post polio syndrome: examination , evaluation and interventions Flashcards
how was acute poliomyelitis exposed
thru the GI tract (coughing , breathing it in)
what kind of virus was polio
benign viral illness
for <5% it invaded the ___ with a ___ spinal tap bu no clinical ___
CNS
+
paralysis
wbat cells and neurons does acute polio affect
anterior horn cell
lower motor neuron cells (final pathway to mm)
for 1-2% of people with polio it causes a variable degress of what ?
paralysis to the limbs/trunk , swalling/breathing or both
when were vaccine made that virtually eradicated polio
late 1950
when did india and nigeria deem polio free
2013
2019
deemed polio free mean no new cases of polio for 3 whole years
who will continue to have wild polio
afghanistan and pakistan
what kind of countries are at high risk of resurgence
under developed
how long is the period of recovery for polio patients
weeks to years depending on how severe the polio is
how long is the period of neurological stability for polio
greater then 15 years
being with plateau of maximum neurologic and functional recovery
the period of neurological stability lasts indefinitely in ___ of those with paralytic polio
50%
what is post polio sequelae
when someone with polio develops a functional limitions and then adapts to that over the years
for 20-50% of people with polio the period of neurological stability ends with the onset of new ___ and other post polio ___ symptoms
weakness
syndrome
what does someone have to have with the onset of post polio syndrome
new weakness
when is post polio syndrome typically start
30-50 years after polio and has to have new weakness
for the diagnosis of post polio syndrome there must be what 3 things
a history of polio
a period of neurological recovery followed by period of stability lasting at least 15 years
no other medical problems that can cause similar issues
how do u determine if someone has a history of polio
spinal tap acutely and there will be polio in CSF
GIANT motor units by EMG (this is where u will see the reinnervation)
no pattern in weakness
motor deficits ONLYYYYY
what is the hallmark of pilio related weakness
that there is no patten
who is the risk higher for in PPS
people with more severe permanent impairments
women
length of time since acute illness
what is the PPS pathophysiologu
degenerating terminal axonal sprouts and an inability to maintain the increased metabolic deman from the giant motor unit
what is persistent poliovirus
people who has polio with PPS
how will people wiht polio compensation to increase their function
use their weak mm at high level of capacity
substitution of strong mm with increased energy expenditure
use of ligaments for stability with resulting in hypermobility
in the clinic we will see pilio survivors with/without PPS who have
___ pain complained that need intervention
have ___ pain complaints
require re education for functional activities
require ___ conservation education
desire exercise programs in or out of water
require follow up after bracing
acute
chronic
energy
what is the hand held dynamometry limited to
200N
low reproducibility over time
unable to detect small declines in strength