phys dis module 6 Flashcards

1
Q

No know caused may be localized or generalized  DIP and first CMC joints most often involved  Incidence increases with age

A

Primary OA-

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2
Q

 Has identifiable cause such as trauma  Any age

A

 Secondary OA-

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3
Q

Defined as a gradual loss of articular cartilage due to degenerative joint disease and chemical factors

A

OA

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4
Q

Commonly effected joints include  Hip, knee, IP jts, CMC jt and spine

A

OA

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5
Q

Two part process  Deterioration of articular cartilage  Reactive bone formation (osteophyte formation)

A

OA

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6
Q

Heberden’s nodes

A

(DIP jts)

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7
Q

Bouchard’s nodes

A

(PIP jts)

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8
Q

CMC jt commonly effected  Characterized by thumb adduction and subluxation from the trapezium, MP hyperextension, IP joint flexion

A

OA

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9
Q

OT Treatment for OA

A

Patient Education  Weight management  Joint protection techniques  Adaptive equipment  Environmental modifications  Energy conservatio

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10
Q

Joint protection techniques

A

 Respect pain  Minimize force and load on joints  Balance rest and activity  Use larger, stronger joints  Good body mechanics  Avoid positions of deformity  Decrease effort and resistance  Maintain ROM

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11
Q

OT interventions

A

 Modalities (PAMs)- depending on your state and facility  Pain-free ROM  AROM exercises  Isometric strengthening  General conditioning  Avoid pinch strengthening  Splinting  Thumb – Short opponens splint  Custom, prefab, neoprene

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12
Q

OA Surgical Treatment

A

 Joint replacements  Hip (THR)  Knee (TKR)  Shoulder (Total shoulder, Reverse Total shoulder)  CMC jt-Ligament Reconstruction Tendon Interposition (LRTI)  Gold standard treatment  Trapezium removed, tendon “anchovied

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13
Q

Etiology is not well understood, synovial membrane of a joint becomes inflamed and wears away at the bone, cartilage and soft tissues which contributes to joint deformities

A

RA

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14
Q

Joint involvement is often symmetrical and bilateral throughout the body

A

RA

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15
Q

Characterized by remissions and exacerbations

A

RA

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16
Q

Joint swelling and inflammation, no destructive changes, possible presence of osteoporosis on X-ray “flare” 

A

phase 1, acute

17
Q

synovium begins to invade the soft tissues producing tenosynovitis and limiting joint movement (no joint deformities yet), adjacent muscle atrophy

A

Stage 2, Moderate Phase (Proliferative)

18
Q

synovial activity “burnt out” fibrosis or bony ankylosis

A

Stage 4, Terminal phase (Chronic):

19
Q

synovial erosion causes irreversible changes including joint deformities, loosening of ligamentous insertions, impairment of tendon function, muscle atrophy and joint disorganization. X-ray evidence of cartilage and bone destruction and osteoporosis

A

Stage 3, Severe Phase (Destructive):

20
Q

PIP hyperextension with DIP extension lag, due to erosion of the PIP volar plate, rupture of lateral band or FDS rupture

A

Swan-neck deformity:

21
Q

PIP flexion contracture with DIP hyperextension (caused by rupture or lengthening of the central slip of the EDC

A

Boutonniere deformity:

22
Q

OT treatment for RA

A

 Education  Prevent joint deformities  Sleep and Rest  Modalities (PAMs)  Therapeutic exercise  Adaptive equipment  Splinting  For rest, deformity, function  Joint Protection  Fatigue management  Energy Conservation