phys dis module 6 Flashcards
No know caused may be localized or generalized DIP and first CMC joints most often involved Incidence increases with age
Primary OA-
Has identifiable cause such as trauma Any age
Secondary OA-
Defined as a gradual loss of articular cartilage due to degenerative joint disease and chemical factors
OA
Commonly effected joints include Hip, knee, IP jts, CMC jt and spine
OA
Two part process Deterioration of articular cartilage Reactive bone formation (osteophyte formation)
OA
Heberden’s nodes
(DIP jts)
Bouchard’s nodes
(PIP jts)
CMC jt commonly effected Characterized by thumb adduction and subluxation from the trapezium, MP hyperextension, IP joint flexion
OA
OT Treatment for OA
Patient Education Weight management Joint protection techniques Adaptive equipment Environmental modifications Energy conservatio
Joint protection techniques
Respect pain Minimize force and load on joints Balance rest and activity Use larger, stronger joints Good body mechanics Avoid positions of deformity Decrease effort and resistance Maintain ROM
OT interventions
Modalities (PAMs)- depending on your state and facility Pain-free ROM AROM exercises Isometric strengthening General conditioning Avoid pinch strengthening Splinting Thumb – Short opponens splint Custom, prefab, neoprene
OA Surgical Treatment
Joint replacements Hip (THR) Knee (TKR) Shoulder (Total shoulder, Reverse Total shoulder) CMC jt-Ligament Reconstruction Tendon Interposition (LRTI) Gold standard treatment Trapezium removed, tendon “anchovied
Etiology is not well understood, synovial membrane of a joint becomes inflamed and wears away at the bone, cartilage and soft tissues which contributes to joint deformities
RA
Joint involvement is often symmetrical and bilateral throughout the body
RA
Characterized by remissions and exacerbations
RA
Joint swelling and inflammation, no destructive changes, possible presence of osteoporosis on X-ray “flare”
phase 1, acute
synovium begins to invade the soft tissues producing tenosynovitis and limiting joint movement (no joint deformities yet), adjacent muscle atrophy
Stage 2, Moderate Phase (Proliferative)
synovial activity “burnt out” fibrosis or bony ankylosis
Stage 4, Terminal phase (Chronic):
synovial erosion causes irreversible changes including joint deformities, loosening of ligamentous insertions, impairment of tendon function, muscle atrophy and joint disorganization. X-ray evidence of cartilage and bone destruction and osteoporosis
Stage 3, Severe Phase (Destructive):
PIP hyperextension with DIP extension lag, due to erosion of the PIP volar plate, rupture of lateral band or FDS rupture
Swan-neck deformity:
PIP flexion contracture with DIP hyperextension (caused by rupture or lengthening of the central slip of the EDC
Boutonniere deformity:
OT treatment for RA
Education Prevent joint deformities Sleep and Rest Modalities (PAMs) Therapeutic exercise Adaptive equipment Splinting For rest, deformity, function Joint Protection Fatigue management Energy Conservation