Perio-Endo and Periodontal Abscess Flashcards

1
Q

What are the different abscesses of the periodontium

A

Gingival abscess
Periodontal abscess
Periocoronal Abscess
Endodontic periodontal lesion

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2
Q

What is a gingival abscess

A

An abscess Localised to gingival margin in space between tooth and gum

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3
Q

What is a periodontal abscess

A

Infection in a periodontal pocket which can be acute or chronic and asymptomatic if freely draining

usually related to pre-existing deep pocket also associated with food packing and tightening of gingival margin post HPT, forms in periodontal pocket

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4
Q

What is a pericoronal abscess

A

An abscess associated with partially erupted tooth most commonly 8s

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5
Q

What is a endodontic periodontal lesion

A

when the tooth is suffering from varying degrees of endodontic and periodontal disease

“EPL is a pathological communication between the endodontic and periodontal tissues of a given tooth”

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6
Q

How much of dental emergincies is for periodontal abscesses

A

approximately 7.7–14.0%

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7
Q

What happens in a periodontal abscess

A

Rapid destruction of periodontal tissues, with a negative effect on the prognosis of the affected tooth

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8
Q

What % of teeth found with a perio abscess are extracted

A

45%

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9
Q

What are the signs and symptoms of perio abscess

A

Swelling
Pain
Tooth may be TTP in lateral direction
Deep periodontal pocket
Bleeding
Suppuration
Enlarged regional lymphnodes
Fever
Tooth usually vital
Commonly pre-existing periodontal disease

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10
Q

What are the SDCEP guidlines when dealing with a perio abscess

A

Carry out careful sub-gingival instrumentation short of the base of the periodontal pocket to avoid iatrogenic damage; local anaesthesia may be required.

If pus is present in a periodontal abscess, drain by incision or through the periodontal pocket.

Recommend optimal analgesia.

Do not prescribe antibiotics unless there are signs of spreading infection or systemic involvement.

Recommend the use of 0.2% chlorhexidine mouthwash until the acute symptoms subside.

Following acute management, review and carry out definitive periodontal instrumentation and arrange an appropriate recall interval.

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11
Q

When would you use systemic antibiotics in a perio abscess case and what antiobiotics would you use and how much

A

Only if signs of spread and systemic effects or if symptoms do not resolve with local measures

Careful RSD

Penicillin V 250mg(preferred) or Amoxicillin 500mg 5 days
Or
Metronidazole 400mg 5 days

MUST only be used in conjunction with mechanical therapy in order to reduce the bacterial load and disrupt the biofilm

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12
Q

What are the signs and symptoms of a perio-endo lesion

A

deep periodontal pockets reaching or close to the apex

negative or altered response to pulp vitality tests

bone resorption in the apical or furcation region

spontaneous pain

Pain on palpation and percussion

Purulent exudate

tooth mobility

sinus tract

crown, and gingival colour alteration

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13
Q

In a perio Endo lesion what are the possible routes of communication

A

Exposed dentinal tubules
Lateral and accesssory canals
Furcal canals
Apical foramen
Perforation
Developmental groove

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14
Q

How do the possible routes of communication in an Perio Endo lesio work

A

They allow bi-directional spread of infection and/or inflammation

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15
Q

What are the classifications of a perio endo lesion

A

A carious lesion affecting the pulp and secondarily the periodontium

Periodontal destruction that secondarily affects the root canal

or by both concomitantly

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16
Q

How do dentinal tubules act as a possible route of communication

A

Dentin is highly permeable with the tubules as the major channels for diffusion of material across dentin,

application of soluble material from bacterial plaque to exposed dentin can cause pulpal inflammation (Bergenholtz and Lindhe) suggesting there is a way for bacterial plaque to cross through the tubules

17
Q

What are the stats for teeth with accessory canals

A

30–40% of all teeth have lateral and accessory canals, majority are found in the apical third of the root.

17% of teeth presented multiple canal systems in the apical third of the root, about 9% in the middle third and fewer than 2% in the coronal third

18
Q

Do all furcal canal reach the pulp chamber

A

No

The incidence of furcal canals may vary from 23% to 76%. Not all of these canals extend the full length from the pulp chamber to the floor of the furcation

19
Q

Pulp inflammation and a furcal canal can lead to what

A

Pulpal inflammation may cause an inflammatory reaction in the interradicular periodontal tissues

20
Q

What else can associate with endo-perio lesions and list them

A

Trauma and iatrogenic factors

root/pulp chamber furcation perforation (e.g. because of root canal instrumentation or to tooth preparation for post‐retained restorations)

root fracture or cracking (e.g., because of trauma or tooth preparation for post‐retained restorations)

external root resorption (e.g., because of trauma)

pulp necrosis (e.g., because of trauma) draining through the periodontium

21
Q

What are the SDCEP guidlines for treatment of a perio endo lesion

A

Carry out endodontic treatment of the affected tooth.

Recommend optimal analgesia.

Do not prescribe antibiotics unless there are signs of spreading infection or systemic involvement.

Recommend the use of 0.2% chlorhexidine mouthwash until the acute symptoms subside.

Following acute management of the lesion, review within ten days and carry out supra- and sub-gingival instrumentation if necessary and arrange an appropriate recall interval

22
Q

What is the treatment of a perio endo lesion

A

Primary endodontic therapy first

Then peridontal therapy (OHI etc)

and then surgical investigation and treatment

23
Q

Does endodontic disease affect periodontal health?

A

When the pulp becomes infected, it elicits an inflammatory response in the periodontal ligament at the apical foramen and/or adjacent to openings of the small portals of exit

24
Q

Does periodontal disease affect endodontics?

A

Pulp is usually not significantly affected by periodontal disease until recession affects a lateral or accessory canal to the mouth.

Bacteria access through the lateral or accessory canal into the pulp causing chronic inflammation and possibly pulp necrosis.

If the lateral or accessory canals are protected by cementum, necrosis usually does not occur. If the blood supply from the apical foramen is still intact, the pulp will remain vital

Pathogenic invasion and secondary inflammation and necrosis of the pulp can occur if during scaling, curettage or periodontal surgery accessory canals are severed and/ or opened to the oral environment.