Pt 4 Ventricular Dysrhythmias Flashcards

* PVC's * V Tach * Torsades de Pointes * V Fib * Asystole * PEA

1
Q
  • PVC’s
  • V Tach
  • Torsades
A
  • V fib
  • Asystole
  • PEA
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2
Q

Ventricular dysrhythmias are potentially more life-threatening than atrial dysrhythmias b/c the __ __ pumps oxygenated blood throughout the body to perfuse vital organs & other tissues

A

left ventricle

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3
Q

?

Is a contraction coming from an ectopic focus in the ventricles

A

premature ventricular contraction (PVC)

> Is the premature (early) occurrence of a QRS complex

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4
Q

A PVC is wide & distorted in shape compared to a QRS complex coming down the normal conduction pathway

A

PVCs that arise from different foci appear different in shape from each other & are called multifocal PVCs; PVCs that have the same shape are called unifocal PVCs

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5
Q

When every other beat is a PVC, the rhythm is called __ __

When every third beat is a PVC, it’s called __ __

A

ventricular bigeminy

ventricular trigeminy

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6
Q

Two consecutive PVCs are called a couplet

A
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7
Q

__ __ occurs when there are 3 or more consecutive PVCs

A

Ventricular tachycardia

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8
Q
  • R-on-T phenomenon occurs when a PVC falls on the T wave of a preceding beat
  • Especially dangerous b/c the PVC is firing during the relative refractory phase of ventricular repolarization
A
  • Excitability of the cardiac cells inc during this time, & risk for PVC to start ventricular tachycardia or ventricular fibrillation is great
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9
Q
  • HR varies according to intrinsic rate & # of PVCs
  • Rhythm is irregular b/c of premature beats
  • P wave is rarely visible & is usually lost in the QRS complex of the PVC
A
  • Retrograde conduction may occur, & the P wave may be seen following the ectopic beat
  • P-R interval is not measurable
  • QRS complex is wide & distorted, lasts > 0.12 sec
  • T wave generally large & opposite in direction to major direction of QRS complex
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10
Q
  • Associated w/stimulants, electrolyte imbalances, hypoxia, & heart dz
  • Not harmful w/normal heart but CO reduction, angina, & HF in diseased heart
A
  • Assess apical-radial pulse deficit
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11
Q

Treatment

  • Correct causes (i.e., oxygen therapy for hypoxia, electrolyte replacement)
A
  • Anti-dysrhythmics
    > Beta-adrenergic blockers, procainamide, or amiodarone
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12
Q

?

A run of 3 or more PVCs defines __ __

Rate is 150-250 bpm

Rhythm may be regular or irregular

A

ventricular tachycardia (VT)

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13
Q
  • AV dissociation may be present, w/P waves occurring independently of the QRS complex
  • Atria may be depolarized by the ventricles in a retrograde fashion
A
  • P wave is usually buried in the QRS complex, and the P-R interval is not measurable
  • QRS complex is distorted in appearance & wide (>0.12 sec in duration)
  • T wave is in the opposite direction of the QRS complex
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14
Q
  • Ectopic foci take over as pacemaker
  • Monomorphic, polymorphic, sustained, & non-sustained
  • Considered life-threatening b/c of dec CO & the possibility of deterioration to ventricular fibrillation
A
  • VT may be sustained (longer than 30 sec) or non-sustained (<30 sec)
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15
Q

___ VT occurs when the QRS complexes gradually change back & forth from 1 shape, size, & direction to another over a series of beats

A

Polymorphic VT

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16
Q

___ VT has QRS complexes that are the same in shape, size, & direction

A

Monomorphic VT

17
Q

?

Is a polymorphic VT associated w/a prolonged Q-T interval of the underlying rhythm

! Typically treated w/magnesium sulfate or 150mg amiodarone

A

Torsades de Pointes

18
Q

?

Associated w/heart dz, electrolyte imbalances, rx’s, CNS disorder

  • Can be stable (pt has a pulse) or unstable (pulseless)
  • Sustained __ causes severe dec in CO
    > Hypotension, pulm edema, dec cerebral blood flow, cardiopulmonary arrest
A

Ventricular tachycardia

VT

19
Q
  • Precipitating causes must be identified & treated (e.g., hypoxia, electrolyte imbalances, ischemia)
  • VT w/pulse (hemodynamically stable) treated w/antidysrhythmics or cardioversion (if rx therapy ineffective)
A
  • Pulseless VT treated w/CPR & rapid defibrillation, followed by admin of vasopressors (epinephrine) & antidysrhythmics
20
Q

?

Is a severe derangement of the heart rhythm characterized on ECG by irregular waveforms of varying shapes & amplitudes

> Represents firing of multiple foci in the ventricle

> Mechanically, the ventricle is “quivering”, w/no effective contraction, & consequently no CO occurs

! Is a lethal dysrhythmia

A

Ventricular fibrillation

21
Q

Ventricular Fibrillation

  • HR is not measurable; rhythm is irregular & chaotic
  • P wave is not visible, & PR interval & QRS interval are not measurable
A
22
Q
  • Associated w/MI, ischemia, dz states, procedures
  • Unresponsive, pulseless, & apneic
A
  • If not treated rapidly, death will result

! Treat w/immediate CPR & ACLS
> Defibrillation
> Drug therapy (epinephrine, vasopressin)

23
Q

?

Represents the total absence of ventricular electrical activity

No ventricular contraction; pt unresponsive, pulseless, apneic

Must assess in more than 1 lead

A

Asystole

24
Q

Asystole

  • Usually a result of advanced cardiac dz, severe conduction disturbance, or end-stage HF
A
  • Treat w/immediate CPR & ACLS measures
    > Epinephrine &/or vasopressin
    > Intubation
  • Poor prognosis

> Strip shows VFIB to asystole

25
Q

?

Is a situation in which organized, electrical activity is seen on the ECG, but there’s no mechanical activity of the ventricles & pt has no pulse

A

Pulseless Electrical Activity (PEA)

! Prognosis is poor unless the underlying cause is quickly identified & treated

26
Q

PEA - H’s & T’s (mnemonic)

  • Hypovolemia
  • Hypoxia
  • Hydrogen ion (acidosis)
  • Hyper-/hypokalemia
  • Hypoglycemia
  • Hypothermia
A
  • Toxins
  • Tamponade (cardiac)
  • Thrombosis (MI & pulmonary)
  • Tension pneumothorax
  • Trauma
27
Q

PEA - Treatment

  • CPR followed by intubation & IV epinephrine
A
  • Correct the underlying cause