Pleural Flashcards
Hydrostatic and oncotic pressure in pulmonary circulation favor fluid accumulation on _______ surfaces and absorption at _____ surfaces
Accumulation at parietal pleural surfaces, absorption at visceral.
Higher hydrostatic pressure in vessels does what to fluid
Pushes it out
Higher hydrostatic pressure outside vessels does what to fluid
Pushes it in
Oncotic pressure - lower causes what in vessels
Water leaves
Oncotic pressure- higher causes what in vessels
Water influx/stay
Higher oncotic pressure in insterstitial space leads to what
Water leaves vessels
Lower oncotic pressure in interstitial space leads to what
Water goes into vessels
Filtration coefficient/ permeability effect on vessel
More permeable= mild changes in starling forces have greater effect on fluid accumulation
Which vessels are more leaky pleural vessels vs lung vessels
Pleural not very leaky, lung leaky but oncotic pressures so narrow that they drive fluid resorption so hard to get fluid build up inside lung from oncotic pressure changes. More possible outside lung
When do clinical signs of fluid build up become apparent
> 60 mL/kg
What are the signs of fluid build up
Tachypnea (1st) –> inspiratory dyspnea, cyanosis, orthopnea
Why does fluid accumulation cause dyspnea
Inspiratory effort increased due to lack of ability to move negative pressure
What are the clinically auscultable signs of pleural effusion
Dull/absent lung sounds ventrally (lung sounds heard dorsally) - TAP THEM! Thoracocentesis- not radiographs
What drugs can be used in patients with respiratory distress
Anxiolytic, not sedative (unless needed for tap)
What does thoracocentesis cytology test for?
Total protein, cell count, differential cell count, cell morphology assessment - all to classify the fluid