peri implant health and disease - periodontitis Flashcards
what is implant?
used to replace individual teeth or support a fixed bridge or removable denture
implant to epithelial surface
creation of biological seal
functions as barrier
between implant and oral cavity
sulcus lined by SE and JE (just like natural tooth)
components of the implant
implant body: the root of the implant on the alveolar bone
abutment post: made from titanium, through gingival tissue and support the crown
prosthesis: single crown, bridge or denture
implant to connective tissues
significantly different to natural tooth
CT fibres support gingiva against the abutment - orientated parallel or encircling the implant
there are no PDL fibres
keratinised gingival tissue may/may not be present around the implant
what is osseointegration?
is the directed contact of bone with the implant surface. determine the success of the implant. but it does not happen over night, around 4 weeks when the new bone form and the implant connected to the bone. it will be success if:
no mobility
absence of inflammation of tissues
no discomfort or pain when functioning
no increase bone loss or radiolucency
what are the implant health and conditions?
peri implant health
peri implant mucositis
peri implantitis
what is peri implant health?
absence of:
erythema, BOP, swelling and suppuration
probing depth may be deeper at a healthy implant site compare with a health tooth site.
what is peri implant mucositis?
plaque biofilm-induced inflammation of the soft tissues with no loss of supporting bone
it is reversible
diagnosis requires: visual signs of inflammations; presence of BOP/suppuration; increase probing depths: absence of bone loss
what is peri-implantitis?
plaque biofilm-induced inflammation of the peri-implant mucosal tissues and progressive loss of supporting alveolar bone
diagnosis visual signs of inflammation: the presence of BOP/suppuration; increased PD; progressive bone loss
probing depth more than 6 mm - consider as peri-implantitis
what is peri-implant soft and hard tissue deficiencies?
Tissue deficiencies at implant sites are common clinical findings. Their presence may lead to an increase in marginal bone loss, soft-tissue inflammation, and soft-tissue recession. These abnormalities can be corrected by grafting the hard or soft tissues. Hard-tissue defects at implant sites encompass intra-alveolar, dehiscence, fenestration, horizontal ridge, and vertical ridge defects.9 Soft-tissue defects include volume and quality deficiencies, i.e. lack of keratinized tissue.
aetiology: bacterial infection
mucositis- plaque/ biofilm is the same as gingivitis
implantitis - overwhelming infection which is same as periodontitis
note; peri mucositis does not always progress to peri implantitis
bacterial infection of peri implants
the rate tissue destructions tends to be more rapid in peri implant tissues than in periodontal tissues.
after 3 weeks of undisturbed plaque accumulation; similar response in dento- gingival and implant mucosal units (not much different, very identical)
after 3 months of undisturbed plaque accumulation; inflammatory infiltrate in implant mucosal unit is 3 times greater
risk factor for the implants
history of periodontal disease
poor plaque biofilm control - the longer plaque, the more destruction of the tissue
lack of maintenance therapy
smoking - it is advance to quit smoking first before put the implant because there is too much failure
residual cement
biomechanical overload - influence by position, the number of the implant and the patient occlusal
clinical monitoring of peri-implant health and disease
probing
assessing clinical attachment levels
assess for bleeding
assess for evidence of suppuration
collect routine radiographic evidence of health/disease
diagnosis of peri implant health, peri implant mucositis and peri implantitis
peri-implant health:
- clinical signs of inflammation: absent
- PD; no increase or < 5 mm
- RBL: no changes after initial remodelling (<2mm)
peri-implant mucositis:
- clinical signs of inflammation: localised erythema, swelling, suppuration, bleeding on probing
- PD; increase compared to baseline
- RBL; no changes after initial remodelling (<2mm)
peri-implantitis:
- clinical signs of inflammation: localised erythema, swelling, suppuration, bleeding on probing
-PD: increased or >6mm (if no baseline)
- RBL: increased beyond initial remodelling or >3mm after 1 year in function