Psychological explanations and treatment Flashcards

1
Q

Family dysfunction

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Schizophrenic Mother - Mother is cold, controlling and rejecting. Father is often passive, Mother is dominant.
Double Bind theory - Mixed messages, child will never know what to do and will see the World as confusing and dangerous.
Expressed emotion - High EE environment –> Higher relapse rate –> Relapse rate doubled (Butzlaff + Hooley, 1998) –> Emotional over involvement, overbearing, anger, hostility, self - sacrifice, verbal criticism.
Family dysfunction - Schizophrenia is due to family experiences of interpersonal conflict, communication problems, criticism and control.
Interpersonal conflict - Conflict between you and other individuals in the home.

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2
Q

The schizophrenogenic Mother

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Fromm Reichmann (1948) - A neo - freudian
Proposed a psychodynamic explanation for schizophrenia. Based on the accounts she heard from her patients about their childhoods.
She noticed that many of her patients spoke about a particular type of parent, which she called the SM.
The schizophrenogenic Mother is cold, rejecting and controlling. She tends to create a family climate characterised by tension and secrecy.
This leads to distrust and later develops into paranoid delusions.

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3
Q

Double - bind theory

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Bateson et al (1972) - Agreed that family climate is important in the development of sz, but emphasised the role of communication style within a family. The developing child regularly finds themselves trapped in a situation where they fear doing the wrong thing, but receive mixed messages about what this is and feel unable to comment on the unfairness of the situations or seek clarification.
When they get it wrong, the child is punished by the withdrawal of love.
This leaves them with an understanding of the world as confusing and dangerous. This is reflected in symptoms like disorganised thinking and paranoid delusions.

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4
Q

Expressed Emotion

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Brown et al (1972) -
The level of particularly negative emotion expressed towards a patient by their carers.
These high levels of EE –> a source of stress for the patient.
Primarily an explanation for relapse in patients with schizophrenia, but may also be a source of stress that can trigger the onset of sz in a vulnerable person.
Verbal criticism - “They just really frustrate me”
Hostility - anger and rejection - “They cause problems for me everyday”
Emotional over involvement/ Self-sacrifice - “I don’t care about myself anymore, I would rather focus on him getting better.”

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5
Q

A03) Evaluation

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Weakness of RM:
P) Although there is a large amount of evidence to support the idea that poor childhood experiences in the family are associated with adult schizophrenia, there is almost none to support the schizophrenogenic Mother or double-bind.
E) Berger (1965) - Found that schizophrenics reported a higher recall of double bind statements by their Mothers than non-schizophrenics.
C) However, other research is less supportive.
Liem (1974) –> Found no difference in parental communication in families with a schizophrenic child compared to normal families.
Whilst Berger’s study supports the association between double bind and sz, this research may not RELIABLE, as patient’ recall may be affected by their sz, or by time.
C) This use of retrospective data, where patients report childhood, lowers the validity, as recall and accuracy may be affected. This may also be subjective, so not scientific data. To combat this, use inter-rater reliability.

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6
Q

A03) Evaluation

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Weakness of RM:
P) Weakness of the family based explanations –> led to parent blaming.
E) Parents, who have already suffered at seeing their child’s descent into schizophrenia, are also likely to bear the lifelong responsiblity for their care and feel responsible for their child’s illness, which causes even greater stress and anxiety.
C) The views are outdated –> no longer tolerated by families –> seen as destructive rather than productive. Highly unethical + adds insult to the injury.
C) Therefore, the shift in the 1980’s from hospital to community care (often parental care), may be one of the factors leading to the decline of the schizophrenogenic Mother and double bind theories, as parents no longer tolerated them.

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7
Q

A03) Evaluation

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Supports A01:
P) Families where communications are commonly to do with criticism, hostility and disapproval are said to have high EE.
E) Brown (1966): People recovering from SZ and discharged from hospital were followed up over a 9 month period. Interviews with the family members were conducted to determine the level of expressed emotion.
Findings: High EE levels - 58% of people returned to hospital for further treatment.
Low EE levels - 10% returned to hospital.
C) Criticism - The sufferer may be influencing the family behaviour patterns rather than the other way round.
Western Cultures - No consideration of other cultures –> imposed etic —> cultural bias.
C) Supports high EE’s role in schizophrenia, but methodology problems.

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8
Q

A03) Evaluation

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Supports A01/ Weakness of RM:
P) Supporting evidence for the effectiveness of family therapy and the role of EE in relapse.
E) Garety et al (2008) –> estimates that relapse rates for individuals who receive family therapy are 25%, compared to 50% for those who receive standard care alone. The effectiveness of family therapy, based on reducing expressed emotion, supports the family dysfunction approach, as by reducing the expressed emotion, the rate of relapse reduces.
C) However, Altorfer et al (1998), found that 1/4 of patients they studied showed NO PHYSIOLOGICAL RESPONSES to stressful comments from their relatives. This suggests that NOT ALL patients who live in high EE families relapse.
L) Therefore, there are individual differences in response to high EE, which lowers the validity, as the explanation may not apply to all schizophrenia sufferers. However, expressed emotion does still play a role in the return of schizophrenia symptoms, but individual differences need to be considered.

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9
Q

Psychological treatments: Family therapy

A

Family therapy - A range of INTERVENTIONS aimed at the family of someone with schizophrenia.
Improve communication and interaction, reduce stress, reduce re hospitalisation, used with drug therapy and clinical care. The main aim of family therapy is to provide SUPPORT for carers, in an attempt to make family life LESS STRESSFUL and so REDUCE
RE-HOSPITALISATION.
Family intervention - Has developed as a result of STUDIES of the family environment and its potential role in affecting the course of sz.
Research has consistently shown that the long-term outcome for an individual with sz has much to do with the RELATIONSHIP between the individual and carers.
Poor relationships tend to result in poor outcomes eg. a greater chance in relapse.

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10
Q

How does family therapy help?

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Pharoah et al (2010) - Identified a range of strategies by which family therapists aim to improve the functioning of the family with a member suffering from schizophrenia.
- Forming a therapeutic ALLIANCE with all family members.
- Reducing the levels of EXPRESSED EMOTION and STRESS of caring for a relative with schizophrenia.
- Reduction of anger + guilt in family members.
- Improving the ABILITY of the family to ANTICIPATE and SOLVE PROBLEMS.
- Helping family members achieve a BALANCE between caring for the individual and MAINTAINING their OWN LIVES.
- Improving families beliefs about behaviour towards schizophrenia.
- Psycho education - Helping the person and their carers to understand and be better able to deal with the illness.

Pharoah –> These strategies work by REDUCING STRESS LEVELS and EXPRESSED EMOTION, while also INCREASING the chances of patients COMPLYING with MEDICATION. This combination tends to result in reduced relapse rates and re- admission to hospital.
Family therapy activity -
- Psycho education - Educate them on sz + how to communicate with patients.
- Talk about triggers to anticipate and solve problems.
- Identify expressed emotion, talk through it, teach communication techniques.
- Helplines and other resources to help stress.
- Deal with double bind (mixed messages).

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11
Q

NICE Guidance

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National Institute for Clinical Excellence - An organisation
NICE recommend that family therapy should be offered to ALL individuals with schizophrenia, who are IN CONTACT with or LIVE with family members.
They stress that such interventions should be considered a PRIORITY where there are PERSISTENT symptoms or a HIGH risk of relapse.

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12
Q

Duration of family therapy

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  • Typically offered for a period of between 3-12 months and at least 10 sessions.
  • Garety et al (2008) - Estimated those who received family therapy had 25% relapsed rate, compared to those who received standard care alone had a 50% relapse rate.
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13
Q

A03) Family therapy

A

Challenges A01)
P) Family therapy aims to improve the patients’ quality of life and make sz more manageable.
E) It helps by reducing stress in the family and the patient themselves.
C) Whilst this is worth doing, it does not cure sz.
C) Therefore, whilst providing coping strategies for patients, which they can use to manage their current and future symptoms is effective, family therapy still isn’t an independent successful cure for sz.

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14
Q

A03) Family therapy

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Weakness of RM)
P) Methodological limitations of FT studies was shown by Pharoah et al (2010), who reviewed the evidence for the effectiveness of FT compared to anti-psychotics alone.
E) Findings –> There was a reduction in the risk of relapse and hospital readmission, during treatment and in the 24 months after.
C) However, whilst some studies showed an improvement in the overall mental state of patients, others did not, yet there was an increased compliance with medication. Additionally, FT did not appear to have much of an effect on CONCRETE outcomes, such as living independently or employment.
C) This suggests that FT significantly REDUCES
RE HOSPITALISATION, and somewhat improves the quality of life for patients and their families, but the findings are INCONSISTENT. So, the evidence to support the effectiveness of FT is fairly WEAK.

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15
Q

A03) Family therapy

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Challenges A01)
P) Weakness of FT –> Idea proposed by Pharoah et al (2010) –> suggests that it may have less to do with improvements in functioning and more to do with the fact that it increases medical compliance.
E) Patients are more likely to reap the benefits of medication, because they are more likely to COMPLY with their MEDICATION REGIME.
C) Therefore, anti-psychotics may be the more effective treatment for schizophrenia and family therapy may ONLY BE NECESSARY to the extent that it encourages patients to take anti-psychotics.
C) Additionally, Pharoah et al’s META ANALYSIS found METHODOLOGICAL ISSUES.
Although 53 studies claimed to have RANDOMLY ALLOCATED participants to TREATMENT groups, the researchers note that a LARGE NO. of studies used in this review were from the people’s republic of CHINA. Evidence has emerged that there may be some DISCREPANCY as whether there was actually random allocation (Wu et al, 2006). Additionally, this could be seen as CULTURAL BIAS + IMPOSED ETIC, as the majority are in ONE AREA.

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16
Q

A03) Family therapy

A

Supports A01)
P) Family therapy has ECONOMIC BENEFITS.
E) It is associated with significant cost savings, when offered to people with sz, in addition to standard care. Therapy is expensive, but LESS EXPENSIVE than being constantly re-hospitalised, as there are LOWER RELAPSE rates associated with FT.
Further evidence –> relapse rates down for a significant period after completed intervention –> means that cost savings would be even higher.
E) This suggests that family therapy may be an APPROPRIATE TREATMENT for sz, from the perspective of the NHS.
L) Therefore, FT clearly has significant cost savings, which supports its effectiveness, as relapse rates are lowered.
Contrast point: Is FT used for therapeutic benefit or for financial benefit?

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17
Q

A03) Family therapy

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Supports A01)
P) An additional advantage of FT –> it has been shown to improve outcomes for the individual with sz, but it may also have a POSITIVE IMPACT on family members.
E) Lobban et al (2013) –> META ANALYSIS –> analysed the results of 50 family therapy studies that had included an intervention to support RELATIVES.
E) Findings –> 60% of these studies reported a significant positive impact of the intervention on at least ONE OUTCOME category for relatives. For example, coping and problem solving skills.
L) Therefore, FT has evidence to support positive impacts on the family, as well as the patient.

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18
Q

Cognitive Explanation

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Keywords:
Cognitive - Thinking processes
Dysfunctional thought processing - Cognitive beliefs that cause the individual to EVALUATE information INAPPROPRIATELY and produces UNDESIRABLE CONSEQUENCES.
Meta representations - DISRUPTS our ability to recognise our OWN ACTIONS and THOUGHTS as being carried out by OURSELVES, rather than SOMEONE ELSE.
Central control - Sufferers with sz tend to experience DERAILMENT of thoughts and spoken sentences, because each word triggers ASSOCIATIONS and the patient CANNOT SUPPRESS AUTOMATIC RESPONSES.

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19
Q

Dysfunctional thought processing

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Cognitive explanations emphasise the role of dysfunctional thought processing, especially in those who display the POSITIVE SYMPTOMS of sz, such as delusions and hallucinations.
Cognitive explanations of hallucinations:
- Hallucinating individuals focus EXCESSIVE ATTENTION on AUDITORY stimuli (hypervigilance) and so have a HIGHER EXPECTANCY for the occurrence of a VOICE than normal individuals.
Baker + Morrison, 1998 - Hallucinating patients with sz are significantly more likely to MISATTRIBUTE the source of SELF-GENERATED AUDITORY EXPERIENCE to an EXTERNAL SOURCE, than are NON-HALLUCINATING patients with schizophrenia.
Simon et al, 2015 - REDUCED processing of info in the TEMPORAL and CINGULATE gyri is associated with hallucinations. Additionally, REDUCED THOUGHT PROCESSING in the VENTRAL STRIATUM is associated with NEGATIVE SYMPTOMS.

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20
Q

Frith’s Model (1992)

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Proposes that Sz sufferers are NOT ABLE TO DISTINGUISH between actions influenced by EXTERNAL FORCES and those driven by INTERNAL INTENTIONS. He identified 2 types of dysfunctional thought processing, that could underlie symptoms.

Metarepresentation - The cognitive ability to reflect on thoughts and behaviour. Allows us INSIGHT into our OWN INTENTIONS and GOALS, as well as allowing us to INTERPRET the actions of others.
Dysfunction in metarepresentation would DISRUPT our ability to recognise our own actions + thoughts as being carried out by OURSELVES rather than SOMEONE ELSE. This would explain hallucinations of voices and delusions like THOUGHT INSERTION.

Central control - The cognitive ability to SUPPRESS AUTOMATIC RESPONSES while we perform DELIBERATE ACTIONS instead. If someone has the inability to suppress automatic thoughts + speech triggered by other thoughts, this could lead to DISORGANISED SPEECH and THOUGHT DISORDER.
For example, sz sufferers tend to experience DERAILMENT of thoughts and spoken sentences, because each word triggers ASSOCIATIONS and the patient cannot suppress automatic responses to these. Can result in speech poverty + word salad.
Example: Leads to them losing their train of thought and not being able to focus on others.

21
Q

Hemsley (1993,2005)

A

Sz involves a breakdown in the relationship between memory and perception.
People with sz have a DISCONNECT between their SCHEMAS and what they actually SEE + HEAR.
When they encounter new situations, their schemas are NOT ACTIVATED.
Therefore, people with sz experience SENSORY OVERLOAD, because they don’t know what to attend to.

22
Q

A03) Cognitive explanation

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Supports A01:
P) Supporting evidence –> For the relationship between central control dysfunction and schizophrenic symptoms.
E) Stirling et al (2006) –> Compared 30 patients with a diagnosis of Sz, with 18 non patient controls, on a range of cognitive tasks eg. the STROOP TEST.
Patients took over TWICE as LONG to name the ink colours as the control group.
C) However, even if the link between faulty cognitions and symptoms is clear, this does not tell us about the ORIGINS of those COGNITIONS and thus the ORIGINS of SCHIZOPHRENIA.
C) This supporting evidence backs up Frith’s suggestion of a relationship between central control dysfunction and sz symptoms. However, the origins are still unclear, lowering the validity.

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Q

A03) Cognitive explanation

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Supports A01/ RWA:
P) Strength of CBTp –> It has been demonstrated to be effective in the NICE review of treatments for schizophrenia (2014).
E) When compared to anti psychotic medication, CBTp was MORE EFFECTIVE in REDUCING SYMPTOMS SEVERITY and IMPROVING LEVELS of SOCIAL FUNCTIONING.
C) Counter criticism –> CBTp requires more motivation and takes longer.
C) This supports the idea that the origin of sz may be in faulty cognitions, as changing these cognitions has evidence to support it reducing the symptoms of schizophrenia.

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Q

Psychological treatments: CBTp

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Cognitive Behavioural Therapy is the MAIN psychological treatment for schizophrenia. However, antipsychotics are often used ALONGSIDE CBT to reduce PSYCHOTIC thought processes, ensuring CBT is most effective and to reduce patient dropout rate (Kuipers et al, 1997).

Aims of the therapy:
- To help the patient to identify delusions. To challenge and modify delusory beliefs (make irrational thoughts rational) also by looking at evidence.
- To help the patient begin to test the REALITY of the evidence.
- Recognise negative thoughts.
- Delusions can also be CHALLENGED, so that a patient can come to learn that their beliefs are not based on reality.
- Set BEHAVIOURAL assignments to improve general levels of functioning eg. educating yourself on your delusions.

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CBTp
NICE recommend at least 16 sessions of CBTp!!! Criticism: Cost and motivation Procedure: Assessment - Assessing WHAT is wrong with them and HELPING them find what the CAUSE is. Engagement - ATTENDING sessions, therapist's job to keep them ENGAGED. The ABC model - Involves IDENTIFYING activating events and the resulting beliefs, identify EMOTIONAL and BEHAVIOURAL consequences from the event. These beliefs can then be DISPUTED and CHANGED through CCA, leading to the effect of RESTRUCTURED BELIEFS. Normalisation - Therapist shares with the patient that many people have UNUSUAL experiences, such as hallucinations and delusions. This REDUCES ANXIETY, by making the patient feel LESS STIGMATISED. Critical collaborative analysis - Therapist uses GENTLE questioning to help the patient to UNDERSTAND and CHALLENGE their ILLOGICAL CONCLUSIONS eg. "if your voices are real, why can no one else hear them?" Developing alternative explanations - Rather than getting rid of Sz, CBTp helps patients COPE BETTER with their symptoms, because it REDUCES DISTRESS.
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ABCDE model
Activation event Beliefs Consequences Dispute the beliefs Effective new beliefs
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A case example - Turkington et al, 2004
Patient: The Mafia are observing me to decide how to kill me. Therapist: You are obviously very frightened ... there must be a good reason for this. (Normalisation) Patient: Do you think it's the Mafia? Therapist: It is a possibility, but there could be other explanations. (alternative explanations) How do you know that it's the Mafia? (Disputing) Why don't you do some research about the Mafia and their past? (Behavioural assignment)
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A03) CBTp
Supports A01) P) Evidence to support the benefits of CBTp for the treatment of schizophrenia. E) The NICE review of treatments for sz (2014) found CONSISTENT evidence that when compared with standard care (AP's alone), CBTp was EFFECTIVE in reducing rehospitalisation rates up to 18 months, following the end of treatment. It was also shown effective in reducing symptom severity and improving social functioning. C) However, most studies have been conducted with patients who were treated with antipsychotics at the same time. Therefore, it's difficult to assess the effectiveness of CBTp SOLELY. C) Overall, there seems to be modest support for the effectiveness of CBTp and this support suggests that it may be more effective LONG TERM than drug therapy, as it provides COPING STRATEGIES.
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A03) CBTp
Weakness of RM) P) Weakness --> CBTp requires motivation which patients may not have. E) Length of the therapy --> Means that patients have to be MOTIVATED and COMMITTED to multiple sessions. It also requires AWARENESS and a WILLINGNESS to engage with the process. E) However, the positive symptoms lead to a lack of awareness and the negative symptoms lead to a reluctance/inability to engage. The length of the therapy also leads to patients dropping out at times of severe episodes. L) Overall, this suggests that CBTp may not be an appropriate treatment for all patients with sz.
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A03) CBTp
Supports Lang - AP's are "chemical straitjackets" Fernando - Institutionalised racism
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A03) CBTp
Weakness of RM) P) Weakness --> Only aims to make sz more MANAGEABLE and improve the patients' quality of life. E) Example --> Allows patients to make sense of and challenge some of their symptoms through CCA. This does NOT CURE schizophrenia. Therefore, whilst CBTp may provide patients with coping strategies, it doesn't effectively cure sz. E) Additionally --> There are also ethical issues. CBTp may involve challenging a person's paranoia. At what point does this impact an individual's freedom of thought? L) Weakness --> Challenging their beliefs may become unethical and CBTp doesn't cure the sz.
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A03) CBTp
Challenges A01) P) A challenge of CBTp is that it may be more effective at CERTAIN STAGES of the disorder. E) Addington + Addington (2005) --> Claim that self- reflection is not particularly appropriate during the initial ACUTE phase of sz. Following stabilisation of the psychotic symptoms with AP's, individuals may THEN benefit more from group based CBTp. This can help to normalise their experience by meeting others with similar issues. C) Additionally, research has found that it is those with more experience of their sz and a greater realisation of their symptoms that benefit more from individual CBTp. C) Therefore, CBTp is effective, but may be more appropriate in the LATTER STAGES of the treatment, rather than INITIALLY.
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Psychological treatments: Token Economy
Keywords: Operant Conditioning - Using + and - reinforcement to encourage desirable behaviour. Token economy - A form of behavioural therapy used in the management of schizophrenia. It is used to shape and manage behaviour, so patients in long stay hospitals are easier to manage. Widely used in the 60's and 70's, although nowadays this treatment has been replaced with other forms of interventions, such as social + life skills training. Rewards (tokens) - Given as secondary reinforcers immediately, when patients engage in desirable behaviours eg. getting dressed in the morning. Selective - Reinforcing desirable behaviours that aim to be REPLICATED eg. getting dressed. Primary - Immediate reward eg. a walk Secondary - Not a direct reward, can be EXCHANGED for a direct reward eg. tokens.
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Token Economy
- Negative symptoms --> apathy + social withdrawal --> these individuals typically lack interest in those aspects of a normal, healthy life eg. washing + eating. --> the use of a token economy system, was developed as a way of dealing with these negative symptoms and encouraging more + behaviours. eg. speech poverty --> if they speak 5 sentences a day --> token --> 5 minute walk. How does it work? - The principles of operant conditioning describe the relationship between a behaviour and environmental events. Positive reinforcement is the increase of a desirable behaviour, when it is followed by a desirable event. - Tokens --> Given immediately to patients when they have carried out a desirable behaviour that has been targeted for reinforcement. This IMMEDIACY of reward is important, because it PREVENTS DELAY DISCOUNTING. - Rewards --> Tokens can be exchanged later for more TANGIBLE rewards. Tokens are secondary reinforcers and originally neutral, because they only have value once the patient has learned that they can be used to obtain rewards. These rewards are primary reinforcers and might be in the form of materials such as sweets or magazines or walks.
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Process of token economy
Assigning value --> To give the neutral token value, it needs to first be REPEATEDLY presented alongside or immediately before the reinforcing stimulus eg. a walk. By pairing the neutral tokens with the reinforcing stimulus, the token eventually acquires the same reinforcing properties. Reinforcing target behaviours --> The more items or rewards the token can be exchanged for, the more powerful the token becomes. Sran + Borrero (2010) --> Compared behaviours reinforced by tokens that could be exchanged for one single highly preferred edible item with tokens that could be exchanged for a variety of preferred edible items. Findings: All participants had HIGHER RATES of responding in those sessions where tokens could be exchanged for a VARIETY of items. The "trade" --> An important part of token economy is the exchange of tokens for backup rewards, chosen by the clinician eg. sweets, watch a movie. During the early stages, FREQUENT exchange periods mean that patients can be QUICKLY REINFORCED and TARGET BEHAVIOURS can then increase in frequency. Kazdin (1977) --> The effectiveness of the token economy may DECREASE if MORE TIME passes between presentation of the token and exchange for the backup reinforcers.
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Real World Example
Ayllon and Azrin (1968) --> - Used a token economy on a ward of female sz patients. - Given plastic tokens, each embossed with the words "one gift" for behaviours such as making their bed. - These tokens were then exchanged for privileges eg. being able to watch a movie. - The researchers found that the use of a token economy with these patients INCREASED the number of desirable behaviours every day. - Modifying these habits DOES NOT CURE sz, but it improves the patient's QUALITY OF LIFE and makes it MORE LIKELY they can live OUTSIDE a hospital.
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A03: Token Economies
Weakness of RM) P) Weakness of token economies --> Only aim to make sz more manageable and improve the patients quality of life. E) They help by making patient's behaviour more SOCIALLY ACCEPTABLE, so that they can better REINTEGRATE into society. C) However, while this is worth doing, it should not be confused with curing schizophrenia. TE's do not address symptoms of sz, so they are not technically a treatment. C) Therefore, whilst token economies may provide patients with coping strategies to manage their current symptoms, they DO NOT cure schizophrenia.
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A03: Token Economies
Weakness of RM) P) Weakness of token economy --> Raises ethical issues. E) Example --> In order to make reinforcement effective --> clinicians may exercise control over important primary reinforcers eg. food, privacy and hobbies to alleviate boredom. C) However, it is generally accepted that all human beings should have basic rights to things like food and privacy. These should not be violated regardless of the positive consequences that might be achieved by manipulating them. Additionally --> primary reinforces becomes MORE available to patients with MILD symptoms than those with SEVERE symptoms that prevent them from complying with desirable behaviours. C) Therefore, the most severely ill patients suffer discrimination and some families have challenged the legality of this.
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A03: Token Economies
Supports A01) P) Supporting evidence --> Dickerson et al (2005) reviewed 13 studies of the use of TE systems in the treatment of schizophrenia. E) Findings: 11 of these studies --> beneficial effects that were directly attributable to the use of TE's. This supports the effectiveness of TE's in increasing the adaptive behaviours of those with schizophrenia. C) However, they did caution that many of the studies they reviewed had significant METHODOLOGICAL SHORTCOMINGS that limited their impact in the overall assessment of TE's. C) Yet, Dickerson's findings still support the effectiveness of TE's, but do have some methodological issues to consider.
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A03: Token Economies
Weakness of RM) P) A weakness of token economies is that they have only really been shown to work in a HOSPITAL setting. There are problems administering the method with outpatients who live in the community. E) Within a psychiatric ward setting, inpatients receive 24 hour care and so there is better control for staff to monitor and reward patients. In the community, outpatients only receive day treatment for a few hours, so the method can only be used for part of the day. Therefore, even if TE did produce positive results within the ward setting, this may mot be MAINTAINED beyond that environment, limiting the long term effectiveness of TE as a method to manage schizophrenia. C) Another major problem --> Assessing the effectiveness of token economies --> Studies tend to be uncontrolled. When a token economy system is introduced into a psychiatric ward, typically all patients are put onto the programme, rather than having a control group that does not. This means that patients improvements can only be compared with their past behaviours, rather than a CG. This may be misleading, as other factors eg. an increase in staff attention could be causing the improvement, rather than the TE. C) Therefore, the research evidence can't provide as strong support for the effectiveness of TE's and lack ecological validity.
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The Interactionist Approach (biosocial)
Keywords: Diathesis - Genetic vulnerability Genetic predisposition - Genes making you more vulnerable to sz. Environmental factors - Anything within the environment that makes you more susceptible to sz. Relapse - An individual who suffers from that illness again (once cured). The interactionist approach looks at the COMBINATIONS of DIFFERENT THERAPIES as the best form of treatment. So, it uses both therapy (psychological) and drugs (biological). This is a combination of CBTp and AP's. Explanation: Genetic predisposition and environment = schizophrenia Diathesis + Stress = Illness - An interaction between biological and environmental influences causing SZ.
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Diathesis
- Schizophrenia --> A genetic component in terms of vulnerability. - Tienari et al 2004 --> Supporting the genetic role for sz, are the findings that the identical twin of a person with the disorder is at GREATER RISK of developing it than a sibling or fraternal twin. Adoptive relatives do not share the increased risk of biological relatives. - However, in about 50% of identical twins in which one twin is diagnosed with schizophrenia, the other NEVER MEETS THE DIAGNOSTIC CRITERIA. This dis concordance among identical twins indicates that ENVIRONMENTAL FACTORS must also play a role in determining whether a biological vulnerability actually develops into the disorder.
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Stress
- Stressful life events can trigger schizophrenia. - These take various forms such as childhood trauma, stress of living in a highly urbanised environment. - Varese et al (2012) --> Found that children who experienced severe trauma before the age of 16 were THREE TIMES as likely to develop sz in later life, compared to the general population. There was a relationship between the level of trauma and the likelihood of developing schizophrenia. - Severely traumatised = greater risk - Vassos et al (2012) meta analysis --> Found that the risk of sz in the most urban environments was estimated to be 2.37 times higher than in the most rural environments. - Higher level of urbanisation = increased risk of developing a range of different psychoses eg. sz. - Possible that the more ADVERSE living conditions of densely populated urban areas may be a factor. - Many people live in densely populate urban areas, but only a tiny minority will develop schizophrenia. Therefore, a genetic disposition may exist for sz.
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Additive nature of diathesis and stress
- A combination of diathesis and stress can lead to the onset of schizophrenia. For example, relatively minor stressors may lead to the onset of the disorder for a highly vulnerable individual. Or, a major stressful event may cause a similar reaction of a person with low vulnerability. - Additivity - Diathesis + Stress add together to produce the disorder.
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Diathesis Stress Models
Meehl's model (1960) - Proposed the OG D-S model - Believed diathesis was entirely genetic, the result of a single schizogene. - Leads to schizotypic personality - If a person doesn't have the schizogene, then no amount of stress would lead to it. Modern day diathesis - Many genes increase genetic vulnerability. - There is no single schizogene. - Include a range of factors beyond the genetic, including psychological trauma. - Read (2001) - Early trauma alters the developing brain. For example, HPA becomes over active, the person becomes more vulnerable to later stress. Toxoplasmosis - In the womb, if Mum has illness/trauma, this can impact neurotypical development and has lead to sz before.
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A03) Interactionist approach
Supports A01) P) Support for the usefulness of adopting an interactionist approach from studies, who compared the effectiveness of combinations of bio and psycho treatments for sz, versus bio treatments alone. E) Tarrier et al (2004) --> Randomly allocated 315 patients to a medication + CBT, medication group and supportive counselling group or a control group (medication only). E) Findings --> Patients in the two combination groups showed lower symptoms levels than those in the control group. Contrast point: There was no difference in rates of hospital readmission. C) Suggests that there is a CLEAR PRACTICAL advantage to adopting an interactionist approach in the form of superior treatment outcomes. This highlights the importance of taking this approach.
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A03) Interactionist approach
Weakness of RM) P) A weakness of the older diathesis - stress model is that it may be overly simplified, as diathesis may not be exclusively genetic and stress may involve anything that risks triggering schizophrenia. E) The increased risk for sz can also result from brain damage, caused by environmental factors. For example --> Verdoux et al (1998) --> estimated that the risk of developing schizophrenia later in life was FOUR TIMES GREATER for those who had experienced prolonged labour and oxygen deprivation, than those who hadn't experienced such complications. E) Additionally, cannabis may be a stressor, as it increases the risk of sz by up to SEVEN TIMES, depending on the dose. This is probably because it interferes with the dopamine system. Although, most people do not develop sz after smoking cannabis, which suggests that there must be one or more triggering stressful factors. This means that from the model it is difficult to predict exactly WHO will develop schizophrenia. Both suggest that the old idea of diathesis-stress may be overly simple. L) However, it still supports that an interactionist approach to sz is important, in that vulnerabilities and triggers seem to ADD TOGETHER in some way, to produce schizophrenia.
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A03) Interactionist approach
Weakness of RM) P) A weakness of this approach is that it's difficult to determine the stress that triggered sz, which may lower the effectiveness of treatment. E) Example --> It is possible that stressors earlier in life could influence how people respond to later stressful events, and increase their susceptibility to the disorder. E) Maladaptive methods of coping with stress in childhood means that the individual fails to develop effective coping skills, which in turn compromises their resilience and increases vulnerability. This may make life more stressful for the individual and thus triggers schizophrenia. L) Therefore, it is extremely difficult to determine the CAUSAL STRESS that triggered schizophrenia, which may negatively impact the effectiveness of treatment.
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A03) Interactionist approach
Supports A01) P) There is supporting evidence for the dual role of genetic vulnerability and stress in the development of schizophrenia. E) Tienari et al (2004) --> studied 145 children who had at least one biological parent with schizophrenia and were adopted into a new family and compared them with 158 adoptees without this genetic risk. The adopted parents were assessed for child-rearing style and the rates of schizophrenia across the two groups was compared. E) Findings: A child rearing style characterised by high levels of criticism, high levels of conflict and low levels of empathy were implicated in the development of schizophrenia, but ONLY for those in the HIGH GENETIC RISK group. High genetic risk adoptees reared in families with low scores on the scale were significantly less likely to have developed schizophrenia. This suggests that BOTH genetic vulnerability and family-related stress are important in the development of sz, as genetically vulnerable children are more sensitive to parenting behaviour. L) This is STRONG DIRECT SUPPORT for the importance of adopting an interactionist approach to schizophrenia, including that poor parenting is a possible source of stress.