Psychopathology Flashcards

1
Q

(AO1) definitions of abnormality: statistical infrequency

A

*anything in the top or bottom 2% of a certain characteristic is deemed abnormal

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2
Q

(AO3) definitions of abnormality: statistical infrequency

A

*Strength. there is real life application in the diagnosis. all assessments of patients with mental disorders include measurement of how severe the symptoms are. therefore useful as part of clinical assessment
*Limitation. does not help distinguish between desirable and undesirable behaviour. some abnormal behaviour is desirable. using this method alone wouldn’t inform clinic if treatment was needed. reduces usefulness as a measure of classification.

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3
Q

(AO1) definitions of abnormality: failure to function adequately

A

abnormally judged as unable to deal with the demands of everyday living.
-distress and anxiety
-observer discomfort
-stops them from working
-stops keeping hygienic
-causing them or others harm
-dangerous behaviour

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4
Q

(AO3) definitions of abnormality: failure to function adequately

A

*Strength. takes into account individual experiences of the patient. allows us to view mental disorder from the pov of the person. easy to judge objectively. therefore, treatment can be specific to patients needs.
*Limitation. hard to judge unconventional behaviour. ppt may feel like they are coping just fine. weakness is that it depends who is judging.

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5
Q

(AO1) definitions of abnormality: deviation from ideal mental health

A

absence of signs of good mental health used to judge abnormally. jahoda (1958)
-accurate perception of reality
-positive attitude to himself
-self actualisation
-resistance to stress
-environmental mastery
-independent

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6
Q

(AO3) definitions of abnormality: deviation from ideal mental health

A

*strength. focuses on positives over negatives. also focuses on desirable over undesirable behaviour. therefore can be argued it takes more of a positive approach to defining abnormality.
*Limitation. sets high standards for mental health. hard to generalise. also hard to measure. could be argued this definition is not useable as it is too hard to achieve.

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7
Q

(AO1) definitions of abnormality: deviation to social norms

A

sees any behaviour which differs from that society expects as abnormal. passed on through socialisation. social norms can change over time and across cultures.

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8
Q

(AO3) definitions of abnormality: deviation to social norms

A

*Strength. distinguishes between desirable and undesirable behaviour. also takes into account the effect that the behaviour has on others. high validity
*Limitation. doesn’t consider cultural relativism. the DSM is based on western social norms and ignores Eastern deeming them abnormal. therefore can’t be used as a universal explanation of abnormality.

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9
Q

(AO1) emotional characteristics of phobias, OCD and depression

A

phobias - fear, anxiety
OCD - embarrassment, shame
depression - sadness, abolition

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10
Q

(AO1) cognitive characteristics of phobias, OCD and depression

A

phobias - irrational beliefs, cognitive distortion
OCD - obsessive thoughts
depression - negative memory bias

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11
Q

(AO1) behavioural characteristics of phobias, OCD and depression

A

phobias - avoidance, panic
OCD - compulsive behaviour, avoidance
depression - insomnia, change in appetite, reduction in energy

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12
Q

(AO1) behavioural approach to explaining phobias
*****

A

*Mowrer (1960) two process model. Phobias acquired through classical conditioning and maintained through operant conditioning.
*Classical conditioning - Watson and Reiner (1920) Little Albert. UCS=UCR. UCS+NS=UCR. CS=CR
*Operant conditioning - reinforced or punished.

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13
Q

(AO3) behavioural approach to explaining phobias
*****

A

*Strength. Link between bad experiences and phobias. De Jongh et al (2006) found that 73% of people with fear of dental treatment had a past traumatic experience compared to a control group with only 21%.
*Limitation. Not complete explanation. Bounton (2007) highlights evolutionary factors could play a role in phobias especially if the result of a particular stimulus could cause pain/death to ancestor. Therefore suggests that some phobias are not learnt and are actually innate.
*Limitation. Ignores role of cognitions. Argue phobias develop as a result of irrational thinking and not just learning. CBT argued more effective than behaviourist treatments demonstrating its importance.

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14
Q

(AO1) behavioural approach to treating phobias *****

A

*systematic desensitisation. therapist and client work together to make an anxiety hierarchy. List goes from least to most frightening. Clients are taught relaxation techniques and understand that it is possible to undergo reciprocal inhibition. (relax and stress same time). client exposed to least feared level and when relaxed they move up.
*flooding. immediate exposure to very frightening. without the option of avoidance, the client quickly learns that the phobia stimulus is harmless. clients could also become so exhausted of being in presence of phobic stimulus that they become relaxed.

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15
Q

(AO3) behavioural approach to treating phobias *****

A

*Strength of systematic desensitisation. Gilroy et al (2003) followed up 42 people who had SD for spider phobia. At both 3 and 33 months, the SD group less fearful than control group
*Strength of flooding. Cost effective and time effective. Only recquires 1 session at 2-3 hours long.
*Limitation of flooding. Traumatic. Schaumer et al (2015) found ppts and therapists rated flooding stressful. sometimes could drop out and phobia becomes worse than originally believed.

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16
Q

(AO1) cognitive approach to explaining depression *****

A

*Beck (1967) negative triad. Faulty information processing. Negative self schema. Negative triad (world, self, future)
*Ellis ABC model (1962) claimed anxiety and depression is a result of irrational thoughts.
A- activating event (external event)
B- beliefs (range of irrational beliefs)
C- consequences (emotional and behavioural consequences)

17
Q

(AO3) cognitive approach to explaining depression *****

A

*Limitation. too reductionist. states negative thinking develops depression and ignores biological research. therefore cognitive approach too simplistic
*Limitation. ignores situational factors like family problems or life events. these aren’t considered with this explanation
*Strength. faulty cognitions linked to depression. Grazioli and Terry (2000) studied 65 pregnant women. women judged higher cognitive vulnerability and more likely end with depression. supports cognitive thinking leads to depression. increases validity.

18
Q

(AO1) cognitive approach to treating depression

A

*Client and therapist work together to clarify client problems and identify goals.
*Becks cognitive therapy - negative triad (the world, the self, the future) Once identified these thoughts must be challenged and this is the central component of therapy. Client is set homeowork to increase motivation.
*Ellis’ rational emotive behaviour therapy (REBT) extends ABCDE model. D - dispute. E - effect. central technique is to challenge irrational thoughts. This is to break the link between negative life effects and depression

19
Q

(AO3) cognitive approach to treating depression

A

*Strength. Evidence for effectiveness. March et al (2007) tested 327 depressed clients with CBT, drugs, or use of both. Found after 36 weeks, 81% improved on CBT and drugs. 86% increased when used both
*Limitation. Ali et al (2017) assessed depression in 439 clients each month for 12 months and found that 42% of clients relapsed into depression after 6 months and 53% after a year. CBT must be repeated periodically
*Limitation. Many clients with depression experience extreme abolition. CBT requires a lot of meetings and talking which might be too much. Might struggle with the idea of homework. Sturmey (2005) suggests any form of psychotherapy including CBT is not suitable for those with learning difficulties

20
Q

(AO1) biological approach to explaining OCD : genetic

A

*OCD can be explained as family pass on genetic vulnerability. Due to the nature of the diathesis stress model, it only means the offspring will pick up OCD if there is environmental stress.
*candidate gened involved with development of OCD. For example, 5HT1-D beta implicated with transport of serotonin across synapse.
*OCD is polygenic. Taylor (2013) found 230 different genes associated with the condition
*OCD is aetiologically heterogeneous meaning that genes from 1 person can vary to another b ut both still have OCD.

21
Q

(AO3) biological approach to explaining OCD : genetic

A

*Strength. Nestadt et al (2010) twin studies found 68% MZ twins shared OCD compared to 31% DZ. MZ 100% dna. However, could be due to MZ twins being treated similarly in envrionment.
*Strength. Lewis (1936) observed OCD patients. 37% had parents with OCD and 21% had siblings with OCD.
*Limitation. OCD doesn’t focus enough on environmental risk factors causing the condition. Cromer et al (2007) found that over half of his patients with OCD had experienced some form of traumatic event in their past.

22
Q

(AO1) biological approach to explaining OCD : neural

A

*Serotonin and dopamine. Low levels of serotnin reduces mood.
*The worry circuit:
1) OFC sends signal to thalamus
2) caudate nucleus suppresses important signals
3) when the caudate nucleus is damaged, the thalamus is signalled too much causing compulsions

23
Q

(AO3) biological approach to explaining OCD : neural

A

*Strength. Menzies et al (2007) found OCD has low low grey matter in key brain regions. Grey matter plays crucial role in allowing you to function normally from day to day.

24
Q

(AO1) biological approach to treating OCD *****

A

*SSRI - increase level of serotonin in the synapse which continue to stimulate post-synaptic neruon. Often used effectively with CBT (March et al) Example of SSRI = fluoxetine
*SNRI - used for people who don’t benefit from SSRI. increases levels of serotonin and noradrenaline in the synapse

25
Q

(AO3) biological approach to treating OCD *****

A

*Strength. cost effective and non disruptive to peoples lives. In comparison to CBT, tablets are quick and easy to take to reduce symptoms
*Strength. Soomro et al (2009) did 17 studies comparing SSRI and placebo effects. Found that SSRI was significantly better and reduced symptoms 70% of the time. For the other 30%, often other drugs or a combination was effective to reduce symptoms
*Limitation. Potentially serious side effects. Some people may receive no benefit from drugs ad experience weight gain, loss of sex drive, blurred vision and this could outweigh the symptoms of OCD. SNRI tend to have worse/ more severe effects and that is why it is uses as a back up. Also, Ashton (1997) claimed drugs are only effective for 4 weeks.