Psychiatric emergencies Flashcards

1
Q

How are psychiatric emergencies classified?

A
major, minor and emergenices related to medical causes Around 30% of emergency patients are suicidal, 10% are violent, 40% require hospital admission
Major:
Suicidal 
Agitated and violent 
Minor:
Grief reaction 
Rape
Disaster
Panic attack
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2
Q

What are common medical emergencies?

A
Delirium
Neuroleptic malignant syndrome
Serotonin syndrome
OD of common psychiatric medications
OD and withdrawal from addicting substance
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3
Q

Whta is delirium?

A

Transient, potentially reversible cerebral dysfunction with acute or subacute onset that manifest with fluctuating mental state abnormalities
Global cognitive impairment associated with behavioural abnormalities
10-30%
10-15% elderly people admitted to hospital, another 10-40% develop it during their stay
60% of delirium is superimposed on dementia
Due to alterations in Ch or NA neurotransmitter systems, or interruption of the BBB

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4
Q

What is the ICD-10 criteria for delirium?

A

Clouding of consciousness- reduced awareness of environment, reduced focus, shifting attention
Disturbed cognition- Impaired immediate recall and recent memory disorientation in time, place or person
Variable anxiety levels, increased reaction time, altered speech flow, enhanced starlet reaction
Insomnia, daytime drowsiness, reversed sleep-wake cycle, disturbing dreams/nightmares

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5
Q

What are the types of delirium?

A
Hyperactive- heightened arousal, restless, agitated aggressive 
Hypoactive- withdrawn, quiet, sleepy 
Mixed
Delirium superimposed on dementia 
persistent delirium
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6
Q

What are raw causes of delirium?

A

INFECTION - HIV, Sepsis, pneumonia
WITHDRAWAL - alcohol, barbiturates, sedatives
ACUTE METABOLIC - acidosis, alkalosis, electrolyte imbalance, hepatic failure, renal
failure
TRAUMA - head injury, heat stroke, post-op, severe burns
CNS PATHOLOGY - abscess, haemorrhage, SDH, stroke, seizures, tumours,
meningitis, encephalitis, syphilis
HYPOXIA - anaemia, CO poisoning, hypotension, CCF
DEFICIENCIES - B12, folate, niacin, thiamine
ENDOCRINE - hyper/hypoadrenocorticism, hyper/hypoglycaemia, hyperparathroid
ACUTE VASCULAR - hypertensive encephalopathy, stroke, arrhythmia, shock
TOXINS/DRUGS - illicit, prescription, solvents
HEAVY METALS - lead, managnese, mercury

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7
Q

What is the assessment for delirium?

A
History - patient, collateral and notes
Consider physical causes - infection, constipation, dehydration, medications, alcohol
withdrawal
Risk assessment
Complete a BASELINE MoCA
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8
Q

What are the investigations for delirium?

A
Infection - culture, urinalysis, FBC, CRP, CXR
Medication review
Metabolic/endocrine
Cardiac - ECG
Hypoxia - ABGs
Neuro - CT brain
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9
Q

What is the management for delirium?

A

Identify and treat precipitating cause
Provide a calm environment and supportive measures
Involve family and carer
May refer to psychiatric team
Avoid sedation unless severely agitated
Review patient regularly
Prognosis- average duration is 7 days, increased risk of mortality- increased by 75% in elderly population

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10
Q

What is acute behavioural disturbance?

A

Develop in patients with mental health illnesses at any time in the course of their illness, or as a manifestation of an underlying organic illness
Patients become agitated/aggressive during an acute episode of illness, eg mania/schizophrenia
- May be due to psychotic symptoms
- Due to non-psychotic symptoms
- Use of illicit drugs

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11
Q

What are the basic principles of acute behavioural disturbance?

A

Predictions of risk of agitation
Prevention of behaviour escalating once patient becomes disturbed
Use interventions to ensure safety of patients and staff

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12
Q

What are the warning signs for an increased risk of aggression?

A
Persecutory delusions
Delusions of passivity
Actual threats of violence
Emotional state linked to violence
Behaviour
Limited insight
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13
Q

What are the warning signs for violent behaviour?

A
Angry facial expression
Restless or pacing
Shouting
Prolonged eye contact
Refusal to cooperate
delusions/hallucinations with violent content
Verbal threats/thoughts of violence
Blocking escape routes
Evidence of arousal - SNS activation
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14
Q

How should physical restraint be carried out?

A

Only used when other measures have failed/inappropriate
One member of staff should be in charge and carry out:
Encourage patient to move to a separate room/area
Speak confidently, slowly and clearly
Adopt a non-threatening body posture and verbal communication
Pre-warn the patient
Explore concerns
Develop a rapport with patient
Request patient to put any weapons down

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15
Q

What should be considered when giving rapid tranquillisation?

A

Calm the patient without sedating them and reduce risk of violence and harm
Give orally where possible
Always consult a senior doctor
Use minimum dose of medications
Choose one with a rapid onset, short-acting, minimal side-effects and easily reversible effects

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16
Q

What are the choices of medications for rapid tranquillisation?

A

Benzodiazepines
Lorazepam has quick onset, short duration, safe and tolerable
Midazolam has 15 min onset after IM, 3-5 for IV
Diazepam has a longer half life and when given IM has an erratic and
slow absorption
○ Antipsychotics - haloperidol, olanzapine

17
Q

What are other considerations for rapid tranquillisation?

A

Ensure cardiopulmonary resuscitation facilities are available
Need to carry out physical observation after giving medication - pulse, BP, temp,
RR, oxygen sats
If given IM - maintain patients dignity

18
Q

What are the complications fo benzodiazepines?

A

Acute dystonia- severe painful muscle stiffness
Hypotension- orthostatic or <50 diastolic
Respiratory depression-
Arrhythmias- irregular pulse or pulse rate <50

19
Q

What is the management for the complications of benzodiazepines?

A

Acute dystonia:
Procyclidine 5-10mg IM/IV or benzotropine 1-2mg
Hypotension:
Lie patient flat and raise legs, monitor closely
Respiratory depression:
Give 02, raise legs, if RR <10 give flumazenil 200mg IV over 15 seconds
Arrhythmias:
Monitor closely
Liaise with medical team

20
Q

What are the risk factors for neuroleptic malignant syndrome?

A
Previous NMS
Known cerebral compromise
Alcoholism
Agitation, overactivity, catatonia
Dehydration
IM therapy
Recent/rapid antipsychotic dose increase
Rapid dose reduction/abrupt withdrawal of anticholinergic medication
High doses of antipsychotic medication
21
Q

What are the symptoms for neuroleptic malignant syndrome?

A
Variable presentation 
Fever
Diaphoresis
Rigidity
Confusion
Fluctuating consciousness
Autonomic instability
22
Q

What are the investigations for neuroleptic malignant syndrome?

A

CK is frequently raised >1000 units/L

Leucocytosis and deranged LFTs

23
Q

What is the treatment for neuroleptic malignant syndrome?

A
Withdraw antipsychotic
Monitor tempr, BP and pulse
Consider benzo for sedation
Rehydration
Dopamine agonist - Bromocriptine
24
Q

What is acute dystonia?

A

Reversible extrapyramidal side effect
Muscle spasms, immediate or delayed
Causes distress to patients and can be life-threatening if it includes laryngeal muscles
usually idiosyncratic and unpredictable
More common in high potency D2 receptor medications- e.g haloperidol
Occurs in around 10%- more common in young men

25
Q

What is the treatment for acute dystonia?

A

Dystonia usually responds well to anticholinergic medications, eg
procyclidine
- Can be taken IM or IV
- Check for cyanosis and give oxygen if required

26
Q

What is serotonin syndrome?

A

Rare, potentially life-threatening ondition occurring during initiation/increased dose of serotonergic medication
Caused by:
Increased serotonin synthesis/release
- Reduced serotonin uptake/metabolism
- Direct serotonin receptor activation
Occurs when patients switch antidepressants or when a combination are used
Occurs when taken alongside triptans, Wort, drugs

27
Q

What are the symptoms of serotonin syndrome?

A
Restlessness
Confusion
Agitation
Hyperthermia
GI upset
Tachycardia
Hypo/hypertension
Mydriasis
Myoclonus
Rigidity
Tremors
Hyperreflexia
28
Q

What is the treatment for serotonin syndrome?

A

Stop medication
Rehydration
Benzo for agitation
If OD - gastric lavage

29
Q

What are the differences between neuroleptic malignant syndrome and serotonin syndrome?

A
NMS:
Onset slow (days to weeks)
Progression slow (24-72 hours)
Muscle rigidity severe (lead pipe)
Bradykinesia 
Serotonin syndrome:
Onset rapid
Progression rapid 
Less severe muscle rigidity
Hyperkinesia