PEM - neuro, opthal, ENT, dental, radiology, renal, resus Flashcards
Causes of Raised ICP
Increased CSF fluid
- decreased absoprtion (Obstruction, communicating)
Swollen contents
- infection: meningitis, encephalitis
- Cerebral oedema: metabolic, vasogenic, ischaemic, axonal injury
- Increased venous pressure: venous sinus thrombosis
Space occupying lesion: tumours, haematoma, absess/cyst
Decreased volume: depressed skull fracture
Simple febrile seizure, characteristics
- generalised tonic-clonic
- < 10 min
- Complete recovery in 1 hr
- not reoccuring in same illness
Management of seizure
> 5 min seizure / unknown length
Monitoring, BSL
Benzodiazepine - midazolam 0.1 mg/kg IV/IM (0.3mg/kg buccal)
5 min - repeat benzodiazepine
5 min - levetiracetam 40 mg/kg IV (max 3g) over 5 min
5 min after infusion - phenytoin IV 20mg/kg over 20 min, max 50mg/min
5 min after infusion - intubation
Dose of midazolam in seizures
- 1 mg/kg IM / IV
0. 3 mg/kg IN / Buccal
Predictors of ICU admission in diagnosis of muscular weakness
- bulbar palsy
- Vital capacity < 20 mls/kg
- > 30% reduction in vital capacity
- Flaccid quadriparesis
- Rapidly progressive weakness
- Autonomic cardiovascular instability
Guillian-Barre: clinical findings
Motor weakness
Absent reflexes
Ascending pattern.
Cranial nerve abnormalities
Predictors of intubation in Guillian Barre
Vital capacity <= 20mls/kg
Max insp pressure <= 30 cmH2O or exp pressure <= 40 cmH2O
Tidal volume < 5 mls/kg
Sustained increase of pCO2 > 50
Rising RR or O2 requirement.
Increased accessory muscle use or paradoxical diaphragm movements.
Features of infantile botulism and treatment
Exposure to honey. Bulbar palsy, descending. lack of facial expression.
No antibiotics.
BabyBIG (botulism IG)
NG feeding
Respiratory support
Features of transverse myelitis
Flaccid paralysis –> increased tone.
Sensory level (often midthoracic)
Back pain, neck stiffness / fever.
Bladder / bowel disturbance
DDx of ataxia
Post-viral acute cerebellar ataxia - dysarthria, hypotonia
Poisoning - altered consciousness and vomiting
Tumours - posterior fossa, paraneoplastic
Trauma - NAI, concussion
Metabolic - BSL, Na, Ammonia, B12, IEM
Infectious - CNS, larbrynthitis
Vascular - stroke, vasculitis
Immune - MS, ADEM
Absence of red reflex
large retinal detachment
large vitreous haemorrhage
dense cataract
retinoblastoma
Distinguishing features of orbital cellulitis
Systemic features, sinusitis
Red eye
Painful eye movements / diplopia
Proptosis
Neonatal conjunctivitis management and differential
Swab
IV ceftriaxone (Gonorrhoea, 24-48 hrs)
PO azithromycin or erythromycin (Chlamydia 5-14 days)
Other - staph/strep (D5-7) or HSV
DDx:
congenital nasolacrimal duct obstruction (not red / inflamed)
How do you identify keratitis, causes and management
Corneal opacity, without overlying epithelial defect.
Pain, erythema, photophobia and reduced visual acuity
+/- hypopyon
Staph/strep Pseudomonas (contact lenses) Fungi (garden trauma) HSV (dendritic) Varicella (skin rash)
Mx: ophthalmologist for scrapings prior to abx
Indications for referral in eyelid trauma
Bite trauma Involves lid margin Medial 1/3 of eyelid Levator aponeurosis (proptosis) Orbital fat exposed Significant tissue loss