perio Flashcards
what is the normal bony remodelling with implants
less then 2mm
when you have bony remodelling when are you thinking peri- implantiitis
more than 2mm in one year
pregnancy and perio
increased vascular permeability
3 month recall
menopause and perio
increased but doesn’t decrease with HRT
genetics and perio
can be up to 50% in younger pt
stress and perio
oHI,diet ect
and reduced leukocyte function, altered cytokine profile, salivary changes
medications and gingival hyperplasia
Anti-epileptic drugs- Phenytoin 1-84%(50 average)
Calcium-channel blockers-Nifedipine - 33%- 70% in kids
Immunosuppressants-Cyclosporin- 42%
verapamil, amlodipine, felodipine, diltiazem-5%
ohi- better but won’t prevent
history and ghp
increased fibroblasts and college and vascularity
pathology and ghp
unknown but it was thought more fibroblast proliferation
perio and diabetes
increased AGE/RAGE promote inflammation and increased inflmmatory cytokines
diabetes mechanism
Microvascular damage, affecting leukocyte/nutrient delivery
Altered collagen turnover→ ↑periodontal breakdown/↓ periodontal healing
PMN dysfunction
Heightened production of inflammatory mediators (PGE2, cytokines, MMPs)
Persistent hyperglycaemia results in glycosyation of proteins, forming AGEs
AGEs (advanced glycation end products) responsible for much of the above.
eds tyes and perio
type 8 is the worse (sever-tooth losss before 30 yrs) followed by 4
downs syndrome and perio
to be 58%-96%
mouth breathing/mouth breathing/poor tooth to crown ratio
Alterations in immune response, neutrophil chemotaxis/phagocytosis
leukaemia
proliferation of leukaemic
thrombocytopenia -gingival bleeding
neutropenia
rolonged fall in circulating neutrophils•Multiple forms-congenital or acquired•Variable periodontal manifestations, including:◦Ulceration/necrosis of marginal gingivae◦Gingival bleeding◦Generalised severe periodontal disease
neutropenia
fall in neutrophils
- Ulceration/necrosis of marginal gingivae
- Gingival bleeding
- Generalised severe periodontal disease
Chediak-Higashi syndrome•Autosomal recessive condition
Impaired neutrophil function
Severe periodontal disease even at a young age
Leukocyte adhesion-deficiency syndrome
Leukocyte adhesion-deficiency syndrome•Single gene defect•Clinical features◦Impaired wound healing◦Prone to severe infections◦Periodontitis and acute gingivitis
Hypophosphatasia
increase alkaline phosphates and therefore poor bone and tooth formation loss mention early nd also prone to hypoplasia
what is supportive pd therapy
it is reviews after pintail therapy including pocket depths scaling and some localised psi- 3 months at first and then different intervals later
