Peripheral venous and arterial disease Flashcards

1
Q

describe the lower limb venous anatomy.

A
  • superficial and deep veins.
  • superficial located in subcutaneous tissue.
  • deep veins underneath deep fascia with major arteries.
    MOVEMENT SUPERFICIAL TO DEEP.
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2
Q

give some examples of superficial and deep veins.

A
  • superficial : great and short saphenous vein.

- deep : popliteal, femoral, anterior/ posterior tibial, peroneal vein.

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3
Q

why is the calf muscle called ‘the peripheral heart’ ?

A
  • because the soleus and gastronemius contribute to push blood against gravity back towards heart.
  • contain valves to prevent retrograde movement when closed.
  • perforating valves open again to allow filling from superficial veins and venous pressure in foot reduced during exercise.

*faints due to pooling of blood at gravity.

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4
Q

what is the peripheral venous disease varicose veins?

A
  • ineffective valves and slow blood movement/ reversed saphenous veins causes walls of veins to weaken and varicosities to develop and valve cusps tO separate.
  • veins tend to be tortuous and twisted.

*risk factors : age, familial history, female, occupation and standing, number of births.

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5
Q

what is the symptomatic presentation of varicose veins?

A
  • heaviness and aching, muscle cramps and throbbing.
  • leg cramps, ankle swelling, haemorrhage.
  • increased risk of DVT.
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6
Q

Why might someone develop chronic venous insufficiency? any other complications?

A
  • due to reflux and or obstruction leading to venous hypertension. ( harder for venous return )
  • lipodermatosclerosis ( fat layer under skin inflamed and thickened ).
  • haemosiderin staining.
  • venous ulceration.
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7
Q

why might venous eczema and ulceration occur?

A
  • as a result of venous pooling and hypertension causing leaking of material into interstitium, irritation leads to chronic, painful ulcers in hard nodular areas like medial malleoulus.
  • chronic, itchy red and swollen can lead to lipodermatosclerosis which is hard to touch.
  • TREAT CAUSE!
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8
Q

why does the calf muscle fail to pump and who is at risk from this?

A
  • plantar flexion in ankle joint needs to be used for calf muscle to pump.
  • deep vein incompetence causes retrograde flow and superficial incompetence causes calf muscle pump to overflow.
  • risk : immobile, obese, injured.
  • ligation and vein stripping helps treat.
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9
Q

what is the risk with the lower limp and thrombosis?

A
  • arterial thrombosis : most common cause is atheroma, platelet rich accumulation of plaque.
  • venous thrombosis : STASIS and trauma/ medication/ dehydration/ chemo/ inflammation in fibrin rich env.
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10
Q

what is DVT?

deep vein thrombosis.

A
  • clotting of blood in deep vein (calf) impairing venous return and hyper-coagulability.
  • due to inflammation response following thrombosis.
  • well’s score : pre-test probability for suspected DVT.

*presents as calf tenderness, warmth, distended and visible superficial veins, oedema, unexplained pyrexia, asymmetry. (differential : soft tissue trauma, cellulitis, lymphatic obstruction).

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11
Q

how would you reduce risks of surgery and DVT?

A
  • promote mobility after surgery.
  • prophylaxis to reduce DVT associated with surgery like anticoagulants, physical activity, gradient stockings.
  • risk of PE!
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12
Q

what is acute limb ischaemia?

A
  • happens due to acute occlusion which doesnt allow for time for collaterla development.
  • due to trauma or embolism : A-fib, popliteal artery aneurysm, rupture of plaque.
  • presents as 6p’s : pain, pallor, perishing cold, pulselessness, paraestheia, paralysis.

*needs imaging at vascular surgery unit : angioplasty, thrombectomy, intra-arterial thrombolysis or amputation.

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13
Q

what is chronic peripheral arterial disease?

A

( similar to coronary artery disease intermittent claudication of lower limb )

  • claudication : cramping pain in leg induced by exercise.
  • caused atherosclerosis and is induced by exercise like stable angina, pain goes at rest.

*management : exercise, smoking cessation, anti-platelet drugs, angioplasty, bypass graft.

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14
Q

describe the pathology of claudication.

A
  • most common presentation of atheroma = atherosclerosis of superficial femoral artery that present s with calf claudication.
  • if untreated ISCHAEMIA.

*stenosis location dictated area of claudication and where pulses palpated (if any). easier above occlusion.

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15
Q

what can be used to assist diagnostics of peripheral artery disease?

A
  • Ankle-brachial index.

- divide ankle systolic by branchial systolic and if ABI i less than 0.9 indicated peripheral artery disease.

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16
Q

what is critical limb ischaemia?

A
  • progression of chronic peripheral vascular disease.
  • shows as pain at rest which is relieved by hanging foot off bed which causes pooling.
  • needs strong pain killers.
  • in untreated leads to ulceration and gangrene.
  • angioplasty, thrombectomy, intra-arterial thrombolysis or possibly amputation if severe.
17
Q

where do you palpate lower limb pulses?

A
  • femoral pulse : mid-inguinal point midway between anterior, superior iliac spine and pubic symphysis.
  • popliteal pulse : deep in popliteal fossa.
  • dorsalis pedis pulse : just lateral to extensor hallucis longus tendon.
  • posterior tibial pulse : behind medial malleolus.
18
Q

why is doppler ultrasonography so important in these conditions?

A
  • to measure real time flow and velocity.
  • echoes produced from moving blood detected and computed into flow direction and velocity.
  • echoes from stationary tissues are same from pulse to pulse so able to differentiate echoes from moving objects like blood.

*velocity increases through stenosis which can be measured using doppler, as the turbulence travels further.