Pulmonary Exam 2 Flashcards
active cycle of breathing
quiet breathing 2-3 cycles 3-4 thoracic expansions, hold 3 sec then sigh out quiet breathing 2-3 cycles repeat several times follow with forced expiratory technique
forced expiratory technique
1-2 huffs followed by relaxed controlled diaphragmatic breathing
autogenic drainage
can’t usually teach to children
often used in chronic bronchitis and CF
three levels: unsticking (from ERV to 1/2 ERV), collecting (VT+1/2ERV), evacuating (from 1/2 ERV to insp. capacity)
positive expiratory pressure
exhale through resistance, keeps airways open and helps air get behind mucus
after 10 PEP exhalations, perform several huffs, followed by several coughs
repeat 4-6 times for 10-20 min total
Acapella and flutter
PEP + high frequency oscillations
works sort of like internal percussion
acapella can be done in any position
flutter valve frequency changes at different angles
assist control ventilation
no spontaneous breaths occur
predetermined number of machine breaths
pt can initiate additional breath at set tidal volume and flow rate
intermittent mandatory ventilation (IMV, SIMV)
provides predetermined breaths, but pt can initiate additional spontaneous (non mechanical) breaths, more as they get better
minute ventilation = spontaneous + mechanical breaths
control ventilation
machine totally controls ventilation
assist ventilation
patient initiates, but breath is mechanical
resp rate is determined by patient
PEEP
positive end expiratory pressure
lungs remain partially distended to prevent airway collapse and facilitate collateral ventilation
given with mechanical breath
CPAP
continuous positive airway pressure
like PEEP but with spontaneous breath
pressure support
used when weaning from ventilator
ventilator delivers set pressure when patient triggers it
not forcing any air in, just giving pressure to help with flow rate
dyspnea definition
subjective!
sensation of difficulty breathing
chronic bronchitis lung changes
mucus clogging airways
emphysema lung changes
destruction of alveolar walls or overly dilated alveoli leading to poor gas exchange
cor pulmonale
right heart failure due to pulmonary pathology
training in COPD
train diaphragm for strength - endurance is fine
train peripheral muscles for endurance, they have more atrophy than you would expect from just being sedentary
exercise with COPD
quicker to produce lactic acid
increased CO2
central chemoreceptors increase activity
hyper-respirate to blow off CO2
intrinsic asthma
affects adults > kids not allergic reaction no family history attacks don't respond to treatment history of sinus infections poor prognosis
extrinsic asthma
affects kids>adults, often grow out of it
usually family history
attacks related to antigen
bronchiectasis
permanent abnormal dilation and distortion of medium sized bronchi and bronchioles due to destruction of elastic and muscular components of bronchial walls
associated with cystic fibrosis and infections
BODE index
predicts mortality in COPD BMI degree of obstruction (FEV1) Dyspnea Exercise capacity (6 min walk test)
cystic fibrosis
disorder of exocrine glands
abnormal secretions in resp tract, sweat glands, small intestine, pancreas, bile ducts
leads to frequent resp. infections and COPD
treatment for cystic fibrosis
*bronchial hygiene* exercise meds nutrition lung or heart/lung transplant