PSY311 Midterm 3 Flashcards

1
Q

What was personality disorder before DSM-5?

A

There was classification (like most common mental health disorder, medical issues) and they were not well investigated

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2
Q
  1. What is the definition of personality?

2. When does a disorder occurs?

A

1.Characteristics that describe how a person behaves and thinks.
2. Interferes with a person’s relationships and
Causes distress or impairment in daily life activities

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3
Q

What is a personality disorder?

A

A persistent pattern of emotions, cognitions, and behaviors that results in enduring emotional distress for the person affected and/or others and may cause difficulties with work or relationships
-> This is a global definition, there is more than 1 personality disorder (there are 10 in total)

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4
Q

What is the chronic course of a personality disorder?

A

Stems from childhood and continues on (difficult to treat something chronic ). also it does not mean they are diagnosis at childhood (very rare because you do not know if it is persistent or not) - the signs can be there. A major hint is childhood environment.

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5
Q

Personality disorder is comorbid with…?

A

depression (very important) and anxiety. This is why this is difficult to see betond that, that there is a pathology.

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6
Q

Name all the personality disorder of cluster A ‘‘odd cluster’’ eccentric cluser’’
→ social awkwardness, distorted thinking

A

paranoid personality disorder
schizoid personality disorder
schizotypal personality disorder

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7
Q

Name the key terms of paranoid personality disorder

A

Unjustified distrust
Suspicious unfounded
Can be argumentative and sensitive to criticism
Can be short-tempered: able to attack someone if needed

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8
Q

what is the etiology of paranoid personality disorder? they are common…

  1. Biological contribution
  2. Psychological contribution
  3. Sociocultural contribution
A

Biological contribution
Weak genetic contribution

Psychological contribution
Weak-moderate contribution
Early traumatic childhood experiences → distorted schemas that develop in childhood when ask about it

Sociocultural contribution
Prisoners, refugees, people with hearing impairments more likely to develop this disorder
We don’t really know why
little research about it, not much

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9
Q

What is the treatment for paranoid personality disorder?

A

Unlikely to seek professional help
Difficulty in developing a trusting relationship with the therapist
Usually present to clinic following a crisis or for other disorders
Cognitive therapy
Change distorted schemas “everyone is untrustworthy”
No documented effectiveness

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10
Q

Name the key terms of schizoid personality disorder

A

“Loner”
Detached and lack emotional expression
Do not appear affected by praise or criticism
“Observers” - they are not team work player, prefer to observe then participate
Do not have unusual thought processes: not like paranoid personality

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11
Q

Schizoid personality disorder rtiology: 2 key points different from other personality disorder

A
  • connexion with Autism spectrum disorder (little evidence)

- Low dopamine receptors (when underdeveloped)~ detachment: low reward, low motivation

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12
Q

What is the treatment for schizoid personality disorder?

A
Rarely seek treatment
Psychotherapy
Increase value of social relationships
Social skills training
No documented effectiveness
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13
Q

Schizotypal personality disorder is possibly on a continuum with…

A

Schizophrenia BUT in less severe form.

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14
Q

Schizotypal personality disorder: etiology

Breakdown biological contribution

A

Weak genetic contribution
Connection with Schizophrenia
E.g. Catechol-O-methyltransferase (COMT) → enzyme involved in breakdown of various NT such as dopamine, norepinephrine. alteration in the COMT genes.
the brain region that runs on dopamine seems to be overactive in limbic region, that is why most of the time, when you think about schizophrenia, the major treatment is an antipsychotic . but not overactive in the frontal regions, not enough dopamine - not much regulation…
Brain alterations
E.g. deep in temporal lobe (volume is reduce) → Odd speech, odd beliefs

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15
Q

What are the treatments for Schizotypal personality disorder.

A

30-50% of individuals seeking help for other symptoms
Psychotherapy:
Social skills training: Modestly effective
Medication
Antipsychotics
Not always effective- more effective for schizophrenia
High drop outs
Negative side effects
Huge difficulties with medication compliance

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16
Q

What are the personality disorder in cluster B which is characteristic ‘‘dramatic, emotional thinking and behavior, impredictable’’

A

Antisocial personality
Borderline Disorder
Histrionic Personality
Narcissistic Disorder

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17
Q

What are some criteria of antisocial personality disorder?

A

A pervasive pattern of disregard for and violation of the rights of others, occurring since age 15 years, as indicated by ≥3 of the following:
- Failure to conform to social norms with respect to lawful behaviors -Deceitfulness, - Impulsivity or failure to plan ahead - Irritability and aggressiveness -Lack of remorse,

The individual is at least age 18 years.- adulthood
There is evidence of conduct disorder with onset before age 15 years.
The occurrence of antisocial behavior is not exclusively during the course of schizophrenia or bipolar disorder.

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18
Q

What is a conduct disorder? what does it precedes?

A

It precedes antisocial personality disorder. It develops in childhood.

  • agression to people and animals
  • destruction of property
  • deceitfulness or theft
  • serious violations of rules
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19
Q

what is psychopathy subset?

Stats among prisionner

A

Psychopathy: subset of individuals with antisocial personality disorder with all of the following additional characteristics

Glibness -smooth talk, work people-, superficial charm
Grandiose sense of self-worth
Proneness to boredom, need for stimulation
Pathological lying
Conning, manipulative
Lack of remorse
80% of prisonnier are diagnosed with antisocial disorder but out of the 80%, only 20% display are the characteristics of psychopathy

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20
Q

what is the etiology of antisocial personality?

  • genetic influences
  • Gene x environment
  • Neurobiological influences
  • Neuropsychological influences
  • Psychological influences
  • Social influences
A

Moderate genetic influences (32% higher than other disorder)
Family, twin, adoption studies support a genetic link
↑ ASPD rate in adopted children of biological mothers with criminal history

Gene x environment
↑ ASPD rate in adopted children of biological mothers with criminal history x who spent more time in an orphanage (a year or two there)

Neurobiological influences
Underarousal hypothesis (not enough)
Low levels of arousal - to get to the proper level, they need to have more stimulation.
Need more stimulation 🡪 sensation-seeking and risk-taking (+ lack of long term planning so just jumping into stuff, not much restrain, inhibition)

Fearlessness hypothesis
Higher threshold for experiencing fear (it takes them much more to feel fear). Inability to learn and respond from negative consequences. No remorse, no empathy

Neuropsychological influences
BIS-BAS disbalance
Weak Behavioral Inhibition System vs Overactive Behavioral Activation System. ↓ prominent anxiety and ↓behavioral inhibition (impulsivity)

Psychological influences
Do not let go of goals
Emotionally distant and mistrustful ~ ↑ violent crime

Social influences
Parental attitudes early in life (psychodynamic theory)
Low parental involvement
Inconsistent discipline
Stress
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21
Q

what are the treatment about antisocial personality disorder?

A

Few documented treatments
CBT
Violent acts reduced 5 years later, except for those who are particularly “selfish, remorseless”
Psychotherapy in childhood
Parent training
Children less responsive to treatment if high stress and family dysfunction

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22
Q

what are Borderline personality disorder?

A

A pervasive pattern of instability of interpersonal relationships, self-image, and affects, and marked impulsivity, beginning by early adulthood and present in a variety of contexts
- Frantic efforts to avoid real or imagined abandonment - A pattern of unstable and intense interpersonal relationships - Identity disturbance - Recurrent suicidal behavior - Chronic feelings of emptiness
More common than other personality disorders
Worldwide lifetime prevalence: ~2%

More common in women
Prognosis better than other personality disorders
88% remission after 10 years of therapy
Core: impulsivity, emotional instability
High comorbidity with mood disorders
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23
Q

Bordeline personality disorder have high comorbidity with…?

A

mood disorder

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24
Q

what is the etiology about antisocial personality disorder?

Biological influences
Psychological influences
Social influences

A

Biological influences
Moderate genetic influences
Possible shared link with mood disorders

Psychological influences
Memory bias

Social influences
Early trauma (more common in women)
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25
Q

what are the treatment for antisocial personality disorder?

A

Few studies on treatment efficacy
Complicated by comorbidity

Medication
Lithium
Antidepressants
Antipsychotics

Dialectical Behavior Therapy (DBT)
Identify and regulate emotions
Cope with stressors that trigger suicidal ideation

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26
Q

Name some of the histrionic personality disorder: criteria

A

A pervasive pattern of excessive emotionality and attention seeking, beginning by early adulthood

  • Is uncomfortable in situations in which he or she is not the center of attention
  • Interaction with others is often characterized by inappropriate sexually seductive or provocative behavior.
  • Displays rapidly shifting and shallow expression of emotions.
  • Consistently uses physical appearance to draw attention to self.
  • Has a style of speech that is excessively impressionistic and lacking in detail.
  • Shows self-dramatization, theatricality, and exaggerated expression of emotion.
  • Is suggestible.
  • Considers relationships to be more intimate than they actually are
  • More common in women: Uterus used to be blamed
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27
Q

Histrionic personality disorder: treatment

A

Few studies on treatment efficacy

Psychotherapy:
Try to minimize attention-getting behaviors
Try to improve interpersonal relationships

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28
Q

Name some of the narcissistic personality disorder: criteria

A

A pervasive pattern of grandiosity, need for admiration, and lack of empathy, beginning by early adulthood and present in a variety of contexts, as indicated by ≥5 of the following:

  1. Has a grandiose sense of self-importance.
  2. Is preoccupied with fantasies of unlimited success, power, brilliance, beauty, or ideal love.
  3. Believes that he or she is “special” and unique and can only be understood by, or should associate with, other special or high-status people or institutions.

4.Requires excessive admiration.
5.Has a sense of entitlement.
6.Is interpersonally exploitative.
7.Lacks empathy: is unwilling to recognize or identify with the
feelings and needs of others.
8.Is often envious of others or believes that others are envious of him or her.
9.Shows arrogant, haughty behaviors or attitudes.

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29
Q

Narcissistic personality disorder: etiology

A
Weak genetic influences
Psychological influences
     Failure of empathetic mirroring
     Self-centered
     Stemming early in life
Social influences
    Parenting (overindulgent or over-controlling) attitudes
   Societal pressure on individualism, competitiveness, and success
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30
Q

Narcissistic personality disorder: treatment

A

Psychotherapy

Address grandiosity, sensitivity to evaluation, and lack of empathy
Remove fantasies
Treat and prevent depression

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31
Q

How can you describe the avoidant personality disorder?

A

anxious, fearful cluster.

Inhibition, feelings of inadequacy, and hypersensitivity to negative evaluation, beginning by early adulthood

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32
Q

What is the etiology for avoidant personality disorder?

A

Biological influences: Moderate genetic influences &
behavioral inhibition temperament

Psychological influences: Behavioral inhibition temperament & low self-esteem

Social influences: Parenting (rejecting, neglecting, unaffectionate) attitudes → no attachment

Connection with social anxiety disorder?

  • Similar characteristics, shared traits
  • Ego platonic: seems normal…so it is normal that -people judge you - avoidant personality.
  • It distress, know it is not normal - anxiety disorder:
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33
Q

What are the treatment for avoidant personality disorder?

A

Psychotherapy

  • Address thoughts of rejection through exposure to feared situation
  • Improve social skills
  • Role playing
  • CBT: 50-60 % useful but high relapse
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34
Q

Dependent personality disorder, what are some criteria?

A

A pervasive and excessive need to be taken care of that leads to submissive and clinging behavior and fears of separation
such as…
Has difficulty making everyday decisions without an excessive amount of advice and reassurance from others. Has difficulty initiating projects or doing things on his or her own.

Goes to excessive lengths to obtain nurturance and support from others, to the point of volunteering to do things that are unpleasant.

Feels uncomfortable or helpless when alone because of exaggerated fears of being unable to care for himself or herself.

Urgently seeks another relationship as a source of care and support when a close relationship ends.

Is unrealistically preoccupied with fears of being left to take care of himself or herself.

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35
Q

What is the etiology for dependent personality disorder?

A

Moderate genetic influences

Psychological influences:

  • Personality trait of sociotropy→traits characterized by excessive investment in positive social interactions (strong needs for acceptance of others)
  • Opposite of independent, no autonomy
  • Social influences: Trauma in childhood
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36
Q

What are the treatments for dependent personality disorder?

A

Few studies on treatment efficacy

Psychotherapy

  • Increase independence
  • Client can be dependent of the therapist
37
Q

What is OCPD?

Obsessive-compulsive personality disorder

A

preoccupation with orderliness, perfectionism, and mental and interpersonal control, at the expense of flexibility, openness, and efficiency.

Only slightly related to OCD: Generally, lacking specific obsessive thoughts and
compulsions (like symmetry, washing hands, etc)

38
Q

What is the etiology for OCPD?

A

More often in men
Biological influences weak
Psychological influences:Perfectionism
Potential social influences: Trauma faced during childhood- ex. If you do THIS then THIS will happen

39
Q

What are the treatments for OCPD?

A

Few studies on treatment efficacy

Psychotherapy: Changing maladaptive behaviors into healthier productive behaviors

40
Q

What are psychoactive substances for substance use and addictive disorder? what are some reasons to use it?

A

Psychoactive substances
Chemical compound
Altering mood, behavior, consciousness…

Reasons: medical, recreational (euphoria, to feel that high), entheogen (spiritual reason, spiritual experience, yoga, ritual, etc. meditation -not relaxation), purposeful (performance-related, it overlap with medical… ex.steroids ), research

41
Q

Substance use and addictive disorder

what are the levels of involvement?

A

Use
Intoxication
Abuse
Dependence

42
Q

Substance use and addictive disorder

define use and intoxication

A

Levels of involvement: use
Ingestion of psychoactive substances that does not significantly interfere with social, educational, or occupational functioning

Levels of involvement: intoxication

Intoxication
-Slang terms: getting high, booze, tipsy
-Reactions to use substances: more extraverted, trouble to speak, 
-Includes changes, varying according to substances and individuals, in:
    Mood
    Speech
    Energy
    Heart rate: morphin, heroin…
    Motor ability
43
Q

Substance use and addictive disorder

define abuse

A

Levels of involvement: abuse
Substance abuse
How significantly it interferes with the user’s life
E.g.
Driving drunk
Unable to get a job
Do not attend class
in DSM-4: different point of view. you could say that
the person is displaying substance abuse as soon as
you see harmful consequences OR dependence
(you can have both).

44
Q
Substance use and addictive disorder
define dependence (4 points)
A
  1. Dependence (negative effect but keep doing it to avoid the negative symptoms. for addiction = compulsive use. The reason: you take it for the positive effect - the rush, adrenaline…you ready to do anything for it) .today with DSM-5, you won’t have substance dependence it would be : you name the substance in the disorder
  2. Psychological dependence → emotional-motivational withdrawal symptoms (dysphoria, anxiety, anhedonia, dissatisfaction)
  3. Physical dependence → physical-somatic symptoms (faitgue. vomiting, chills, pain diarrhea)
  4. Drug-seeking behaviors of dependence/addiction
    - Repeated use of a drug
    - Desperate need to ingest more
    - Likely to resume the use after a period of abstinence
45
Q

What are some changes in DSM concerning substance abuse?

A

Changes DSM-IV → DSM-5 include:
DSM-IV separated substance abuse and substance dependence → combined in DSM-5 as substance use disorders

DSM-IV 1 symptom for substance abuse and ≥2 symptoms for substance dependence → described in DSM-5 in terms of severity: mild (2-3), moderate (4-5), severe (≥6)

DSM-5: each specific substance addressed as a separate
use disorder: cannabis dependence…

46
Q

Substance and addictive disorder have some comorbidity with…?

A

Comorbidity: mood disorders, anxiety disorders, etc.
1. Intoxication and withdrawal cause other symptoms
Depression, anxiety, or psychosis
Increases risk taking behaviors
2. Mental health disorders cause the substance use disorders

47
Q

Substance use disorder: diagnostic criteria

A
  1. often taken in larger amounts or over a longer period
  2. persistent desire or unsuccessful efforts to cut down or control substance use.–> lack of success
  3. time is spent in activities necessary to obtain the substance use the substance, or recover from its effects.
  4. Craving, or a strong desire or urge
  5. use despite having persistent or recurrent social or interpersonal problems
  6. Activities are given up or reduced because of the substance use.
  7. Use is continued despite knowledge
  8. Tolerance
  9. Withdrawal,
48
Q

Substance use disorder: classification in DSM-5

A
Sedatives, hypnotics, or anxiolytics
Inhalants
Alcohol
Stimulants
Tobacco
Caffeine
Opioids
Hallucinogens
Cannabis
Other substances
49
Q

What are the sedatives, hypnotics and anciolytics about substance use disorder:?

A

Decrease activity of Central Nervous System
E.g. Promoting GABA (it calms you down)

Relieving anxiety, insomnia

Include: barbiturates, benzodiazepines (very common)
Inhalants/inhaling th(e.g. aerosol, glue): depressant
effect, and potential precursor for alcohol (mis)use
in adulthood.

50
Q

What are the alcohol effect about substance use disorder:?

A

Predominantly, depressant property
Various factors influence the extent of alcohol-related effects, including
– Prenatal alcohol exposure
– Family history
– Age when a person first started drinking and for how long a person has been drinking
– How much and how often individual drinks
– Blood alcohol content (BAC) (0.08 = can drive a car - muscle coordination and attention which you need both the drive)
(BAC increases, so does impairement)

51
Q

what is Cirrhosis? what are the brain damage of alcohol effects?

A

Cirrhosis: body cannot to replace its tissue due to abuse

Brain damage: lot of effect on the frontal lobe

52
Q

what is Wernicke-Korsakoff syndrome?

A

Wernicke’s encephalopathy:

    • Mental confusion
    • Oculomotor disturbances: trouble with eyes nerve
    • Difficulty with coordination

Korsakoff’s psychosis:

    • Learning and memory impairments: trouble with new , antego amnesia (new). some with that walking to your apartment might not be able to go back to his place because they won’t remember how they got there in the first place
    • both condition goes hand in hand, don’t have to but they do
53
Q

what are the alcohol statistics?

A

Most drink in moderation
Men drink more than women
16% men vs. 4% women heavy drinkers
~9% drinkers report some problem with alcohol
Most likely to have alcohol problems are single males with lower income + of younger age
Worldwide lifetime prevalence of alcohol use disorder:
~18-21%
Most people’s drinking pattern fluctuates
~20% of individuals with alcohol abuse present spontaneous remission
Progressive pattern through levels of involvement
Might lead to violence

54
Q

what are some stimulants?

A
    • Increase CNS activity: Blocking the reuptake of dopamine, norepinephrine (NT related to motivation, reward, rush)
    • Increases levels of alertness, energy
    • Include: (meth)amphetamine (more dangerous than cocaine), cocaine
      amphetamine: it is prescribed a lot, also commonly use to treat ADHD
    • Nicotine from tobacco plants, with mild stimulant and depressant properties→ increase heart beat, increase alertness, BUT could be used to reduce stress, relaxant properties… It has a class of its own.
    • Caffeine (like coffee, tea, chocolate…everywhere, so where is the line?), a “gentle” stimulant; no officially recognized related disorder: it is a class; there is no official disorder for it because it is everywhere. It is an exception in DSM-5
55
Q

what are Opiod?

A

From the opium poppy
Relieve pain, along with euphoria and relaxation: E.g. promoting the activity of opioid receptors

Include: heroin, opium, morphine
if too much of it: affect the breathing (decrease.. can lead to coma or even death),
Increased risk for HIV infections due to needle use, not the only use but it is highlighted

56
Q

what are hallucinogens?

A

– Alter sensory perception
–Include: psilocybin(the magic mushroom), LSD (acid), PCP
– Use in clinical because: Low to no withdrawal symptoms
– Tolerance builds slowly
– emotion, dilated pupils, mystical experience…
– Some of them are used in clinical trials → often
combined with psychotherapy (psilocybin, MDNA-
cocaine) short term because it alters sensory
perception. It may lead the patient to talk about
traumatic experience without experience the
emotional trauma that comes with it (PTSD) - it helps
the process.
– common site of action: Chemical similarity to serotonin (partial agonist of serotonin receptor)
– there is a cannabis disorder that is recognized
– acts on cannabinoid that is in our body

57
Q

what is cannabis?

A

it is complex
more psychological addiction rather then physical
– No part of hallucinogens
– From Cannabis sativa
feeling the ‘’high’’ = THC
feeling relaxe = CBD
– Low to no withdrawal symptoms
– Tolerance builds quickly (useful in the war, less
appetite, less fatigue)
– Endocannabinoid system
– Depressant, stimulant, hallucinogenic effects
– More adolescent, young adult mostly use it
– Medicinal purposes

58
Q

what are other substances?

A

E.g.: anabolic steroids: synthetic hormones, develop muscle growth of muscle, use to weight loss, use illegally by athletes, induce puberty (it helps people late on puberty), therapeutic use: chronic condition, cancer…
– various side effect after heavy use: nausea,
abdominal pain, insomnia, hair loss, liver & kidney &
heart trouble

59
Q

what are the biological approach for substance use disorder?

A

Genetic and hereditary influences (esp. for alcoholism)
Twin, family, and adoption studies support this claim
High genetic influence in men
gene and environment play both an important role

No single gene determining substance use → contribution of endophenotypes- characteristic that has a genetic basic, ex impulsivity- and combination of genes
Impulsivity, seeking high activity
Low sensitivity to alcohol
Shared genetic background with antisocial
personality disorder,
mood disorders, schizophrenia
major inhibitory GABA receptor gene, Dopamine
receptor gene, ALDH gene(alcohol metabolize
enzyme) = a lot involved at the same time.
Neurobiological influences
Substances affect brain reward pathways (euphoria,
rush)
Cause pleasurable experience
a lot of substance affect levels of dopamine
tolerance will build over time, desentization if
agonist

60
Q

what are the psychosocial approach for substance use disorder?

A

Behavioral conditioning
Positive reinforcement: The “high” of the drug -
addiction
Negative reinforcement: Remove pain, unpleasant
feelings- dependence (maybe you’ll increase the
dose to take away the pain)

Cravings
Influenced by cues = conditioned stimuli
cues: getting into a bar and look at the bottles
(seeing = crave starts, temptation)… external cue
mood: fatigue, anxiety, wanting to celebrate,
conflict… the feeling = internal cues

61
Q

what are the social approach for substance use disorder?

A

Exposure to substances through: family peers, media (seeing drinking, smoking..) now sensitization in high shool

Poor adult supervision: neglect, no support, no awareness (not limited to parents or immediate family)

Societal views:
–Ex. drinking can be perceived as something very bad
–past what ages it can be consumed
–what excessive means
–Brain disease theory vs. Moral weakness theory
there is a research saying that the way you phrase certain words can lead to stigma to… the group of the dependence believed more about the brain disease but the group called drug abuser believe more in the moral weakness (their fault) that is why we use the term substance use disorder (and then to precise it: heroin…) and not abuse or dependant it is to prevent stigma

62
Q

Integrated model - important for exam

A

image on page 25

63
Q

what are biological treatment for substance use disorder?

A
  • agonist substitution

Safer drug with similar makeup, working on the same receptors, stimulate
E.g. Methadone (opioids) instead of heroine
it works on the same receptors… Some can stay on methadone for life, some might decrease gradually where it is no longer needed (it takes years to get there).

antagonist treatment

Block or counteract the effects of a substance
E.g. Naloxone for heroin abuse
it is in the paramedic kit… if someone is in an overdose = you use naloxone to overcome it. you want to be quick on that, you cannot use it in the long run.
block on the BBB

aversive treatment

Make ingesting the drug unpleasant
E.g. Disulfiram’s Antabuse effect= it comes in a form of a powder, easily solvable in alcohol, it inhibited the LDA (enzyme that metabolize alcohol).. so alcohol is not breaking down and the effects will accumulate and you are going to feel hungover right away, immediately. Headache, nausea, vomiting… it makes the digestion of alcohol quite unpleasant
least popular one: rare that the patient will stick to that treatment.. might start it but it won’t last.

64
Q

what are psychosocial treatment for substance use disorder?

A

Biological treatments alone usually insufficient
Inpatient facilities: Help with initial withdrawal, control environment
Expensive, not everyone can make it there. Rehabs

Alcoholics Anonymous and its variations (e.g. NA (narcotic anonyme = any other drugs)–> AA = 12 steps for it

Aversive therapy: Substance use paired with an unpleasant stimulus: control use, control environment, pharmacological - rare

Contingency management

-Make goals and rewards for attaining those goals
-Frequent monitoring of the target behavior
control environment
-Social support is important when the patient is with their family… how was your behavior outside of the hospital let’s say. the more closer to the target behavior = more reward (more family visit)
-reinforce behavior
-a lot of it at the hospital

Community reinforcement approach and family training (CRAFT)
Family-based therapy- major component. Give them knowledge. How to support the individual which will help to prevent a relapse
ABCs of substance abuse
Antecedents (triggers), behavior (drug use), consequences

Relapse prevention

  • Target positive beliefs about drug use
  • Target consequences of drug use
  • Develop strategies to deal with cravings: medication, avoiding certain places, distress management, social support, etc. Keep going even if you relapse
  • Relapse seen as something that individual can recover from
65
Q

Harm reduction

A

Main goal is to minimize harm
–unlike ‘’say no to drugs’’ compain in the US
–More promising than ‘’say no to drugs’’
can end in abstinence

Relapse prevention

  • Target positive beliefs about drug use
  • Target consequences of drug use
  • Develop strategies to deal with cravings
  • Relapse seen as something that individual can recover from
66
Q

Gambling disorder: Statistic (only addictive disorder is official in the DSM-5)

A

-Prevalence: ~2%
-More prevalent in men than in women
- AOO: 20s
Consequences:
14% experience job loss
19% declared bankruptcy
21% arrested
Considered a behavioral addiction or an addictive
disorder

67
Q

Gambling disorder: Criteria

A

Persistent and recurrent problematic gambling behavior leading to clinically significant impairment or distress

  • —restless or irritable when attempting to cut down or stop
  • —made repeated unsuccessful efforts to control, cut back
  • —often preoccupied with gambling.
  • — gambles when feeling distressed.
  • —-After losing money gambling, often returns
  • —Relies on others
  • —jeopardized or lost

B. The gambling behavior is not better explained by a manic episode: impulsivity (excessive spending)

68
Q

what about internet use?

A

Internet use is not a recognized disorder in DSM-5 but maybe in DSM-6. (around 10 years gap between each DSM)

69
Q

SCZ: early descriptions

Kraepelin’s and Bleuler

A

Kraepelin’s form of psychosis (1856-1926)
—Manic-depressive illness
—-Dementia praecox– .precautious
madness, or precocious dementia =
cognitive deterioration(speak and talk)
later it is set as schizophrenia
(delusional belief, motor disturbances)
—-
Bleuler (1857-1939)
–Did not believe in early onset
–Did not believe in inevitable progress
towards dementia
–Coined term Schizophrenia

70
Q

SCZ in the media

A

“People with schizophrenia are dangerous”
Not true
Individuals with schizophrenia are not more likely to commit future crimes than individuals with no schizophrenia

Complex psychotic disorder characterized by major disturbances in thinking, perception, emotion, language, and/or behavior, disconnected

71
Q

Diagnosing SCZ

A

Heterogeneity

People with schizophrenia can differ from each other more than do people with other disorders

Contribution of comorbid disorders: depressive, substance use, anxiety… apathie?

72
Q

Statistics SCZ

A

Onset: late adolescence or early adulthood

Course: chronic -> Typically, several acute episodes of their symptoms, with
less severe symptoms in between

Full recovery is rare
Comorbidity: MDD, substance use disorders, anxiety disorders
Also: almost 10% commit suicide

73
Q

Development SCZ

A

Children show abnormal signs before they develop SCZ, but difficult to know.. (ex. imaginary friend)
Older adults show fewer positive symptoms

74
Q

SCZ diagnostic criteria

A
  1. Delusions
  2. Hallucinations
  3. Disorganized speech
  4. Disorganized or catatonic behavior
  5. Negative symptoms

disturbance persist for at least 6 months

Schizoaffective disorder and depressive or bipolar disorder with psychotic features have been ruled out

The disturbance is not attributable to the physiological effects of a substance or a medical condition

75
Q

Aside SCZ…

A

Schizophreniform disorder

Same criteria as schizophrenia, except:
Symptoms for fewer months → then disappear
“Why” mostly unknown

76
Q

SCZ: clinical symptoms

positive symptoms

A

Presence of too much of a behavior
Experienced by 50%-70% of individuals with SCZ
- Delusions
- Hallucinations
Erroneous beliefs despite clear contradictory evidence

Usually involves misinterpretation of reality

Can also occur during manic episodes, during depressive episodes with psychotic features, etc.

77
Q

what are the delusions types?

A

persecution

reference

body control

delusion of Grandeur

delusion about one’s own thoughts (broadcastin, insertion and withdrawal)

Capgras syndrome: Belief that someone a person knows has been replaced by an imposter
Cotard’s syndrome: Belief that one has lost body parts or even that one has died

78
Q

what are the hallucinations types?

A

Most dramatic distortions of perception

Sensory experiences in the absence of stimulation from the environment
Types: auditory*, visual, gustatory, olfactory, or tactile (e.g. formication)

Like delusions, hallucinations can be very frightening experiences

79
Q

SCZ: clinical symptoms

disorganized symptoms

A

Least studied and understood
Prevalence is unclear

Includes
Inappropriate affect
Disorganized speech

80
Q

SCZ: clinical symptoms

disorganized symptoms -> inappropriate affect

A

Emotional responses are out of context: Likelihood to shift rapidly from one emotional state to another for no discernible reason

This symptom is quite rare, but its appearance is of considerable diagnostic importance because it is relatively specific to SCZ

81
Q

SCZ: clinical symptoms

disorganized symptoms -> disorganized speech

A

Include: Loose associations & Incoherence

Evidence indicates that the speech of many people with SCZ is not disorganized and that the presence of disorganized speech does not discriminate well between SCZ and other psychoses

Loose associations or derailment
1. “I was going down to the store because it is October and the people near the street didn’t tell me what was happening since I don’t like to walk too fast. So, after the time when it was happening I decided that no one knew who I was and needed to buy some kind of bicycle or other type of train.”

Incoherence or “word salad”
2. “Well, lettuce is a transformation of a dead cougar that suffered a relapse on the lion’s toe. And he swallowed the lion and something happened. The… see, the… Gloria and Tommy, they’re two heads and they’re not whales. But they escaped with herds of vomit, and things like that.”

82
Q

Catatonia

A

Several motor abnormalities

Unusual increase in overall level of activity: Can gesture repeatedly

Unusual decrease in overall level of activity
  Stupor: total immobility
  Catalepsy: unusual postures
   Waxy flexibility: limbs can  be 
      manipulated into strange positions
83
Q

SCZ: clinical symptoms

negative symptoms

A

Present in 25% of individuals with SCZ
Behavioral deficits
Avolition: lack of will, energy
Alogia: poverty of speech or content
Anhedonia: lack of interest, pleasure
Asociality: few friends, poor social skills
Flat (or reduced) Affect*: lack of emotional expressiveness

Alogia
“Why do you think that people believe in God?”

“Well, first of all, because, he is the person that is the personal savior. He walks with me and talks with me. And uh, the understanding that I have, a lot of people, they don’t really know their personal self. Because they just don’t know their
personal self. They don’t know that he uh, seems to like us. A lot of them don’t
understand that he walks and talks with them.
And uh, show’em their way to go…”

84
Q

what are the 4 subtypes of SCZ in DSM-4 ?

A
Paranoid
Disorganized (hebephrenic)
Catatonic
Undifferentiated
Residual
85
Q

SCZ Dsm-4 to DSM5, what about it?

A

DSM-5 work group proposed:
Removing all of the subtypes
Major argument for discontinuing the subtypes
Low reliability and poor validity (APA, 2013)
Instead, DSM-5 enables clinicians to consider the heterogeneity in symptom expression

86
Q

SCZ: Etiology

Biology approach

A

Twin studies
Role of genes and environment in the development of SCZ
Identical twins do not always both have the disorder
“Genain” quadruplets
All developed SCZ → Genetics
Different time, severity → Non-shared environment?

Nora Iris Myra Hester- all four had SCZ by 24 years old (quadruplet). Different course, severity → same family environment but what else? Non-shared environment? peer pressure?

Adoption studies
Even when raised away from biological parents, adopted children have an increased risk for developing SCZ
Good home environment lowered these risks

Genetic linkage and association studies
Several genes implicated in the development of SCZ
E.g. 5-HT2A gene, DRD3 gene

Dopamine theory: System may be too active in individuals with SCZ

Brain
Larger ventricles observed in some individuals with SCZ
Larger in individuals who have had SCZ longer.
Less brain tissue, less space for frontal lobe

87
Q

SCZ: psychosocial and social influences

A

tress
Stressful life events appear to precipitate the onset of the disorder
↑ Life stress ~ ↑ relapse

Sociogenic hypothesis: living in lower social class is stressful

Social support: Appears to be a protective factor and improve prognosis

Transcranial magnetic stimulation (TMS), page30

88
Q

SCZ psychosocial intervention

Social learning ward
Milieu-therapy ward
Routine hospital management
Social skills training
 Family therapy
A

Social learning ward

  • token economy
  • addressing aspects of living

Milieu-therapy ward

  • therapeutic community
  • acting responsibily
  • kept busy 85% of the time

Routine hospital management

  • Regular custodial care
  • Antipsychotics

Social skills training

  • Learn skills in various domains
  • Does not necessarily improve global functioning

Family therapy

  • Goal: reduce emotional expressivity
  • Communication skills
  • Education

Psychodynamic therapy: insight into the role of the past
Cognitive behavioral therapy (CBT): decrease symptom intensity, relapse, reduced more negative symptoms

Early intervention affects prognosis