Perf Tech 3 Test 6 Flashcards

1
Q

Dissection vs. Aneurysm

A

dissection- occurs when blood penetrates the intimate of the aorta, expanding medial layers, true lumen gets smaller, branching vessels may not be affected
aneurysm- dilation of all 3 layers

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Aortic Dissections

A

=Intimal Tear! - at weak spots or areas with high shear force

  • risk factors HTN (90%), age (>60), male, Marfan’s, CHD, prego
  • causes- high physical activity, emotional stress, trauma (deceleration injury- MVA—most common occurring distal to left subclavian at lig. arteriosum, 2nd most common at ascending aorta distal to aortic valve), cannulation/CPB
  • propagation- spreading driven by pulse pressure and ejection velocity
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

DeBakey Classification

A

-based on intimal tear origin and sections involved
Type 1- tear in asc. aorta, includes rest of aorta
Type 2- tear in asc. aorta, stays in asc. aorta
Type 3a- tear in desc. aorta, dissection stays above diaphragm
Type 3b- tear in desc. aorta, dissection goes below diaphragm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Standford (Daily) Classification

A
  • doesn’t matter the origin, just where it is
    Type A- if it involves the asc. aorta (regardless of how far it propagates
    Type B- distal aorta (past left subclavian)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Dissection causes of death

A
  • rupture of false lumen into pleural or pericardium space!

- or HF, MI, stroke, bowel gangrene

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Aneurysm Etiology and Shape

A

-is site dependent
-Asc. = congenital (marfans), post-stenotic dilation
-arch= isolated (atherosclerosis)
-desc.= atherosclerosis (intimal disease)
Shape= fusiform (circumference gets bigger), saccular (sac bulges out-most common)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Crawford Classification

A
  • to classify thoracoabdominal aortic aneurysms
    Extent 1- desc. thoracic aorta and upper abdominal aorta (above diaphragm)
    Extent 2- desc. thoracic aorta to infrarenal
    Extent 3- distal second half of desc. aorta and down
    Extent 4- abdominal aorta and down
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Indications for surgery- Ascending aorta

A
  • dissection= acute type A (high mortality)

- aneurysm= pain, MI, angina, expanding, >5-5.5 cm

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Indications for surgery- Aortic Arch

A
  • dissection= acute, limited to arch (rare)

- aneurysm= persistant symptoms, > 5.5-6 cm, expansion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Indications for surgery- Desc. aorta

A
  • dissections= HTN, pain, enlargement, neurological deficit, renal/GI ischemia
  • aneurysm= >5-6 cm, expanding, leaking, chronic pain
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

CPB with DHCA and ACP

A
  • DHCA= blood less filed, uncluttered field, BUT brain ischemia and accumulation of waste, must monitory temps and brain
  • Drugs= Mannitol 25 g and steroids before going off= cerebral protection
  • cannulation= axillary and bicaval
  • cooling= 10 degree drop in temperature = reduces O2 consumption rate by 50% and 20-25% increase blood viscosity
  • hemodilute HCT<25% (keep low until rewarming)
  • at firbrillation= give retrograde CPG
  • cool for at least 25 min before circ arrest, want brain temp 18-20, NO lower than 15 until no EEG= give pentobarbital, head in ice and Trendelenburg
  • flow off= drain patient
  • innominate artery is snared= ACP at 10 ml/kg/min at 90 mmHg= NEED suction at distal arch (because some blood will flow back into left carotid and subclavian
  • after DHCA= there will be low platelets and hypothermia
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Retrograde Cerebral Perfusion

A

-cerebral cooling, was out air, debris, wastes, prevent aggregation, delivery of oxygen and nutrients
=goes retrograde up venous line into SVC
=pressure no higher than 25 mmHg

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Antegrade Cerebral Perfusion

A

-most popular right now= maintains jugular venous sats and cerebral oxygen extractions

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Complications of Aortic Surgery and DHCA

A

air, clots, LV dysfunction, MI, renal failure, respiratory failure, coagulopathy, hemorrhage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

TEVAR

A

Thoracic Endo Vascular Aortic Repair

  • femoral access, graft, fluoroscopy
  • advantages= lowered death, less blood loss, quicker recovery
  • complcations- bad if you need to convert to open heart if something goes wrong, bleeding, endo-leakage, stroke
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Left Heart Bypass

A
  • shunts around aneurysm
  • venous cannula LA, cent pump (2/3), arterial in descending aorta
  • heart is still beating
  • still use lungs to oxygenate
  • no reservoir, H/E, filters
17
Q

Marfan’s Syndrome

A

connective tissue disorder= weak arteries and aorta= can dilate or tear

18
Q

Aortic Debranching and Endovascular Repair

A

-to repair arch aneurysm

make graft from aorta to head vessels and deploy endograft in arch and occult head vessels

19
Q

Embolic Events with CPB

A

50% of CABG patients have cerebral infarct BEFORE surgery

  • 30% new infarcts after CPB
  • stroke post CPB 1-5%
20
Q

Embolus

A

carried by blood and occludes vascular system

  • deformable (conformal)= GME, fat
  • non-deformable= bone chip, calcific particle
  • sources= circuit component, drugs, blood/surface interactions, perfusionists/surgeon (major cause!)
  • types= foreign material (cotton fibers, plastic, filter, tubing, metal, bone wax), gaseous (cannula, de-airing, low reservoir, damaged circuit, ,suction, vacuum), biological (blood born-thrombin, aggregates, proteins, bone, muscle, fat)
21
Q

Biologic Emboli areas for risk and circuit

A

areas for risk= low flow, stagnant, turbulence, cavity, rough surface
areas in circuit= connections, oxygenators, AF, reservoir

22
Q

Opportunity for Embolic Events

A
  • greatest period of risk= insertion of arterial cannula, initiation of bypass, cross clamp
  • BRAIN is at greatest risk (15-20% don’t have circle of willis)
23
Q

Prevention of Biological, Foreign Material, and Gaseous Emboli

A
  • Biological= transfusion filters, anticoagulants properly, decrease circuit SA, lower WBC activation
  • Foreign Material= good circuit, prime, prebypass filter, reservoir filters
  • Gaseous= components secure, co2 flush, retrograde ALF priming, safety device, purge lines, pressure valves, watch for air, vents, trendelendburg, TEE, suction,
  • –X clamp types= Bahnson (, Fogarty L
24
Q

Safety Devices

A

low level alarm, bubble detector, filters, one way valves, communication protocols, checklists

25
Q

Treatment of Massive Air Emboli

A
  • retrograde cerebral perfusion (w/ induced hypothermia)
  • hyperbaric chambers- most effective
  • protocol in place
26
Q

Embolic Take Home Message

A

preventing embolic events remain the best hope from improving neurological outcomes after CPB