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Drug Profiles > Pharm Test 1 > Flashcards

Pharm Test 1 Flashcards

1
Q

Aspirin MOA

A

Small Doses block thomboxane A2 (platelet aggregate and vasoconstrictor) (used for MI)

Decreased platelet aggregation

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2
Q

Amiodarone Class

A

Anti-arrhythmic agent

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3
Q

Amiodarone MOA

A

Multiple effects on Na, K, and Ca channels
Prolongs action potential, refractory period
Ventricular automaticity (K)
Slows membrane depolarization and impulse conduction (Na)
Negative chronotropic activity in nodal tissue (antisympathetic activity)
Dilates coronary artery, Ca channel and alpha-adrenergic blocking action

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4
Q

Channels Amiodarone effects

A

Na, K, Ca

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5
Q

Lidocaine Indications

A

Suppression of ventricular arrhythmias (vtac, vfib, pvcs)
Prophylaxis against recurrence after conversion from vtac or vfib
Pain management after IO insertion in conscious patients

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6
Q

Lidocaine MOA

A

Decrease automaticity by slowing rate of spontaneous phase 4 depolarization. Terminates re-entry by decreasing conduction in re-entrant pathways. Increases vfib threshold.

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7
Q

Sodium Bicarb Incompatibilities

A

Incompatible with other drug infusions

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8
Q

Why does Nitro dilate coronary arteries?

A

To stop infarct from happening

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9
Q

Nitroglycerin MOA

A

Smooth muscle relaxant (vascular, uterine, bronchial, intestinal)
Reduces workload on heart by causing blood pooling (decreased preload)
Arteriolar vasodilation (decreased afterload)
Coronary Artery vasodilation
Increase blood flow to myocardium
Decrease myocardial 02 demand

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10
Q

Atropine MOA

A

Blocks acetylcholine at muscarinic receptor sites in smooth muscle, secretory glands and CNS.
Blocks parasympathetic response, sympathetic takes over.
Increase cardiac output and drying of secretions

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11
Q

Clinical MOA of Atropine

A

CV: Increased heart rate, increased conduction velocity, increased force of contraction, increase cardiac output
Resp: Decreased mucus production, bronchodilation
GI: Decrease GI secretion and motility
GU: Decrease urinary bladder tone
Misc: Mydriasis (pupillary dilation), decreased sweat production

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12
Q

Atropine and beta blockers

A

Not effective with beta blockers

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13
Q

Used for organophosphate poisoning

A

Atropine

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14
Q

Etomidate Class

A

Sedative/hypnotic agent

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15
Q

Etomidate MOA

A

Produces hypnosis rapidly causing CNS depression and anesthesia. No analgesic effect.

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16
Q

Is Sodium Bicarb for other med acidicy?

A

No

17
Q

Furosemide MOA

A

Inhibits electrolyte reabsorption in the ascending loop of henle. Promotes excretion of Na, K, Cloride. Vasodilation increases venous capacitance and decreases afterload

Diuresis.

18
Q

Wolff Parkinson White Syndrome complications

A

Can’t give adenosine or verapamil

19
Q

Swoop from P wave to QRS

A

Wolff Parkinson White Syndrome

20
Q

Verapamil Indications

A

SVT

A-Fib and A flutter with rapid ventricular response

21
Q

Verapamil Adverse Reactions

A 
Extreme Bradycardia
Asystole
AV Block
Hypotension
CHF
22
Q

Verapamil dosage and delivery

A

Adult: 2.5 - 5 mg slow IV push (2-3min) Rebolus every 15-30 min with 5 - 10mg until max of 30 mg

23
Q

Diphenhydramine Class

A

Antihistamine, anticholinergic

24
Q

Diphenhydramine MOA

A

Blocks cellular histamine receptors (does not prevent histamine release) results in decreased capillary permeability and decreased vasodilation, prevent bronchospasm
Some Anticholinergic effects

25
Q

Diphenhydramine Indications

A

Anaphylaxis (2nd line)
Phenothiazine reactions (extrapyramidal symptoms)
Antiemetic

26
Q

Dopamine Class

A

Sympathomimetic

27
Q

Dopamine MOA with 1-2 mcg/kg/min

A

Acts on dopaminergic receptors to stimulate cerebral, renal, mesenteric vasculature to dilate. HR & BP usually unchanged

28
Q

Dopamine MOA with 2-10 mcg/kg/min

A

B1 stimulant action is primary effect. Increased cardiac output and only modest increase in systemic vascular resistance

29
Q

Dopamine MOA with 10-20 mcg/kg/min

A

a-adrenergic effects predominate resulting in renal, mesenteric and peripheral arterial and venous vasoconstriction with marked increase in systemic vascular resistance, pulmonary vascular resistance and further increase preload

30
Q

Dopamine MOA with >20 mcg/kg/min

A

Produces hemodynamic effects similar to norepinephrine; may increase HR and O2 demand to undesirable limits

Drug Profiles (1 decks)

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