Pneumonia, PE, oedema, pneumothorax Flashcards

1
Q

S+S PE

A

SOB, pleuritic CP, syncope, tachycardic, potentially have fever, low sats

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2
Q

1st line investigations for PE

A

CXR
Wells score:over 4 = PE likely, under 4 = unlikely
If over 4 = LMWH + CTPA
Under 4 = D dimer.
If positive, CTPA
If they have an allergy to contrast, do V/Q perfusion ABG (mildly alkalotic)

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3
Q

Acute management of PE

A

O2, vascular access, NSAIDs or weak opioid
LMWH unless massive thrombolysis (then fluid challenge first)
Warfarin within 24hrs
LMWH for 5 days until INR over 2

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4
Q

Long term management of PE

A

3 month warfarin then assess

Continue if unprovoked PE

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5
Q

1st line investigations + results for pneumonia

A

CXR (consolidation), ECG, bloods (raised CRP, WCC, urea)
Culture bloods + sputum
ABG
CURB65 = ICU >3
Pneumococcal + legionella urinary testing
Pneumonia = S+S of LRTI + radiological signs

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6
Q

What is CURB65?

A
Confusion <8 AMTS
Urea >7
RR >30
BP <90/<60
Age >65

30 day mortality

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7
Q

1st line management for low severity CAP

A

Amoxicillin
Allergic: clindamycin or tetracycline
5 days

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8
Q

1st line management for moderate-high severity CAP (Curb >2)

A

Amox + macrolide (clarithromycin) 7-10 days - ADMIT

High severity: clarithromycin + cefotaxime IV - ADMIT

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9
Q

1st line management for HAP

A

Ceftriaxone + metronidazole

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10
Q

What are the organisms that cause CAP?

A
Gram positive:
Strep pneumoniae
H influenza - commonly COPD
Staph
Mycoplasma
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11
Q

What are the organisms that cause HAP?

A
Gram negative:
Pseudomonas
Mycoplasma
Legionella
Chlamydia pneum
Staph aureus
Pneumonia developing >48hrs after admission
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12
Q

What are the S+S of legionella?

A

Flu, dry cough, bradycardic

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13
Q

How to manage legionella?

A

Clarithromycin or erythromycin for 14 days

Report to PHE

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14
Q

How to distinguish aspiration pnemumonia

A

Sudden onset in someone with hx of vomiting or swallowing problems
May look like pulmonary oedema on CXR
Distinguish by fever, bloods

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15
Q

Management of severe croup

A

Nebulised adrenaline 1:1000

Dex or pred

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16
Q

Investigations for ?asthma

A

ABCDE
ABG - high CO2 + low O2 (when breathing slows)
CXR, ECG, bloods
Fractioned exhaled nitric oxide for diagnosis

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17
Q

Pneumothorax S+S

A

Abrupt SOB
Dull heavy chest pain
Hyperresonant
Reduced air entry (absent breath sounds)

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18
Q

Management of tension pneumothorax

A

Needle decompression (large bore cannula 2nd ICS mid clav) - then CXR + chest drain (5th IC space, mid axillary line)

19
Q

Presentation of HF with pulmonary oedema, and what heart sounds are produced?

A

Acute SOB
Pink frothy sputum
Chest pain
Triple/ gallop rhythm

20
Q

Management of pulmonary oedema

A

O2
If wheezy: salbutamol nebs, ipratropium bromide + hydrocortisone
Diamorphine 2.5mg, anti-emetic, furosemide GTN

21
Q

Diagnostic criteria for acute respiratory distress syndrome

A

Hypoxaemia

Bilateral CXR infiltrates

22
Q

S+S of acute respiratory distress syndrome

A

SOB
RR
Bilateral crackles

23
Q

What is type 1 resp failure?

A

Hypoxaemia (eg asthma)

24
Q

What is type 2 resp failure?

A

Hypoxia + hypoventilation causing high CO2

25
Causes of type 2 resp failure
COPD, opiate OD
26
Management of resp failure
O2 in venturi mask, nebs, steroidsCPAP
27
Organisms causing pneumonia + characteristics
Strep pneumoniae = most common, high fever, rapid onset, pleuritic chest pain, herpes labialis H influenza = common in COPD Staph aureus = follows influenza infection Mycoplasma pneu = dry cough, atypical x ray Legionella = hyponatraemia + lymphopenia Klebsiella = alcoholics, causes cavitation Jiroveci = HIV Chlamydia psittaci = pet bird dead Coxiella bunetii = Q fever - people working in animal houses
28
Simple vs tension pneumothorax
``` Simple = air in pleural cavity, not under pressure Tension = air drawn into pleural space with each breath ```
29
Causes of pneumothorax
``` Primary: Spontaneous (tall young males) Trauma Secondary: Chronic disease - COPD (bullae), pneumonia, cancer, connective tissue diseases, asthma Iatrogenic - from PICC lines ```
30
Management of primary pneumothorax
Aspirate if >2cm, discharge if <2cm  | If failed, chest drain (5th IC space, mid axillary line) + admit
31
Management of secondary pneumothorax
If >2cm, insert chest drain (5th IC space, mid axillary line) If 1-2cm, aspirate then admit
32
How to determine between primary + secondary pneumothorax?
Primary = spontaneous, <50 y/o, no comorbidities
33
CXR for pneumothorax
Black - no lung markings
34
Follow up for pneumonia
CXR in 6 weeks
35
When else is d dimer raised?
Pregnancy Cancer PE Renal disease
36
What is a PERC score?
Pulmonary embolism rule out criteria
37
How does LMWH work?
LMWH - Factor 10a inhibitor - affects clotting cascade 
38
When can you get a stroke from a DVT?
If you have PFO | All stroke pts have an echo to check for PFO
39
Clinical signs (chest expansion, trachea, percussion, breath sounds, vocal resonance) for pneumothorax
chest expansion: reduced on affected side trachea: shifted to affected side in simple + away from affected side in tension percussion: hyperresonant on affected side breath sounds: reduced on affected side vocal resonance: reduced on affected side
40
Clinical signs (chest expansion, trachea, percussion, breath sounds, vocal resonance) for pneumonia
chest expansion: can be reduced on affected side trachea: central or shifted towards side of lobar collapse percussion: dull over affected area breath sounds: reduced, coarse creps, bronchial breathing vocal resonance: increased
41
Clinical signs (chest expansion, trachea, percussion, breath sounds, vocal resonance) for pleural effusion
chest expansion: reduced on affected side trachea: central or shifted away from affected side if large percussion: stony dull breath sounds: reduced vocal resonance: reduced
42
Clinical signs (chest expansion, trachea, percussion, breath sounds, vocal resonance) for pulmonary oedema
``` chest expansion: equal trachea: central percussion: dull bases breath sounds: fine inspiratory creps vocal resonance: reduced ```
43
Ix for TB
Sputum Early morning urine Tissue biopsy = needs to be in saline (some to micro, some to pathology)