psychopathology Flashcards

1
Q

what are the four definitions of abnormality?

A
  1. deviation from social norms
  2. failure to function adequately
  3. statistical infrequency
  4. deviation from ideal mental health
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2
Q

describe deviation from social norms

A

when someone’s behaviour differs from how we expect them to behave (e.g. wearing pjs in office)

abnormal behaviour is even it offends people’s sense of what’s acceptable/ normal.

is culturally and time specific (e.g. homosexuality in victorian times)

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3
Q

outline the evaluation for deviation from social norms

A

criticism - susceptible to abuse
Ev- Muslims in china = abnormal by Chinese Gov + put into re-education camps. Same as homosexuality in victorian times.

Ex- Q’s whether it accurately identifies abnormality that needs treatment or way of Gov’s justifying societal prejudice.

L- but differentiates between normal and abnormal unlike DIMH

criticism - culturally relative
Ev- dependent upon culture e.g. DSM-V uses DSN and is based on white, western cultures but generalises to all.

Ex- so someone = abnormal dependent upon the culture they live in rather than whether it causes them a ‘problem’, like FTF does. Surely if beh abnormal, should be no matter where.

L- but can avoid cultural bias - e.g. DSM-V considers cultural differences when diagnosing abnormality - but is still a subjective measure within each culture.

Criticism- depends upon the context and degree of the DSN

Ev- e.g. wearing a swimming costume on the beach=normal, wearing it in a classroom=abnormal

Ex- if abnormal should be abnormal irrespective of the context or degree of beh

L- but even objective def like statistical infrequency has subjective judgements (e.g. where is the cut off for abnormality- 1%, 5%, 10%?)
all def gave subjective judgement issue.

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4
Q

describe failure to function adequately

A
  • unable to cope with demands of every day life
  • unable to look after themselves (hold down job, interact with others meaningfully)
  • counter-productive to individuals (e.g, not getting up, self-harm
  • causes observer discomfort
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5
Q

outline evaluation for failure to function adequately

A

criticism- who judges what’s abnormal?
Ev- someone’s deciding what functioning is, subjective, effected by class and ethnicity
Ex- individuals receive different diagnoses of abnormality,e.g. Sz more likely in men, depression women. (male not functioning more serious?)
L- more susceptible to abuse - not correctly diagnose someone. maybe be a beh that psychologist doesn’t like, so says not functioning.
Whereas SI based on data, so not.

criticism- what is defined as functioning/not?
Ev- what the individual seems as functioning may be different to the psychologist (e.g. self harm)
Ex- could be functional to individual, but seems not so observer discomfort.
stopping this beh may lead to more serious consequences like suicide so functional.
L- FTF can be utilised by having more than one psychologist decide abnormality, decreases bias.

strength- considers individuals subjective experience
Ev- allows us to view mental disorder from the view of person with it. How beh is affecting everyday life, so unique to individual.
Ex- not overgeneralising abnormality to all ppl with same beh, unlike DSN. basing abnormality of whether beh is adaptive or not to individuals.
L- looking at beh from neg perspective, as to what person can’t do.
DIMH has more pos outlook oh beh.

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6
Q

describe statistical infrequency

A

uses statistical data based on a normal distribution to decide who’s abnormal.

anyone outside statistical normal is abnormal, and any behaviour trait that is rare - e.g. IQ test - 100 average, 140+ seen as exceptionally smart, less than 80 abnormal

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7
Q

outline evaluation for statistical inferequency

A

strength- objective - based on statistical data
Ev- uses normal distribution and statistical significance to define abnormality -e.g. 1%, 5%, 10% significant difference from normal pop.
Ex- eliminates unconscious bias/human error. Less subjective, more reliable def of abnormality (can be applied consistently to all ppl)
L- still requires judgement of whether to use a 1%,5% or 10% cut off for being abnormal.

criticism - doesn’t distinguish between abnormal being functional or not
Ev - would consider a higher IQ (over 120) to be just as abnormal as a low IQ (below 80- learning disability)
Ex - doesn’t distinguish if infrequency is desirable or not. assumes both high and low IQ needs treatment.
L - does distinguish diff between normal and abnormal, unlike DIMH.

criticism- culturally relative
Ev- depends on which normal population data is being used. normal distribution is culturally specific (e.g. IQ test=western test and normed in USA)
Ex- abnormality is not defined universally so may be invalid to define abnormal from another culture.
L- can ensure gather normal population data from every culture and define within culture data gathered from.

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8
Q

describe deviation from ideal mental health

A

abnormality is the absence of criteria for good mental health:
- positive self-esteem
- self-actualisation
- ability to cope with stressful situations
- independence
-accurate perfection of reality

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9
Q

outline evaluation for deviation from ideal mental health

A

strength - positive approach to defining abnormality
Ev - Looks at what’s desirable
rather than undesirable. She accepted that the person is in control of working on themselves to develop in the 6
categories.
Ex - defines normality and not abnormality. Compares people against what it is to be normal, and if does not meet the criteria then abnormal.
L - doesn’t state how many of criteria are needed to be considered normal. so vague definition unlike FTF.

criticism- unrealistic
Ev - criteria hard to measure as vague, e.g. self-actualisation, and accurate perception of reality.
Ex-not effective def of ab - everyone ab as no one meets all criteria, so not useful as no distinct between normal and abnormal.
L- but is positive approach, and humanistic - views everyone as unique and we all need help; as no such thing as ‘normality’.

criticism - compares physical illness to a mental illness
Ev - Physical health is observable and can be felt and measured objectively. Mental health cannot be directly observed or measured.
Ex - criteria for DIMH isn’t observable or easily measurable, e.g., self-actualisation or self- esteem.
L - However, this is a criticism
relevant to all definitions. (and are all culturally relative)

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10
Q

define phobias

A

a persistent extreme + irrational fear of an object which disrupts your everyday life.

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11
Q

name some behavioural characteristics of phobias

A

-avoidance (running away)
- panic behaviour (fight/flight, e.g. sweating, increased heart rate

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12
Q

name some emotional characteristics of phobias

A
  • anxiety - worried when facing phobic stimulus
  • fear
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13
Q

name some cognitive characteristics of phobias

A
  • cognitive distortions - person is aware their fear is excessive
  • irrational beliefs - unrealistic thoughts
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14
Q

what is the behavioural explanation of phobias?

A

the two process model (classical creates phobias and operant maintains it)

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15
Q

how does classical conditioning explain phobias?

A

UCS —> UCR
(loud noise —>fear)

UCS+NS —> UCR (loud noise + rat —> fear)

CS —> CR (rat—> fear)

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16
Q

how does operant conditioning explain phobias?

A

negative reinforcements - take away fear to increase the chances of further avoidance of phobia

makes person feel calmer by avoiding it

but is teaching the person that there is something to fear, so increases the fear

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17
Q

describe the case study of little albert

A
  • baseline check- showed a variety of animals and chose the animal
    Little Albert liked the most (white rat)
  • Paired the white rat with a loud noise (hammer striking a metal bar)
  • Created a phobia of white rats (discrimination)
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18
Q

what are the 2 behavioural approaches treatments of phobias?

A

systematic desensitisation

flooding

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19
Q

outline systematic desensitisation

A

UCS (relaxation techniques) = UCR (calm)

UCS (relaxation techniques) + NS (phobia) = UCR (calm)

CS (phobia) = CR (calm)

  • teach client relaxation techniques (eg meditation, deep breathing)
  • Develop a hierarchy of needs with the client (from least fearful to most fearful)
  • The client goes through each step of the hierarchy using the relaxation techniques. Once calm they can then move onto the next step in the hierarchy.

This uses 3 techniques:
a. In vivo = direct contact with the phobic stimulus
b. In vitro = no direct contact with the phobic stimulus
c. Modelling = client watches another person interact calmly with phobic
stimulus.

  • if become fearful, can go back a step where they’re calm and repeat process.
  • goes at pace of client and is a gradual process.
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20
Q

outline flooding

A

one session

Directly exposes client to phobic stimulus (in vivo exposure) without the ability to withdraw. NOT GRADUAL

Client is exposed to phobic stimulus until fear is eradicated (e.g., client is back in a parasympathetic state (calm)

Client recognises that fear levels have dropped + that the phobic stimulus is harmless = extinction of phobia

(removal of negative reinforcer - operant conditioning) - cannot avoid fear

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21
Q

outline the A03 for the behavioural approach for treating phobias

A

_strength (research evidence)_
Ev- 75% patients responded to SD. Used several exposure techniques- in vivo/vitro, modelling.

Ex- SD = effective in treating phobias.
in vivo/vitro allows gradual exposures and sudden exposures
modelling shows how else to respond

L - but evidence shows in vivo more successful in SD - supports flooding. study found flooding more effective and quicker in treating.

criticism - flooding = unethical
Ev- immediately faced with fear, no right to withdraw, treatment ‘done’ to client.

Ex - causes high level of distress (can harm those with medical conditions). less likely to give consent. very few psychologists conduct due to this. SD better as gradual, at clients pace, so ethical.

L- but flooding does screen for medical condition to avoid harm and one session so aren’t exposed to fear as long as SD

criticism - not effective for all types of phobias
Ev - study found SD and flooding may not be effective against phobias which have underlying evolutionary survival component.

Ex - criticises as not all phobias learned through association + maintained through reinforcement.
Evolutionary - resistant to treatment. (could explain 25% didn’t respond)

L - SD and flooding only treat symptoms not cause, so ‘talking therapies’ could be better

22
Q

what are the definitions of depression?

A
  • Mood disorder

Major depressive disorder = severe but short-term change in mood

Persistent depressive disorder = long term or reoccurring depression
Behavioural

23
Q

give some examples of behavioural characteristics of depression

A

changes in sleep patterns (insomnia/hypersomnia)

changes in eating habits (eating more/less)

social withdrawal

24
Q

give some examples of emotional characteristics of depression

A

low mood
loss of interest
anger

25
name some cognitive characteristics of depression
poor concentration negative schemas of yourself (low self worth) suicidal thoughts
26
what are the 3 explanations of depression? (cognitive approach)
- Ellis’ ABC model - Beck’s cognitive triad - mustabatory thinking and negative automatic thoughts.
27
describe Ellis’ ABC model
- Activating Event - e.g., failing an assessment - Belief (“I am a failure, I’ll never pass this course, I am not clever enough”) - consequence - stop revising, avoid future assessments, fail real exams The consequences reinforce the irrational thinking when a similar activating event occurs = negative automatic thoughts (NATs)
28
describe Beck’s cognitive triad.
- the self (i am worthless, a failure) - the future (i’ll never amount to anything, ill have no decent job or money) - the world (everyone thinks I’m a failure, nobody values me)
29
describe mustabatory thinking and negative automatic thoughts (NATs)
mustabatory thinking - Ellis’ ABC model (irrational belief based on ‘should and musts - assumption must be true for them to be happy) e.g. i must do very well or i am worthless. NATs - Becks cognitive triad - People are prone to overly self defeating and negative thoughts that are difficult to challenge. Both NATs and Mustabatory thinking lead to low mood and passivity in behaviour.
30
outline the AO3 for the cognitive approach for explaining depression
**_Support: depressed clients have more irrational thinking_** - **Ev** - Depressed patients made more logical errors than non-depressed patients. Depressed patients given more NATs became more depressed. -**Ex** - Suggests irrational thinking causes depression, as depressed are more likely to report irrational thinking, such as Mustabatory thinking and NAT’s. -**L** - However, not causational. Irrational thinking may be a symptom of depression rather than a cause. People with depression are realists, as gave more accurate estimations of the likelihood of disasters than “non-depressed”. ‘sadder but wiser effect’. **_Support: CBT effective in treating depression, hence supports cognitive as cause_** -**Ev** - Study found CBT effective, especially when combined with drug treatment. -**Ex** - This suggests that challenging irrational thinking treats depression. Hence irrational thoughts cause depression. Means that the cognitive explanation is correct in explaining depression. -**L** - However, it assumes therapies treat cause of depression (treatment fallacy). Attachment issues may cause depression and alter cognition. E.g., maternal deprivation **_Criticism: Ignores biological explanations of depression_** -**Ev** - Research supports correlation – Depressed people 10 times more likely to have gene (5-HTT) mutation. -**Ex** - Implies depression is heredited through genes and that irrational thinking is a symptom of a biological cause for depression. -**L**- Therefore, an interactionist explanation of both biological and cognitive, hence explains why combined drugs and CBT is most effective treatment of depression
31
outline Ellis’ ABCDEf: rational Emotive Behavioural Therapy (REBT) (treatment of depression)
activating event (A) e.g. failing an assessment challenging irrational beliefs to be more rational Disputing irrational thoughts (D) Consequence (C) • Stop revising • Avoid future assessments • Fail the real exams Belief (B) • “I am a failure” Effect on Feelings (F) • Feel motivated Effect on behaviour (E) • Revise harder
32
outline the AO3 of the cognitive approach in treating depression
**_Strength - Research support for the effectiveness of CBT_** -**Ev** - Ellis claimed a 90% success rate for REBT, average of 27 sessions. -**Ex** - Shows CBT is effective – challenging irrational to rational thoughts through logical and empirical disputing reduces depression. So, REBT is a good treatment for depression. -**Ex** - but depends on competence of therapist. The more competent the more successful the outcomes of REBT. It also depends on the motivation of the client. **_ Criticism - individual differences in effectiveness of CBT_** -**Ev** - CBT = less effective where clients have high levels of stress and realistic stressors they cannot control; high level of irrational beliefs & they are resistant to change. -**Ex** - so, REBT does not work with all people. Due to some being more resistant to empirical disputing as in high stress situations. They have lots of evidence to prove their many irrational beliefs. Hence more resistant to change. **_ Criticism – ignores alternative biological treatments for depression_** -**Ev** - Study found that CBT with anti- depressants (SSRIs) are more effective than CBT or drugs alone. -**Ex** - The drug therapy reduces the symptoms of depression to increase the depressed patient's motivation to attend the 27 sessions and engage actively in the therapy sessions. Thus, reducing the resistance to REBT. -**L** - In summary, research suggests that there are small differences in effectiveness of alternative treatments for depression. Therefore, they all have efficacy. So an interactional approach to treatment is best (drugs & CBT)
33
what are emotional characteristics of OCD?
- anxiety - depression - guilt and disgust
34
what are cognitive characteristics of OCD?
obsessive thinking hyper vigilant selective attention
35
what are the behavioural characteristics of OCD?
- compulsions - e.g. repetitive hand washing - avoidance beh of anxiety stimulus (e.g. germs)
36
what are the 2 biological explanations of OCD?
genetic explanation neural explanation
37
outline the genetic explanation of OCD
OCD = inherited . SERT gene • decreased level of serotonin • increases obsessions (lack of inhibition of thought processes) COMT gene faulty • increased level of dopamine • increases compulsions by rewarding the compulsive behaviour.
38
outline AO3 for the biological explanation of OCD: genetic explanation.
**_Strength - research support for candidate genes causing OCD_** -**_Ev_**- Meta-analysis of longitudinal twin studies. Found MZ twin = 68% concordance rate; DZ = 31% concordance rate. -**_Ex_** - so, OCD is genetic – SERT & COMT gene. MZ twins = 100% genes compared to DZ = 50%. Therefore, the more genetic similarity have the higher the concordance rate for OCD are more similar as they share the same OCD gene. E.g., MZ higher concordance than DZ and have higher genetic similarity. -**_L_** - but, OCD could be env. MZ twins have higher concordance as have more environmental similarity in how they are reared (e.g., dressed the same, same friends) than DZ (who look different in appearance). Therefore, the meta-analysis does not control enough for the environment and OCD may therefore not be genetically caused. **_Criticism – OCD could be environmentally caused and not biological_** -**_Ev_**- Study found ½ of OCD patients had experienced a traumatic experience. OCD was more severe in those who had experienced a traumatic event. -**_Ex_** - Study suggests that OCD is due to life experiences (e.g., MZ twins may have experienced the same childhood trauma unlike DZ). Therefore, OCD is not genetics. Explains why brain may be hypervigilance to future threats, from having past threats. -**_L_** - but, DSM may be a better exp as only 50% of people with OCD had trauma. Could be a genetic predisposition for OCD that is triggered by a traumatic event from the env bringing out the OCD symptoms. Therefore, an interaction of nature & nurture. -**_L_** - but, the modern DSM argues that the env can switch on or off our genetics (such as the SERT & COMT gene; epigenetics). So, is it that people with OCD already have a genetic predisposition that is being triggered by childhood trauma, or is it the childhood trauma that causes the genetic abnormality?
39
outline the role of the caudate nucleus in the neural explanation of ODC
regulates attention. There is a decrease in serotonin in people with OCD. Therefore, they attend more to threats (hypervigilance). This is linked to the obsessions.
40
outline the role of the parahippocampal gyrus in the neural explanation of ODC
regulates unpleasant emotions (e.g., anxiety). In those with OCD it is overactive, thus creating high anxiety linked to the obsessions.
41
outline the role of the orbitofrontal cortex (OFC) in the neural explanation of ODC
worry centre (responsible for planning and decision making to avoid the high anxiety). In those with OCD there are high dopamine levels. This leads to abnormal decision making to avoid irrational threats and anxiety.
42
outline the role of the basal ganglia in the neural explanation of ODC
has increased dopamine. It causes the repetitive motor movement, known as compulsions.
43
outline the role of the thalamus in the neural explanation of ODC
high in dopamine in those with OCD, rewards the compulsive behaviour causing the to the OFC to say the “threat” has been dealt with successfully. Increasing the chances of the same action being taken again.
44
outline AO3 for the biological explanation of OCD: Neural explanation.
**_Strength – research support for neurochemical explanation of OCD_** -**_Ev_** - Treatment using SSRIs (selective serotonin reuptake inhibitors – which increases serotonin levels), OCD symptoms decrease; various studies range from 50-80% improvement in symptoms of OCD. -**_Ex_** - Suggests that low serotonin causes OCD symptoms, as when serotonin levels are increased by SSRIs that there is a reduced in OCD symptoms. -**_L_** - However, only 50% improve, so 50% of people with OCD taking SSRIs do not improve in their symptoms. Therefore serotonin cannot be the only factor ‘causing’ OCD. If it was the success rate would be closer to 100%; but the results are too mixed, as a large range. **_Strength – support for neurophysiological & neurochemistry causing OCD_** -**_Ev_** - PET scan – found too low serotonin & hypersensitivity in caudate nucleus. Also found an exaggerated response in basal ganglia & OFC. -**_Ex_** - Implies that caudate nucleus has too little serotonin in people with OCD, so does not inhibit the function of the caudate nucleus to detect threats. The OFC and basal ganglia are then overactive and creating compulsions to reduce the anxiety created by the caudate nucleus. Therefore, OCD is biologically caused by the function and structure of the brain. -**_L_** - However, correlational, cannot assume that abnormal brain activity in specific areas are the cause of OCD. PET scans does not measure serotonin levels directly. **_On the other hand, there is mixed evidence proving the correlation between the basal ganglia and OCD_** -**_Ev_** - E.g., one study found no difference between OCD and non-OCD patients in the structure of the basal ganglia. -**_Ex_** - If the basal ganglia was linked to OCD there would be a unique difference between those with OCD and without. Therefore, it appears OCD is not linked to the basal ganglia. -**_L_** - However, there is evidence supporting the basal ganglia. A case study of person who suffered basal ganglia damage due to a head injury, developed OCD. However, could be the trauma of the head injury event causing OCD than biological brain damage
45
what do Selective Serotonin Reuptake Inhibitors (SSRIs) do?
1.Blocks the reuptake process in the pre-synapse. 2.Trapping serotonin in the synaptic gap. 3.With each new action potential more serotonin is released into the synaptic gap but cannot be reabsorbed. 4.Hence serotonin slowly builds in the synaptic gap over time, allowing more neurotransmitters to bind to the receptor sites on the post synapse.
46
what do Tricyclics & SNRI’s do?
Block the reuptake and reabsorption of noradrenaline and serotonin through reuptake.
47
what’s do MAOI’s do?
stop the breakdown of monoamine oxidases (noradrenaline, serotonin & dopamine)
48
what is the effectiveness and side effects of SSRI’s?
Takes 3-4 months to work, and 50 – 80% improvement. Indigestion, sterilisation, dry mouth.
49
what is the effectiveness and side effects of SNRI’s and tricyclics?
small improvement compared to SSRI’s Fewer side effects and better tolerability, only symptom = changes in sexual function.
50
what is the effectiveness and size effects of MAOIs?
56 – 60% responded well to treatment, similar effect as the SSRI’s. Can cause fatal increases in blood pressure when mixed with certain foods.
51
outline A03 for the biological approach for treating OCD
**_Strength - Drugs are effective at treating OCD_** **_Ev_** - One study found 50-80% improvement in symptoms using SSRI’s. Another meta-analysis compared SSRIs to placebo and all 17 studies found a 70% reduction in OCD symptoms for SSRIs. **Ex_** - Shows robust evidence from multiple studies that SSRIs are effective in treating OCD by increasing serotonin levels. **_L_** - However, drug only treats symptoms not the cause. Study found 90% relapse rate when stopped taking drugs. Therefore, only works whilst on the SSRIs, so only a ‘sticking plaster’ and not treating the cause of OCD. **_Criticism - side effects of drugs_** **_Ev_** - side effects of SSRI’s - Indigestion, sterilisation, dry mouth. **_Ex_** - This a negative compared to other psychological therapies. Effects quality of life of adults, particularly those in intimate relationships as it affects their sexual functioning and can even be fatal (MAOIs). Whereas psychological therapies have no side effects. **_L_** - However, psychological therapies are more time consuming and expensive. They require commitment to turn up to sessions and this may be anxiety provoking for the person with OCD. Whereas drug therapies require very little commitment or motivation, can be taken at home and does not disrupt daily life. Therefore, drug therapies are better practically for OCD. **_Criticism - biologically reductionist_** **_Ev_** - Most treatment uses a combination of CBT and drug therapy. The drugs reduce the symptoms of anxiety and enable to person to access the more committed CBT sessions. **_Ex_** - Therefore, it is reductionist to consider only drug therapy as it is not a long-term solution. However, when considered through interactional treatment methods it is more effective and holistic. **_L_** - Therefore, in summary to reduce long term symptoms of OCD both biological and psychological treatments are needed. One to deal with the neurophysiological and neurochemical changes in the brain and the other to deal with coping strategies and ways of managing their OCD. This gives the best