Peripheral Vestibular Disorders Pt.2 Flashcards

1
Q

what is an acoustic neuroma?

A

slow-growing tumor derived from Schwann cells of the vestibulocochlear nerve or internal auditory canal

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2
Q

describe the clinical presentation of an acoustic neuroma

A
  1. Gradual or sudden onset of tinnitus, hearing loss, vertigo or disequilibrium
    • hearing loss, tinnitus most common 1st symptom
    • balance, vertigo, disequilbirum (~50%)
  2. Symptoms tend to depend on location of tumor
    • small tumor → hearing may be preserved, pt may be asymptomatic
    • as it grow, it can compress CN V or VII
    • can cause pressure on brainstem or cerebellum if large enough
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3
Q

how is an acoustic neuroma diagnosed?

A
  1. MRI w/contrast - gold standard
  2. Audiogram
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4
Q

What are some interventions for acoustic neuroma?

A
  1. Surgical removal
  2. Radiation
  3. Monitoring (depening on size/location)
  4. Vestibular rehab only once tumor has been fully resected
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5
Q

what is a perilymph fistula?

A

an opening in between middle and inner ear that is caused by rupture of the oval window resulting in perilymph leaking into middle ear

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6
Q

what are typical causes of perilymph fistula?

A

most commonly associated with head trauma

  • usually trauma involves direct blow to the ear
  • other causes:
    • ear trauma
    • objects perforating ear drum
    • rapid increases in ICP (weighlifting, childbirth)
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7
Q

describe the clinical presentation of perilymph fistula

A
  1. sudden onset of:
    • vertigo
    • motion intolerance
    • ear fullness
    • fluctuating hearing
    • severe HA, N/V can also occur
  2. Symptoms worsen w/activity but improve w/rest
  3. Tulio’s phenomenon
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8
Q

what types of things worsen symptoms of perilymph fisutla?

A

changes in altitude

valsalva-eliciting activities

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9
Q

what is Tulio’s phenomenon?

A

use of one’s own voice or musical instrument will cause dizziness

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10
Q

how is perilymph fistula diagnosed?

A

A confident dx is difficult

  1. Fistula test
    • pressurizing test while recording eye movements
  2. Valsalva test
    1. pt holds breath while recording eyes
  3. Audiogram → helps determine involved side
  4. ENG/VNG
  5. MRI (rule out CNS)
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11
Q

List some intervention strategies for perilymph fistulas

A
  1. Smaller fistulas → conservative management
    • rest
    • avoid lifting or any activity that would increase ICP
    • if pt can take it easy, most cases resolve on their own
  2. Surgical repair
    • exploratory tympanotomy
    • vestibular nerve section
  3. Vestibular Rehab post-surgical repair often warranted
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12
Q

what is SCC Dehiscence?

A

the bony labyrinth that surrounds the membranous labyrinth of our canals degrades over time

fistual due to lack of temporal bone covering of superior SCC

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13
Q

which canal is most common affected by SCC dehiscence?

A

superior/anterior canal

(aka SCDS)

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14
Q

what is the incidence of SCC dehiscence?

A
  1. middle age disorder
  2. up to 60% can report premorbid trauma that shifted symptoms
  3. 25% bilateral
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15
Q

describe the clinical presentation of SCC dehiscence

A
  1. Transient vertigo precipitated by coughing, loud noises and pressure changes in ear
  2. Pressure sensitivity
    • sneezing, coughing, Valsalva, lifting, bowel movement
  3. Sound sensitivity
    • Internal → heel strike w/gait, eye movement, hear beat, own voice
    • External
  4. Imbalance
    • may be constant
    • accentuated w/head movement, dark environment
  5. Hearing loss, tinnitus, aural fullness
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16
Q

how is SCC dehiscence diagnosed?

A
  1. Bedside eval
    • Test for Tulio’s phenomena
    • Valsalva test
    • Bone conduction sensitivity test
      • tuning fork on lateral malleoli (+test = pt hears sound in inner ear)
  2. Radiographic imagining (CT scan)
  3. Audiogram
17
Q

What are some intervention options for SCC Dehiscence?

A

Will not benefit from rehab

  1. Conservative
    • avoid offending stimuli
      • noise (earplug)
      • exertion
  2. Surgery
    • canal “plugging”
    • resurfacing surgery
  3. Rehab possibly after surgery
18
Q

what causes bilateral vestibular hypofunction?

A
  1. Idiopathic in 50% of cases
  2. Typically caused by otoxic agents
  3. Other etiologies include:
    • autoimmune inner ear disease
    • chemo agent → Cisplatin
    • sequential vestibular neuritis
    • meniere’s disease
    • meningitis
    • neurodegenerative conditions
19
Q

describe the clincial presentation for bilateral vestibular hypofunction

A
  1. primary complian → severe oscilopsia
    • particulary during walking (imbalance, possibly due to hearing loss)
  2. Vertigo only if loss is sequential in nature
  3. functional tasks become inefficient and exhausting
20
Q

how is bilateral vestibular hypofunction diagnosed?

A
  1. Clinical exam
    • +HIT (bilaterally)
    • +Dynamic Visual Acuity
    • +Caloric testing
  2. Rotary chair test - gold standard
21
Q

List some interventions for bilateral vestibular hypofunction

A
  1. Medical management
    • meds typically not helpful
    • vestibular suppressant meds typically worsen symptoms
  2. no surgical intervention available to restore vestibular function
  3. Vestibular rehab
22
Q

what is the impact of vestibular rehab on bilateral vestibular hypofunction?

A
  1. improvement in postural control and gaze stability w/predictable > unpredictable head movements noted
  2. sig functional improvements noted by pts
    • however, typically w/residual impairment
      • difficulty driving due to oscillopsia, especially at night
      • imbalance w/moving about in dark environment