Post-Partum Complications Flashcards

1
Q

High risk, prolonged, or difficult delivery warrants increased PP monitoring because of increased risk of ______ , _____ and _________

A

Hemorrhage, infection and PPD

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2
Q

What is the foci of nursing care of a PP woman?

A

BUBBLE…

Breasts, Uterus, Bowel, Bladder, Legs, Emotions AND baby.

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3
Q

According to what we’re focusing our assessments on on a post-partum mom (BUBBLE), what common complications can arise?

A

B - mastitis, U - PPH, B- constipation, B- urinary retention, L- thrombophlebitis, E- PPD

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4
Q

When might an “early” PPH occur? Late?

A

<24 hours. >24 hr - 6 wk.

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5
Q

What are the “4 T’s” of risk factors for PPH? An example of each?

A

Tone: uterine atony. Trauma: forceps delivery. Tissue: retained POC. Thrombin: DIC

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6
Q

What is the most common cause of PPH?

A

Uterine atony

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7
Q

What is uterine atony?

A

Relaxation of the uterus, blood vessels cannot be pinched off where the placenta detached from and the woman bleeds.

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8
Q

What can cause uterine atony? What should we encourage?

A

Full bladder. Urinating q2 hours.

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9
Q

What are our interventions if our patient experiences uterine atony? (6)

A

Fundal massage, express clots from uterus, lower HOB, VS, IV NS/RL, oxytocin.

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10
Q

What does our PPH assessment look like? How often are we assessing mom?

A

Fundus (placement/timing), lochia (amount/clots/timing/color), Perineum, VS, pallor/fatigue/SOBOE, Voiding? q 15 mins.

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11
Q

What is the normal amount of lochia expected in a PP woman? What does lochia usually smell like?

A

6 in or less on peri pad in 1 hr. Odourless.

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12
Q

How often are we changing mum’s peri pad?

A

q 3 hr

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13
Q

In order of how we would give IV medications for PPH, what do we start with first and what would we use last? Give a short description of each.

A

Oxytocin (synthetic hormone causes contraction of the uterus - used to augment labour and control PPH). Mesoprostol (synthetic prostaglandin that causes contraction of the uterus). Ergometrin (ergot alkaloid causing smooth muscle contraction of uterus). Hemabate (used for abortion in first and second trimesters- given in PPH when conventional methods have not worked)

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14
Q

What are four risk factors for PPI?

A

> 24 hr ROM, retained POC, PPH/anemia, internal FHR monitoring.

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15
Q

When do signs of PPI usually come about? Day 3/4 what should we not mix up infection with? What are 5 things we might see that indicate PPI? What is something we normally look at that indicates infection that wont be useful in a PP woman?

A

3-4 days after delivery. Milk comes in on day 3/4 and mom might feel feverish/sweaty. Uterine pain, malaise, foul smelling lochia/discoloured lochia, increased temp, increased PV losses. WBC count usually is elevated after pregnancy - but if ++ elevated can indicate infection.

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16
Q

What are our interventions for a mom with PPI (4)? What drug might be necessary for involution (shrinkage) of the uterus?

A

Teaching about SS of infection because mom will likely have already gone home by day 3-4 when an infection will pop up. Abx, analgesia, infection control. Oxytocin.

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17
Q

Foci of nursing care for a high-risk newborn (7)?

A

Respiratory, temperature, extrauterine circulation. Fluids/lytes, nutrition/waste, preventing infection, bonding/attachment.

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18
Q

Normal vital signs for a newborn?

A

Resp: 30-60. T: 36.5-37.4. PR: 110-160. BG: 3.2-6.

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19
Q

What does the APGAR tell us? What should we NOT use this to determine?

A

Tells us how babe is adjusting to extrauterine life. Should not use this to determine whether or not a babe need resuscitation.

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20
Q

What will our assessment be of a newborn? (6)

A

Review pregnancy/labour/birth history, review mom’s history (smoking, GBS, gestational DM, HIV etc), review APGAR, VS, physical assessment, review feeding/elimination patterns.

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21
Q

What can happen to a baby if they are exposed to group B strep located in the mom’s vagina? When are mom’s tested for GBS? What does their care look like during labour?

A

Meningitis. Test mom at 35 wk gestation, if positive ABX will be given IV during labour.

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22
Q

What does TORCH stand for?

A

Toxoplasmosis (cat feces), Other (syphilis, chicken pox), Rubella, Cytomegalo virus, Herpes.

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23
Q

If mom’s titre for rubella is low, can we give mom a vaccine while pregnant?

A

No cause it will cause brain injury to baby as it is a live vaccine. Give to mom before leaving hospital.

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24
Q

If mom has active herpes lesions, can she deliver vaginally? What can we give mom?

A

No, baby could get herpes. Give mom acyclovir. Deliver baby via C/S.

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25
Q

How often are we checking APGAR?

A

1 min, 5 min, and 10 min after birth.

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26
Q

If babe’s APGAR is <5 at 1 min… what do we do?

A

Monitor more closely.

27
Q

If babe’s APGAR is <7 at 5 min… what do we do?

A

Transfer to NICU.

28
Q

If babe’s APGAR is <2-3 at 1 min what do we do?

A

Transfer to NICU.

29
Q

If an infant is born healthy, what are the only things we need to do for them when they come out?

A

Dry them off, and bundle them up.

30
Q

What are factors that lead to respiratory depression in preemies (6)?

A

Decreased surfactant/lung maturity, susceptibility to cold and stress/metb disturbances, immature organ development, immature CNS, reduced fat deposits/no brown fat, reduced ability to eat/absorb nutrition.

31
Q

What 2 main valves closed after birth? What did those valves in the heart do?

A

Ductus arteriosus and foramen ovale. Shunted blood away from the lungs while in utero.

32
Q

As a neonate, they are vulnerable to hypoglycemia and cold stress (especially if high risk), what can result from cold stress or hypoglycemia? How does this affect the infant?

A

Ductus arteriosus and foramen ovale might stay OPEN and not CLOSE, therefore blood will be routed around the lungs, not to the lungs. This can result acidosis and return to fetal circulation resulting in respiratory failure.

33
Q

What is important to remember for temperature regulation of an infant? What things can we do to keep babe warm?

A

All of the ways heat can be lost: radiation, conduction, convention. Bundle, incubator, dry linen, plastic wrap, warmed stethoscope.

34
Q

Does an infant lose blood during birth? What can happen easily to infants? How many mL can make an impact?

A

Yes through detachment of the placenta. Dehydration/FVD. 15 mL.

35
Q

How do we assess an infant for FVD?

A

Anterior fontanels (will be sunken), urine/feces, feeding

36
Q

Baby’s with respiratory distress are also at risk for what?

A

Fluid/lyte imbalance because of increased insensible fluid losses.

37
Q

A neonate can have FVE occur easily if they have an IV in situ. Why is this? What will occur?

A

Immature kidneys. Return to fetal circulation (opening of ductus arteriosus).

38
Q

How many times per dad does a baby usually feed? How long per feed?

A

8-12. 15-40 min.

39
Q

An infant born to a mother with gestational DM is at risk for what? What is the nursing care around this? What is usually necessary until breastmilk supply has fully been established?

A

Hypoglycemia due to a cut off of sugars from mom after birth and insulin that was produced remains in system for some time after birth. Frequent BG checks and ensuring adequate intake. Supplementation with formula.

40
Q

What are 4 indications a baby’s blood glucose might be low?

A

Shaking/jittery, flappy tone, reduced reflexes and cannot maintain temperature.

41
Q

How does macrosomia and respiratory distress go together?

A

A macrosomic infant’s heart and lungs have difficulty keeping up with having to supply a larger amount of O2 rich blood.

42
Q

If a macrosomic infant is not feeding well in the first few hours of life - what is the risk?

A

Decreased BG leading to acidosis and impeded respiratory function.

43
Q

What is the nursing care associated with a macrosomic infant who might develop respiratory distress?

A

Frequent BG check, supplemental feeds, possible IV and O2

44
Q

What are benefits of breastmilk for the baby’s immune system?

A

Breast milk has immunologic properties (antibodies)

45
Q

True or false: no matter how complex the infant’s condition, parent’s are partners in care for the infant?

A

True

46
Q

Are babies always encourages skin-skin whenever possible?

A

Yes

47
Q

What are 8 manifestations of neonatal abstinence syndrome?

A

Irritability, disturbed sleep pattern, frequent sneezing, shrill/high-pitched cry, possible hyperreflexia and clonus, convulsion, inc RR, vomiting/diarrhea

48
Q

______ and ____ influence ABCs of infant

A

Thermoregulation and blood sugars

49
Q

Keeping baby warm thinking about all of the ways heat is lost… conduction, evaporation, radiation, convection… give example for each.

A

Conduction - don’t put baby on cold surface. Evaporation - dry baby right after birth. Radiation - don’t put baby near window. Convection - draft takes heat away from baby.

50
Q

Eight risk factors putting pregnant women at risk for DVT?

A

Inactivity in labor, legs in stirrups for long time, history of thrombophlebitis, inactivity in pregnancy, pre-existing varicose veins, PPI, increase parity, cigarettes.

51
Q

What three factors predispose a pregnant woman to DVT development?

A

Increased fibrinogen, increased estrogen levels, and dilation of lower extremity veins causing pooling.

52
Q

Five strategies to prevent a DVT in a perinatal period?

A

Side-lying or back-lying during birth, hydrate, ambulate ASAP after birth, quit smoking, do leg/feet exercises while in bed.

53
Q

Why is it dangerous to massage the skin over a clotted area?

A

Can loosen clot and cause PE or cerebral embolism.

54
Q

Five ways to prevent the development of mastitis?

A

Position baby correctly and help grasp nipple properly, baby releases grasp before removing breast, wash hands before touching breasts, expose nipples to air, Vit E oil to soften nipples.

55
Q

What organisms are associated with nosocomial mastitis?

A

Staph aureus and candidiasis

56
Q

5 findings associated with mastitis?

A

Unilateral, fever, scant breast milk production, painful and swollen.

57
Q

4 medical/nursing interventions to treat mastitis? Where is most of the treatment done (hospital/home?)

A

Abx, cold compress, continue to feed from affected breast (more milk=more bacteria growth), supportive bra. Done at home.

58
Q

Why are women prone to UTIs after delivery?

A

Catheterization. Also maybe decreased sensation leading to urinary retention.

59
Q

Assessment findings of UTI (5)? Management of UTI after delivery?

A

Dysuria, frequency, hematuria, low-grade fever, discomfort in lower abdo. Abx, hydrate (flush out bacteria from bladder), tylenol.

60
Q

If a child is born with a physical challenge/illness when should the child be shown to parents? Why at this time? What are some common responses to delivering a child with an illness or physical challenge? What are some helpful nursing interventions and responses for a family whose newborn has died?

A

Immediately so problem/illness can be described and a prognosis given. Grief response, shock. Reinforce/review information, open line of communication. Allow parents to see deceased baby, remain with them but allow to tough. They might want to take a photo. Also need to sign certain forms. Provide them with a private room for family to give them enough time they need to grieve and visit freely.

61
Q

What 4 kinds of babies are at risk for RDS? When does surfactant usually form? What are SS of RDS (6)? Describe action of CPAP/PEEP oxygen administration? What is a risk of O2 admin for very immature infants?

A

Preterm, born to DM mom, C/S (no pressure to expel fluid from lungs like vag birth), decreased perf of lungs (MAS baby). Surfactant forms 34 wk gestation. SS: diff initiating resps, decreased temp, nasal flaring, sternal/subcostal retractions, inc RR, cyanotic mucous memb. CPAP/PEEP following surfactant admin exerts pressure on alveoli to keep open. Retinopathy of prematurity (ROP) or bronchopulmonary dysplasia.

62
Q

What is the cause of TTN? Normal RR? Rate seen in TTN? Why is feeding difficult when newborn has TTN? Which babies are more at risk (3)? What nursing actions are required? When should TTN resolve?

A

Retained lung fluid. Normal = 30-60. TTN = 80-120. Infant can’t complete suck/swallow/breath cycle effectively when RR is high. C/S, mom received ++ IVF during labor, preemie. Monitor infant for increasing resp. efforts/tiring, administer glucosteroid to decreased resp tract inflm. Will begin to resolve after 36 hours of life, typically gone by 72 hours of life.

63
Q

What causes MAS? Why is it a concern… causes resp distress in what three ways? What SS will the nurse note (5)?

A

Hypoxia leading to vagus nerve stimulation and rectal spincter relaxation. Causes severe resp distress in three ways: inflammation of bronchioles, block small bronchioles by mechanical plugging, and causes a decrease in surfactant production due to lung trauma. Difficulty est. resps at birth, dec APGAR, inc RR, retractions, cyanosis.