: 35-45 mmHg
- HCO3: 22-26 mEq/L
- SaO2: 95% or above
Without adequate respiration x
- heart and brain become irritable almost immediately
- brain damage within 4 min
- permanent brain damage within 6 minutes
- irrecoverable brain damage within 10 minutes
Ventilation-perfusion mismatch
V/Q mismatch or V/Q defect
*Occurs when lungs receive oxygen,but not adequate blood flow
-or-
when the lungs receive blood flow, but inadequate oxygen
*could be a ventilator problem or perfusion problem
Mallampati score (used for oral intubation)
Class I: entire tonsil clear
Class II: upper half of tonsil visible
Class III: soft and hard palate visible
Class IV: only hard palate visible
LEMONS (for difficult airway)
L- look externally E- evaluate 3-3-2 rule M- mallampati score O- obstruction N- neck mobility S- saturation’s
Oxygen cylinder sizes and contents
D cylinder: about 350-L ; 0.16
E cylinder: about 625-L ; 0.28
M cylinder: about 3,000-L ; 1.56
Atonal respiration
Slow, shallow, infrequent breaths
Indicates brain anoxia
Biot’s respiration
Irregular pattern of rate and depth and periodic apnea
Indicates increased ICP
Central neurological hyperventilation
Deep, rapid respiration’s
Indicates increased ICP
Cheyenne-Stokes respiration’s
Progressively deeper and faster breaths, changing to slower and shallow breaths
Indicates brain injury
Kussmaul respirations
Deep, gasping breaths
Indicates possible DKA
Rales (crackles)
Fine, bubbling sound on inspiration
Indicates fluid in lower airways
Rhonchi
Coarse sounds on inspiration
Indicates inflammation or mucus in lower airways
Wheezes
High-pitched sound on inspiration or expiration
Indicates bronchoconstriction
Snoring
Indicates partial airway obstruction from the tongue
Stridor
High-pitched sound
Indicating significant upper airway obstruction (ex:foreign body, angioedema, anaphylaxis)
Gurgling
Indicates fluid in the upper airway
Pleural friction rub
Sounds like dried pieces of leather rubbing together
SpO2
Pulse oximetry
ETCO2
Capnography
Continuous Positive Airway Pressure (CPAP)
- Indications: alert and spontaneously breathing patients, at least 12 years, in significant respiratory distress. (Sleep apnea, COPD, pulmonary edema, CHF, pneumonia); tachypnea, SpO2 below 94%, use of accessory muscles
- Contraindications: apnea, unable to follow verbal commands, suspected pneumothorax, chest trauma, tracheostomy, vomiting, GI bleeding and hypotension
Infarct
Area of necrosis or death
Pulse CO-oximetry (SpCO)
Non-invasive measurement of carbon monoxide saturation of hemoglobin
Pulse oximetry (SpO2)
Non-invasive measurement of oxygen saturation of hemoglobin
Capnography
Measure or monitoring of exhaled CO2
Sudden drop of ETCO2 to zero
- esophageal intubation
- ventilation defect or disconnect
- defect in CO2 analyzer
Sudden decrease of ETCO2 (not to zero)
- leak in ventilator; obstruction
- partial disconnect in ventilator circuits
- partial airway obstruction (secretions)
Exponential decrease of ETCO2
- pulmonary embolism
- cardiac arrest
- sudden hypotension
- severe hyperventilation
Change in CO2 baseline
- calibration error
- water droplet in analyzer
- mechanical failure (ventilator)
Sudden increase in ETCO2
- accessing an area of lung previously obstructed
- release of tourniquet
- sudden increase in BP
Gradual lowering of ETCO2
- hypovolemia
- decreasing cardiac output
- decreasing body temp; hypothermia; drop in metabolism
FBAO
BLS: Conscious - Abdominal thrusts, alt back blows & chest thrust
Unconscious - CPR
ALS: *attempt to remove foreign body with laryngoscope and McGill forecps
*Attempt ETT insertion to try passing tube through obstruction or forcing it into right mainstem
ARDS - Acute Respiratory Distress Syndrome
- a form of pulmonary edema NOT caused by poor left ventricle function
- causes: sepsis, trauma, OD, drowning, toxic inhalation
- s/s: decline in respiratory status; tachypnea, tachycardia, decrease in SpO2
- mgmt: monitor SpO2, sit pt upright with legs dangling, CPAP, PEEP
COPD
- cause: smoking and environmental toxins
* incl emphysema & chronic bronchitis, increased mortality
-s/s: hx of smoking, cough with increased mucus, right heart failure, JVD & pedal edema, decrease SpO2, clubbing, Ronchhi lung sounds
Asthma
- cause: chronic inflammatory airway disease
- s/s: dyspnea, wheezing, cough, tachypnea, tachycardia, decrease SpO2, pulses paradoxus (decrease in systolic BP of at least 10mmHg during inspiration
- mgmt: monitor expiratory flow rates (PEFR), aggressive use of bronchodilators to reverse bronchospasm
- status asthmaticus: not reversible with bronchodilator medications, may have absent lung sounds, respiratory arrest is eminent
Pneumonia
*lung infection
- s/s: patients with a history of CP with associated fever, chills, cough
- weakness, dyspnea, pleuritic CP, abnormal lung sounds
-mgmt: dehydration is common, consider use in IV fluids
Pulmonary embolism
-patho: blockage in pulmonary artery that decreases blood flow
- s/s: possible indications of DVT (warm, swollen,lower extremity with pain upon palpitation)
- tachypnea, tachycardia, acute unexplained dyspnea, cough, pleuritic CP
-mgmt: O2 therapy, prepare for sudden cardiac arrest & rapid transport
Spontaneous pneumothorax
- patho: not related to blunt or penetrating trauma, reoccurrence rate high (50%); common in male smokers
- s/s:acute onset of sharp pleuritic CP or shoulder pain, localized diminished lung sounds, tachypnea, possible subcutaneous emphysema
- mgmt: monitor SpO2, O2, transport in a position of comfort
Hyperventilation syndrome
- hyperventilation considered significant until confirmed otherwise
- s/s: tachypnea, CP, anxiety, possible carpal pedal spasms due to alkalosis & hypocalcemia
- causes: anxiety, metabolic & respiratory disorders, pulmonary embolism, cardiac & CNS disorders, medication (aspirin)
- mgmt: monitor SpO2, O2, transport
Gradual increase in ETCO2
- rising body temp
- hypoventilation
- CO2 absorption
- partial airway obstruction (fbao); reactive airway disease
Processes of gas exchange
- ventilation
- diffusion
- perfusion
Residual volume
Air that remains in the lungs at all times maintaining the patency of the alveoli
Inspiratory reserve volume
Additional volume of air beyond the volume inspired during quiet respiration
Expiratory reserve volume
Amount of air that can be forcibly expired out of the lung after a normal breath
Diffusion
Process by which gases move between the alveoli and the pulmonary capillaries
Most important determinant of the ventilatory rate
Arterial PCO2
*Increase in arterial PCO2 results in a decrease in the pH of the blood
Perfusion
Circulation of blood through the lungs (pulmonary capillaries)
Lung perfusion is dependent on three conditions:
- adequate blood volume
- Intact pulmonary capillaries
- Efficient pumping of blood by the heart