Post-Anesthesia Care Flashcards
6 major concerns with transport to PACU
- Oxygenation
- Ventilation
- Circulation
- Upper airway support
- Nausea and vomiting
- Systemic HTN
What to monitor during transport:
Pulse ox
Adequate ventilation
Minute ventilation increases by how much for every 1mmHg increase in arterial PCO2
2L/min
6 treatments to alveolar hypoventilation
- Supplemental O2
- Raise head of bed 30degrees
- Normalizing the PaCO2
- External stimulation of pt to wakefulness
- Reversal of opioid or benzodiazepine
- Mechanical ventilation
Should you always transport with supplemental oxygen?
YES
Does N2O make a difference with transportation?
Diffusion hypoxia
What does elimination of N2O at the end of anesthesia produces in the reduction of concentrations of the accompanying volatile agents, contributing to speed of emergence?
Significant acceleration
What 2 things does N2O do?
Adds to MAC
Provides analgesia
4 paradoxical breathing patterns
- Retraction of eternal notch
- Exaggerated abdominal muscle activity
- Collapse of chest wall
- Protrusion of abdomen with inspiratory effort (rocking horse motion)
When does upper airway obstruction become evident with residual muscle relaxant?
Once calmly resting in PACU
Gold standard for assessing residual blockade?
Sustained 5sec head lift
Treatment for laryngospasm:
- Jaw thrust with CPAP (40cm H2O)
2. If fails, sux (.1-1 mg/kg IV)
Do to attempt to pass at Rachael tube through glottis that is closed due to laryngospasm?
NO
5 possible causes to pulmonary edema:
- IV fluid volume overload
- CHF
- Sepsis
- Transfusion-related pulmonary edema
- Post-obstructive pulmonary edema
Exaggerated negative pressure generated by inspiration against closed glottis
Post-obstructive pulmonary edema