Pressure sores Flashcards

1
Q

What is the epidemiology of pressure sores?

A

Cost the UK £1.4-2 billion per year
A comparative study of patients with pressure ulcer in the UK, US & Canada demonstrated prevalence in hospitalised patients of 4.7% to 32.1%
In patients in community care the prevalence ranged from 4.4% to 33.0% and was 4.6% to 20.7% in patients in nursing homes
Among elderly patients being seen by a GP in the UK, an overall incidence rate of 0.58 ulcers per 100 person- rates were considerably higher in people >85yrs

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2
Q

What is the definition of a pressure sore?

A

Pressure sore- a localised injury to the skin and/or underling tissue over a bony prominence as a result of pressure, or pressure in combination with shear shear forces occur due to the constant pull of gravity against the body, and results in the patients’ body gradually slipping down the bed, chair or trolley

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3
Q

What is a stage 1 pressure sore?

A

Non-blanchable erythema
o Intact skin over a non-blanchable redness of a localised area usually over a bony prominence
o Darkly pigmented skin will appear purple or bluish rather than red may not have visible blanching
o Area may be painful, firm, soft, warmer, bluish tinge
o Patients should be considered at risk regardless of the Braden score
o Must commence a skin bundle and avoid positioning the patient on the affected area whenever possible

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4
Q

What is a stage 2 pressure sore?

A

Partial thickness
o Partial thickness loss of dermis presenting as a shallow open ulcer with a red/pink wound bed, without slough
o May also present as an intact or ruptured serum-filled blister
o Presents as a shiny or dry shallow ulcer without slough or bruising (indicates deep tissue injury)
o This stage should not be used to describe skin tears, tape burns, incontinence associated dermatitis or excoriation

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5
Q

What is a stage 3 pressure sore?

A

Full thickness skin loss
Full thickness tissue loss- subcutaneous fat may be visible, but bone, tendon or muscle are not exposed
Slough may be present, but does not obscure the depth of tissue loss, may include undermining and tunnelling
The depth of a stage 3 pressure ulcer varies by anatomical location
Bone/tendon is not visible or directly palpable

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6
Q

What is a stage 4 pressure sore?

A

Full thickness tissue loss
Full thickness tissue loss with exposed bone, tendon or muscle, slough or eschar may be present
Ulcers can extend into muscle and/or supporting structures (eg. fascia, tendon or joint capsule) making osteomyelitits likely
Often includes undermining or tunnelling
The depth of a stage 4 pressure ulcer varies with anatomical location

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7
Q

When is a deep tissue injury suspected?

A

Purple or maroon localised area of discoloured intact skin or blood-filled blister due to damage of underlying soft tissue from pressure and/or shear
The area may be preceded by tissue that is painful, firm, mushy, boggy, warmer or cooler as compared to adjacent tissue
Deep tissue injury may be difficult to detect in individuals with dark skin tones- evolution may include a thin blister over a dark wound bed, the wound may further evolve and become covered by thin eschar
Evolution may be rapid exposing additional layers of tissue even with optimal treatment

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8
Q

What is a moisture lesion?

A

Caused by chronic exposure to urine/faecal matter leading to skin appearing macerated
extremely painful for the patient

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9
Q

What are the features of a moisture lesion?

A

Shining wet skin
May be over a bony prominence or in skin folds, anal cleft, peri-anal redness/skin irritation
Diffuse superficial spots or irregular shape. Linear shape in cleft and skin folds
Superficial depth
No necrosis or eschar present
Diffuse, irregular edges
Redness that isn’t uniformly distributed. Pink ro white maceration

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10
Q

How is the skin tested?

A

Using the blanch test caused by chronic exposure to urine/faecal matter leading to skin appearing macerated extremely painful for the patient
Assess for:
Pain or oedema in the area
Warmer or cooler temperature over a bony prominence
Hardened area
Broken skin

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11
Q

What are intrinsic risk factors?

A

Reduced mobility or immobility
Vascular disease (reduced blood flow)
Sensory impairment (neurological disorders, spinal cord injury lead to lack of stimulus to relieve pressure
Severe chronic or terminal illness
Reduced level of consciousness
Either the very young or the old (poorer circulations)
Previous history of pressure damage (weak skin)
Malnutrition
Dehydration
Older people undergoing hip replacement surgery
Acute illness

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12
Q

What are the extrinsic risk factors?

A

All factors that are involved that can cause injury, pressure, shearing and friction will eventually lead to tissue damage
Exacerbating conditions- medication (hypnotics, sedatives, inotropes) or moisture to skin

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13
Q

What is the Braden risk assessment tool?

A

all adult services within 2hrs of admission- reassess at least weekly and upon change of condition
Score 16 or less- high risk – 2hrly repositioning (red skin bundle)
Score 17-20- medium risk (amber skin bundle)
Score 21-23- low risk
Sensory perception
Moisture
Activity
Mobility
Nutrition
Friction and shear
1-4: 1- worst, no 4 for friction and shear

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14
Q

What are the other risk assessment tools for pressure sores not in the elderly?

A

Glamorgan scale: all children’s services within 2hrs of admission- score 10 or over are at risk
Cubbin & Jackson: all critical care services within 6hrs of admission- score 40 or less are at risk

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15
Q

What is pressure ulcer assessment?

A
Cause of ulcer
Site/location
Photography
Dimensions of ulcer
Pain and/or odour
Exudate & signs of local infection
Stage – fistulae/sinus
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16
Q

How are pressure sores prevented (SSKIN)?

A

Support surface- provide a mattress & cushion
Skin evaluation- assess on repositioning, record blanch test
Keep moving- record repositioning frequency and code
Incontinence- assess for moisture lesions
Nutrition- complete MUST tool

17
Q

What are the methods for preventing pressure sores?

A

Repositioning 30o tilt: when patients are positioned correctly the sacrum should be clear of the mattress, if patients have a tendency to roll back, then keep them in position using pillows and check them more frequently
Heels: off-loaded using pillows/dermal heel pads/orthotic boots
Repositioning regimes: 2hrly, if isn’t marking use 3hrly
Seating: not sit out of bed for longer than 2hrs, pressure relieving cushions
Incontinence: pad and pants should be checked at every positioning
Nutrition: poor nutrition is directly linked to skin breakdown and will compromise wound healing also consider blood results that could impact on wound healing (albumin, anaemia)

18
Q

What are the types of dressing used?

A

Alginates made from calcium alginate, highly absorbent e.g. Sorbsan, Tegaderm: All wounds with moderate to heavy exudate
Cadexomer iodine: Iodine acts as antiseptic and Cardomer absorbs
wound exudate e.g. Povidone Iodine dressing: Sloughy infected with heavy exudate
Capillary - action: Absorbent core of hydrophilic fibres in between low
adherent wound contact layers: Only for heaving exuding sloughy
wounds
Films: Use on epithelializing wounds with low
exudate
Foams-Contains hydrophilic polyurethane foam: Best used on granulating wounds

19
Q

Which dressings are used for infected wounds?

A

Honey-can be used as a topical or in combination with Alginates
Silver- antimicrobial effect
Soft polymers e.g. Mepitel. Do not in heavy bleeding: granulating wounds

20
Q

What are hydrocolloids used for?

A

Hydrocolloid layer on a vapour-permeable film or foam pad. Also promote granulation e.g. Comfeel Plus.
Not suitable for infected wounds but
most types with low to moderate exudate.

21
Q

When should pressure sores be reported?

A

Inherited: patients are admitted with pressure damage from home, care home or another trust
stage 3&4 are also inherited in occur within 72hrs
Acquired: skin damage has occurred since admission to Trust and must be reported-if occurred on previous ward make sure it has been documented – if not an incident form needs to be completed