pHTN and PVS Flashcards

1
Q

2D pHTN findings

A

1) Diminished or absent “a” wave of the pulmonary valve
2) Midsystolic closure of the pulmonary valve
3) Enlarged chambers of the right heart
4) D-shaped LV cavity

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2
Q

Transtricuspid pressure gradient

A

4(TRV)^2

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3
Q

What does tricuspid regurgitation velocity reflect?

A

The pressure difference during systole between the RV and the RA.

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4
Q

PASP

PA systolic pressure

A

In the absence of a gradient across the pulmonic valve or RVOT, RVSP is equal to PASP

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5
Q

When might the TRV appear low?

A

In the presence of markedly increased RA pressure due to RV infarct, RV failure, or severe TR.

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6
Q

RVSP

A

RA pressure + peak TR velocity

RA + 4(V)^2

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7
Q

How should TR velocity be measured?

A

Held-expiration, to avoid respiratory variation

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8
Q

PAEDP

A

4(PREDV)^2 + RAP

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9
Q

MPAP

A

4(Peak PRV)^2

or

1/3(PASP) + 2/3(PAEDP)

or

79 - 0.45(AcT) (For RVOT flow velocity)

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10
Q

Why is there a dip in the PA-RV pressure gradient?

A

PRV usually reflects small pressure differences between the PA and the RV, ATRIAL CONTRACTION with increased RV pressure creates a unique DIP in the velocity curve.

NB: When PA pressure is high, RA contraction usually does not make a notable change in the PA-RV pressure gradient. NO DIP!

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11
Q

Mahan’s equation for MPAP

A

MPAP = 79 - 0.45(AcT) (For RVOT flow velocity)

* Dependent on CO and HR, adjust for 100bpm.

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12
Q

Acceleration time

A

The time interval between the beginning of the flow and its peak velocity

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13
Q

PVR (Cath Lab)

A

(MPAP - PCWP)/CO

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14
Q

PVR (Doppler)

A

10 (TRV/RVOT TVI) + 0.16

  • A cutoff value of 0.2 for TRV/RVOT TVI separates a group with PVR greater than 2 Wood units
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15
Q

How can you assume pHTN is due to a pulmonary process?

A

If mitral inflow shows a nonrestrictive diastolic filling pattern.

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16
Q

What happens to hepatic venous flow in pHTN?

A

There is prominent atrial flow reversal in the hepatic vein caused by increased diastolic pressure and decreased compliance of the RV.

***There is very little respiratory variation of atrial flow reversal in pHTN, unlike the variation seen in restrictive CM or constrictive pericarditis.

17
Q

How to quantify RV dysfunction?

A

1) RV dilation
2) Paradoxical septal motion
3) TRV > 2.8m/s

18
Q

Congenital pulmonary veins

A

From one to all four PVs can be connected with or drain into the right side of the heart.

Best seen with the suprasternal short-axis view

One of the PVs may drain into the VERTICAL VEIN, which connects with the innominate vein.

19
Q

PV Stenosis: Etiology and Rx

A

May be congenital or due to PVI (catheter ablation)

The best treatment is percutaneous dilatation or stent placement.

20
Q

Causes of increased RV pressure

A

1) RV infarct
2) pHTN
3) Constrictive pericarditis