Periodontal Abscess and Furcations Flashcards

1
Q

What is an abscess?

A

A localized collection of pus in a cavity formed by disintegration of tissues

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2
Q

What is formation of pus termed?

A

Suppuration

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3
Q

What are the different types of abscesses?

A

Endodontic, periapical, dentoalveolar, pericoronal (pericoronitis), trauma, and foreign body

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4
Q

How is a dental abscess identified clinically?

A

Through signs and symptoms such as pain, redness and swelling, regional lymph node enlargement

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5
Q

What is an abscess that only involves soft tissues termed?

A

Cellulitis

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6
Q

What is an abscess that involves bone termed?

A

Osteitis

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7
Q

What is an abscess that involves bone marrow termed?

A

Osteomyelitis

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8
Q

What do dental abscesses expand through?

A

Those tissues providing least resistance, forming a sinus tract

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9
Q

What is a periodontal abscess?

A

Localized accumulation of pus located within gingival wall of periodontal pocket

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10
Q

What is a periodontal abscess also known as?

A

A lateral periodontal abscess

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11
Q

What is the most common cause of tooth loss during periodontal maintenance?

A

Periodontal abscess

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12
Q

What is the severe systemic consequence of a periodontal abscess?

A

Ludwig’s angina

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13
Q

What is the pathology of a periodontal abscess?

A

Bacterial invasion of soft tissues around periodontal pocket, inflammatory process (PMN – Cytokines – destruction of connective tissues), encapsulation of bacterial infection and production of pus

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14
Q

What is drainage of a periodontal abscess most likely to take place through?

A

Periodontal pocket since this is usually path of least resistance

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15
Q

What is the iatrogenic pathology of a periodontal abscess?

A

Foreign bodies such as calculus and food debris may be pushed into gingival tissue during debridement and cause abscess formation

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16
Q

What are the symptoms of a peridontal abscess?

A

Pain, tenderness of gingiva, swelling ovoid elevation in gingiva along lateral part of root/tooth “elevation”, suppuration on probing/sampling was common (66– 93%) whereas a fistula was not, deep periodontal pocket (7.3– 9.3 mm), bleeding on probing (100%), increased tooth mobility (56.4– 100%), bone loss was normally seen, and extraoral findings were uncommon: facial swelling (3.6%), elevated body temperature, malaise, regional lymphadenopathy (7–40%) or increased blood leukocytes (31.6%)

17
Q

What do all studies on periodontal abscesses suffer from problem of?

A

Mixing abscess flora with that of periodontal pocket

18
Q

What are the causes of periodontal abscess in a periodontal patient?

A

Acute exacerbation and after different treatments

19
Q

When can acute exacerbation of periodontitis occur?

A

In untreated periodontitis, “refractory” periodontitis, and in periodontal maintenance

20
Q

What treatments can lead to a periodontal abscess in periodontitis patients?

A

Scaling and RSD, surgical periodontal therapy, systemic antimicrobial intake, without subgingival debridement, and use of other drugs: e.g., nifedipine

21
Q

What causes periodontal abscess in a non-periodontitis patient?

A

Foreign body impaction (floss, toothpick), habits (nail biting, wire biting), inadequate orthodontic forces, gingival enlargement, and alteration of root surface (invaginated tooth, grooves, perforations, fractures, external resorption)

22
Q

What is the differential diagnosis of periodontal abscess?

A

Other odontogenic abscesses (dento-alveolar abscesses, pericoronitis, endo-periodontal abscess)/other acute conditions (lateral periapical cyst and postoperative infection), tumour lesions, including metastatic tumoral lesions, odontogenic myxoma, non-Hodgkin ́s lymphoma, squamous cell carcinoma, metastatic carcinoma, other oral lesions: pyogenic granuloma, osteomyelitis, odontogenic keratocyst, eosinophilic granuloma, self-inflicted gingival injuries, and sickle cell anaemia

23
Q

What is the treatment of a periodontal abscess?

A

Incise abscess and create drainage, irrigation with antiseptic solutions, and after abscess resolution ,periodontal infections should continue to be treated by subgingival debridement

24
Q

When are antimicrobial agents indcated?

A

Only when patient shows signs systemic involvement (fever, malaise, lymphadenopathy)

25
Q

What is the aim of systemic antibiotic treatment of dental abscesses?

A

Preventing bacterial spreading and serious complications

26
Q

What is furcation involvement?

A

Bone loss to bi/trifurcation region of multirooted teeth

27
Q

What is furcation usually as a result of?

A

Periodontal disease

28
Q

What is the root complex?

A

CEJ to apex

29
Q

What is the root trunk?

A

CEJ to furcation

30
Q

What is the root cone?

A

Beyond the furcation

31
Q

What is grade I furcation?

A

Incipient early lesion, early bone loss, and less than 1/3 of furcation ‘width’

32
Q

What is grade II furcation?

A

More than 1/3 of furcation ‘width’ but NOT through and through

33
Q

What is grade III furcation?

A

Through and through lesion identified most accurately with a Nabers probe

34
Q

What is the non-surgical treatment of furcation defects?

A

Aim is for cleanliness (by us and by patient)

35
Q

What are the outcomes of non-surgical treatment of furcation defects?

A

Less promising

36
Q

What is the surgical treatment of furcation defects?

A

‘Furcationplasty’, tunnel preps, root resection, and GTR

37
Q

What are the indications for root resection?

A

Isolated defect around 1 root of a multi-rooted tooth, caries, failed root filling, and furcation