Periodontium in Health: Fight Against Invasion Flashcards

1
Q

List features of a healthy periodontium:

List features of an unhealthy periodontium:

What is the relevance to BOP?

A

Healthy: pink, stippled, knife edge margin, papillae exactly fill ID space, no BOP from base of pocket

Unhealthy: red, swollen/inflammed gingiva with loss of knife edge margin, papillae under/over-fill interdental space, loss of stippling, BOP from base of pocket

BOP indicates active disease at that site at that time

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2
Q

List some facts of Gingivitis:

List some points regarding Periodontitis:

A

Gingivitis: primary cause is plaque-induced inflammation

  • no LOA
  • base of pocket is at/coronal (above) the ACJ
  • false pockets

This condition is reversible: no permanent damage

Periodontitis: plaque-induced, can be influenced by smoking/diabetes

  • LOA: base of pocket below ACJ i.e. on root surface

LOA measured from ACJ to base of pocket –> true pockets

Condition is irreversible: permenant damage, loss of bone and gingival recession

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3
Q

Where does plaque bacteria build up, even in health?

What is the gingival sulcus/crevice?

What is at the base of the gingival sulcus?

What problem does this pose?

A

Plaque builds up at the gingival margin quickly following OH

Gingival crevice/sulcus: small groove between gingival margin and enamel

At base of sulcus, there is junctional epithelium

Junctional epithelium: contains large spaces between epithelial cells so plaque bacteria and their toxins can leak into the underlying gingival connective tissue causing inflammation

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4
Q

What is the concept of physiological immune surveillance?

A
  • always a low level of inflammatory infiltrate (mainly neutrophils and some B cells) in the cinnective tissue underlying junctional epithelium

Symbiotic relationship - good balance between good bacteria and host bacteria maintaining health

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5
Q

What is Gingival Crevicular Fluid? (GCF)

How much is secreted in clinical gingival health?

What is the dentogingival plexus?

A
  • a serum-like fluid formed from the post-capillary venules of dentogingival plexus
  • very small volume of GCF in health - aroud 0.2microlitres/hour = 1ml per day to saliva

The dentogingival plexus is a dense network of blood vessels in the gingival connective tissue sub-adjacent to junctional epithelium

GCF flows through the junctional epithelium into gingival crevice and into the mouth where it mixes with saliva

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6
Q

What are the components of GCF?

A
  • plasma proteins
  • defence cells/proteins: neutrophils, antibodies, complement
  • growth factors
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7
Q

List some host mechanisms to prevent bacterial invasion:

A
  • outward flow of GCF into gingival sulcus (washing effect)
  • high turnover of JE cells: any invading bacteria attached to epithelial cells are rapidly shed into gingival sulcus
  • killing of micro-organisms by inflammatory and immune cells: neutrophils migrating through JE
  • destruction of micro-organisms by immunoglobulins and/or activation of complement
  • normal bacterial flora restricts micro=organisms growth
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