Psych board review

Question 1

Modafinil mechanism of action

Answer 1

on histamine neurons in the reticular activating system in the pons.

Question 2

Metachromatic leukodystrophy

Answer 2

Arylsulfatase A

recessive

Question 3

Tay Sachs

Answer 3

Hexosaminidase A

Question 4

Cravings associated with SAD

Answer 4

Carbohydrate cravings

Question 5

Diagnosis of SAD

Answer 5

depressive symptoms occurring at a certain time of the year, 2 consecutive years, with full remission in between episodes

Question 6

Treatment of SAD

Answer 6

light therapy

Question 7

Biogen amines (6)

Answer 7

Epinephrine, NE, serotonin, histamine, dopamine, Acetylcholine

Question 8

Catecholamines

Answer 8

NE, epinephrine and dopamine

derived from tyrosine

Question 9

Lesch Nyhan

Answer 9

Hypoxantine-guanine-phosphoribosyl transferase

x linked recessive

Question 10

OSA diagnosis in adults

Answer 10

5 apneic episodes or hypopneas per hour of sleep, must last for 10 seconds or longer, accompanied by one other sxs OR 15 or more apneas / hypopneas per hour of sleep regardless of accompanying symtoms

Question 11

What makes an atypical antipsychotic atypical

Answer 11

5HT2A antagonism

Question 12

Artery most commonly affected in epidural hematoma

Answer 12

middle meningeal artery

Question 13

Artery most commonly affected in subdural hematoma

Answer 13

meningeal bridging veins, most common in alcoholics

Question 14

CSF findings in aseptic (viral) meningitis

Answer 14

Slightly increased lymphocytes, normal glucose, mildly elevated protein

Question 15

CSF findings in bacterial meningitis

Answer 15

Highly increased neutrophils, decreased glucose, very high protein

Question 16

CSF findings in TB meningitis

Answer 16

mild lymphocytic pleocytosis, mildly decreased glucose, and markedly increased protein

Question 17

CSF findings in fungal meningitis

Answer 17

moderate lymphocytic pleocytosis, mildly decreased glucose, and mildly increased protein

Question 18

CSF findings in Guillain Barre

Answer 18

Absent or few lymphocytes, normal glucose, very high protein

Question 19

Organophosphate poisoning mechanism

Answer 19

Cholinergic toxicity

Question 20

Ganser syndrome

Answer 20

not listed in DSM-V. voluntary production of symptoms that involve giving approximate answers or talking past the point.

Question 21

Mechanism of action of clonidine

Answer 21

presynaptic alpha 2 agonist, decreases norepinephrine release

Question 22

LAte onset schizophrenia age

Answer 22

Spiegel:45

DSM-V :40y/o

Question 23

infarcts of the internal capsule

Answer 23

result in a pure contralateral motor hemiparesis

Question 24

Anterior cerebral artery ischemia

Answer 24

contralateral hemiparesis of the leg preferentially

Question 25

Middle cerebral artery territory ischemia

Answer 25

dominant hemisphere: aphasia

Nondominan: hemineglect, anosognosia, visual and tactile extinction, aprosody of speech, and contralateral limb apraxia.

Question 26

Vertebrobasilar territory

Answer 26

ischemia would be expected to affect the cerebellum and/or brain stem. Classic posterior circulation ischemic symptoms include ataxia, nystagmus, vertigo, dysarthria, and dysphagia. An ipsilateral Horner’s syndrome can occur if the descending oculosympathetic fibers are disrupted. Crossed weakness (ipsilateral facial paresis, contralateral limb paresis) is indicative of brain-stem involvement above the level of the area of decussation of the pyramids in the medulla

Question 27

Tests most helpful to evaluate right hemisphere disease

Answer 27

Line orientation test